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1 ous glycerol became the major contributor to acylglycerols.
2 nd should be generally applicable to other 2-acylglycerols.
3 l-modified acylglycerols as a new structured acylglycerols.
4 e reaction involving glycerol 3-phosphate, 1-acylglycerol 3-phosphate, and dihydroxyacetone phosphate
5 eparation necessary to isolate the labeled 2-acylglycerol [(3)H]2-AG resulted in only 4% of the rearr
6 cids arising from the dephosphorylation of 1-acylglycerol-3-P followed by the deacylation of monoacyl
7 glycerol acetyltransferase 1), and Agpat3 (1-acylglycerol-3-phospate O-acyltransferase 3), and lipoly
8           The effects of overexpression of 1-acylglycerol-3-phosphate acyltransferase (AGAT)-alpha, w
9                                        The 1-acylglycerol-3-phosphate acyltransferase 1 haplotypes we
10              Loss-of-function mutations in 1-acylglycerol-3-phosphate O-acyltransferase (AGPAT) 2 in
11                  AGPAT6 is a member of the 1-acylglycerol-3-phosphate O-acyltransferase (AGPAT) famil
12 patic insulin resistance in mice that lack 1-acylglycerol-3-phosphate O-acyltransferase 2 (AGPAT2).
13 nt mutations of the gene (AGPAT2) encoding 1-acylglycerol-3-phosphate O-acyltransferase 2 in 20 affec
14               UBXD8 binds to and activates 1-acylglycerol-3-phosphate O-acyltransferase 3 (AGPAT3), w
15 5-AzaC also strongly induced expression of 1-acylglycerol-3-phosphate O-acyltransferase 9 (AGPAT9) an
16 ipins 2/3/5, hormone-sensitive lipase, and 1-acylglycerol-3-phosphate O-acyltransferase ABHD5.
17 rms for the same enzymes, specifically for 1-acylglycerol-3-phosphate O-acyltransferases (AGPATs), ha
18                           Mutations in the 1-acylglycerol-3-phosphate-O-acyltransferase 2 (AGPAT2) ge
19    Eight different genetic loci, including 1-acylglycerol-3-phosphate-O-acyltransferase 2, Berardinel
20  phospholipid antigen contained a C18:0 lyso-acylglycerol, a C16:0-acylated inositol, and an unsubsti
21                                              Acylglycerols (AGs) rich in n-3 were produced by extract
22 ming both all acids of the same type on each acylglycerol and all acids randomly distributed on the a
23  results present high resolution data on the acylglycerol and cholesterol ester species that were aff
24 uantitation of fatty acid acyl chains in the acylglycerol and FFA portions.
25 k, the effect of the main precursors, namely acylglycerols and chlorinated compounds, on the formatio
26  temporary storage of neutral lipids such as acylglycerols and steryl esters.
27 r colipase with a diacylphosphatidylcholine, acylglycerols, and free fatty acid was investigated by m
28 idative stability of distigmasterol-modified acylglycerols as a new structured acylglycerols.
29 ospectroscopy allowed us to locally identify acylglycerols as the main constituents of the pattern di
30 ult that both hydrolyzed fatty acids and the acylglycerol backbone are re-esterified to form TG.
31 osphorylates diacylglycerol, ceramide, and 1-acylglycerol but not sphingosine.
32 hese data, we conclude that the acylation of acylglycerols by DGAT1 is important for dietary fat abso
33 from 35 % to 52 % of omega3 mass fraction in acylglycerols, by losing 12.1 % of omega3 as ethyl ester
34 d TG resynthesis, occurring because released acylglycerols cannot be used for phospholipid synthesis.
35 lycerols (SQDGs), sphingolipids, di- and tri-acylglycerols (DAGs and TAGs), and sterol derivatives.
36  on acid distribution affects the results of acylglycerols fraction composition.
37 sumptions showed strong discrepancies on the acylglycerols fraction compositions predictions, demonst
38 mportant when an accurate description of the acylglycerols fraction is desired.
