ライフサイエンスコーパス: active neuron

1 sleep need into the depolarization of sleep-active neurons.

2 be dominated by a small population of highly active neurons.

3 airwise recordings of rat striatal tonically active neurons.

4 activity based on the passage of Mn(2+) into active neurons.

5 elp to decode combinations of simultaneously active neurons.

6 vents in sleep apnea permanently damage wake-active neurons.

7 the firing rate of the entire population of active neurons.

8 es the long-lasting genetic tagging of c-fos-active neurons.

9 de of MT neurons and not the identity of the active neurons.

10 by enhancing BDNF signaling in electrically active neurons.

11 ely enhancing the growth and connectivity of active neurons.

12 ng the fine control of blood distribution to active neurons.

13 ) is that it supplies the metabolic needs of active neurons.

14 those studying neural populations with a few active neurons.

15 d flow and delivery of oxygen and glucose to active neurons.

16 rganoids that contain electrophysiologically active neurons.

17 reliably stored in networks of synchronously active neurons.

18 nd oppositely modulated fear- and extinction-active neurons.

19 t and identifying subgroups of spontaneously active neurons.

20 analyze the network formed by the identified active neurons.

21 though those are rationally in the middle of active neurons.

22 creases in somatic neural activity in weakly active neurons.

23 -human performance in detecting locations of active neurons.

24 ional neural network to identify and segment active neurons.

25 cells, new adult-born neurons, and recently active neurons.

26 mapping them onto larger numbers of sparsely active neurons.

27 ansitioned rapidly between different sets of active neurons.

28 t on the regulation of blood flow to nourish active neurons.

29 Ns) are thought to be identical to tonically active neurons.

30 oduct of catabolism that is also released by active neurons.

31 al hyperemia, brings oxygen and nutrients to active neurons.

32 ivations that correspond to UP states within active neurons.

33 aneous firing persists in many "autonomously active" neurons.

34 onses, with weak or inhibitory responses in 'active' neurons.

35 was a bursting discharge pattern in >75% of active neurons (33 of 44).

36 during rhythmic whisker movement, 54 of 115 active neurons (47%) represented self-motion.

37 w adequate supply of oxygen and nutrients to active neurons, a process termed neurovascular coupling

38 Finally, highly active neurons acquired topological characteristics that

39 ivo, finding significant turnover within the active neurons across days, with only few neurons that r

40 ssue move toward this goal via 3D imaging of active neurons across the entire mouse brain.

41 ft is induced by co-activation of previously active neurons along with neurons with high excitability

42 icrom slices, there were fewer spontaneously active neurons, although these neurons had a higher mean

43 ges in pressure are encoded by the number of active neurones and not graded changes in the discharge

44 distribution, with a small number of highly active neurons and an overabundance of low rate neurons

45 d as dominant reference points for most task-active neurons and anchored the spatial code in RSC.

46 he current transfer from more active to less active neurons and by shunting currents from active neur

47 The identification of active neurons and circuits in vivo is a fundamental cha

48 o direct microglial processes towards highly active neurons and injuries in the brain.

49 natal hippocampus, develop into electrically active neurons and integrate into neuronal networks with

50 Suppression of fear-active neurons and recruitment of extinction-active neur

51 iatal cholinergic interneurons are tonically active neurons and respond to sensory stimuli by transie

52 e an efficient method for repeatedly mapping active neurons and synapses in cell culture, slice prepa

53 ation intensity increases both the number of active neurons and the average level of activity per neu

54 The faster oscillation frequency of active neurons and the slower theta LFP, underlying phas

55 te balance between the high energy demand of active neurons and the supply of oxygen and nutrients fr

56 y dependent, so that TMS suppresses the most active neurons and thereby changes the balance between e

57 pause in firing of these otherwise tonically active neurons and to the striatal dopamine/acetylcholin

58 us (MnPN) of the hypothalamus contains sleep-active neurones, and sleep-related Fos-immunoreactivity

