ライフサイエンスコーパス: acquired immune response

1 endritic cell (DC) is a critical step of the acquired immune response.

2 levels in tissues is not due to an impaired acquired immune response.

3 re may contribute to the polarization of the acquired immune response.

4 or to the onset of IFN counteraction and the acquired immune response.

5 o define the magnitude and the nature of the acquired immune response.

6 t specific components of the host innate and acquired immune response.

7 ys a crucial role in both the innate and the acquired immune response.

8 nnate immunity that profoundly influences an acquired immune response.

9 owing a second inoculation, indicative of an acquired immune response.

10 viable bacteria together with an attenuated acquired immune response.

11 ), mediate innate responses, and initiate an acquired immune response.

12 e miRNAs in the regulation of the innate and acquired immune response.

13 eficient bone formation and activation of an acquired immune response.

14 e an antigen-specific T helper type 1 (Th1)--acquired immune response.

15 on TCR stimulation and thus may modulate the acquired immune response.

16 ing immunoglobulin gene diversity during the acquired immune response.

17 t TNF- alpha is a necessary component of the acquired immune response.

18 Less is known regarding the role of the acquired immune response.

19 he innate immune response in addition to the acquired immune response.

20 the innate as well as the early phase of the acquired immune response.

21 most certainly preceded the emergence of the acquired immune response.

22 e required for the induction of a Th1 T-cell acquired immune response.

23 ism by which motile bacteria can initiate an acquired immune response.

24 everal agonists to CD40 have shown to induce acquired immune responses.

25 etory pathway is crucial for both innate and acquired immune responses.

26 y issues arising from anti-vector innate and acquired immune responses.

27 cytokines and to exert helper functions for acquired immune responses.

28 id metabolism and trafficking and influences acquired immune responses.

29 IL-37 suppresses innate and acquired immune responses.

30 nate cytokine production, leading to altered acquired immune responses.

31 rpreting activation states of the innate and acquired immune responses.

32 ugh to interaction with signals derived from acquired immune responses.

33 omodulatory cytokine that affects innate and acquired immune responses.

34 lopment of colitis by activating deleterious acquired immune responses.

35 roteins during the transition from innate to acquired immune responses.

36 n of the airways and induction of innate and acquired immune responses.

37 igration, a key process governing innate and acquired immune responses.

38 e of PPARgamma in regulating both native and acquired immune responses.

39 r, insulin sensitivity, bone metabolism, and acquired immune responses.

40 strongest balancing selection from naturally acquired immune responses.

41 ost defense mechanisms leading to innate and acquired immune responses.

42 Host innate and acquired immune responses activated following RSV infect

43 nflammation and the modulation of innate and acquired immune responses after binding to their G prote

44 ncode molecules that lie at the heart of the acquired immune response against infectious diseases.

45 to the central role of DCs in initiating the acquired immune response against M. tuberculosis infecti

46 bscess formation, yet in humans, a naturally acquired immune response against the CRD did not persist

47 ulated proteins elicit protective innate and acquired immune responses against a wide range of pathog

48 Naturally acquired immune responses against human cancers often in

49 nd play an important role in both innate and acquired immune responses against infectious diseases an

50 ion may enhance Mphi-mediated innate and Th1-acquired immune responses against intracellular infectio

51 jor role in the induction of both innate and acquired immune responses against pathogenic invaders.

52 Absence of a properly functioning acquired immune response allows MAC persistence within m

53 infection is not necessarily dependent on an acquired immune response and can occur despite the prese

54 nesis is dependent upon inflammation, a Th-1 acquired immune response and hormonal changes including

55 ive vaccine that improves upon the naturally acquired immune response and induces rapid and long-last

56 gnificant impact on our understanding of the acquired immune response and may provide insight concern

57 by ANCA may contribute to modulation of the acquired immune response and to granuloma formation.

58 -18) is an important regulator of innate and acquired immune responses and plays an important role in

59 , it represents a potent target for the host acquired immune response, and constitutive expression of

60 he host innate immune response, modulate the acquired immune response, and evade intracellular destru

61 uces both a strong inflammatory and a strong acquired immune response, and Mtb then actively regulate

62 nce the sensitization phase of at least some acquired immune responses, and can contribute to the pat

63 t Candida infection involves both innate and acquired immune responses, and cytokines produced by mon

64 Thus, the acquired immune response appeared to be functional in My

65 nate/proinflammatory and some aspects of the acquired immune response are up-regulated to maintain a

66 The acquired immune responses are crucial to the survival of

67 Our data indicated that vigorous innate and acquired immune responses are elicited, activated, and r

68 ammatory pathways, and the host's innate and acquired immune responses are leading to identification

69 of macrophages as participants in innate and acquired immune responses are regulated by the specific

70 CNS inflammation associated with adaptive or acquired immune responses are uncertain.

71 In summary, CCR4 modulates both innate and acquired immune responses associated with chronic fungal

72 , development, expression, and regulation of acquired immune responses, both those associated with Ig

73 HLA class I molecules are essential to the acquired immune response, but they are also important in

74 drivers of tissue remodeling in established acquired immune responses, but also may contribute to th

75 ue environment essential for generating this acquired immune response by reversing the LN defect in l

76 es during the innate response may modify the acquired immune response by T cells.

