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1 nosine receptors instead of administering an adenosine receptor agonist.
2 lymeric (PEG-b-PLA) nanoparticles for use as adenosine receptor agonists.
3 eukin-1 (IL-1) in the presence or absence of adenosine receptor agonists.
5 mobilization stimulated by the nonselective adenosine receptor agonist 5'-(N-ethylcarboxamido)adenos
8 ily A(2B) receptors because the nonselective adenosine receptor agonist 5'-N-ethylcarboxamidoadenosin
9 es supplemented with adenosine, but not with adenosine receptor agonist 5'-N-ethylcarboxamidoadenosin
10 lls were stimulated by focal ejection of the adenosine receptor agonist 5'-N-ethylcarboxamidoadenosin
16 t evidence that a small molecule, such as an adenosine receptor agonist, accelerates wound healing in
21 ignificantly altered by dialysis delivery of adenosine receptor agonists and antagonists to the prefr
24 n unexpected synergistic interaction between adenosine receptor agonists and phosphodiesterase (PDE)
25 diators of preconditioning, e.g., adenosine, adenosine receptor agonists, and adenosine triphosphate-
30 ns, VEGF expression is slightly increased by adenosine receptor agonists but adenosine A(2) or A(1) r
32 red the effect of topical application of the adenosine receptor agonist CGS-21680 (2-p-[2-carboxyethy
33 epithelial (HK-2) cells with a selective A1 adenosine receptor agonist, chloro-N(6)-cyclopentyladeno
37 loyed to study the functional consequence of adenosine receptor agonists directly on neuronal membran
38 tive chronotropic responses to muscarinic or adenosine receptor agonists do not require activation of
39 hat infusion of either adenosine (ADO) or an adenosine receptor agonist during reperfusion after hypo
40 osine (EC50: 0.5 microM) and NECA (universal adenosine receptor agonist; EC50 0.1 microM) stimulated
41 h clinically relevant agents (eg, NO donors, adenosine receptor agonists, endotoxin derivatives, or o
43 R such as forskolin, beta2-adrenergic or A2B-adenosine receptor agonists failed to activate rescued d
46 demonstrate that in conscious rabbits the A3 adenosine receptor agonist IB-MECA confers a powerful pr
49 onamide (IB-MECA), a potent and selective A3 adenosine receptor agonist, in models of myocardial stun
50 sine receptors by adenosine and selective A1 adenosine receptor agonists mimics the protective effect
51 of benzyloxy and phenoxy derivatives of the adenosine receptor agonists N(6)-cyclopentyl adenosine (
52 was equivalent to that obtained with the A1-adenosine receptor agonist N6-cyclopentyladenosine (1.56
53 the presence or absence of the nonselective adenosine receptor agonist NECA (5'-N-ethylcarboxamidoad
54 The effect of inosine was mimicked by the adenosine receptor agonist NECA and the A2B receptor ago
58 sed to assess the impact of adenosine and/or adenosine receptor agonists on proliferation, apoptosis,
59 GABA in the inhibitory actions of A1 and A2a adenosine receptor agonists on rat cerebral cortical neu
61 secondary to hypoxia was reversed in part by adenosine receptor agonists or reconstitution with solub
62 eta-EP was elevated following treatment with adenosine receptor agonist phenyl-isopropyl adenosine (P
63 (2-phenylisopropyl) adenosine (PIA), an A(1)-adenosine receptor agonist; preconditioned hearts pretre
66 orted that prior treatment with selective A1 adenosine receptor agonists results in a rapid uncouplin
67 f ischemia of the heart with adenosine or A1 adenosine receptor agonists results in uncoupling of A1
70 we tested the P-gp interaction of some A(3) adenosine receptor agonists that are being developed for
71 Thus, small, highly permeable drugs (such as adenosine receptor agonists) that transactivate TrkB rec
72 rated that administration of a selective A2A adenosine receptor agonist to CD73-deficient mice result