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1 irments could be ameliorated by blocking the adenosine A1 receptor.
2 tion than that produced by activation of the adenosine A1 receptor.
3 a subunits rescued presynaptic inhibition by adenosine A1 receptors.
4 ction by ethanol is mediated through myocyte adenosine A1 receptors.
5 ry effect appears to be due to activation of adenosine A1 receptors.
6 europrotection through activation of central adenosine A1 receptors.
7 more heterogeneous activation of presynaptic adenosine A1 receptors.
8 could transiently be provoked after blocking adenosine A1 receptors.
9 opamine D2, metabotropic glutamate mGlu5 and adenosine A1 receptors.
10 r/middle lateral RA had significantly higher adenosine A1 receptor (2.7+/-1.7-fold; P<0.01) and GIRK4
11 tency (hA1, Ki=7 nM) and selectivity for the adenosine A1 receptor (915-fold versus adenosine A2A rec
12                                              Adenosine A1 receptor (A1-AdoR) function in rat ventricl
13 ic and diuretic action of a highly selective adenosine A1 receptor (A1AdoR) antagonist, 1,3-dipropyl-
14 the hypothesis that a partial agonist of the adenosine A1 receptor (A1AdoR) may cause a greater atten
15      In this study, we targeted the cochlear adenosine A1 receptor (A1AR) by trans-tympanic injection
16              In this study, we show that the adenosine A1 receptor (A1AR) protects against cisplatin
17                                              Adenosine A1 receptor (A1AR) subtype present on renal pr
18                     Allosteric modulation of adenosine A1 receptors (A1ARs) offers a novel therapeuti
19 duced long-term depression (LTD) mediated by adenosine A1 receptor (A1R) activation at corticostriata
20                                              Adenosine A1 receptor (A1R) agonist N6-cyclopentyl-adeno
21                                CPA, a stable adenosine A1 receptor (A1R) agonist, did not affect KDR
22 l blockade of A1 receptor signaling using an adenosine A1 receptor (A1R) antagonist, 8-cyclopentyl-1,
23 ne induces cortical actin polymerization via adenosine A1 receptor (A1R) induction of a Rho GTPase CD
24                                          The adenosine A1 receptor (A1R) is a key mediator of the neu
25  neuronal activity through activation of the adenosine A1 receptor (A1R), resulting in antinociceptio
26 els around synapses resulting in an enhanced adenosine A1 receptor (A1R)-dependent protective tone de
27  adenosine alleviates pain by activating the adenosine A1 receptor (A1R).
28 rogeneous expression of If, INa currents and adenosine A1 receptors (A1R) across SAN pacemaker-conduc
29 on upregulate adenosine concentration and/or adenosine A1 receptors (A1R) in the brain.
30                    Activation of presynaptic adenosine A1 receptors (A1Rs) causes substantial synapti
31                                              Adenosine A1 receptors (A1Rs) in human and rodent brains
32           Because very little is known about adenosine A1 receptors (A1Rs) in the spinal cord, we det
33        In hippocampal neurons, activation of adenosine A1 receptors (A1Rs) or GABA(B) receptors on sy
34  plasticity by experimental manipulations to adenosine A1 receptors (A1Rs).
35 seizures in mice by increasing activation of adenosine A1 receptors (A1Rs).
36 ce of heterosynaptic plasticity; blockade of adenosine A1 receptors abolished it.
37 ce of heterosynaptic plasticity: blockade of adenosine A1 receptors abolished weight dependence, whil
38                                              Adenosine A1 receptor activation also impaired the expre
39 algesic effect of acupuncture is mediated by adenosine A1 receptor activation at the acupuncture poin
40                 The inhibitory modulation of adenosine A1 receptor activation by hydroxylamine sugges
41                                              Adenosine A1 receptor activation depresses excitatory tr
42                                              Adenosine A1 receptor activation dose-dependently and se
43 P inhibited excitatory neurotransmission via adenosine A1 receptor activation in spinal cord slices f
44 this model of trigeminovascular nociception, adenosine A1 receptor activation leads to neuronal inhib
45 een reported in response to agonist-mediated adenosine A1 receptor activation, the implications of A2
46 -induced mechanical allodynia via peripheral adenosine A1 receptor activation.
