ライフサイエンスコーパス: acetylcholinesterase

1 etylcholinesterase) or nonexisting (synaptic acetylcholinesterase).

2 sly validated for imaging cerebral levels of acetylcholinesterase.

3 resulted in a comparable enhanced release of acetylcholinesterase.

4 he essential oil was the most active against acetylcholinesterase.

5 , octopamine synapses, and the inhibition of acetylcholinesterase.

6 tor neuron function and stability, including acetylcholinesterase.

7 components of decay, even in the presence of acetylcholinesterase.

8 re homologous to the dimerization helices of acetylcholinesterase.

9 via a mechanism that depends on both p53 and acetylcholinesterase.

10 f cholinergic transmission via inhibition of acetylcholinesterase.

11 s, in addition to inhibiting the activity of acetylcholinesterase.

12 Acetylcholinesterase 1 (AmAChE1) of the honey bee, Apis

13 potential label-free detection system, using acetylcholinesterase (acetylcholine acetylhydrolase; EC

14 icity assessment, including determination of acetylcholinesterase (AChE) activity, genotoxicity and o

15 s acid (HOCl) assays, and their potential as acetylcholinesterase (AChE) and butyrylcholinesterase (B

16 or their potential inhibitory effect on both acetylcholinesterase (AChE) and butyrylcholinesterase (B

17 en advantage of the distinct localization of acetylcholinesterase (AChE) and butyrylcholinesterase (B

18 tinus L. was investigated via inhibition of acetylcholinesterase (AChE) and butyrylcholinesterase (B

19 pounds with nanomolar potencies toward human acetylcholinesterase (AChE) and butyrylcholinesterase (B

20 The cholinesterases, acetylcholinesterase (AChE) and butyrylcholinesterase, a

21 based on poly(o-phenylenediamine) (PoPD) and acetylcholinesterase (AChE) and choline oxidase (ChO) en

22 by co-immobilizing covalently, a mixture of acetylcholinesterase (AChE) and choline oxidase (ChO) on

23 tion is constructed by immobilizing enzymes, acetylcholinesterase (AChE) and choline oxidase (ChO), o

24 wed by chemical cross-linking of the enzymes acetylcholinesterase (AChE) and choline oxidase (ChO).

25 We show that miR-608 targets acetylcholinesterase (AChE) and demonstrate weakened miR

26 ouble stranded RNA (dsRNA) homologous to the acetylcholinesterase (AChE) and ecdysone receptor (EcR)

27 sites of bovine carbonic anhydrase (BCA) or acetylcholinesterase (AChE) and inhibit their catalytic

28 atform for simple and sensitive detection of acetylcholinesterase (AChE) and its inhibitor using a ca

29 erapeutics for AD through dual inhibition of acetylcholinesterase (AChE) and microRNA-15b biogenesis.

30 targets for Alzheimer's disease (AD), i.e., acetylcholinesterase (AChE) and monoamine oxidase B (MAO

31 The chemistry described here uses acetylcholinesterase (AChE) and produces an unambiguous

32 this work we investigated the expression of acetylcholinesterase (AChE) and the density of myelinate

33 he molecular interactions between the enzyme acetylcholinesterase (AChE) and two compound classes con

34 The catalytic subunits of acetylcholinesterase (AChE) are anchored in the basal la

35 Choline acetyltransferase (ChAT) and acetylcholinesterase (AChE) are the decisive enzymatic a

36 isplays the identical binding mechanism with acetylcholinesterase (AChE) as its more potent counterpa

37 pectrometric detection and a high throughput acetylcholinesterase (AChE) assay was developed.

38 and effect-directed analysis (EDA) using the acetylcholinesterase (AChE) bioassay and metabolomics.

39 Acetylcholinesterase (AChE) biosensor was developed thro

40 A sensitive electrochemical acetylcholinesterase (AChE) biosensor was successfully d

41 OPs inactivate acetylcholinesterase (AChE) by covalently modifying its

42 Ligand binding sites on acetylcholinesterase (AChE) comprise an active center, a

43 Acetylcholinesterase (AChE) converts ACh to choline, whi

44 Towards this, a highly sensitive acetylcholinesterase (AChE) cyclic voltammetric biosenso

45 fect of temperature on the kinetics of brain Acetylcholinesterase (AChE) during adaptation to subleth

46 de organophosphate insecticides that inhibit acetylcholinesterase (AChE) enzyme activity in the salmo

47 l Organic Framework (MOF-Basolite Z1200) and Acetylcholinesterase (AChE) enzyme served as an excellen

48 e (PHA), which mimics the performance of the acetylcholinesterase (AChE) enzyme.

