ライフサイエンスコーパス: acaricide

1 ompounds comprising these pheromones with an acaricide.

2 ed reduced irritancy and repellency to MET-I acaricides.

3 ress T. urticae, growers often apply various acaricides.

4 ils mostly attributed to insecticides and/or acaricides.

5 es aggravated by tick resistance to chemical acaricides.

6 howed preferential oviposition away from the acaricides.

7 trains for physiological resistance to these acaricides affected the behavioral response of T. urtica

8 ol remains dominated by the use of synthetic acaricides, although resistance and treatment failure ar

9 lters (n = 2) and, using a factorial design, acaricide and HDM impermeable bedding covers in isolatio

10 Acaricides and vaccines have been used to try to keep ti

11 een proven difficult given the resistance to acaricides and vaccines observed in ticks.

12 cides, 36 herbicides, 25 insecticides and/or acaricides, and two safeners) in 47 soils sampled across

13 Of the interventions studied to date, acaricides appear to be the most promising, although the

14 Amitraz is a formamidine acaricide applied to hives to manage Varroa destructor,

15 en proposed and validated to determine seven acaricides (atrazine, chlorpyrifos, chlorfenvinphos, alp

16 Used as recommended, acaricide barrier sprays do not significantly reduce the

17 tabolism of coumaphos, a widely used in-hive acaricide, by approximately 60%.

18 odione, procymidone and vinclozolin) and one acaricide (dicofol) in still and fortified wines was dev

19 Here, we show that the acaricide etoxazole inhibits chitin biogenesis in Tetran

20 n on the behavioral responses elicited after acaricide exposure.

21 MET-I acaricides (fenazaquin, fenpyroximate, and pyrabiden) an

22 other insecticides, fungicides, herbicides, acaricides, growth regulators and veterinary drugs in ho

23 The preparation of an acaricide has been developed as an example of synthetic

24 studies, a single springtime application of acaricide has been shown to kill 68%-100% of ticks.

25 able plants worldwide, and its resistance to acaricides has quickly developed.

26 opportunity to develop Varroa mite-selective acaricides, hence, expedited translational processes.

27 Finally, of the seven acaricides investigated in several honey samples only ta

28 One of the key acaricides is the organophosphorus (OP) proinsecticide c

29 e of HDM impermeable bedding covers (n = 4), acaricides (n = 2), high-efficiency particulate air filt

30 mode of action of the novel insecticide and acaricide nodulisporic acid.

31 ex I (MET-I) and mite growth inhibitor (MGI) acaricides on multiple T. urticae strains.

32 corporating neutering/spaying and widespread acaricide programs may also prove beneficial.

33 Chemical acaricides remain the main control method, but repeated

34 Most studies on acaricide resistance have focused on the acute contact t

35 r, their contribution to host adaptation and acaricide resistance in arthropods, such as T. urticae,

36 Multiple acaricide resistance in Tetranychus urticae continues to

37 nduced genes that are strongly implicated in acaricide resistance in the respective strain.

38 Worrying observations include increasing acaricide resistance in the varroa population and sinkin

39 chnology might lead to environmental damage, acaricide resistance in tick populations and a possible

40 Acaricide resistance of the intermediate host, paucity o

41 We tested the effect of acaricide resistance on contact toxicity, irritancy and

42 is study highlights negative consequences of acaricide resistance that can potentially affect T. urti

43 However, the role of CPR on the formation of acaricide-resistance in T. cinnabarinus is still unclear

44 genome-wide transcriptional responses in an acaricide resistant strain of the spider mite Tetranychu

45 results clearly show that reestablishment of acaricide-resistant B. microplus in the United States wo

46 l activities against the different stages of acaricide-resistant Rhipicephalus annulatus (Say).

47 wide climatic changes, human activities, and acaricide-resistant tick strains, necessitates the devel

48 development of honey bee-safe and selective acaricides targeting the Varroa mite-specific neuropepti

49 toxins produced by fungi; antimicrobials and acaricides that are introduced by beekeepers; and fungic

50 h public health authorities recommend use of acaricides to control tick populations in yards, the eff

51 ncountered pesticides in-hive in the form of acaricides to control Varroa destructor, a devastating p

52 The limited number of conventional acaricides to reduce Varroa mites and prevent disease in

53 sting ticks was significantly lower (63%) on acaricide-treated properties, there was no difference be

54 ch population was managed using one of three acaricide treatment regimes: always amitraz, always spin

55 These were: (i) the absence of acaricide use (OR 5.61; 95% CI 2.97-10.94); (ii) the pre

56 Multivariable analysis found species, acaricide use, and period of sampling were significant f

57 Residues of acaricides used for sanitary treatments, coumaphos and t

58 ented to quickly develop resistance to these acaricides which directly cause control failures.

59 t offers a sustainable approach by combining acaricides with biological controls, vaccines, resistant

60 ave focused on the acute contact toxicity of acaricides with little or no information on the behavior

61 Highly selective acaricides with low toxicity to bees are used internatio

62 We also tested whether acaricides with similar physiological target site/mode o