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1 he medial vestibular nucleus and the nucleus abducens).
2 nce of the target nucleus from the accessory abducens.
3 t in the saccadic premotor pathway), whereas abducens activation by the pretectum-vestibular pathway
4 t is typically attributed to agenesis of the abducens and facial cranial nerves.
5 terminals projected axons to the ipsilateral abducens and oculomotor nuclei, respectively.
6 g., oculomotor, trochlear, trigeminal motor, abducens, and vagal motor nuclei) contain protocadherin-
7  fibers travel independent of the trochlear, abducens, and Vidian nerves, but, otherwise, they use th
8 ent allows for the possibility that in vitro abducens conditioning is generated by coincident CS-US d
9 m incorporates the oculomotor, trochlear and abducens cranial nerve nuclei as well as the parabigemin
10 cleus prepositus hypoglossi, all vestibular, abducens, cuneate, and lateral reticular nuclei, labeled
11 s in the physiologic signals conveyed by the abducens internuclear (eye velocity and eye position) an
12                                          The abducens internuclear and ascending tract of Deiters (AT
13                                              Abducens internuclear and ascending tract of Deiters (AT
14 orphology and soma-dendritic distribution of abducens internuclear and ATD synaptic endings are corre
15  pre/postsynaptic membrane profile, both the abducens internuclear and ATD synaptic endings are label
16                                              Abducens internuclear neurons are responsible for conjug
17  of which were VEGF immunopositive, and that abducens internuclear neurons expressed the VEGF recepto
18                                              Abducens internuclear neurons of host animals showed a c
19 haracteristics and synaptology of axotomized abducens internuclear neurons, which mediate gaze conjug
20 motoneurons receive two main pontine inputs: abducens internuclear neurons, whose axons course throug
21 entials in comparison to those evoked by the abducens internuclear pathway as determined electrophysi
22 a synaptogenic compensatory mechanism of the abducens internuclear pathway that could lead to the obs
23                                          The abducens internuclear pathway through the medial longitu
24 rograde horseradish peroxidase labeling, the abducens internuclear projection is predominantly, if no
25                              The majority of abducens internuclear synaptic endings contact distal de
26                                              Abducens internuclear synaptic endings furthermore have
27 the excitatory neurotransmitters utilized by abducens internuclear synaptic endings whose burst-tonic
28         With the exception of oculomotor and abducens MNs, the survival of all other populations of s
29 AL genes and likely plays a critical role in abducens motoneuron development.
30 had ipsilateral projections terminating near abducens motoneurons and collateralized extensively with
31 tudy investigates whether different types of abducens motoneurons exist that become active during dif
32            During horizontal head movements, abducens motoneurons form the final element of the refle
33  lack of retrogradely delivered VEGF renders abducens motoneurons into an axotomy-like state.
34 re, we studied a possible differentiation of abducens motoneurons into subtypes by evaluating their m
35 equencies and velocities allowed subdividing abducens motoneurons into two subgroups, one encoding th
36 e-nerve stimulation or stimulation of single abducens motoneurons or axons.
37    Our data revealed that intact, noninjured abducens motoneurons retrogradely deprived of VEGF exhib
38 e-stabilizing eye movements are commanded by abducens motoneurons that combine different sensory inpu
39 neurons in turtle respond in the same way as abducens motoneurons to horizontal rotations, an unusual
40 n the firing activity and synaptic inputs of abducens motoneurons were completely restored after VEGF
41 shes inhibitory postsynaptic potentials onto abducens motoneurons within 2 days postinjection, and tr
42 ntralateral projections collateralizing near abducens motoneurons, consistent with a role in disconju
43 ter) densely covering the surface of control abducens motoneurons.
44 so affect the physiology of otherwise intact abducens motoneurons.
45 d contralateral projections terminating near abducens motoneurons; these cells collateralized extensi
46 f several transcription factors critical for abducens motor neuron identity, including MAFB, or by he
47 l and contralateral eye movements across the abducens motor neuron pool may provide a basis for learn
48  cell type specific antibodies, that somatic abducens motor neurons and a small subset of OPCs arise
49 ry and trigeminal nerve synaptic inputs onto abducens motor neurons are in spatial proximity because
50 contribute to the selective vulnerability of abducens motor neurons in this disorder.
51 ing experiments demonstrated that individual abducens motor neurons receive inputs from both nerves a
52 remain in the cell cycle and fail to produce abducens motor neurons, and OPCs are entirely lacking in
53                          However, 66% of the abducens motor neurons, which innervate the ipsilateral
54 cal spinal cord neurons closely connected to abducens motor neurons.
55 tons that convey the conditioned stimulus to abducens motor neurons.
56 om eye movements elicited by stimulating the abducens motor neurons.
57 CS or US information on retrogradely labeled abducens motor neurons.
58 primarily the soma and proximal dendrites of abducens motor neurons.
59 tions from auditory and trigeminal nerves to abducens motor neurons.
60 eptor 4 (GluR4) AMPA receptor subunit in the abducens motor nuclei, but not with NMDAR1 or GluR1.
61 e elicited via the red nucleus and accessory abducens motorneurons.
62                                          The abducens nerve (CN6) was traced anteriorly from the deep
63  motor nerves were typically small, with the abducens nerve (cranial nerve [CN]6) often nondetectable
64       Neural discharge was recorded from the abducens nerve after a single shock unconditioned stimul
65  random sinusoidal cycles, we stimulated the abducens nerve and observed the resultant eye movements.