49 biosensor that employs genetically modified acetylcholinesterase (AChE) enzymes B394, B4 and wild ty

50 activity of angiotensin-converting (ACE) and acetylcholinesterase (AChE) enzymes.

51 Like its homologs in the acetylcholinesterase (AChE) family, ChEL possesses two c

52 A series of inhibitors of acetylcholinesterase (AChE) have been screened by back-s

53 olive oil, based on a genetically-engineered acetylcholinesterase (AChE) immobilized in a azide-unit

54 aking advantage of the crystal structures of acetylcholinesterase (AChE) in complex with galantamine

55 y binds to aromatic residues of OP-inhibited acetylcholinesterase (AChE) in orientations that are non

56 showed improved inhibitory properties toward acetylcholinesterase (AChE) in relation to tacrine.

57 The synaptic form of acetylcholinesterase (AChE) in skeletal muscle ColQ-AChE

58 The expression of acetylcholinesterase (AChE) in skeletal muscle is regula

59 ber of studies detailing the distribution of acetylcholinesterase (AChE) in the hippocampal formation

60 xicants is due to irreversible inhibition of acetylcholinesterase (AChE) in the nervous system.

61 reased during the initial period and that of acetylcholinesterase (AChE) increased during a later tim

62 ldoximes are used as antidotes to reactivate acetylcholinesterase (AChE) inhibited by organophosphoru

63 al-missions of Abeta-targeting clearance and acetylcholinesterase (AChE) inhibition in AD therapy.

64 valuated using a previously established AOP, acetylcholinesterase (AChE) inhibition.

65 herein that RS67333 is also a submicromolar acetylcholinesterase (AChE) inhibitor and therefore, cou

66 , that bis(heptyl)-cognitin, a novel dimeric acetylcholinesterase (AChE) inhibitor derived from tacri

67 Acetylcholinesterase (AChE) inhibitors are commonly used

68 Acetylcholinesterase (AChE) inhibitors are potentially l

69 ganophosphorus (OP) and methylcarbamate (MC) acetylcholinesterase (AChE) inhibitors.

70 ich sources of phenolic compounds, exhibited acetylcholinesterase (AChE) inhibitory activity and also

71 anti-cancer, anti-viral, anti-parasitic, and acetylcholinesterase (AChE) inhibitory activity, were de

72 is-furans and one dihydropyran isomer showed acetylcholinesterase (AChE) inhibitory activity.

73 In vitro, 15 compounds displayed excellent acetylcholinesterase (AChE) inhibitory potencies and int

74 Human acetylcholinesterase (AChE) is a significant target for

75 Acetylcholinesterase (AChE) is crucial for degrading ace

76 ransmitters in the human body, by the enzyme acetylcholinesterase (AChE) is fundamental for the termi

77 Acetylcholinesterase (AChE) is highly expressed at sites

78 In the central nervous system, acetylcholinesterase (AChE) is present in a tetrameric f

79 By utilizing the acetylcholinesterase (AChE) mediated hydrolysis of acety

80 The vast majority of newly synthesized acetylcholinesterase (AChE) molecules do not assemble in

81 ) and anthocyanins (r = 0.312 and 0.301) for acetylcholinesterase (AChE) or butylcholinoesterase (BuC

82 sensing strategy involves reacting ACh with acetylcholinesterase (AChE) to form choline that is in t

83 frozen, serially sectioned, and stained for acetylcholinesterase (AChE) to identify the NMJs.

