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1 tempt to differentiate it from RBBB and LAHB aberrancy.
2 t for IRE1alpha's responsiveness to membrane aberrancy.
3 e cycling to promote integration rather than aberrancy.
4 c replication stress, and signs of telomeric aberrancy.
5 d safety and subgroup analysis based on TP53 aberrancy.
6 M) that results in oncogenic transcriptional aberrancies.
7 ey also exhibit morphological and functional aberrancies.
8 tion near the N terminus results in multiple aberrancies.
9 rrest and exhibited 1 to 11 immunophenotypic aberrancies.
10 issues are also vulnerable to glucocorticoid aberrancies.
11                 Although numerous epigenetic aberrancies accumulate in melanoma, their contribution t
12 e investigated the frequency of EGFR pathway aberrancies among African American patients with NSCLC,
13  may arise from transcriptional or chromatin aberrancies and engage prostate tumorigenic mechanisms.
14 d upregulation of aurora kinases, centrosome aberrancies, and aneuploidy.
15 o metaphase chromosome misalignment, spindle aberrancy, and eventual cell death.
16 omena as Wolff-Parkinson-White syndrome, QRS aberrancy, and multiple reentrant pathways that were cru
17 agnosis of supraventricular tachycardia with aberrancy, and the use of this technique can help the op
18 ins unknown, however, whether such molecular aberrancies are causally related to arrhythmogenesis in
19 disease, but the mechanisms underlying these aberrancies are unknown.
20 lones of cells that have any type of somatic aberrancy associated with an increased propensity to bec
21 ation, among others, are among the principal aberrancies driving CRPC growth.
22 al cell populations and recognize phenotypic aberrancies, even when present in a small proportion of
23                          Furthermore, tissue aberrancies generated from oncogenic Hras, and even muta
24 and therapeutic targeting of this epigenetic aberrancy has not been fully explored.
25                                              Aberrancies in AMPAR-mediated signaling are associated w
26                                              Aberrancies in IgA1 glycosylation have been linked to th
27 lecular factors underlying these age-related aberrancies in immune signaling are unclear.
28 hogenic consequences of repeat expansions as aberrancies in normal gene regulation.
29 studies have addressed intestinal microbiota aberrancies in pediatric CD, but the results are inconsi
30                                              Aberrancies in regulation lead to corruption of signal t
31 and hence no indication of nonsense-mediated aberrancies in splicing.
32 fixed clinical factors with the influence of aberrancies in subsequent physiology has superior perfor
33                       Our findings highlight aberrancies in task-evoked modulation of SN and FPN conn
34  such diseases is to identify characteristic aberrancies in the biological pathways and molecular net
35                Crucially, pDNN models reveal aberrancies in the geometry of manifold structure, provi
36 asmablastic features and is characterized by aberrancies in the molecular networks controlled by IRF4
37      The majority of human malignancies have aberrancies in the Retinoblastoma (Rb) pathway.
38                                              Aberrancies in the tumor microvasculature limit the syst
39 al, and functional brain imaging studies for aberrancies in these networks and their dynamic temporal
40             Our studies further suggest that aberrancies in this delicate degradation pathway might c
41 rtant role of DNA methylation in DLBCL where aberrancies in transcription factors were frequently obs
42                                              Aberrancy in cell cycle progression is one of the fundam
43                        We confirmed the same aberrancy in IgA1 secreted by the human DAKIKI B cell li
44 nous IRE1alpha protein to recognize membrane aberrancy in mammalian cells.
45  systemic lupus erythematosus (SLE) involves aberrancy in multiple components of the immune system in
46 on-mediated PCR) assay and found significant aberrancy in promoter methylation patterns compared with
47 Waf1/Cip1) showed normal development with no aberrancy in their cell cycles, and antisense p21(Waf1/C
48           Skin epithelial stem cells correct aberrancies induced by oncogenic mutations.
49 ation that isolated the response to membrane aberrancy induced by palmitate from unfolded protein str
50 omes the multitude of genetic and epigenetic aberrancies inherent in human cancers to restore normal
51                            The most frequent aberrancy is the "common deletion" that involves a 4,977
52 review of the mechanics of the UPS, describe aberrancies leading to cancer, and give an overview of c
53  is unknown, but is critical, as hemodynamic aberrancies may cause severe clinical deterioration.
54 ms and insights into ways in which molecular aberrancies occur are becoming better understood through
55 ic lethal, whereas haploinsufficiency caused aberrancies of adult skin, but not a frank cEDS-like phe
56                       Unlike in primary MDS, aberrancies of chromosome 5 and 20 were infrequent.
57 ndrogen synthesis as well as the most common aberrancies of the AR confirm that prostate cancer retai
58 ptor (EGFR) -targeting agents and in genetic aberrancies of the EGFR pathway according to ethnic back
59 thesis and biological methylation reactions, aberrancies of which have all been implicated in carcino
60                        Certainly, epigenetic aberrancies play a fundamental role in propagation of th
61                                              Aberrancy produced via tryptophan depletion results in a
62  N-glycan content and the timing of N-glycan aberrancy relative to disease onset were compared in hea
63      On receiver operator curve analysis, an aberrancy scale feature computed by comparison with cont
64 er-lived plasma cells as well, perhaps as an aberrancy stemming from its evolutionary roots from an a
65 alous arterial patterning may cause vascular aberrancies that often require surgical treatment.
66 functionally group variants and link complex aberrancies to phenotypic severity, serving as a resourc
67                                Glycosylation aberrancy was comparable in patients with and those with
68 ly differentiating LPF-VT from RBBB and LAHB aberrancy was developed.
69                                   The growth aberrancy was normalized after restoring the distal enha
70  QT duration, were unchanged, although minor aberrancies were noted in 20% (16/80) of the G628S mice
71                                        These aberrancies were remedied by nCUR+INS or INS but not by
72  cell-cycle control can lead to oncogenesis, aberrancies within the UPS pathway can result in a malig
73 E1alpha also directly recognize the membrane aberrancy wrought by lipid perturbation.