ライフサイエンスコーパス: acute phase

1 ) cells into the inflammatory lesions at the acute phase.

2 mproving Global Outcomes criteria during the acute phase.

3 had a second troponin measurement during the acute phase.

4 ntended to curtail renal inflammation in the acute phase.

5 esting prior to revascularization during the acute phase.

6 years), strong connections were found in the acute phase.

7 hepatitis C virus (HCV) infection during the acute phase.

8 ted with life-threating complications in the acute phase.

9 ifest restricted joint movements at the post-acute phase.

10 physiological reactivity were central in the acute phase.

11 th abbreviated courses of therapy during the acute phase.

12 rasite load within the myocardium during the acute phase.

13 characterized by vascular alterations in the acute phase.

14 asymptomatic compared with 4.4 % during the acute phase.

15 to die at a higher frequency during the sub-acute phase.

16 with the highest difference occurring at the acute phase.

17 ized persons with mild illness course in the acute phase.

18 c symptoms with negative serology during the acute phase.

19 7 kcal) was significantly higher than during acute phase (1,765 +/- 456 kcal) (p = 0.037).

20 In the acute phase, 1-3 days after BDL, BS concentrations in bi

21 Evidence is sparse and conflicting for acute-phase activation in catatonia, and whether this fe

22 Conclusion: Bile microinfarcts occur in the acute phase after BDL in a limited number of dispersed h

23 er the dramatic loss of vaginal FBLN5 in the acute phase after injury (12-48 h), but resulted in resc

24 is of native T1 maps already revealed in the acute phase after MI substantial discrepancies in myocar

25 the association between PTSD symptoms in the acute phase after trauma and the chronic phase.

26 nge, 0.3 to 17.9 years) were included in the acute-phase analysis.

27 a significant morbidity and mortality in the acute phase and an increased risk of developing End Stag

28 c resonance imaging was performed during the acute phase and at 1 month.

29 important to identify the pathogen(s) in the acute phase and determine appropriate treatment to minim

30 inflammation was significantly lower in the acute phase and higher in the chronic phase, reflecting

31 onsists of the use of pharmacotherapy in the acute phase and of psychotherapy in its residual phase.

32 how robust and diverse genomic signal in the acute phase and others have found shared biological mech

33 ells (CD3-CD161 +) were increased during the acute phase and progressively declined, whereas CD8+CD16

34 remarkable decrease in parasitemia levels in acute phase and the parasitemia reactivation following i

35 ant genes regulated by NRF2 and those of the acute phase and tissue injury response pathways, in both

36 fection also correlated positively with both acute-phase and chronic viremia.

37 drome encompassing events before, during the acute phase, and subsequently in the chronic phase is pr

38 monocytes to the lungs occurring during the acute phase, and we describe two subsets of interstitial

39 tion of immunoglobulin M (IgM)-specific shed acute phase antigen (SAPA) bands, using qPCR as the gold

40 nisms that regulate IFN levels following the acute phase are unknown.

41 Finally, a positive correlation between the acute-phase area under the curve (AUC) for IL-6 and solu

42 dy we present symptoms registered during the acute phase as well as long COVID, i.e. long-lasting COV

43 An elevated chymase level in acute phase blood samples was highly indicative of later

44 However, whether or how the acute-phase changes promote liver tissue resilience duri

45 The association of acute-phase complete CIV control (from first chemotherap

46 CIV control was more likely in patients with acute-phase complete CIV control (RR, 1.19; 95% CI, 1.06

47 Acute-phase complete CIV control and antiemetic regimen

48 and antiemetic regimen were associated with acute-phase complete CIV control in pediatric patients r

49 t was associated with a higher likelihood of acute-phase complete CIV control versus ondansetron alon

50 Acute-phase complete CIV control was less likely in olde

51 Association of the same factors and acute-phase complete CIV control with complete CIV contr

52 munity and activated B cell frequency in the acute phase correlated with prior KFDV vaccination, and

53 aemic stroke that occurs in three phases: an acute phase (Day 2) characterized by increased signal tr

54 The acute phase, defined as the first 96 hours after burn, i

55 , our studies provide evidence of OAS in the acute-phase dengue virus immune response, providing a ba