84 rseradish peroxidase and surface coated with acetylcholinesterase (AChE) were attached to gold screen

85 -oxime reactivation rates for OP-inactivated acetylcholinesterase (AChE) were lower compared to 2-PAM

86 can be induced in humans through blockade of acetylcholinesterase (AChE) whereas antidepressant-like

87 to investigate inhibition of human BChE and acetylcholinesterase (AChE) with metaproterenol, isoprot

88 tivators of chemical warfare agent inhibited acetylcholinesterase (AChE) with promising in vitro pote

89 ing bovine serum albumin and conjugated with acetylcholinesterase (AChE), an enzyme specific for acet

90 g the N-methyl-d-aspartate receptor (NMDAR), acetylcholinesterase (AChE), and monoamine oxidase B (MA

91 his work, the selected target was the enzyme acetylcholinesterase (AChE), and the AChE-ICERs produced

92 It is selectively reduced by the addition of acetylcholinesterase (AChE), and thus appears to involve

93 ucleus, distinct in cytochrome oxidase (CO), acetylcholinesterase (AChE), and vesicular glutamate tra

94 Inhibitory activity of the conjugates toward acetylcholinesterase (AChE), butyrylcholinesterase (BChE

95 ontain several cholinergic factors including acetylcholinesterase (AChE), choline acetyltransferase (

96 onjugates (MB-gCs) against human erythrocyte acetylcholinesterase (AChE), equine serum butyrylcholine

97 immunocytochemical stains were performed for acetylcholinesterase (AChE), nicotinamide adenine dinucl

98 whether APP is involved in the regulation of acetylcholinesterase (AChE), which is a key protein of t

99 variable and insensitive to perturbations of acetylcholinesterase (AChE), while slow non-alpha7 recep

100 iated with an abnormal sprouting response of acetylcholinesterase (AChE)-positive fibers, a phenotype

101 tly binding to the active site of the enzyme acetylcholinesterase (AChE).

102 ow antioxidant properties, and inhibit human acetylcholinesterase (AChE).

103 sly block the active and peripheral sites of acetylcholinesterase (AChE).

104 enisn-converting enzyme (ACE) and the enzyme acetylcholinesterase (AChE).

105 as the result of irreversible inhibition of acetylcholinesterase (AChE).

106 Among its other biological roles, acetylcholinesterase (AChE, EC 3.1.1.7), encoded by two

107 We reported previously that insect acetylcholinesterases (AChEs) could be selectively and i

108 f synaptic acetylcholine (ACh) by tetrameric acetylcholinesterase (AChEt).

109 ge (thiobarbituric acid reactive substances, acetylcholinesterase, acid phosphatase), no significant

110 lcholine levels resulting from inhibition of acetylcholinesterase act on such receptors.

111 P = 0.13), but not significant for cortical acetylcholinesterase activity (F = 0.3, P = 0.75).

112 ed a significant regressor effect for limbic acetylcholinesterase activity (F = 10.1, P < 0.0001), bo

113 roteins, reactive oxygen species, and higher acetylcholinesterase activity are also strongly associat

114 c (hippocampal and amygdala) and neocortical acetylcholinesterase activity as well as striatal monoam

115 midinopropane) dihydrochloride and inhibited acetylcholinesterase activity by 85%.

116 and encapsulated lutein was able to inhibit acetylcholinesterase activity even in an aqueous medium.

117 In addition, Smu. 1393c protects acetylcholinesterase activity from being inhibited by tw

118 ation test scores correlated positively with acetylcholinesterase activity in the hippocampal formati

119 O-glycosyl flavonoids on the antioxidant and acetylcholinesterase activity of these juices has been e

120 ecreased the levels of SE-bound CD63/CD9 and acetylcholinesterase activity respectively.

121 The anti-acetylcholinesterase activity was higher in butanol extr

122 nic acetylcholine receptor was increased and acetylcholinesterase activity was reduced versus control

123 cetylcholine type 2 receptor) receptors, and acetylcholinesterase activity were evaluated in RV (n=11

124 ing of the sacral cord reveals expression of acetylcholinesterase activity, ability to synthesize ace

125 using ultra-centrifugation, and analyzed for acetylcholinesterase activity, total proteins, drug conc

126 lcholine, and paraoxon-induced inhibition of acetylcholinesterase activity.

127 minimal in the shell in part due to elevated acetylcholinesterase activity.