39 I-EDP-CID was applied for the analysis of 57 acylglycerols in linseed oil with complete DB position c
40 hesis of glycogen and the glycerol moiety of acylglycerols in skeletal muscle of animals with high pl
41 to incorporation into the glycerol moiety of acylglycerols in the liver.
42 alysis of five lipids (4 phospholipids and 1 acylglycerol) in complex mixtures using MALDI-TOF-MS wit
43 glyceride species (alkyl, acyl- and alkenyl, acylglycerols) in rat mesangial cells, a smooth muscle-l
44 undance of non-chlorinated compounds, namely acylglycerols, in the first stages of the treatment sugg
45                      The analyses pointed to acylglycerol kinase (AGK) and general transcription fact
46 munoblot studies, we validated mitochondrial acylglycerol kinase (AGK) as a new direct target for miR
47 inase receptor, type 3 (NTRK3) and fusion of acylglycerol kinase (AGK) with BRAF.
48 rization of a novel lipid kinase, designated acylglycerol kinase (AGK), that phosphorylates monoacylg
49 ncodes a functional DGAT and that changes in acylglycerol lipid metabolism disrupt normal egg chamber
50 found in vitro that they are nucleated at an acylglycerol lipid-water interface.
51            This dovetails with evidence that acylglycerol lipids are involved in hemozoin nucleation
52 al lines of evidence point to involvement of acylglycerol lipids in the nucleation process.
53 s hydrolysis (forskolin) suggesting a shared acylglycerol-mediated mechanism.
54  through short path distillation (SPD) of an acylglycerol mixture (containing 67% MAGs) produced by e
55 roductive cell signaling; interaction by the acylglycerol moiety of GPIs is also required.
56 t in rat adipocytes, probably by hydrolyzing acylglycerols or acyl-CoA esters to the respective free
57 epatic lipogenesis, the glycerol backbone of acylglycerols originates from one of three sources: gluc
58 t, the late endosome-specific lipid bis(mono)acylglycerol phosphate (BMP) 44:12 was markedly decrease
59 cyltransferase), and plsC (yhdO) (acyl-ACP:1-acylglycerol-phosphate acyltransferase) function in phos
60                                 Medium-chain acylglycerol-phosphates were found to self-assemble into
61 ps allowed for the selection of longer-chain acylglycerol-phosphates.
62 gents leads to the formation of a library of acylglycerol-phosphates.
63 nvolve TG-derived acyl groups rather than an acylglycerol precursor.
64                                      Partial acylglycerols showed greater ability, than did triacylgl
65 cylation activity at the level of acyl-CoA:1-acylglycerol-sn-3-phosphate acyltransferase.
66                                     Among 33 acylglycerol standards, selective ionization for acylgly
67 s used to identify lipids (free fatty acids, acylglycerols, sterols, sterol esters, glycolipids, phos
68 stingly, the presence of ascorbic acid in an acylglycerol structure protected alpha-tocopherol agains
69 ical cations (M(+.)) and [M-H](+) cations of acylglycerols termed Electron Deficient Precursor Ions (
70 sts of a number of stereo- and regioisomeric acylglycerols, their components remain challenging analy
71 d the acyl-CoA independent transacylation of acylglycerols, thereby facilitating energy mobilization
72 amily that possesses triglyceride lipase and acylglycerol transacylase activities.
73 psilon, iPLA2zeta, and iPLA2eta also possess acylglycerol transacylase activity utilizing mono-olein
74 ese results identify three novel TAG lipases/acylglycerol transacylases that likely participate in TA
75 ats, and the glycerol moiety from hydrolyzed acylglycerols was analyzed by (13)C NMR.
76 ch substrate source to glycerol in rat liver acylglycerols was determined using (13)C-enriched substr
77 trate to glycogen and the glycerol moiety of acylglycerols was evaluated.
78  of hemozoin, was consistently induced at an acylglycerol-water interface via their {100} crystal fac
79 glycerol standards, selective ionization for acylglycerols with >=1 DB was observed, where acylglycer
80 cylglycerols with >=1 DB was observed, where acylglycerols with <=3 DB formed mainly [M-H](+) ions an