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1 r forms the basis for developing more potent active immunotherapy.
2 ight synergize with, rather than antagonize, active immunotherapy.
3 pecific antigen and an attractive target for active immunotherapy.
4 as (idiotype [Id]) can serve as a target for active immunotherapy.
5 on tumor cells is critical for the design of active immunotherapy.
6 responses and acts as a potent adjuvant for active immunotherapy.
7 splaying C3dg warrant further development as active immunotherapies.
8 enhance the function of DNA immunization for active immunotherapy, a panel of cytokines was added as
9 gents in breast cancer more effectively with active immunotherapy and potentially other anticancer th
11 mmunotherapies, focusing on both passive and active immunotherapy approaches that have entered clinic
12 ced the therapeutic activity of adoptive and active immunotherapy, delaying tumor growth and prolongi
16 To overcome this challenge, we developed an active immunotherapy for personalized treatment based on
17 ular immunization may enhance the effects of active immunotherapy for the treatment of synucleinopath
18 somers are potential compounds in developing active immunotherapy for treatment of VEGFR bearing tumo
20 ed clinically significant treatment effects, active immunotherapy has not yet become an established c
21 ide epitopes that can be used as targets for active immunotherapy have been identified within melanoc
22 ls, currently underway, evaluating antitumor active immunotherapies in patients with prostate cancer.
25 that when a Bcl-2 inhibitor is combined with active immunotherapy in humans, such as the use of a vac
26 ls has been used as a target for passive and active immunotherapy in patients with malignant melanoma
27 anced, nonviral genetic immunization for the active immunotherapy of cancer and indicated that using
31 s could overcome some of the shortcomings of active immunotherapy or in vivo cytokine treatment, wher
32 hese results suggest that a longer period of active immunotherapy, or passive immunization, may be re
33 ch [idiotype (Id)] can serve as a target for active immunotherapy.Promising results have been obtaine
34 eted therapies, these successes suggest that active immunotherapy represents a path to obtain a durab
36 specific CTLs or to develop antigen-specific active immunotherapy strategies for glioma patients.
37 r conditions acceptable for use in adjuvant, active immunotherapy strategies for surgically treated m
38 nstrate for the first time the ability of an active immunotherapy strategy to generate antigen-specif
41 hat the brain is not a barrier to successful active immunotherapy that uses gene-modified autologous
42 tion, antibiotics, and, recently, the use of active immunotherapy to enhance the immune response towa
44 assemblies were investigated as therapeutic active immunotherapies, which may offer advantages over
45 mmunotherapy of CRC patients with mAb and in active immunotherapy with anti-idiotypic Abs mimicking t
46 erved in patients who received postoperative active immunotherapy with Canvaxin therapeutic cancer va