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1 and was augmented after administration of an adenosine antagonist.
2 y provide leads for the development of novel adenosine antagonists.
3 s for the development of novel, selective A3 adenosine antagonists.
4 f the potent and highly selective racemic A1-adenosine antagonist 1,3-dipropyl-8-[2-(5,6-epoxynorborn
5   After reinstatement, application of the A1 adenosine antagonist 1,3-dipropyl-8-cyclopentylxanthine
6 IBMX; phosphodiesterase type 5 inhibitor and adenosine antagonist, 10 micromol/L) and phorbol myrista
7 aken together, our findings suggest that the adenosine antagonist 125l-3-(4-azidophenethyl)-1-propyl-
8                      Coadministration of the adenosine antagonist 3,7-dimethyl-1-propargylxanthine (D
9    Group 1 received a 10-min infusion of the adenosine antagonist 8-sulfophenyltheophylline (0.9 mg/k
10                                          The adenosine antagonist 9-chloro-2-(2-furanyl)[1,2,4]triazo
11                                          The adenosine antagonist 9-chloro-2-(2-furanyl)[1,2,4]triazo
12  as increase in affinity for the nonxanthine adenosine antagonist 9-chloro-2-(furyl)[1,2,4]triazolo[1
13 acophore was introduced with overlap into an adenosine antagonist arylindenopyrimidine core.
14  strains also differed in the ability of the adenosine antagonist caffeine (50 mg/kg, i.p.) to prolon
15                 Conversely, the nonselective adenosine antagonist caffeine and the A(2A)R antagonist
16 ade of A1 adenosine receptor action with the adenosine antagonist caffeine can prevent hypoxia-induce
17                           Treatment with the adenosine antagonist CGS15943 reversed PEO1 cell-mediate
18 havioral synergy observed following combined adenosine antagonist-D1 dopamine agonist and combined D1
19 onist ZM 241385 (10 nmol/L) but not the A(1) adenosine antagonist DPCPX (5 micromol/L) abolished the
20 gents, including vasopressin antagonists and adenosine antagonists, hold promise for the future, and
21             The effects of administration of adenosine antagonists, such as the methylxanthines antag
22 ctural features of xanthine and non-xanthine adenosine antagonists to BTH4 (7) suggests a high degree
23 llyl-8-cyclohexylxanthine (DAX) are xanthine adenosine antagonists which activate chloride efflux fro
24                          The selective A(2A)-adenosine antagonist ZM 241385 (10 nmol/L) but not the A