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1 eatment of the neurological complications of Zika virus infection.
2  of neurological symptoms, suggesting recent Zika virus infection.
3 nd two spontaneous abortions associated with Zika virus infection.
4 interaction and proteasome degradation after Zika virus infection.
5 ogic fetal outcomes during murine congenital Zika virus infection.
6  to confirm microcephaly and assess previous Zika virus infection.
7 sis and disease outcomes in a mouse model of Zika virus infection.
8  and kinetics with those elicited by natural Zika virus infection.
9 r vesicles (EVs) in semen and saliva inhibit Zika virus infection.
10 d December, 2017, 368 were confirmed to have Zika virus infection.
11 gnized by human antibodies following natural Zika virus infection.
12 st NeSt1 might protect people against severe Zika virus infection.
13 t for the prevention of mosquito-transmitted Zika virus infection.
14 acid-editing deaminase), inherently restrict Zika virus infection.
15 (EC(68): 300-600 nM) and in a mouse model of Zika virus infection.
16 ethal mosquito-borne-but not needle-injected-Zika virus infection.
17 es suspected of resulting from a symptomatic Zika virus infection.
18  organoid development, culture protocols and Zika virus infection.
19 l Zika syndrome and serological evidence for Zika virus infection.
20  imaging findings associated with congenital Zika virus infection.
21 ine the association between microcephaly and Zika virus infection.
22 ay be the only initial finding in congenital Zika virus infection.
23 inhibitors to identify several inhibitors of Zika virus infection.
24 this neuronal population as a consequence of Zika virus infection.
25 ociation between microcephaly and congenital Zika virus infection.
26  studies supported a causal association with Zika virus infection.
27 resumed or laboratory-confirmed intrauterine Zika virus infection.
28 il, which was later attributed to congenital Zika virus infection.
29 rocephaly epidemic is a result of congenital Zika virus infection.
30  on the incidence and prevalence of prenatal Zika virus infection.
31 ogic correlation in a patient with the acute Zika virus infection.
32 o seen as common outcomes of first trimester Zika virus infections.
33 boratory tests and no controls had confirmed Zika virus infections.
34 nital infections or are unique to congenital Zika virus infection: (1) severe microcephaly with parti
35  samples from 54 people with suspected acute Zika virus infection, 76 infants with suspected microcep
36  (95% CI 13.0-infinity) for microcephaly and Zika virus infection after adjustments.
37 id and systematic review about links between Zika virus infection and (a) congenital brain abnormalit
38                  Due to associations between Zika virus infection and a range of fetal maladies(1,2),
39 tion of available evidence from outbreaks of Zika virus infection and clusters of microcephaly is tha
40 gs provide strong evidence of a link between Zika virus infection and different congenital central ne
41  is how dengue virus (DENV) immunity impacts Zika virus infection and disease.
42 ned questions about the relationship between Zika virus infection and each of the two clinical outcom
43 ing the spread of emerging diseases, such as Zika virus infection and Ebola virus disease.
44 ded both for appraisal of the evidence about Zika virus infection and for the next public health thre
45 tion of available evidence from outbreaks of Zika virus infection and GBS is that Zika virus infectio
46 nfant laboratory evidence of possible recent Zika virus infection and outcomes reported in the contin
47 tor that contributes to host protection from Zika virus infection and virus-induced neuropathological
48                                              Zika virus infections and suspected microcephaly cases h
49    The ocular manifestations and sequelae of Zika virus infection are not well known.
50 g findings as primary outcomes of congenital Zika virus infection are variable regarding inclusion cr
51  imaging findings associated with congenital Zika virus infection as found in the Instituto de Pesqui
52 NA decay and RNA splicing are compromised by Zika virus infection as well as by sfRNA alone.
53 activity against Chikungunya, West Nile, and Zika virus infection, as demonstrated with a novel cocul
54 trated to aid the radiologist in identifying Zika virus infection at imaging.
55 e infecting primary flavivirus; with primary Zika virus infection being most protective from Zika vir
56  with laboratory evidence of possible recent Zika virus infection, birth defects potentially related
57 ts with microcephaly or laboratory-confirmed Zika virus infection but not for all infants potentially
58 ome sequences from three people positive for Zika virus infection by real-time reverse transcription
59 or infants with birth defects after maternal Zika virus infection by trimester of infection and mater
60                                              Zika virus infection can be prenatally passed from a pre
61                            We also find that Zika virus infection can boost DENV immunity and produce
62                                   IMPORTANCE Zika virus infection can cause mild fever, rash, and mus
63 irst in vivo evidence that prior exposure to Zika virus infection can enhance Dengue infection, which
64 nt of ZIKV-3'UTR-LAV is warranted for humans.Zika virus infection can result in congenital disorders
65 is is the first study providing evidence for Zika virus infection causing Guillain-Barre syndrome.
66          24 (80%) of 30 mothers of cases had Zika virus infection compared with 39 (64%) of 61 mother
67                                              Zika virus infection could also cause Guillain-Barre syn
68 none of 62 controls had laboratory-confirmed Zika virus infection; crude overall OR 55.5 (95% CI 8.6-
69                                              Zika virus infection during pregnancy can cause congenit
70 anial US and laboratory findings of maternal Zika virus infection during pregnancy confirmed with RNA
71 fection and clusters of microcephaly is that Zika virus infection during pregnancy is a cause of cong
72 ng the risk of birth defects associated with Zika virus infection during pregnancy may help guide com
73                         Laboratory-confirmed Zika virus infection during pregnancy was defined as det
74 aneiro, Brazil, who presented with suspected Zika virus infection during pregnancy were referred to o
75 ing the association between microcephaly and Zika virus infection during pregnancy.