47 is study, we report a novel dual role of the adenosine A1 receptor (Adora1) as an E2/ERalpha target a
48 ssion of a potential glucagon inhibitor, the adenosine A1 receptor (Adora1), is gradually diminished
49 sponses are thought to require activation of adenosine A1 receptors (adorA1Rs) and release of transmi
50 -D-aspartate receptor antagonist MK-801; the adenosine A1 receptor agonist 2-chloro-N6-cyclopentylade
51 the GABA(B) receptor agonist baclofen or the adenosine A1 receptor agonist 2-chloroadenosine, short-t
52               These data are consistent with adenosine A1 receptor agonist actions on REM sleep media
53 the NO donor sodium nitroprusside (SNP), the adenosine A1 receptor agonist CCPA (2-chloro-N6-cyclopen
54 osine A2a receptor agonist APEC, but not the adenosine A1 receptor agonist CHA, attenuated c-Fos indu
55 r-bearing mice was lowered to 32 C using the adenosine A1 receptor agonist N(6)-cyclohexyladenoxine t
56               Intrastriatal microinfusion of adenosine A1 receptor agonist N6-cyclohexyladenosine (CH
57                        Pretreatment with the adenosine A1 receptor agonist N6-cyclohexyladenosine (CH
58 of the BF cholinergic neurons, the selective adenosine A1 receptor agonist N6-cyclohexyladenosine, ad
59 ditioning was mimicked by perfusion with the adenosine A1 receptor agonist N6-cyclopentyladenosine an
60                            Microinjection of adenosine A1 receptor agonist or an inhibitor of adenyly
61                       Direct injection of an adenosine A1 receptor agonist replicated the analgesic e
62 lisopropyl)-adenosine (R-PIA [50 nmol/L], an adenosine A1 receptor agonist), or 4 beta-phorbol 12-myr
63                   The selective low efficacy adenosine A1 receptor agonist, GR190178 (30-1000 microg/
64 enous administration of the highly selective adenosine A1 receptor agonist, GR79236 (3-100 microg/kg)
65 adenoson bialanate, a first-in-class partial adenosine A1 receptor agonist, has the potential to impr
66                    N6-Cyclopentyl adenosine (adenosine A1 receptor agonist, Kd = 1 nmol/l) did not af
67          Systemic injection of the selective adenosine A1 receptor agonist, N(6)-cyclohexyladenosine
68 lure was studied with the use of a selective adenosine A1 receptor agonist, N6-cyclohexyl-2'-O-methyl
69               In Experiment 1, the selective adenosine A1 receptor agonist, N6-cyclopentyladenosine (
70 A, 48.4+/-5.5% with SfA; P<0.001), CCPA (the adenosine A1-receptor agonist, 200 nmol/L) (24.9+/-4.5%
71  the effects of capadenoson (CAP), a partial adenosine A1-receptor agonist, on left ventricular (LV)
72                                The selective adenosine A1 receptor agonists 2-chloro-N6-cyclopentylad
73 flammatory sites was signficantly reduced by adenosine A1 receptor agonists injected through intrathe
74 is a considerable literature to suggest that adenosine A1 receptor agonists may have anti-nociceptive
75 onists, nitric oxide synthase inhibitors and adenosine A1 receptor agonists.
76   The results support development of partial adenosine A1-receptor agonists for the treatment of chro
77                                 Therapy with adenosine A1-receptor agonists, however, is limited by u
78                           Overexpressing the adenosine A1 receptor also led to increased protection a
79                  Allosteric enhancers of the adenosine A1 receptor amplify signaling by orthosteric a
80 amine D2 receptor, dopamine transporter, and adenosine A1 receptor and decreased adenosine A2A recept
81 al inhibition was blocked in the presence of adenosine A1 receptor and GABAB receptor blockade.
82 entry in RA areas with highest expression of adenosine A1 receptor and its downstream GIRK (G protein
83 denosine (2CA) and baclofen, agonists of the adenosine A1 receptor and the GABAB receptor, respective
84 previously produced up-regulation of central adenosine A1 receptors and created a state of opiate dep
85 e examined the manner in which activation of adenosine A1 receptors and GABAB receptors modulates syn
86 ynthesis, which was prevented by blockade of adenosine A1 receptors, and decreased expression of gene
87 f ATP-sensitive potassium (K(ATP)) channels, adenosine A1 receptors, and the effects of different lev
88  attenuated by adrenergic alpha 2-receptors, adenosine A1-receptors, and histamine H3-receptors.
89                                    Moreover, adenosine A1 receptor antagonism had larger effects on t
90 s to cardiac arrhythmias and seizures due to adenosine A1-receptor antagonism.