59 ted the hypothesis that MnPN and vlPOA sleep-active neurones are GABAergic by combining staining for

60 at)-GFP], we then show that >50% of PZ sleep-active neurons are inhibitory (GABAergic/glycinergic, VG

61 ives up activity in the stimulated area, but active neurons are saturating; (3) noise generation--TMS

62 However, the sleep-active neurons are spatially intermingled with wake-acti

63 Persistently active neurons are thought to be a mechanism to maintain

64 Sleep-active neurons are under the control of homeostatic mech

65 lely due to a presynaptic inhibition of wake-active neurons as previously hypothesized but rather is

66 sensory information using a small number of active neurons at any given point in time.

67 Here, we tagged active neurons at different stages of motor task perform

68 em cells develop into electrophysiologically active neurons at heterogeneous rates, which can confoun

69 spase-9 accelerates the rate of apoptosis in active neurons back to control levels.

70 Cholinergic interneurons, or tonically active neurons, become responsive to the CS and show dra

71 period progresses, the network formed by the active neurons becomes less modular, and the hubs switch

72 representational codes that rely on very few active neurons, but also to allocate its energy resource

73 The control of sleep-active neurons by locomotion circuits suggests that slee

74 itions in C. elegans, sleep requires a sleep-active neuron called RIS.

75 egins to inhibit these neurons so that sleep-active neurons can become active.

76 present, but whether topographic patterns of active neurons change between laminae is unknown.

77 e in rapid succession so that the pattern of active neurons changed dramatically while the spatial in

78 in rapid succession, so that the pattern of active neurons changed dramatically within each interval

79 Whole-brain reconstruction and analysis of active neurons (colocalized tdTomato/NeuN) were performe

80 We report here that the active neurons composing these ensembles change in a sti

81 s proportion is similar to the percentage of active neurons defined electrophysiologically.

82 own how wakefulness is translated into sleep-active neuron depolarization when the system is set to s

83 Sleep-active neurons depolarize during sleep to suppress wakef

84 erneurons, whereas the activity of tonically active neurons differed from cortical activity with ster

85 eover, DCX expression was observed in adult, active neurons, differentiated projection neurons, and b

86 The most active neurons during the slow oscillation are excitator

87 Active neurons exert a mitogenic effect on normal neural

88 Seventy four percent of these wake-active neurons exhibited moderate or strong activation i

89 Finally, spontaneously active neurones exposed to nor-binaltorphimine switched

90 stent with the hypothesis that spontaneously active neurons expressing GABA are most susceptible to a

91 t perturbed synaptic potassium released from active neurons for Pathway 1, astrocytic transmembrane c

92 ining the saccade magnitude is the number of active neurons for the small saccades.

93 previously that damage to a cluster of sleep-active neurons (Fos-positive during sleep) in the ventro

94 Pairs of active neurons frequently fire action potentials or "spi

95 ttenuated mitochondrial Ca(2+) elevations in active neurons from 6- to 12-months-old female and male

96 ical layer can be used to accurately segment active neurons from another layer with different neuron

97 h a second nonlinearity that prevents weakly active neurons from contributing inhibition.

98 In contrast, plastic changes to highly active neurons from the same ensemble that paradoxically

99 capacity to reflect the elevated needs of an active neuron, guards against future increased demand an

100 d in amplitude and to shift so that the most active neurons had higher preferred speeds.

101 Even in V1, only a small fraction of active neurons had sensory-like responses time-locked to

102 uitment, the tissue-level phenomenon whereby active neurons harvest resources from their surroundings

103 cogenetic activation of c-Fos-labelled sleep-active neurons has been shown to induce sleep.