128 One clue could be that acetylcholinesterase, also present in amyloid plaques, i

129 aimed to evaluate anti-cholinesterase (anti-acetylcholinesterase and -butylcholinesterase), anti-dia

130 sts of alpha-glucosidase, pancreatic lipase, acetylcholinesterase and 15-lipoxygenase were performed.

131 sting, and [(11)C]methylpiperidyl propionate acetylcholinesterase and [(11)C]dihydrotetrabenazine (DT

132 ased segmentation, and focal misalignment of acetylcholinesterase and AChRs.

133 cally used cholinomimetics that both inhibit acetylcholinesterase and also interact directly with and

134 Acetylcholinesterase and butyrylcholinesterase accumulat

135 These hybrids are potent inhibitors of human acetylcholinesterase and butyrylcholinesterase in vitro

136 zed and evaluated for binding potency toward acetylcholinesterase and butyrylcholinesterase using enz

137 biological significance (alpha-glucosidase, acetylcholinesterase and butyrylcholinesterase) and free

138 emon juice and chokeberry controls inhibited acetylcholinesterase and butyrylcholinesterase, and this

139 classification of carbon 11-labeled [11C]PMP acetylcholinesterase and caudate nucleus [11C]DTBZ monoa

140 e hypothesis that time-of-day differences in acetylcholinesterase and choline acetyltransferase expre

141 s produced by the enzymatic reaction between acetylcholinesterase and hypoxanthine oxidized antioxida

142 ibitors, with high selectivity against human acetylcholinesterase and low to no toxicity in human cel

143 ta, that is a potent reversible inhibitor of acetylcholinesterase and NMDA receptors, could mitigate

144 iber stained preparations, supplemented with acetylcholinesterase and parvalbumin immunohistochemistr

145 rylated state, which allowed comparison with acetylcholinesterase and rationalization of its ability

146 tion of target site resistance genes such as acetylcholinesterase and the GABA receptor.

147 ferase-positive neurons, as well as, reduced acetylcholinesterase and vesicular acetylcholine transpo

148 s very similar to the primary target of OPs (acetylcholinesterase) and a unique N-terminal alpha-heli

149 1(R) allele expressing the insensitive G119S acetylcholinesterase, and a resistant allele of an unkno

150 r enzymes: alpha-glucosidase, alpha-amylase, acetylcholinesterase, and butyrylcholinesterase.

151 le, ascorbic acid content, antioxidant, anti-acetylcholinesterase, anti-inflammatory and cytotoxic ac

152 nt pathogens, a radical scavenging assay, an acetylcholinesterase assay as well as in situ and ex sit

153 orm capabilities by an in-depth study of the acetylcholinesterase assay, including kinetic parameter

154 method of fabrication of a highly sensitive acetylcholinesterase biosensor and its application to de

155 A highly sensitive acetylcholinesterase biosensor was developed for detecti

156 ed acetylcholinesterase using the reversible acetylcholinesterase blocker donepezil.

157 ment, [(11)C]methyl-4-piperidinyl propionate acetylcholinesterase brain positron emission tomography

158 t potent inhibitory activities against human acetylcholinesterase, butyrylcholinesterase, and BACE-1,

159 rmeability and multiple activities in vitro (acetylcholinesterase, butyrylcholinesterase, beta-site a

160 reened for their inhibitory activity against acetylcholinesterase, butyrylcholinesterase, lipoxygenas

161 ybdenum assay) and enzyme-inhibitory assays (acetylcholinesterase, butyrylcholinesterase, tyrosinase,

162 nd flavonoid compounds were screened against acetylcholinesterase, butyrylcholinesterase, urease, and

163 Moderate inhibition of acetylcholinesterase by extracts of all investigated Ros

164 ilarly to previously described inhibition of acetylcholinesterase by rivastigmine and other carbamate

165 revealed when tissue was stained for SMI32, acetylcholinesterase, calbindin, or calretinin.

166 m sections stained for Nissl bodies, myelin, acetylcholinesterase, calbindin, or cytochrome oxidase,

167 solutions of acetic acid as a route to sense acetylcholinesterase-catalyzed hydrolysis of acetylcholi

168 They inhibit acetylcholinesterase causing cholinergic crises that lea

169 ion for NADPH diaphorase-expressing, but not acetylcholinesterase-, choline acetyltransferase-, or tr

170 cose oxidase-catalyzed oxidation of glucose, acetylcholinesterase/choline oxidase-mediated hydrolysis

171 protein or its peptide resulted in increased acetylcholinesterase concentration and activity in the A

172 e ColQ deficient mouse, a model of end-plate acetylcholinesterase deficiency.