91 BTI), or exogenous adenosine, or a selective adenosine A1 receptor antagonist (8-cyclopentyl-1, 3-dim
92                                          The adenosine A1 receptor antagonist 1,3-dipopylcyclopentylx
93 (19.6 +/- 0.9%) by systemic injection of the adenosine A1 receptor antagonist 8-CPT (2.5 mg kg(-1)) a
94  was largely reversed in the presence of the adenosine A1 receptor antagonist 8-cyclopentyl-1, 3-dipr
95                                          The adenosine A1 receptor antagonist 8-cyclopentyl-1,3-dipro
96 denosine as a mediator of IPC, the selective adenosine A1 receptor antagonist 8-cyclopentyl-1,3-dipro
97                                The selective adenosine A1 receptor antagonist 8-cyclopentyl-1,3-dipro
98                                     Both the adenosine A1 receptor antagonist 8-cyclopentyl-1,3-dipro
99  (NOS) inhibitor l-NAME (Group 1, n = 8) and adenosine A1 receptor antagonist 8-cyclopentyl-1,3-dipro
100 roduced by the administration of a selective adenosine A1 receptor antagonist but not by a selective
101        Prior administration of the selective adenosine A1 receptor antagonist dipropylcyclopentylxant
102  GR79236 could be inhibited by the selective adenosine A1 receptor antagonist DPCPX (300 microg/kg; P
103 fore and after administration of a selective adenosine A1 receptor antagonist DPCPX (8-cyclopentyl-1,
104 value of 0.4 nM for antagonism of CPA by the adenosine A1 receptor antagonist DPCPX (8-cyclopentyl-1,
105                                          The adenosine A1 receptor antagonist DPCPX did not affect th
106       Intracerebroventricular infusion of an adenosine A1 receptor antagonist produced a similar decr
107                       Here, we show that the adenosine A1 receptor antagonist rolofylline (KW-3902) i
108 Controlled Randomized Study of the Selective Adenosine A1 Receptor Antagonist Rolofylline for Patient
109 is comparable to that of BG9928, a selective adenosine A1 receptor antagonist that is currently in cl
110 yl-1,3-dipropylxanthine (DPCPX), a selective adenosine A1 receptor antagonist that is permeable to th
111              Imidazoline 14 is a competitive adenosine A1 receptor antagonist with a pA2 value of 8.8
112 -i]purin-5(4H)-one, is a particularly potent adenosine A1 receptor antagonist with good selectivity o
113                In the search for a selective adenosine A1 receptor antagonist with greater aqueous so
114 e presence of SPWs in slices treated with an adenosine A1 receptor antagonist, a finding that links t
115 ices treated with either a glial toxin or an adenosine A1 receptor antagonist.
116 blocked by protein kinase A inhibitor and an adenosine A1 receptor antagonist.
117 s with ADHF and volume overload, KW-3902, an adenosine A1-receptor antagonist, enhances the response
118 e hypothesis that the use of rolofylline, an adenosine A1-receptor antagonist, would improve dyspnea,
119  fibrillation were abolished or prevented by adenosine A1 receptor antagonists (50 mumol/L theophylli
120                The inhibition was blocked by adenosine A1 receptor antagonists and by agents that dis
121 vel tricyclic imidazoline antagonists of the adenosine A1 receptor are described.
122 cardial ischemia-reperfusion injury and that adenosine A1 receptors are required for this protective
123                  These results indicate that adenosine A1 receptors are selectively upregulated durin
124                                              Adenosine A1 receptor blockade abolished the protection
125 67156) inhibition of nucleotide breakdown or adenosine A1 receptor blockade and reduced by apyrase in
126                                              Adenosine A1 receptor blockade with 4.5 mg/kg DPCPX admi
127 ht to evaluate the dose-dependent effects of adenosine A1-receptor blockade on diuresis and renal fun
128 yclopentyl-1, 3-dipropylxanthine (DPCPX) (an adenosine A1 receptor blocker).
129  manipulated by microperfusing nephrons with adenosine A1 receptor blocker, A1-agonist, or 5'-nucleot
130 gated by TRPV4 channel, GABAB, as well as an adenosine A1 receptor blocker.
131 iated by increased activation of presynaptic adenosine A1 receptors by endogenous adenosine.
132                                              Adenosine A1 receptor control of the homeostatic regulat
133 antisense oligodeoxynucleotide targeting the adenosine A1 receptor desensitized the animals to subseq
134 haracterize SAN structure, SAN function, and adenosine A1 receptor expression in control (n=17) and 4
135 ption of caffeine during adulthood increased adenosine A1 receptor expression in the NAc, but no othe
136 d that its anti-nociceptive actions required adenosine A1 receptor expression.
137  were conducted to examine the importance of adenosine A1 receptors for the acquisition and expressio
138 chemia might be attributable to the enhanced adenosine A1 receptor function on synaptic transmission,
139 lized in lateral RA regions with the highest adenosine A1 receptor/GIRK4 expression.
