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1 inistration of PG(Bb) in BALB/c mice elicits acute arthritis.
2 nd T cell activation were evident but before acute arthritis.
3 o inflammatory activity in a rabbit model of acute arthritis.
4 tory disorders, including the development of acute arthritis.
5 c character of the inflammatory condition of acute arthritis.
6 bitor of apoptosis during the development of acute arthritis.
8 spirochete Borrelia burgdorferi, results in acute arthritis and carditis that regress as a result of
9 ies have shown that parvovirus B19 can cause acute arthritis and occasionally a chronic arthropathy,
13 The mice with the least retinol developed acute arthritis earlier and more severely than those wit
14 del of experimental ovalbumin (OVA)-mediated acute arthritis has been established in which autoimmuni
18 rile peritonitis, they are protected against acute arthritis induced by passive transfer of monoclona
22 Borrelia burgdorferi-infected mice develop acute arthritis that undergoes Ab-mediated resolution.