56 EM has a poor prognosis during the acute phase, despite a publication bias that could have

57 r; 95% CI, 0.96 to 0.98 per year) and longer acute-phase duration (RR, 0.89 per day; 95% CI, 0.84 to

58 apy block) with age, sex, race, cancer type, acute-phase duration, and antiemetic regimen was examine

59 Younger age, shorter acute-phase duration, and antiemetic regimen were associ

60 The acute phase (first hours to days) is characterized by an

61 sessions of CBASP or SP for 20 weeks in the acute phase, followed by 8 continuation sessions during

62 Studies of adults who had MRI in the acute phase following moderate or severe traumatic brain

63 An effective treatment during the acute phase following SCI could potentially have a posit

64 CD14 + + classical monocytes isolated in an acute phase from DENV-infected pediatric patients correl

65 nificantly stronger at 12 months than in the acute phase (global strength values, 6.57 vs 7.60; paire

66 of the infract in the fundus seen during the acute phase gradually resolved in about 2-3 weeks, leavi

67 ced an increased risk of new-onset HF in the acute phase (hazard ratio, 1.62; 95% confidence interval

68 t can be treated with drugs during the early acute phase; however, effective therapy against the chro

69 e, small ischemia studies show reductions in acute-phase hsCRP production with the IL-1 receptor anta

70 Patients were examined at 2 to 3 days (acute phase I) and 8 to 10 days (acute phase II), and so

71 to 3 days (acute phase I) and 8 to 10 days (acute phase II), and some of them at 4 to 6 months (chro

72 During acute phase II, favorable recovery from neglect was asso

73 r surface inflammation was common during the acute phase in both SJS and TEN.

74 Indeed, mortality reported during the acute phase in hospitalized patients is approximately 4%

75 decreased numbers of CD45+ cells during the acute phase in PB of aSAH patients compared with healthy

76 n RNAs by RNA-binding proteins (RBPs) and an acute phase in which expansion RNAs exceed RBP sequestra

77 evels were IFN responsive, increasing during acute phase infection and decreasing during a more quies

78 n together, we have identified signatures of acute-phase inflammation and oxidative stress in ifosfam

79 extended this analysis to show that PTX3, an acute phase inflammatory glycoprotein, is one such facto

80 in liver function, which correlated with an acute-phase inflammatory response gene signature.

81 In the acute phase, innate immune cells invade brain and mening

82 Its acute phase is associated with high parasitism, myocardi

83 of this model increased tissue parasitism to acute-phase levels and induced neutrophilic skeletal mus

84 nificant differences in ADIR and ADBR in the acute phase (LSMD: +0.11%; 95% confidence interval [CI]:

85 edictive equations among ICU patients during acute phase (<= 5 d), late phase (6-10 d), and chronic p

86 In its acute phase lung injury induced by tissue or bacterial p

87 The combination of B.b.cfDNA detection and acute-phase MTTT improves clinical sensitivity for diagn

88 C-lowering drug, has not been studied in the acute phase of ACS.

89 Following the acute phase of AFM, patients typically have substantial

90 efore, the optimal cut-off by CMR during the acute phase of an MI to predict viability was </=75% TEI

91 ance (CMR) for defining viability during the acute phase of an MI, using </=50% TEI at follow-up as t

92 However, in the acute phase of an ST-segment elevation myocardial infarc

93 ctional network connectivity obtained in the acute phase of CA were independently associated with FO

94 we show that TNF signaling is active in the acute phase of cardiac inflammation, which is characteri

95 hymase was significantly elevated during the acute phase of disease in DHF or Severe dengue, defined

96 of the hippocampal network, at least in the acute phase of epilepsy.

97 roduced by tmACs and sAC is required for the acute phase of extracellular signal regulated kinase 1/2

98 active CD8 T cells are infrequent during the acute phase of HIV-1 infection.

99 lable on the cellular immune response in the acute phase of human ZIKV infection, and its role in the

100 1) and/or plasmapheresis (n = 13) during the acute phase of HUS had comparable outcomes.

101 e detected strong upregulation of tPA in the acute phase of illness and in PUUV-infected macaques and

102 s with higher initial viral loads during the acute phase of illness had poor prognosis at the post-ac

103 he hypothesis that excitotoxicity during the acute phase of illness leads to reduced glutathione and

104 oads similar to those in patients during the acute phase of IM and a cell-mediated immune response th

105 they resembled those in patients during the acute phase of IM in timing and quality, were never as e

106 were as high as those in patients during the acute phase of IM, whereas the cell-mediated responses,

107 bance like those seen in patients during the acute phase of IM.