66                                      Stalled abducens nerve bundles did not reach the orbit, resultin
67 l correlate of this response recorded in the abducens nerve can be conditioned entirely in vitro usin
68 , we demonstrate that selectively disrupting abducens nerve development is sufficient to cause second
69 d that in vitro classical conditioning of an abducens nerve eye-blink response is generated by NMDA r
70 o imaging of Chn1KI/KI mice revealed stalled abducens nerve growth and selective trochlear and first
71      Postmortem studies of DRS have reported abducens nerve hypoplasia and aberrant innervation of th
72 with subarachnoid CN3 hypoplasia, occasional abducens nerve hypoplasia, and subclinical ON hypoplasia
73 ontine abducens nuclear defects, rather than abducens nerve involvement.
74 anifestations of IIH such as papilledema and abducens nerve palsy are well recognized, but less commo
75 imaging of patients with isolated unilateral abducens nerve palsy without other ocular motility disor
76  for human eye movement dysfunctions such as abducens nerve palsy.
77 Cranial nerve assessment revealed mild right abducens nerve palsy; neurologic examination was otherwi
78                                          The abducens nerve phenotype ranges from complete absence, t
79 e-reversed" responses were poorly defined in abducens nerve recordings.
80                                A conditioned abducens nerve response is generated in in vitro brainst
81  model of the classically conditioned turtle abducens nerve response, we investigated the effect of c
82 t the primary cause of DRS is failure of the abducens nerve to fully innervate the lateral rectus mus
83 mice did not have DRS, and embryos displayed abducens nerve wandering distinct from the Chn1KI/KI phe
84 nce of Listing's law, we microstimulated the abducens nerve with the eye at different initial vertica
85 erized by a failure of cranial nerve VI (the abducens nerve) to develop normally, resulting in restri
86  slope of CR acquisition was recorded in the abducens nerve.
87  nerve in DRS is secondary to absence of the abducens nerve.
88 is linked to Duane syndrome, a defect in the abducens nerve/lateral rectus muscle connection.
89                Intraorbital and intracranial abducens nerves (CN6) were small to absent, particularly
90 ysfunction of the oculomotor, trochlear, and abducens nerves and/or the muscles that they innervate.
91 ted individuals demonstrated small or absent abducens nerves in all four, small oculomotor nerve in o
92 rded from pairs of oculomotor, trochlear, or abducens nerves of an in vitro turtle brainstem preparat
93 f left oculomotor, right trochlear and right abducens nerves were approximately aligned with leftward
94 on of motor columns, loss of the phrenic and abducens nerves, and intercostal nerve pathfinding defec
95 hat include misrouting of the oculomotor and abducens nerves.
96                Two-photon calcium imaging of abducens neurons in control and dscaml1 mutant animals c
97 interactions in distinct motor neuron pools: abducens neurons use bidirectional ephrin signaling via
98 bilateral horizontal gaze palsy from pontine abducens nuclear defects, rather than abducens nerve inv
99 tion from contralateral facial and accessory abducens nuclei and by their synaptic activation from th
100 an anterograde tracer into the oculomotor or abducens nuclei and combined tracer visualization with i
101 rom the Hoxa1 domain, such as the facial and abducens nuclei and nerves as well as r4 neural crest ce
102 and motoneurons in oculomotor, trochlear and abducens nuclei that dictate eye rotations in terms of t
103 rons in hypoglossal, facial, trigeminal, and abducens nuclei.
104 toneurons in the oculomotor-, trochlear- and abducens nuclei.
105 e eyeblink conditioned while their accessory abducens nucleus (ACC), facial nucleus (FN), and surroun
106  anterogradely transported biocytin from the abducens nucleus and the ventral lateral vestibular nucl
107 he CFN to contralateral IBNs and then to the abducens nucleus can account for these effects.
108                               Neurons in the abducens nucleus discharge homogeneously in relation mai
109 F neurons located in a region ventral to the abducens nucleus produced 42 significant SpikeTA effects
110 pontine nuclei, vagus nerve, inferior olive, abducens nucleus, and motor trigeminal nucleus; protein
111 ead burst neuron, tonic neuron, interneuron, abducens nucleus, and oculomotor nucleus, is developed t
112 oneurons distributed intermingled within the abducens nucleus, with MIF motoneurons being smaller and
113 motor-, 72.2% in trochlear- and 78.3% in the abducens nucleus.
114 ly identified MIF and SIF motoneurons in the abducens nucleus.
115 that give rise to somatic motoneurons of the abducens nucleus.
116 f the oculomotor (nIII), trochlear (nIV) and abducens (nVI) cranial nerve nuclei.
117                                              Abducens palsy does not alter normal eye translation dur
118 s revealed lateral incomitance suggestive of abducens palsy not detected by clinical examination.
119 r at distance than at near can be related to abducens palsy or to divergence insufficiency.
120 ial postoperative follow-up in patients with abducens palsy undergoing IRT shows a significant improv
121 bismus, horizontal gaze palsy, and bilateral abducens palsy.
122 n during horizontal duction in patients with abducens palsy.
123  associated with disconjugate gaze mimicking abducens palsy.
124 dren, unlike the adults, likely had a subtle abducens paresis rather than divergence insufficiency.
125 ojected to the contralateral and ipsilateral abducens, respectively, and GABAergic neurons projected
126  travel to the orbit in association with the abducens, trochlear, and Vidian nerves.
127  in corticospinal neurons, exhibited similar abducens wandering that paralleled previously reported g
128 nerve was most severely hypoplastic, but the abducens was also affected.
129 sion of the CR (facial nucleus and accessory abducens) were reversibly inactivated with microinjectio
130 le or muscle unit contraction in response to abducens whole-nerve stimulation or stimulation of singl

 
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