76 matic pregnant women who tested negative for Zika virus infection during pregnancy.
77 hers had polymerase chain reaction-confirmed Zika virus infection during pregnancy.
78 lete protection, which suggests that primary Zika virus infection elicits protective immunity.
79                                   Congenital Zika virus infection has stimulated great international
80 tiveness of this policy in reducing prenatal Zika virus infection has yet to be quantified.
81                                   Dengue and Zika virus infections have been linked to antibody-media
82    Definite cases had laboratory evidence of Zika virus infection; highly probable cases presented sp
83 CI 2.3-3.6) the odds of testing positive for Zika virus infection if they came from the same district
84 rospective cohort of 16 women with suspected Zika virus infection in a highly endemic area, and discu
85       Laboratory evidence of possible recent Zika virus infection in a maternal, placental, fetal, or
86  temporary acute flaccid paralysis caused by Zika virus infection in aged interferon alphabeta-recept
87 are providers should consider a diagnosis of Zika virus infection in children with fever, rash, arthr
88 d to epidemiologic data from 2015 to 2016 on Zika virus infection in Colombia.
89 e included, as well as those with documented Zika virus infection in fluid or tissue (n = 17, confirm
90 tion in southeast Brazil (Natural History of Zika Virus Infection in Gestation cohort).
91 112 with polymerase chain reaction-confirmed Zika virus infection in maternal specimens, 24 infants (
92 The molecular basis dictating specificity of Zika virus infection in neural stem cells (NSCs) remains
93                                 Experimental Zika virus infection in non-human primates results in ac
94                                    Exposure: Zika virus infection in pregnant women.
95 own about the historical and current risk of Zika virus infection in southeast Asia, where the mosqui
96 ring the December 2015-July 2016 outbreak of Zika virus infection in southeast Brazil (Natural Histor
97          Background: Because of the risk for Zika virus infection in the Americas and the links betwe
98                       The control of primary Zika virus infection in the blood was correlated with ra
99 ding Zika virus disease severity is to study Zika virus infection in the context of prior flavivirus
100 ctrum of findings associated with congenital Zika virus infection in the IPESQ in northeastern Brazil
101                                      Despite Zika virus infection in the spinal cord, motor neurons w
102 ers would be useful for medical diagnosis of Zika virus infection in various aptamer-based diagnostic
103  large populations at risk of mosquito-borne Zika virus infection include India (67 422 travellers ar
104                                              Zika virus infection induced detectable Dengue cross-rea
105                                              Zika virus infection induces the degradation of STAT2, a
106 cient evidence to conclude that intrauterine Zika virus infection is a cause of microcephaly and seri
107 eaks of Zika virus infection and GBS is that Zika virus infection is a trigger of GBS.
108 ns from individuals with a typical course of Zika virus infection is low.Zika virus (ZIKV) is present
109 ective, and rapidly scalable vaccine against Zika virus infection is needed.
110 m of adverse reproductive outcomes caused by Zika virus infection is not yet determined, a distinctiv
111 months, the cumulative incidence of prenatal Zika virus infections is likely to decrease by 17% to 44
112            The public health implications of Zika virus infection lend special importance to identify
113  cross-reactive antibodies induced following Zika virus infection might enhance subsequent Dengue inf
114                                              Zika virus infection occurred at 16-33 weeks gestation.
115 he antiviral response, indicated by enhanced Zika virus infection of cells depleted of RAB1B.
116 quitoes during epidemics.IMPORTANCE Although Zika virus infection of pregnant women can result in con
117 an semen inhibited, in a laboratory setting, Zika virus infection of reproductive tissues.
118 fetuses or infants with prenatal exposure to Zika virus infection only in the second or third trimest
119 ective, and durable protection against human Zika virus infections or syndromes.
120 perinfection of Mayaro virus atop a previous Zika virus infection resulted in increased Mayaro virus
121                          In a mouse model of Zika virus infection, Shfl(-/-) knockout mice exhibit re
122 adiologists who were blinded to the maternal Zika virus infection status independently reviewed crani
123 f antibiotics were more potent inhibitors of Zika virus infection than the protease, suggesting they
124 odies isolated from a patient with an active Zika virus infection that potently neutralized virus inf
125            Despite the medical importance of Zika virus infection, the mechanism of viral replication
126 ss both IL-27 and its receptor, and restrict Zika virus infection through IL-27 signaling.
127 potential causes of microcephaly: congenital Zika virus infection, vaccines, and larvicides.
128 and the risk of microcephaly associated with Zika virus infection was 95 cases (34-191) per 10,000 wo
129                In 32 (35%) cases, congenital Zika virus infection was confirmed by laboratory tests a
130 ociation between microcephaly and congenital Zika virus infection was confirmed.
131 alivary factors that modulate mosquito-borne Zika virus infection, we focused on antigenic proteins i
132 luate the prospect of using ferrets to study Zika virus infection, we injected seven pregnant jills w
133                   In November 2015, cases of Zika virus infection were recorded in Cabo Verde (Africa
134                                     Cases of Zika virus infection were reported throughout the year,
135 ed (n = 17) and presumed (n = 28) congenital Zika virus infections were similar, with ventriculomegal
136 nalysed data from individuals with suspected Zika virus infection who presented at hospitals througho
137 ss of mucocutaneous findings associated with Zika virus infection will support its early recognition
138  or those with brain findings suspicious for Zika virus infection, with intracranial calcifications (

 
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