108 irculates in an open conformation during the acute phase of immune-mediated thrombotic thrombocytopen

109 y serologic tests are insensitive during the acute phase of infection and are often cross-reactive wi

110 f HIV-1 replication during the course of the acute phase of infection and the reactivation of silent

111 ZIKV-reactive T cells in the acute phase of infection are detected earlier and in gre

112 e preferential loss of Th17 cells during the acute phase of infection impairs the integrity of the gu

113 tween GAS burden and transmission during the acute phase of infection observed in humans and nonhuman

114 pression of activating FcgammaRIV during the acute phase of infection was also noteworthy.

115 Glucose metabolism also influenced the acute phase of infection when the replicating virus was

116 nor infected, exposed but not infected, pre-acute phase of infection, acute phase of infection, post

117 mmation in the ovary was observed during the acute phase of infection, but this normalized over time.

118 reduced in immunocompromised pigs during the acute phase of infection, but TNF-alpha-specific CD8(+)

119 or Human Immunodeficiency Virus (HIV) during acute phase of infection, initiating an ongoing cascade

120 not infected, pre-acute phase of infection, acute phase of infection, post-acute phase of infection.

121 gs continued fecal virus shedding beyond the acute phase of infection, whereas the majority (7/10) of

122 ations in the RV217 (ECHO) cohort during the acute phase of infection.

123 e responses compared to survivors during the acute phase of infection.

124 ines IL-4 and IL-10, particularly during the acute phase of infection.

125 ontrol of TB in nonhuman primates during the acute phase of infection.

126 of infection, acute phase of infection, post-acute phase of infection.

127 ha(v)beta(6)-BP) in a patient 2 mo after the acute phase of infection.

128 ralizing Abs in COVID-19 patients during the acute phase of infection.

129 high risk of ischemic stroke (IS) during the acute phase of infective endocarditis (IE).

130 les of these cells and chemokines during the acute phase of inflammation in sepsis.

131 In the acute phase of inflammation-(d1), 15-epi-LXA4 primes neu

132 nsive lowering of blood pressure (BP) at the acute phase of intracerebral haemorrhage (ICH) is benefi

133 Conversely, mast cell ablation after the acute phase of IRI had no impact on organ atrophy, tubul

134 cell trafficking can persist long after the acute phase of KD in patients with giant CAA.

135 y few activated B cells were observed in the acute phase of KFD infection.

136 During the acute phase of LF, we observed robust activation of the

137 In conclusion, statin during the acute phase of MI reduces insulin sensitivity in a dose-

138 paB (nuclear factor-kappaB) signaling in the acute phase of MI.

139 In the acute phase of MIS-C, we observed high levels of interle

140 we find that fate decisions made during the acute phase of murine CMV infection can alter the level

141 species exert anti-arrhythmic effects in the acute phase of myocardial ischemia has not been investig

142 often of benefit for recurrent VT after the acute phase of myocarditis.

143 lysis and reactive new bone formation in the acute phase of osteomyelitis.

144 Adult patients surviving the acute phase of PF (exposed group) were matched 1:1 for a

145 proinflammatory cytokines determined in the acute phase of PM.

146 y of life (HR-QOL) of patients surviving the acute phase of purpura fulminans (PF) has not been evalu

147 of antidromic M1 activation at least in the acute phase of STN DBS, the difference in observed antid

148 Changes in HDL proteins during early acute phase of stroke associate with recovery.

149 mmon use of sedatives and anesthetics in the acute phase of TBI management, their effect on glial cel

150 sponse seem to play a beneficial role in the acute phase of TBI.

151 Our results show that during the acute phase of the 2013-14 floods, potential feeding are

152 tients should start, if possible, during the acute phase of the disease and last more than 1 year.

153 rix metallopeptidase-2, were elevated in the acute phase of the disease when microglia were ablated w

154 men and is the only treatment option in the acute phase of the disease, when surgery is contraindica

155 thod to detect viral genetic material in the acute phase of the disease.

156 ce substantially reduces inflammation in the acute phase of the disease.