140                To inhibit K(ATP) channels or adenosine A1 receptors, glibenclamide (0.1 mg/kg icv; n
141 5-cyano-2-thiopyrimidines as selective human adenosine A1 receptor (hA1AR) agonists with tunable bind
142 prior work demonstrated that the blockade of adenosine A1 receptors impairs heterosynaptic plasticity
143 ffects, and we sought to explore the role of adenosine A1 receptors in a model of trigeminovascular n
144                         Stimulation of renal adenosine A1 receptors in animal models has resulted in
145  blood brain barrier, we examine the role of adenosine A1 receptors in mediating cortical blood flow
146 d by endogenous GABAB, serotonin 5-HT1A, and adenosine A1 receptors in neurons coinfected with GIRK1+
147         In vitro studies supported roles for adenosine A1 receptors in promoting fatty acid synthesis
148 ncreases the concentrations of adenosine and adenosine A1 receptors in specific regions of the brain.
149 tissue thermogenesis, and that activation of adenosine A1 receptors in the CNS can block the brown ad
150 al (AAV) vector to focally delete endogenous adenosine A1 receptors in the hippocampus.
151 alothane level but not of K(ATP) channels or adenosine A1 receptors in the preconditioning effects of
152 entylxanthine, indicating the involvement of adenosine A1 receptors in the protection.
153 gical evidence supporting the involvement of adenosine A1 receptors in the regulation of the response
154 response (HVR), and investigated the role of adenosine A1 receptors in these SF effects in conscious
155              This effect requires functional adenosine A1 receptors, in line with the observation tha
156 (l-NAME, 1.5 mM), but was not affected by an adenosine A1 receptor inhibitor, 8-cyclopentyl-1,3-dipro
157 e first time in the mammalian fetus that the adenosine A1 receptor is an important mediator of brain
158 uction or oxygen consumption, suggesting the adenosine A1 receptor is involved in lowering metabolic
159                             We show that, in adenosine A1 receptor knock-out mice, impaired synaptic
160                   Furthermore, activation of adenosine A1-receptors markedly attenuated norepinephrin
161 l natriuretic peptide level, consistent with adenosine A1 receptor-mediated activity, was observed.
162 A3 receptors with Cl-IB-MECA antagonized the adenosine A1 receptor-mediated inhibition of excitatory
163      These data provide good evidence for an adenosine A1 receptor-mediated inhibition of mAChR-media
164       GABA does not appear to be involved in adenosine A1 receptor-mediated inhibition of neuronal fi
165          In addition, hydroxylamine reversed adenosine A1 receptor-mediated inhibition of the evoked
166                          Agents that inhibit adenosine A1 receptors might be useful in the treatment
167                                              Adenosine A1 receptors modulate baseline synaptic transm
168 or agonists directly reduces tremor, whereas adenosine A1 receptor-null mice show involuntary movemen
169                         Activation of either adenosine A1 receptors or GABAB receptors inhibits many
170 olar constriction, a process mediated by the adenosine/A1 receptor pathway.
171 lly, we show that blocking the activation of adenosine A1 receptors prevents the long-term depression
172        This study revealed a 3-fold RA-to-LA adenosine A1 receptor protein expression gradient in the
173                                              Adenosine A1 receptor protein expression was significant
174  had no effect, but those for adrenergic and adenosine A1 receptors reduced firing.
175 er, our data indicate that activation of the adenosine A1 receptor reduces synaptic strength by modul
176             Upon solubilization, the retinal adenosine A1 receptor retained binding characteristics s
177 at caffeine, because of its ability to block adenosine A1 receptors, shares neurochemical properties
178 mals received intravitreal injections of the adenosine A1 receptor stimulant adenosine amine congener
179               The selectivity level over the adenosine A1 receptor subtype for some of the more activ
180 to be mediated by a pathway initiated at the adenosine A1 receptor that transduced signals through a
181 uction in IOP results from the activation of adenosine A1 receptors, the mechanisms responsible for t
182 sites flanking the major coding exon for the adenosine A1 receptor, this AAV-Cre markedly reduced A1
183                               In chronic HF, adenosine A1 receptor upregulation in SAN pacemaker and
184 tivation of a postsynaptic K+ conductance by adenosine A1 receptors was used to determine the rate of
185  for GABA(B) receptors, 5HT1A receptors, and adenosine A1 receptors were essentially absent in mutant
186                   In addition, we found that adenosine A1 receptors were required for astrocyte calci
187               As an example we show that the adenosine A1 receptor, when placed under the influence o
188                            Activation of the adenosine A1 receptor with CCPA or the A3 receptor with

 
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