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1 osis of carditis as a major manifestation of acute rheumatic fever.
2 abscesses and 2 (0.28%) were diagnosed with acute rheumatic fever.
3 mporally with a decrease in the incidence of acute rheumatic fever.
4 atment in timely fashion in order to prevent acute rheumatic fever.
5 n that at which group A streptococcus causes acute rheumatic fever.
6 quelae of group A streptococcal infection in acute rheumatic fever.
7 fulfill the Jones Criteria for diagnosis of acute rheumatic fever.
8 i.e., skin infection and pharyngitis-induced acute rheumatic fever.
9 They had had a total of 20 attacks of acute rheumatic fever.
10 and absence of other major manifestations of acute rheumatic fever.
11 eptococcal throat culture who are at risk of acute rheumatic fever.
12 es: uncomplicated pharyngitis, impetigo, and acute rheumatic fever.
13 carditis, and correlates of mortality after acute rheumatic fever.
15 1000 patient-years), 19 (0.6%) had recurrent acute rheumatic fever (3.49/1000 patient-years), and 20
16 The pathogenic mechanisms for developing acute rheumatic fever after group A streptococcal pharyn
18 The intervals between the last attack of acute rheumatic fever and operation ranged from 2 to 8 y
20 f 7 strategies for the primary prevention of acute rheumatic fever and rheumatic heart disease in chi
21 performance in screening for and diagnosing acute rheumatic fever and rheumatic heart disease needs
23 or studies on the screening and diagnosis of acute rheumatic fever and rheumatic heart disease using
26 with documented GAS infections (pharyngitis, acute rheumatic fever, and severe invasive disease) also
30 nvestigated adverse outcomes for people with acute rheumatic fever (ARF) and rheumatic heart disease
32 well as serious autoimmune sequelae such as acute rheumatic fever (ARF) and subsequent rheumatic hea
38 itis from group A streptococcus, episodes of acute rheumatic fever (ARF), cases of RHD, heart failure
39 l M protein is an epidemiological marker for acute rheumatic fever (ARF)-associated serotypes of grou
40 d States, Kawasaki disease has now surpassed acute rheumatic fever as the leading cause of acquired h
41 We aimed to provide a comprehensive list of acute rheumatic fever-associated GAS isolates and assess
44 nternational guidelines for the diagnosis of acute rheumatic fever by defining high-risk populations,
45 drome was considered part of the spectrum of acute rheumatic fever by some, whereas others stressed t
46 only have the diagnostic Jones criteria for acute rheumatic fever changed, but substantial advances
49 rval (hazard ratio 4.4 [95% CI 1.7-11.2]) at acute rheumatic fever diagnosis were associated with inc
50 ents already had chronic carditis at initial acute rheumatic fever diagnosis, suggesting previous und
51 s a means to diagnose cardiac involvement in acute rheumatic fever, even when overt clinical findings
52 icated that serum samples from patients with acute rheumatic fever have higher levels of antibodies t
53 espite a recent increase in the incidence of acute rheumatic fever in North America, rheumatic heart
54 the historic Jones criteria used to diagnose acute rheumatic fever in the context of the current epid
55 ata highlight the large burden of undetected acute rheumatic fever in these settings and the need for
56 th post-streptococcal glomerulonephritis and acute rheumatic fever indicated that FBP54 is expressed
57 uracy in diagnosing rheumatic heart disease, acute rheumatic fever, or carditis with acute rheumatic
59 ac-related death, and 17 (11%) had recurrent acute rheumatic fever over a median of 4.3 (IQR 3.0-4.8)
60 impact of population-specific differences in acute rheumatic fever presentation and changes in presen
61 ase, acute rheumatic fever, or carditis with acute rheumatic fever (primary outcomes) were extracted
63 We aimed to describe 3-5 year mortality, acute rheumatic fever recurrence, changes in carditis, a
69 of 5 of 13 KS sera, as compared with 5 of 8 acute rheumatic fever sera, contained Ab titers to human
70 d had a different pattern of reactivity than acute rheumatic fever sera, further supporting the assoc
71 coccal homologue in certain diseases such as acute rheumatic fever, suggest that SOK plays an importa
73 4-23 years who were diagnosed with definite acute rheumatic fever using the modified 2015 Jones crit