173 d the utility of (11)C-donepezil for imaging acetylcholinesterase densities in peripheral organs, inc

174 (11)C-donepezil PET is suitable for imaging acetylcholinesterase densities in peripheral organs.

175 donepezil was recently validated for imaging acetylcholinesterase density in the brain and peripheral

176 on and acetylcholine receptor clustering and acetylcholinesterase dispersion seen in the Col13a1-/- m

177 , generated against Electrophorus electricus acetylcholinesterase (EeAChE), inhibits EeAChE and BfACh

178 noencapsulated lutein in the activity of the acetylcholinesterase enzyme.

179 th time-dependent changes in basal forebrain acetylcholinesterase expression.

180 Currently, the four major nerve targets are acetylcholinesterase for organophosphates and methylcarb

181 The acetylcholinesterase found in the venom of Bungarus fasc

182 The subsequent enhanced release of acetylcholinesterase from the extant cells could reflect

183 We compared biological functions of two acetylcholinesterase genes (TcAce1 and TcAce2) in Tribol

184 st promising hybrids, the mechanism of human acetylcholinesterase (hAChE) inhibition as well as their

185 lkylamine reactivators of phosphylated human acetylcholinesterase (hAChE) intended to catalyze the hy

186 Human acetylcholinesterase (hAChE), an enzyme mediating synapt

187 sarin, VX, and paraoxon) conjugates of human acetylcholinesterase (hAChE).

188 se two scenarios: the synaptic form of human acetylcholinesterase (hAChE-S) associates with plaques i

189 Acetylcholinesterase histology has been used for many ye

190 were evaluated using confocal microscopy and acetylcholinesterase histology.

191 tion volume ratio) and thalamic and cortical acetylcholinesterase hydrolysis rate per minute (k3), re

192 Firstly, an effective acetylcholinesterase immobilized electrode was developed

193 cells was monitored by measuring release of acetylcholinesterase in cell perfusates using the Ellman

194 Feeding flies choline or inhibiting acetylcholinesterase in Pn enhances memory, an effect bl

195 lloenzymes that hydrolyze a variety of toxic acetylcholinesterase-inhibiting organophosphorus compoun

196 ntly (p < 0.05) higher alpha-glucosidase and acetylcholinesterase inhibition activities were observed

197 for free fraction, and alpha-glucosidase and acetylcholinesterase inhibition for whole fraction.

198 of dichlorvos based on polydiacethylene and acetylcholinesterase inhibition is developed.

199 teine, two novel reversal drugs that have no acetylcholinesterase inhibition properties.

200 is study investigates, whether pharmacologic acetylcholinesterase inhibition with neostigmine diminis

201 ulating the parasympathetic activity through acetylcholinesterase inhibition, in experimental pulmona

202 s behind this toxidrome in humans, including acetylcholinesterase inhibition, N-methyl-D-aspartate re

203 dynamics to determine ACh's effects, via the acetylcholinesterase inhibitor (AChEI) donepezil, on the

204 naptic morphology and weak resistance to the acetylcholinesterase inhibitor aldicarb, they are signif

205 muscular junction during lethargus using the acetylcholinesterase inhibitor aldicarb.

206 naling mutants and transgenic animals to the acetylcholinesterase inhibitor aldicarb.

207 y of sacral VF neurons in the presence of an acetylcholinesterase inhibitor can be partially ascribed

208 ve M(1) PAM VU0453595 in comparison with the acetylcholinesterase inhibitor donepezil, M(1)/M(4) agon

209 demonstrated by the evolution of an approved acetylcholinesterase inhibitor drug into brain-penetrabl

210 ments of the neonatal rat spinal cord to the acetylcholinesterase inhibitor edrophonium (EDR).

211 consisting of the epidural injection of the acetylcholinesterase inhibitor neostigmine.

