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1 eatment of the neurological complications of Zika virus infection.
2 of neurological symptoms, suggesting recent Zika virus infection.
3 nd two spontaneous abortions associated with Zika virus infection.
4 interaction and proteasome degradation after Zika virus infection.
5 ogic fetal outcomes during murine congenital Zika virus infection.
6 to confirm microcephaly and assess previous Zika virus infection.
7 sis and disease outcomes in a mouse model of Zika virus infection.
8 and kinetics with those elicited by natural Zika virus infection.
9 r vesicles (EVs) in semen and saliva inhibit Zika virus infection.
10 d December, 2017, 368 were confirmed to have Zika virus infection.
11 gnized by human antibodies following natural Zika virus infection.
12 st NeSt1 might protect people against severe Zika virus infection.
13 t for the prevention of mosquito-transmitted Zika virus infection.
14 acid-editing deaminase), inherently restrict Zika virus infection.
15 (EC(68): 300-600 nM) and in a mouse model of Zika virus infection.
16 ethal mosquito-borne-but not needle-injected-Zika virus infection.
17 es suspected of resulting from a symptomatic Zika virus infection.
18 organoid development, culture protocols and Zika virus infection.
19 l Zika syndrome and serological evidence for Zika virus infection.
20 imaging findings associated with congenital Zika virus infection.
21 ine the association between microcephaly and Zika virus infection.
22 ay be the only initial finding in congenital Zika virus infection.
23 inhibitors to identify several inhibitors of Zika virus infection.
24 this neuronal population as a consequence of Zika virus infection.
25 ociation between microcephaly and congenital Zika virus infection.
26 studies supported a causal association with Zika virus infection.
27 resumed or laboratory-confirmed intrauterine Zika virus infection.
28 il, which was later attributed to congenital Zika virus infection.
29 rocephaly epidemic is a result of congenital Zika virus infection.
30 on the incidence and prevalence of prenatal Zika virus infection.
31 ogic correlation in a patient with the acute Zika virus infection.
32 o seen as common outcomes of first trimester Zika virus infections.
33 boratory tests and no controls had confirmed Zika virus infections.
34 nital infections or are unique to congenital Zika virus infection: (1) severe microcephaly with parti
35 samples from 54 people with suspected acute Zika virus infection, 76 infants with suspected microcep
37 id and systematic review about links between Zika virus infection and (a) congenital brain abnormalit
39 tion of available evidence from outbreaks of Zika virus infection and clusters of microcephaly is tha
40 gs provide strong evidence of a link between Zika virus infection and different congenital central ne
42 ned questions about the relationship between Zika virus infection and each of the two clinical outcom
44 ded both for appraisal of the evidence about Zika virus infection and for the next public health thre
45 tion of available evidence from outbreaks of Zika virus infection and GBS is that Zika virus infectio
46 nfant laboratory evidence of possible recent Zika virus infection and outcomes reported in the contin
47 tor that contributes to host protection from Zika virus infection and virus-induced neuropathological
50 g findings as primary outcomes of congenital Zika virus infection are variable regarding inclusion cr
51 imaging findings associated with congenital Zika virus infection as found in the Instituto de Pesqui
53 activity against Chikungunya, West Nile, and Zika virus infection, as demonstrated with a novel cocul
55 e infecting primary flavivirus; with primary Zika virus infection being most protective from Zika vir
56 with laboratory evidence of possible recent Zika virus infection, birth defects potentially related
57 ts with microcephaly or laboratory-confirmed Zika virus infection but not for all infants potentially
58 ome sequences from three people positive for Zika virus infection by real-time reverse transcription
59 or infants with birth defects after maternal Zika virus infection by trimester of infection and mater
63 irst in vivo evidence that prior exposure to Zika virus infection can enhance Dengue infection, which
64 nt of ZIKV-3'UTR-LAV is warranted for humans.Zika virus infection can result in congenital disorders
65 is is the first study providing evidence for Zika virus infection causing Guillain-Barre syndrome.
68 none of 62 controls had laboratory-confirmed Zika virus infection; crude overall OR 55.5 (95% CI 8.6-
70 anial US and laboratory findings of maternal Zika virus infection during pregnancy confirmed with RNA
71 fection and clusters of microcephaly is that Zika virus infection during pregnancy is a cause of cong
72 ng the risk of birth defects associated with Zika virus infection during pregnancy may help guide com
74 aneiro, Brazil, who presented with suspected Zika virus infection during pregnancy were referred to o
82 Definite cases had laboratory evidence of Zika virus infection; highly probable cases presented sp
83 CI 2.3-3.6) the odds of testing positive for Zika virus infection if they came from the same district
84 rospective cohort of 16 women with suspected Zika virus infection in a highly endemic area, and discu
86 temporary acute flaccid paralysis caused by Zika virus infection in aged interferon alphabeta-recept
87 are providers should consider a diagnosis of Zika virus infection in children with fever, rash, arthr
89 e included, as well as those with documented Zika virus infection in fluid or tissue (n = 17, confirm
91 112 with polymerase chain reaction-confirmed Zika virus infection in maternal specimens, 24 infants (
92 The molecular basis dictating specificity of Zika virus infection in neural stem cells (NSCs) remains
95 own about the historical and current risk of Zika virus infection in southeast Asia, where the mosqui
96 ring the December 2015-July 2016 outbreak of Zika virus infection in southeast Brazil (Natural Histor
99 ding Zika virus disease severity is to study Zika virus infection in the context of prior flavivirus
100 ctrum of findings associated with congenital Zika virus infection in the IPESQ in northeastern Brazil
102 ers would be useful for medical diagnosis of Zika virus infection in various aptamer-based diagnostic
103 large populations at risk of mosquito-borne Zika virus infection include India (67 422 travellers ar
106 cient evidence to conclude that intrauterine Zika virus infection is a cause of microcephaly and seri
108 ns from individuals with a typical course of Zika virus infection is low.Zika virus (ZIKV) is present
110 m of adverse reproductive outcomes caused by Zika virus infection is not yet determined, a distinctiv
111 months, the cumulative incidence of prenatal Zika virus infections is likely to decrease by 17% to 44
113 cross-reactive antibodies induced following Zika virus infection might enhance subsequent Dengue inf
116 quitoes during epidemics.IMPORTANCE Although Zika virus infection of pregnant women can result in con
118 fetuses or infants with prenatal exposure to Zika virus infection only in the second or third trimest
120 perinfection of Mayaro virus atop a previous Zika virus infection resulted in increased Mayaro virus
122 adiologists who were blinded to the maternal Zika virus infection status independently reviewed crani
123 f antibiotics were more potent inhibitors of Zika virus infection than the protease, suggesting they
124 odies isolated from a patient with an active Zika virus infection that potently neutralized virus inf
128 and the risk of microcephaly associated with Zika virus infection was 95 cases (34-191) per 10,000 wo
131 alivary factors that modulate mosquito-borne Zika virus infection, we focused on antigenic proteins i
132 luate the prospect of using ferrets to study Zika virus infection, we injected seven pregnant jills w
135 ed (n = 17) and presumed (n = 28) congenital Zika virus infections were similar, with ventriculomegal
136 nalysed data from individuals with suspected Zika virus infection who presented at hospitals througho
137 ss of mucocutaneous findings associated with Zika virus infection will support its early recognition
138 or those with brain findings suspicious for Zika virus infection, with intracranial calcifications (