157 The first survey was conducted during the acute phase of the humanitarian response (October-Novemb

158 to derive estimates for the duration of the acute phase of the infection and effective population si

159 s in the brain that were infected during the acute phase of the infection and survived for approximat

160 o, resulting in higher susceptibility to the acute phase of the infection and they could not cure the

161 t side effects and are only effective in the acute phase of the infection with limited efficacy in th

162 nza virus-infected lungs for >4 h during the acute phase of the inflammation and at least 1 h in the

163 reviewed echocardiographic parameters in the acute phase of the MIS-C and KD groups, and during the s

164 pretation for diagnosing COVID-19 during the acute phase of the pandemic, using the COVID-19 Reportin

165 8(+) central memory T cells (TCM) during the acute phase of the primary response that is dominated by

166 nist isoxazole derivative, was tested in the acute phase of Theiler's murine encephalomyelitis virus-

167 ions and neuroendocrine symptoms, marked the acute phase of trichodysplasia spinulosa, whereas initia

168 terminals are significantly increased in the acute phase of TTS.

169 The development of FID in the acute phase of type B IMH has a poor prognosis owing to

170 therosclerotic mice as systemic drugs in the acute phases of atherosclerotic complications favor the

171 ential therapy that should be limited to the acute phases of ischemic insult and avoided for long-ter

172 During the acute phases of S. pneumoniae infection, these populatio

173 little is known about sex differences in the acute phase, or how disparities in the initial response

174 ulocyte-colony stimulating factor during the acute phase (p < 0.05).

175 Dialysis during the acute phase (P = .01), dialysis duration (P = .01), and

176 res are frequently elevated in sera from the acute-phase patients.

177 B cell repertoire analysis from acute-phase peripheral blood suggested that J9 and J8 fo

178 Serum amyloid A (SAA) is an acute-phase plasma protein that functions in innate immu

179 ein were significantly elevated at the early acute phase postinjury relative to baseline and both con

180 trally associated with other symptoms in the acute phase, potentially pointing to the utility of addr

181 Acute-phase production of MCP-1 correlated with viremia

182 repair mechanisms that persist following the acute phase promote inflammation and fibrosis in the chr

183 ations as a biomarker for sleep restriction, acute phase protein concentrations and malaria infection

184 The human acute phase protein haptoglobin (Hp) protects the host f

185 e-rich alpha-2 glycoprotein (LRG) is a novel acute phase protein involved in inflammation-associated

186 ntified as a carboxy-terminal peptide of the acute phase protein serum amyloid A (SAA) 1.

187 om the formation of amyloid fibrils from the acute phase protein serum amyloid A.

188 Alpha-1 antitrypsin (AAT) is an acute phase protein that possesses immune-regulatory and

189 Fibrinogen is an acute phase protein that will protect exposed cells from

190 Fibrinogen, an acute phase protein, has been shown to interact with the

191 C-reactive protein (CRP) is an acute-phase protein produced in high quantities by the l

192 The acute-phase protein serum amyloid A (A-SAA) was signific

193 sin (AAT) encoded by the SERPINA1 gene is an acute-phase protein synthesized in the liver and secrete

194 We first examined whether the acute-phase protein, alpha-2 macroglobulin (A2M), a majo

195 ly are sensed by an evolutionarily conserved acute-phase protein, serum amyloid A1 (SAA1), that promo

196 isk of ASD in relation to levels of neonatal acute phase proteins (APPs), key components of innate im

197 Acute phase proteins (APPs), plasma proteins synthesized

198 m, longitudinal within-individual changes in acute phase proteins (C-reactive protein [CRP], alpha-1-

199 , hepatic mRNA of inflammatory cytokines and acute phase proteins was upregulated, and liver-infiltra

200 In liver, they regulate the secretion of acute phase proteins.

201 and endotoxin core IgG antibody [EndoCAb]), acute-phase proteins (alpha-2 macroglobulin [alpha-2M],

202 HDL particles were enriched with acute-phase proteins (serum amyloid A, haptoglobin, and

203 talyze fluctuations in the concentrations of acute-phase proteins and certain micronutrient biomarker

204 infiltrating Ly6C(pos) macrophages expressed acute-phase proteins and exhibited an inflammatory profi

205 tion and accompanying increases in levels of acute-phase proteins and markers of inflammation and pro

206 lammation such as inflammatory cytokines and acute-phase proteins are reliably elevated in a signific

207 ables, peak values of 15 cytokines, and nine acute-phase proteins in serum were evaluated as potentia

208 Here, we posit that inflammatory and acute-phase proteins in the circulation increase after O

209 In this context, acute-phase proteins such as Pentraxin-3 (PTX3) are rele

210 clusters: interleukins, adhesion molecules, acute-phase proteins, and chemokines.