77 type-I IFNs can influence the generation of acquired immune responses by modifying T-helper cell dif

78 In addition to the conventional innate and acquired immune responses, complex organisms have evolve

79 To investigate how the acquired immune response could contribute to osteolytic

80 st defense until the slower, more definitive acquired immune response develops.

81 , we investigated the role of the innate and acquired immune responses elicited by Chlamydophila pneu

82 the pathogen to remain one step ahead of the acquired immune response, enabling persistent infection.

83 , including mechanisms related to innate and acquired immune responses following gamma radiation.

84 CpG ODN also enhance the development of acquired immune responses for prophylactic and therapeut

85 ry/autoimmunity disorders trigger innate and acquired immune responses, generating a unique microenvi

86 Evasion of the acquired immune response in African trypanosomes is prin

87 e domain (PvMSP1P-19) elicited high level of acquired immune response in patients.

88 Evidence for a CD8 antigen-driven acquired immune response in psoriatic synovium and blood

89 esent study addresses the causal role of the acquired immune response in the selection of TprK varian

90 rtance of cross-talk between host innate and acquired immune responses in host mucosal defense.

91 of malarial Ags that are capable of inducing acquired immune responses in the fetus.

92 ctivation of key genes involved in adaptive (acquired) immune responses, including major histocompati

93 Activation of the acquired immune response increased osteoclastogenesis an

94 ine, IL-10, that antagonizes both innate and acquired immune responses, interfering with the developm

95 The role of PKCtheta for the acquired-immune responses involved in the development of

96 Development of the acquired immune response is dependent on the signaling o

97 components provides a mechanism by which the acquired immune response may be tailored to specific pat

98 Understanding the basis of these acquired immune responses may be critical in developing

99 factors including receptor heterogeneity and acquired immune responses may drive parasite phenotypic

100 Activation of mammalian innate and acquired immune responses must be tightly regulated by e

101 ve antigens are specifically targeted by the acquired immune response of the host and are able to ind

102 b) is able to manipulate both the innate and acquired immune response of the host.

103 ng molecules and are an integral part of the acquired immune response of vertebrates.

104 ear to be required for the development of an acquired immune response, presumably by functioning in a

105 involved in controlling/resolving innate or acquired immune responses so as to provide a framework f

106 orders and other immunoglobulin E-associated acquired immune responses that it can be difficult to th

107 We propose that, in addition to acquired immune responses, the innate immune system and

108 m, has been implicated in the development of acquired immune responses, though its roles are largely

109 w that IFN-beta influences the generation of acquired immune responses through its regulation of CD40

110 ns and dampens the recruitment of innate and acquired immune responses, thus enabling open-ended inte

111 al chitin, such as involvement in innate and acquired immune responses, tissue remodeling, fibrosis,

112 ciated with DHF than primary infections, the acquired immune response to dengue, both B cells and T c

113 e of CD4(+) helper T cells in modulating the acquired immune response to herpes simplex virus type 1

114 s of this study to dissect the nature of the acquired immune response to infection with Listeria mono

115 r gammadelta receptor-bearing T cells in the acquired immune response to infection with Mycobacterium

116 An effective acquired immune response to infectious agents mediated b

117 c cells (DCs) are crucial for initiating the acquired immune response to infectious diseases such as

118 crucial role of the spleen in the innate and acquired immune response to malaria, there is little inf

119 paB (NF-kappaB) is central to the innate and acquired immune response to microbial pathogens, coordin

120 To investigate mechanisms by which the acquired immune response to Porphyromonas gingivalis cou

121 dicated that deficiency of DbpA/B allows the acquired immune response to restrict early dissemination

122 im of this study was to determine whether an acquired immune response to toxin A, during an episode o

123 ion that play an important role in governing acquired immune responses to a variety of foreign antige

124 IL-10 could have an effect on innate and acquired immune responses to B. burgdorferi and influenc

125 ne population, as well as porcine innate and acquired immune responses to IAV, including recent advan

126 nnate immune responses, but they also affect acquired immune responses to infection.

127 of PA-X in counteracting the host innate and acquired immune responses to influenza virus, an importa

128 pact on viral growth and the host innate and acquired immune responses to influenza virus.

129 Acquired immune responses to malaria have widely been pe

130 e pathogen population, given that hosts have acquired immune responses to multiple antigens of most p

131 In acquired immune responses to parasites or allergens, the

132 ion is only transient due to host innate and acquired immune responses to the virus.

133 cause NS5 may interfere with both innate and acquired immune responses to virus infection, this prote

134 Additionally, the strength of vertebrate acquired immune responses varies dramatically depending

135 Once an acquired immune response was initiated, the P. carinii f

136 Their presence seems to reflect an acquired immune response, which is not translated into a

137 smodium infections trigger strong innate and acquired immune responses, which can lead to severe comp

138 An acquired immune response--with the participation of CD4+