104 Although sleep-active neurons have been identified in other brain areas

105 Sequences of active neurons have distinct spatial structures and are

106 ts indicate that circuits with intrinsically active neurons have rules for information transfer and s

107 is that neurotrophins act preferentially on active neurons; however, little direct evidence supports

108 The phase differences between rhythmically active neurons in a network are thought to arise from th

109 nock-in mouse provides an opportunity to tag active neurons in a region- or cell-type specific manner

110 We again observed increased firing of active neurons in a virtual enriched environment.

111 commensurate changes in the identity of the active neurons in area MT.

112 Upon examining the responses of tonically active neurons in behaving primates, we found that these

113 We found persistently active neurons in both areas.

114 LU (0-40 nA, 20 s) excited all spontaneously active neurons in dorsal (caudate-putamen) and ventral (

115 o perform a brain-wide survey for prenatally active neurons in mice and identified the piriform corte

116 in the baseline firing rate of endogenously active neurons in response to changes in afferent activi

117 Densities of spontaneously active neurons in slices from both mutants were signific

118 hese findings indicate the important role of active neurons in the brain tumor microenvironment and i

119 We recorded from phasically active neurons in the caudate nucleus while monkeys perf

120 al blood flow (CBF) to perfuse metabolically active neurons in the focus.

121 ectively encompass approximately half of the active neurons in the ganglion: (1) second-order sensory

122 oducing noise correlations with persistently active neurons in the hippocampus, PAC neurons shaped th

123 e show that memoranda-selective persistently active neurons in the human medial temporal lobe phase l

124 includes the mutual inhibition of the sleep-active neurons in the hypothalamic ventrolateral preopti

125 of extinction memory, the dominant input to active neurons in the lateral amygdala was from the infr

126 ly been shown to reduce I(h) in rhythmically active neurons in the mammalian brain.

127 and additionally show that the proportion of active neurons in the network increases with the loss of

128 It has been proposed that ensembles of active neurons in the nucleus accumbens could be based o

129 ear extinction memory, the dominant input to active neurons in the PL was from the vHIP, whereas the

130 e manner that reflects the number or type of active neurons in the population.

131 ochemistry have shown the existence of sleep-active neurons in the preoptic area, especially in the v

132 Recently active neurons in the superficial sublayer of stratum py

133 key role of the hypothalamus, we found fewer active neurons in the ventral hypothalamic sleep-promoti

134 We recently found that sleep-active neurons in the ventrolateral preoptic nucleus (VL

135 flurane and halothane increase the number of active neurons in the VLPO, but only when mice are sedat

136 ave a specialized population of rhythmically active neurons in their olfactory organs with the potent

137 tains in each entry the degree of overlap of active neurons in two corresponding time bins.

138 classes, and the percentage of spontaneously active neurons in vincristine-treated rats were not stat

139 ibute to the prolonged ISI seen in tonically active neurons in vivo in monkeys trained to respond to

140 cute mouse brain slices and in spontaneously active neurons in vivo.

141 ain gene expression, the discovery of "sleep active" neurons in the cerebral cortex, the role of the

142 us (MnPN) of the hypothalamus contains sleep-active neurons including sleep-active GABAergic neurons

143 Active neurons increase their energy supply by dilating

144 All AS-active neurons increase their firing rates during period

145 anisms by which neurotransmission from sleep-active neurons induces sleep and determines the duration

146 a device for probing the interaction between active neurons' intracellular contents and EM waves.

147 Data were obtained from spontaneously active neurons known to respond to ACh (5-30 nA) when th

148 more specifically, in prolonged wakefulness-active neurons labeled by Fos.

149 fast (gamma) cortical activity, as "W/PS-max active neurons." Like cholinergic neurons, many GABAergi

150 Sleep-active neurons located in the ventrolateral preoptic nuc

151 neurons are spatially intermingled with wake-active neurons, making it difficult to target the sleep

152 ed toward detecting sparse subsets of highly active neurons, masking important signals carried in low

153 Electrically active neurons may influence OPC function and selectivel

154 ously inhibited during sleep, the VLPO sleep-active neurons may play a key role in silencing the asce

155 e compared the neural activity of phasically active neurons [medium spiny neurons (MSNs), presumed pr

156 other species, and self-inhibition of sleep-active neurons might represent a conserved mechanism for

157 Our findings suggest that, in synaptically active neurons, modest "basal" levels of postsynaptic Ca

158 In spontaneously active neurons, NMDA receptors were clustered at a few s

159 tion in neurovascular coupling could deprive active neurons of adequate nutrients.