212 Acetylcholinesterase inhibitor reversal can cause respir

213 eliorating this cholinergic deficit with the acetylcholinesterase inhibitor rivastigmine would reduce

214 For Alzheimer disease, this includes an acetylcholinesterase inhibitor such as donepezil for mil

215 Galantamine is an acetylcholinesterase inhibitor that also acts as a posit

216 Neostigmine is an acetylcholinesterase inhibitor that ameliorates the effe

217 and functional network enhancements with an acetylcholinesterase inhibitor treatment (donepezil) whe

218 are more likely to cognitively improve with acetylcholinesterase inhibitor treatment.

219 nd no evidence that age, disease severity or acetylcholinesterase inhibitor use influenced rate of de

220 (defined by hospital discharge diagnosis or acetylcholinesterase inhibitor use).

221 ce for influence by age, disease severity or acetylcholinesterase inhibitor use.

222 observed in 6 of 66 patients who received an acetylcholinesterase inhibitor, 65 of 69 patients who re

223 utants also exhibited hypersensitivity to an acetylcholinesterase inhibitor, aldicarb, uncovering def

224 thout an aid, had no previous exposure to an acetylcholinesterase inhibitor, and did not have dementi

225 milar in normal and lesioned animals and the acetylcholinesterase inhibitor, donepezil (1 mg/kg), pro

226 Donepezil, an acetylcholinesterase inhibitor, is an approved drug for

227 ic activation by systemic application of the acetylcholinesterase inhibitor, physostigmine, resulted

228 treatment with donepezil, a centrally active acetylcholinesterase inhibitor, prevented and reversed o

229 macological intervention using donepezil, an acetylcholinesterase inhibitor.

230 With the aim of reducing side effects of acetylcholinesterase inhibitors (AChEIs) during symptoma

231 meta-analysis investigating the influence of acetylcholinesterase inhibitors (AChEIs) therapy on nutr

232 oestrogen replacement therapy (Level A2) and acetylcholinesterase inhibitors (Level B).

233 re also limited by common adverse effects of acetylcholinesterase inhibitors and limited availability

234 Acetylcholinesterase inhibitors and memantine hydrochlor

235 s to examine the safety of NMB reversal with acetylcholinesterase inhibitors and muscarinic anticholi

236 se Research Centre who subsequently received acetylcholinesterase inhibitors and underwent magnetic r

237 Acetylcholinesterase inhibitors are commonly used to tre

238 +/- 6.7 years); 71% of the patients were on acetylcholinesterase inhibitors at baseline; mean Mini-M

239 tors; however, in some cases the efficacy of acetylcholinesterase inhibitors diminishes over time.

240 Acetylcholinesterase inhibitors have also been shown to

241 ion tomography predict treatment response to acetylcholinesterase inhibitors in patients with dementi

242 AD, and enhancing cholinergic signaling with acetylcholinesterase inhibitors is currently the primary

243 The response to acetylcholinesterase inhibitors is often disappointing.

244 racting the detrimental effects of long-term acetylcholinesterase inhibitors on the postsynaptic neur

245 Treatment with acetylcholinesterase inhibitors resulted in worsened con

246 o acquired myasthenia gravis, treatment with acetylcholinesterase inhibitors should be avoided in DOK

247 e specific examples of the therapeutics from acetylcholinesterase inhibitors to recent anti-Abeta imm

248 Use of acetylcholinesterase inhibitors was prohibited.

249 tor agonists) and organophosphate miticides (acetylcholinesterase inhibitors).

250 The beneficial effects of acetylcholinesterase inhibitors, however, are typically

251 nvestigating pharmacological therapies using acetylcholinesterase inhibitors, such as pyridostigmine

252 oup and includes symptomatic treatment using acetylcholinesterase inhibitors, thymectomy and immunoth

253 of age, disease severity and baseline use of acetylcholinesterase inhibitors.