211 d levels of microbial translocation markers, acute-phase proteins, and inflammatory markers were all

212 S and other microbial translocation markers, acute-phase proteins, inflammatory markers, and proinfla

213 ear relation of ferritin concentrations with acute-phase proteins.

214 hypothesis that human fibrinogen (FBG) - an acute phase reactant - inhibits human MMP-2.

215 us MMP-2 inhibitors such as TIMPs and/or the acute phase reactant alpha-2-macroglobulin.

216 Serum amyloid A (SAA) proteins are acute-phase reactant associated with high-density lipopr

217 an indicator of functional ID that is not an acute-phase reactant, but challenges in its interpretati

218 tory investigation revealed the elevation of acute phase reactants and strongly positive cytoplasmic

219 Increases in acute phase reactants are typical of polymyalgia rheumat

220 hat is one of the most dramatically changing acute phase reactants in the circulation.

221 tored closely, and persistent high levels of acute phase reactants should raise concerns about amyloi

222 hycardia, decreasing haemoglobin, increasing acute phase reactants tests, and multiorgan failure.

223 g syndrome (electrolytes and phosphate), and acute phase reactants, and recorded the nutritional ther

224 oncentration and glycosylation state of main acute phase reactants.

225 differential nasal airway response including acute-phase reactants proteins (fibrinogen, haptoglobin

226 tored closely, and persistent high levels of acute-phase reactants should raise concerns about amyloi

227 metabolites, ketone bodies, amino acids, and acute-phase reaction markers.

228 kin 1beta and 6 (IL-1beta, IL-6) mediate the acute phase response (APR).

229 lly, the up-regulated genes were enriched in acute phase response (P < 0.01), pyruvate metabolic proc

230 Response to the burn injury during the acute phase response after burn is substantially differe

231 Inflammation was an acute phase response and chronic inflammation, with litt

232 og[P-value] = 15 vs. 10, respectively) while acute phase response and complement system were affected

233 ription factor known to regulate the hepatic acute phase response and energy homeostasis under stress

234 cardial infarction induces activation of the acute phase response and infiltration of leukocytes to t

235 netic risk factors, its association with the acute phase response and its age of onset and female pre

236 ls, 12 proteins some of which participate in acute phase response and platelet activation (APMAP [adi

237 hepatocytes is important for control of the acute phase response and regulation of liver regeneratio

238 revealed that inflammatory mediators of the acute phase response and the complement cascade were hig

239 ne disease often present with a debilitating acute phase response as a result of Vgamma9Vdelta2 T cel

240 The results show activation of the hepatic acute phase response by 2 hours after TBI, hepatic infla

241 iological ER stress-induced inflammatory and acute phase response in adipocytes, leading to lower cir

242 he IL1B regulator induces a highly conserved acute phase response in each organ assessed for all of b

243 ost bacteria and hypoferremia induced by the acute phase response is a key element of innate immunity

244 The acute phase response of AAT (alpha-1 antitrypsin) to COV

245 implicated in nuclear receptor activations, acute phase response pathway, glutaryl-CoA/tryptophan de

246 he aim of this study was to determine if the acute phase response post burn injury is significantly d

247 The high-levels and representation of acute phase response proteins suggests a functional role

248 y and repair such as coagulation/thrombosis, acute phase response signaling and complement activation

249 d to phagocytosis and signaling (IL-6, IL-8, acute phase response) are upregulated in 5xFAD;CD33(-/-)

250 n SAA, in concentrations resembling a modest acute phase response, was added to platelet-poor plasma

251 The hepatic acute-phase response (APR), which is largely dependent o

252 Brain injury elicits a systemic acute-phase response (APR), which is responsible for co-

253 ription factor known to regulate the hepatic acute-phase response and lipid homeostasis.