160 e significantly fewer spines specifically on active neurons of fear-conditioned mice.

161 We genetically mark active neurons of freely behaving mice at four times of

162 Purkinje cells (PCs) are spontaneously active neurons of the cerebellar cortex that inhibit glu

163 10(10) densely interconnected, continuously active neurons of the human brain?

164 w here what is known about the influences of active neurons on stem cell and cancer microenvironments

165 code that involves the spatial locations of active neurons or synapses and the times at which activi

166 by selectively modulating TrkB receptors at active neurons or synapses without affecting receptors o

167 rly, the noise correlation between tonically active neuron pairs was stronger in the putamen than in

168 The activity of phasically active neurons (PANs) in the striatum covaried with two

169 t against high neuron overlap and changes in active neuron population across sessions.

170 ility of rsCaMPARI for marking and remarking active neuron populations in freely swimming zebrafish.

171 y precise marking, erasing, and remarking of active neuron populations under brief, user-defined time

172 SD-induced iNOS expression in wakefulness-active neurons positively correlated with sleep pressure

173 active neurons and recruitment of extinction-active neurons predicted psilocybin-enhanced fear extinc

174 The firing rate of coherently active neurons predicts the reaction times (RTs) of coor

175 , presumed projection neurons] and tonically active neurons (presumed cholinergic interneurons) acros

176 In release, the active neuron primarily controls the off/on transitions.

177 s-expressing neurons suggests that intensely active neurons provide local signals that trigger reacti

178 In primates, tonically active neurons (putative cholinergic interneurons) exhib

179 eurovascular coupling in vivo, ensuring that active neurons receive an adequate supply of nutrients.

180 re indistinguishable from those of tonically active neurons recorded in vivo.

181 ales, sequentially organized and transiently active neurons reliably differentiated between different

182 Few spontaneously active neurons rely on HCN 'pacemaker' channels for thei

183 elial cell becomes an electrophysiologically active neuron remains unknown.

184 An enlarged core of stable, likely highly active neurons represent rewarded odor at both stages of

185 ed competition between multiple persistently active neurons reproduces this phenomenon.

186 legans, which is induced by the single sleep-active neuron RIS.

187 Automated, fast, and reliable active neuron segmentation is a critical step in the ana

188 In spontaneously active neurones, serotonin abolished the rhythmicity of

189 ch heterogeneous delays between sequentially active neurons shape the spatiotemporal patterns of HVC

190 Furthermore, spontaneously active neurons show exceptional functional resilience to

191 lined rapidly across training, with the most active neurons showing the largest declines in responsiv

192 lastoma xenograft model, we demonstrate that active neurons similarly promote HGG proliferation and g

193 pothesis of blood flow regulation holds that active neurons stimulate Ca(2+) increases in glial cells

194 ce for a distributed network of persistently active neurons supporting working memory maintenance.

195 ss - from pre-patterned neural progenitor to active neuron - takes 3 weeks or less, making it an idea

196 Tonically active neurons (TANs) are known for their responses to u

197 nd that a set of interneurons, the tonically active neurons (TANs) in monkeys' striatum, use temporal

198 Here we show that VMS tonically active neurons (TANs), including putative cholinergic in

199 Tonically active neurons (TANs), the presumed striatal cholinergic

200 We asked what contribution tonically active neurons (TANs), the putative striatal cholinergic

201 Tonically active neurons (TANs)--presumably, striatal cholinergic

202 one neuromodulator group [striatal tonically active neurons (TANs)] from behaving monkeys.