254 lcholine receptor deficiency respond well to acetylcholinesterase inhibitors; however, in some cases

255 Ex vivo determination of the brain acetylcholinesterase inhibitory activity of these compou

256 nds that exert antioxidative, antimicrobial, acetylcholinesterase inhibitory and estrogenic activitie

257 aying both nanomolar dual-binding site (DBS) acetylcholinesterase inhibitory effects and partial 5-HT

258 The hydrolysis of acetylcholine by acetylcholinesterase into choline was monitored in real-

259 Acetylcholinesterase is an enzyme that is intimately ass

260 The overall activity of acetylcholinesterase is found to decline in AD, whereas

261 ncoding the acetylcholine-hydrolyzing enzyme acetylcholinesterase is known to undergo long-lasting tr

262 pite the modest ability to inhibit rat brain acetylcholinesterase, it protects neuronal SH-SY5Y cells

263 5-(11)C-methoxy-donepezil, a noncompetitive acetylcholinesterase ligand, was previously validated fo

264 Functionally, iHPs generate acetylcholinesterase(+) megakaryocytes and phagocytic my

265 he graft-host junction were subjected to the acetylcholinesterase method for the demonstration of cor

266 only in the shell, where higher activity of acetylcholinesterase minimizes nAChR desensitization and

267 The response characteristics of acetylcholinesterase-modified AlGaN/GaN solution-gate fi

268 f fluorescent probes-one to detect the total acetylcholinesterase on erythrocytes (RBC-AChE) and the

269 untermeasures such as antidotes reactivating acetylcholinesterase or scavenging the parent OP.

270 rived butyrylcholinesterase and erythrocytic acetylcholinesterase) or nonexisting (synaptic acetylcho

271 he largest peptidic inhibitors targeting the acetylcholinesterase peripheral site.

272 sferase-positive cells in this region and of acetylcholinesterase-positive fibers throughout the audi

273 toneuron volume and a near-normal density of acetylcholinesterase-positive process arborization, whic

274 Past acetylcholinesterase positron emission tomography (PET)

275 2 and (11)C-methylpiperidin-4-yl propionate acetylcholinesterase positron emission tomography and th

276 mouse model overexpressing a miR refractory acetylcholinesterase-R splice variant showed a parallel

277 s to assess the bioactivity of a fragment of acetylcholinesterase responsible for its non-enzymatic f

278 This exposure inhibits acetylcholinesterase resulting in increased acetylcholin

279 LQ (collagen-like tail subunit of asymmetric acetylcholinesterase; rs7609897-T: P=1.5 x 10(-10), OR=0

280 l candidate inhibitors from a screen against acetylcholinesterase show detectable activity; beyond th

281 9 cells transfected with either p53 siRNA or acetylcholinesterase siRNA.

282 To explore a novel acetylcholinesterase species, ascertain the molecular ba

283 ious genotypes, myenteric plexus presence by acetylcholinesterase staining and embryonic day 12.5 (E1

284 fAChE forms the canonical dimer found in all acetylcholinesterase structures.

285 osedimented with eHAV but not membrane-bound acetylcholinesterase, suggesting that eHAV, and not vira

286 then evoke a subsequent, enhanced release in acetylcholinesterase that could only be derived from the

287 croinjection of neostigmine, an inhibitor of acetylcholinesterase, that evoked rapid increases in ace

288 known naturally occurring hydrolytic enzyme, acetylcholinesterase, the catalytic activity of which ap

289 Donepezil is a high-affinity ligand for acetylcholinesterase-the enzyme that catabolizes acetylc

290 of beta-amyloid with a peptide derived from acetylcholinesterase: the similarity in action suggests

291 choline which is enzymatically hydrolyzed by acetylcholinesterase to myristic acid and choline to pre

292 antimicrobials, estrogen-like activators and acetylcholinesterase/tyrosinase inhibitors.

293 e related to the activity of the immobilized acetylcholinesterase using the reversible acetylcholines

294 re recently, a gene purportedly encoding for acetylcholinesterase was cloned from maize.

295 Acetylcholinesterase was electro-immobilized into a thic

296 We show proof-of-concept where acetylcholinesterase was immobilized on an organosiloxan

297 fos-induced inhibition of its target enzyme (acetylcholinesterase) was only magnified by the heat spi

298 hermal properties.The two molecular forms of acetylcholinesterase were purified from the brain of T.

299 obiase, catalase, xenobiotic metabolism, and acetylcholinesterase) were measured in the adults.

300 Physostigmine is a well known inhibitor of acetylcholinesterase, which can also activate, potentiat