ライフサイエンスコーパス: acute hepatitis B

1 s) using blood samples from 18 patients with acute hepatitis B.

2 plays a central role in the pathogenesis of acute hepatitis B.

3 medicine clinic in the UK was diagnosed with acute hepatitis B.

4 ths after OLT) and the other patient died of acute hepatitis B (7 months after OLT).

5 ly 1992, a 47-year-old woman became ill with acute hepatitis B after undergoing a thymectomy in which

6 ded us to identify the changing incidence of acute hepatitis B (AHB) in children and young adults.

7 As a control, 38 patients with acute hepatitis B (AHB) without liver failure were inclu

8 t patients infected during adulthood develop acute hepatitis B (AHB), which usually results in viral

9 from 134 individuals including patients with acute hepatitis B and C as well as chronic hepatitis B,

10 red 25 inflammatory markers in patients with acute hepatitis B and chronic hepatitis B with hepatitis

11 Reported cases of symptomatic acute hepatitis B and hepatitis C occurring in persons>/

12 t in two (11%) of 19 Caucasian patients with acute hepatitis B and nine (27%) of 33 Caucasian patient

13 lood mononuclear cells from 67 patients with acute hepatitis B, and 12 patients convalescent from acu

14 ibe the results of enhanced surveillance for acute hepatitis B at 7 federally funded sites over a 6-y

15 In liver biopsy specimens from patients with acute hepatitis B, but not chronic hepatitis B or contro

16 using whole-genome sequences (n = 179) from acute hepatitis B cases (n = 1,206) identified through t

17 lness, suggesting that more than half of the acute hepatitis B cases might have been prevented throug

18 In the 6-year period, 2220 acute hepatitis B cases were reported from the 7 sites.

19 to factors potentiating hepatic damage with acute hepatitis B contributed to the outbreak's high mor

20 uring this period, the reported incidence of acute hepatitis B declined by 76.1% from 13.8 cases per

21 quence changes were easily detectable in the acute, hepatitis B e antigen-positive phase of infection

22 nvasive Haemophilus influenzae type b (Hib), acute hepatitis B, hepatitis A, varicella, Streptococcus

23 s targeted since 1980 including hepatitis A, acute hepatitis B, Hib, and varicella.

24 analysis of HBV whole genomes from cases of acute hepatitis B identified from 1998 to 2006 in the Un

25 irectly proportional to tissue injury during acute hepatitis B in humans.

26 n encouraging trend is that the incidence of acute hepatitis B in the United States declined as much

27 riod of decline in the reported incidence of acute hepatitis B in the United States.

28 scribes an uncommon original diagnosis of an acute hepatitis B infection occurring in a vaccinated bu

29 ca. 2002, coinciding with transient rises in acute hepatitis B notification rates among adults; D3 un

30 Death related to acute hepatitis B occurs in approximately 1% of patients

31 ompensated cirrhosis, organ transplantation, acute hepatitis B, pregnancy, coinfection with hepatitis

32 hich a thoracic-surgery resident who had had acute hepatitis B six months earlier assisted.

33 increased level of arginase in patients with acute hepatitis B suppresses the functions of activated

34 In patients with acute hepatitis B, the expansion of activated and prolif

35 ully vaccinated individuals among cases with acute hepatitis B, the proportion of preventable cases i

36 earlier was in favor of an evolution from an acute hepatitis B toward a chronic hepatitis B.

37 f T-bet in virus-specific CD8 T cells during acute hepatitis B virus (HBV) and hepatitis C virus (HCV

38 Some patients with acute hepatitis B virus (HBV) infection develop chronic

39 Clearance of acute hepatitis B virus (HBV) infection is associated wi

40 Recovery from acute hepatitis B virus (HBV) infection occurs in 95% of

41 Acute hepatitis B virus (HBV) infection remains a freque

42 Recovery from acute hepatitis B virus (HBV) infection requires a broad

43 During acute hepatitis B virus (HBV) infection viral loads reac

44 Following an acute hepatitis B virus (HBV) infection, clearance or pe

45 ogical profile suggesting a previous cleared acute hepatitis B virus (HBV) infection, including high

46 Acute hepatitis B virus (HBV) infections either resolve

47 rted a substantial increase in the number of acute hepatitis B virus (HBV) infections in the United S

48 cratic drug-induced liver injury DILI (22%), acute hepatitis B virus infection (12%), autoimmune hepa

49 iral antigens in a transgenic mouse model of acute hepatitis B virus infection.

50 982-1998, enhanced sentinel surveillance for acute hepatitis B was conducted in 4 counties in the Uni

51 A total of 362 of 11 311 (3.2%) cases with acute hepatitis B were vaccinated.

52 patitis B, and 12 patients convalescent from acute hepatitis B, were stimulated with three panels of