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1 ts targeted at the underlying disease (e.g., acute coronary occlusion).
2 al with STEMI and mapped the location of the acute coronary occlusion.
3 te the restoration of cardiac function after acute coronary occlusion.
4 nt detection of the onset of ischemia during acute coronary occlusion.
5 articipate in the arrhythmogenic response to acute coronary occlusion.
6 otherwise healthy men even in the absence of acute coronary occlusion.
7  hemodynamic status identifies patients with acute coronary occlusion.
8      Refractory OHCA patients frequently had acute coronary occlusion (64% versus 47%), especially le
9  tomography/magnetic resonance imaging after acute coronary occlusion and interventional reperfusion.
10 ore intensive resuscitation, higher rates of acute coronary occlusion, and poorer in-hospital outcome
11 ular infarct size was assessed at 24 h after acute coronary occlusion by triphenyltetrazolium chlorid
12                   In 31 open-chest dogs with acute coronary occlusion, dipyridamole (approximately 0.
13 requency, predictors, and clinical impact of acute coronary occlusion in hemodynamically stable and u
14 giography and to determine the prevalence of acute coronary occlusion in resuscitated patients with o
15       Early metoprolol administration during acute coronary occlusion increases myocardial salvage.
16                        However, treatment of acute coronary occlusion inevitably results in ischemia-
17                                              Acute coronary occlusion is a serious manifestation of c
18                                              Acute coronary occlusion is the leading cause of death i
19                                              Acute coronary occlusions leading to ST-segment elevatio
20                                              Acute coronary occlusions leading to STEMI tend to clust
21 e extent of microvascular obstruction during acute coronary occlusion may determine the eventual magn
22 uding volume and pressure overload during an acute coronary occlusion (n = 10).
23                                              Acute coronary occlusion or spasm was not observed at a
24 before vs. 0.93 +/- 0.41 mm Hg.ml(-1) during acute coronary occlusion [p < 0.05] and 7.9 +/- 3.1 m.s(
25  tested in an ambulatory porcine model, with acute coronary occlusion precipitated by stent thrombosi
26 pen-chest dogs were subjected to a 10-minute acute coronary occlusion (proximal left anterior descend
27 arrhythmias similar to those observed during acute coronary occlusion/reperfusion in intact hearts.
28                                              Acute coronary occlusion results in a rapid decrease in
29                          After 10 minutes of acute coronary occlusion, there was an upward shift of t
30 veloped a spatial map of the distribution of acute coronary occlusions to test our hypothesis that pl
31                                              Acute coronary occlusion was associated with an increase
32                                           An acute coronary occlusion was found in 11% of patients in
33  in distance from the ostium, the risk of an acute coronary occlusion was significantly decreased by
34                                              Acute coronary occlusions were found in 19.5% of stable
35  Identification of these high-risk zones for acute coronary occlusions will lead to future advances i
36                We tested the hypothesis that acute coronary occlusion would result in loss of forces