203 and striatal cholinergic neurons (tonically active neurons, TANs) participate in signalling the beha

204 sharpening of the coincidence of spiking in active neurons (temporal coding).

205 sults in a progressive loss of SIRT1 in wake-active neurons, temporally coinciding with lipofuscin ac

206 distribution, with a small portion of highly active neurons (termed Primed Neurons) filling the long-

207 pansion in networks of functionally related, active neurons that are distributed across a single cort

208 n accurately be decoded from ensembles of co-active neurons that are distributed across piriform cort

209 The PF-LHA contains wake-active neurons that are quiescent during non-REM sleep a

210 Sleep requires sleep-active neurons that depolarize to inhibit wake circuits.

211 al immaturities, including a high density of active neurons that display prominent wave-like activity

212 It is induced by conserved sleep-active neurons that express GABA.

213 of SupV BPNs identifies a group of tonically active neurons that function to lower masseter muscle to

214 nism for adjusting control through tonically active neurons that inhibit movement-producing neurons h

215 itionally, as indicated by the percentage of active neurons, the context representation was more spar

216 In networks of spontaneously active neurons, the mean firing rate, the occurrence of

217 sures rapid delivery of energy substrates to active neurons through the blood supply.

218 ke-promoting neurons in turn shut down sleep-active neurons, thus forming a bipartite flip-flop switc

219 signals; (2) preferential activation of less active neurons--TMS drives up activity in the stimulated

220 on of 'object pointers' through hypothetical active neurons to address the 'surface filling-in' proce

221 e show in mice that KET causes spontaneously active neurons to become suppressed while previously sil

222 n animal can use populations of rhythmically active neurons to capture and encode this temporal infor

223 rocess by passing information from currently active neurons to neurons that will become active after

224 geting, and activity modulation of pre-sleep-active neurons to reveal the behaviors preceding sleep i

225 the activity propagations between a group of active neurons to their inactive neuron neighbors in a v

226 active neurons and by shunting currents from active neurons to their less active neighbors.

227 ional and behavioral roles for SIRT1 in wake-active neurons, transgenic whole animal, and conditional

228 Spontaneously active neurons typically fire either in a regular patter

229 emporal delivery of blood-borne nutrients to active neurons via the vast, dense capillary network.

230 The mechanisms by which active neurons, via astrocytes, rapidly signal intracere

231 lows temporally controlled genetic access to active neurons, we find that the temporal association co

232 More active neurons were more discriminative.

233 Surprisingly, about 60% of all active neurons were self-sustained oscillators when disc

234 kefulness into the depolarization of a sleep-active neuron when the worm is sleepy.

235 changing its color from green to red inside active neurons when illuminated with 400 nm light.

236 tically connected and electrophysiologically active neurons, which matured into long-lived functional

237 for the activation of Hcrt, HA, or ACh wake-active neurons, which may underlie the milder cognitive

238 eep is caused by the depolarization of sleep-active neurons, which secrete gamma-aminobutyric acid (G

239 trasted with results from striatal tonically active neurons, which show none of these task-related mo

240 iple items in WM that relies on persistently active neurons whose activation is orchestrated by oscil

241 is made of billions of highly metabolically active neurons whose activities provide the seat for cog

242 Because these are highly active neurons with a large number of Ca2+-permeable syn

243 urons with low membrane conductances than in active neurons with high conductance.

244 However, techniques for tagging only active neurons with high spatiotemporal precision remain

245 hole hippocampus, Camk2a+ neurons, or highly active neurons with phosphorylated ribosomal subunit S6

246 erived NPCs, yielding electrophysiologically active neurons within just 3 wk.

247 rousal systems including HCRT and other wake-active neurons within the PF-LHA and 5-HT neurons in the

248 emical identity of a delimited node of sleep-active neurons within the rostral medullary brainstem.