254 ese genes comprise coexpression networks for acute-phase response and pro-inflammatory processes.

255 BRINDA investigators found that the acute-phase response is so prevalent that it must be ass

256 y analysis suggests that cerebral immune and acute-phase response may play a role in mediating PaO2 o

257 acterize the effects of inflammation and the acute-phase response on nutrient biomarkers.

258 resent because of confounding effects of the acute-phase response on the interpretation of most iron

259 RP) and serum amyloid A (SAA), the prototype acute-phase response proteins, in the context of the var

260 an association between calcification events, acute-phase response signaling, and coagulation and gluc

261 ion regulates the unfolded protein response, acute-phase response, and DDR in hepatocytes.

262 ent endoplasmic reticulum stress, defects in acute-phase response, and increased hepatocellular damag

263 ia, defective B cell memory, and an impaired acute-phase response, as well as skeletal abnormalities

264 al systemic inflammatory biomarkers, but the acute-phase response, enterocyte turnover, monocyte acti

265 set of genes implicated in proteostasis, the acute-phase response, metabolism, and the DNA damage res

266 After adjusting for the acute-phase response, serum ferritin and RBP concentrati

267 n systemic immunity, they are central in the acute-phase response, which floods the circulation with

268 cause RBP concentrations decrease during the acute-phase response.We aimed to assess the relation bet

269 er upregulation of interferon signalling and acute phase responses compared to survivors during the a

270 Acute phase responses, cellular responses to lipopolysac

271 lifying proinflammatory cytokine release and acute phase responses.

272 transcripts associated with inflammatory and acute-phase responses, coagulative activities, and trans

273 resented with recurrent infections, abnormal acute-phase responses, elevated IgE, eczema, and eosinop

274 in the Telemedicine Approaches to Evaluating Acute-Phase Retinopathy of Prematurity (e-ROP) Study and

275 The Telemedicine Approaches to Evaluating Acute-Phase Retinopathy of Prematurity (e-ROP) Study tel

276 om the Telemedicine Approaches to Evaluating Acute-Phase Retinopathy of Prematurity (e-ROP) study was

277 elemedicine Approaches for the Evaluation of Acute-Phase Retinopathy of Prematurity (e-ROP) study.

278 om the Telemedicine Approaches to Evaluating Acute-Phase Retinopathy of Prematurity study, conducted

279 the Telemedicine Approaches to Evaluating of Acute-Phase ROP (e-ROP) Study.

280 ommonly after treatment with bevacizumab for acute-phase ROP than after laser.

281 Acute-phase SARS-CoV-2-specific T cells displayed a high

282 nt gangliosides (ganglioside complex) in the acute-phase sera of some patients with GBS suggested the

283 In comparison, sensitivity of acute-phase serology using modified two-tiered testing (

284 gainst GlpQ which is not highly sensitive on acute phase serum samples.

285 Viral load was quantified in acute-phase serum by real-time reverse transcription pol

286 Acute-phase serum samples from 346 patients with a suspe

287 nal lesions and of the optic disc during the acute phase showed no statistically significant differen

288 ry artery dilatation (z score = 3.15) in the acute phase, showing resolution during early follow-up.

289 gammadelta and CD4(+)CCR7(+) T cells in the acute phase, suggested that these immune cell population

290 s ratio=0.87, 95% CI=0.82, 0.91) of adequate acute-phase treatment and 19% lower odds (adjusted odds

291 ears, but among individuals who survived the acute phase, treatment was associated with a significant

292 side effects (secondary outcomes) with three acute-phase treatments for bipolar II depression.

293 ete CIV control in the delayed phase (end of acute phase until <= 96 hours later) was examined.

294 inhibitor, these cells significantly reduced acute-phase viremia, as well as accelerated HIV suppress

295 n women, activation of CD4(+) T cells in the acute phase was significantly correlated with plasma lev

296 ons with the best agreement and accuracy for acute phase was Swinamer (1990), for late phase was Bran

297 Clinical and laboratory measures during the acute phase were compared between adult and elderly pati

298 se of illness had poor prognosis at the post-acute phase with more restricted joint movement and high

299 mptoms were central to other symptoms in the acute phase, with intrusions and physiological reactivit

300 During the acute phase, ZIKV productively infected the ovary causin