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1 lfactory learning and memory by acting as an adenosine receptor antagonist.
2 p was blocked by theophylline, a nonspecific adenosine receptor antagonist.
3 ither saline or theophylline, a non-specific adenosine receptor antagonist.
4 by the PKA antagonist (R(p))-cAMPS and by an adenosine receptor antagonist.
5 eritoneal injection of 10 mg/kg caffeine, an adenosine receptor antagonist.
6 enyl theophylline (10(-5) M), a nonselective adenosine receptor antagonist.
7 sing ZM 241385, a potent and selective A(2A) adenosine receptor antagonist.
8 h17 responses, we investigated the effect of adenosine receptor antagonists.
9 two subsets of fluorinated nonxanthine A(2B) adenosine receptor antagonists.
10 d, dopamine-deficient mice were treated with adenosine receptor antagonists.
11  were critical for the high affinity of A(3) adenosine receptor antagonists.
12 s constitute a novel class of selective A(3) adenosine receptor antagonists.
13  have been shown to be highly selective A(3) adenosine receptor antagonists.
14  glutamate (mGlu), GABAA, GABAB, glycine and adenosine receptor antagonists.
15 that treatment of obese Zucker rats with the adenosine receptor antagonist 1,3-dipropyl-8-(p-acrylic)
16 r administration of a nonselective A1 and A2 adenosine receptor antagonist 1,3-dipropyl-8-p-sulfophen
17                             Two selective A1 adenosine receptor antagonists (10(-7) or 10(-5) M) had
18 tory activity is reversed by an A2 selective adenosine receptor antagonist 3,7-dimethyl-1-propargyl x
19  phorbol but was completely abolished by the adenosine receptor antagonist 8-(p-sulfophenyl)-theophyl
20 ating effects were entirely prevented by the adenosine receptor antagonist 8-(p-sulfophenyl)-theophyl
21 A were sensitive to the antagonism by the A1 adenosine receptor antagonist 8-cyclopentyl-1,3-dipropyl
22 ith vehicle (control) or the irreversible A1 adenosine receptor antagonist 8-cyclopentyl-3-[3-[[4-(fl
23 , EHNA, and PD81,723 were reversed by the A1 adenosine receptor antagonist 8-cyclopentyltheophylline
24 on of adenosine either with the nonselective adenosine receptor antagonist 8-p-sulfophenyl theophylli
25 locked by pretreatment with the nonselective adenosine receptor antagonist 8-p-sulfophenyl theophylli
26 phonic acid (PPADS; 100 microM), but not the adenosine receptor antagonist 8-phenyltheophylline (8-PT
27  for 5 min) before and after addition of the adenosine receptor antagonist 8-phenyltheophylline (8-PT
28          These responses were blocked by the adenosine receptor antagonist 8-phenyltheophylline (8-PT
29        In the other (n = 5), the nonspecific adenosine receptor antagonist 8-phenyltheophylline (8PT)
30                            The non-selective adenosine receptor antagonist 8-SPT had no further effec
31             Subsequent administration of the adenosine receptor antagonist 8-sulphophenyl theophyllin
32                             By contrast, the adenosine receptor antagonist 8-sulphophenyltheophylline
33                   Topical application of the adenosine receptor antagonist 8-sulphophenyltheophylline
34 ever, in CHU rats, neither the non-selective adenosine receptor antagonist 8-sulphophenyltheopylline
35 boxamido)adenosine (NECA) and blocked by the adenosine receptor antagonist, 8-(p-sulfophenyl) theophy
36 by pretreatment not only with a non-specific adenosine receptor antagonist, 8-phenyltheophylline (8-P
37                                  Because the adenosine receptor antagonist 8PT attenuated this hypere
38 e-dimensional features responsible for A(2B) adenosine receptor antagonist activity.
39 (control) and combined L-NMMA-aminophylline (adenosine receptor antagonist) administration.
40 ngested psychoactive drug and a nonselective adenosine receptor antagonist, alters CB function and ve
41 ttent intratumor injection of a nonselective adenosine receptor antagonist, aminophylline (AMO; theop
42 ed mice; this effect is reversed with an A2A adenosine receptor antagonist and abolished in A2A recep
43 he effects could be antagonized by a charged adenosine receptor antagonist and by adenosine deaminase
44  reduced electrophysiological response to an adenosine receptor antagonist and increased adenosine re
45                            Experiments using adenosine receptor antagonists and cells from A(2B)AR(-/
46             Because caffeine (a nonselective adenosine receptor antagonist) and selective adenosine A
47                 It has been reported that A1 adenosine receptor antagonists are highly effective for
48                                         A(3) adenosine receptor antagonists are sought for their pote
49 kg, i.p.), a centrally active, non-selective adenosine receptor antagonist, attenuated the effects of
50 hophenyltheophylline (8-SPT; a non-selective adenosine receptor antagonist) before the fourth contrac
51                   Pretest treatment with the adenosine receptor antagonist caffeine (7 mg/kg ip) reve
52 tive interaction between chronic exposure to adenosine receptor antagonist caffeine and genetic influ
53             Intranasal administration of the adenosine receptor antagonist caffeine substantially enh
54            Locomotor activity induced by the adenosine receptor antagonist caffeine was significantly
55 k and NBTI were reversed by the nonselective adenosine receptor antagonist caffeine, and the highly s
56     Acute administration of the nonselective adenosine receptor antagonist, caffeine (5-25 mgkg i.p.)
57 linical notion that caffeine, a nonselective adenosine receptor antagonist, can trigger or exacerbate
58 e (VUF8501, VUF8503, VUF8505), the classical adenosine receptor antagonist CGS15943 and the A(1) rece
59           This effect was prevented by a pan-adenosine receptor antagonist CGS15943, but not by A1 or
60                       A mixture of the three adenosine receptor antagonists completely suppressed inh
61              The consumption of caffeine (an adenosine receptor antagonist) correlates inversely with
62 Caffeine (5, 10, or 15 mg/kg), a nonspecific adenosine receptor antagonist, dose-dependently and at h
63     The inhibition was abolished by the A(1) adenosine receptor antagonist DPCPX (8-cyclopentyl-1,3-d
64                      In addition, because A1 adenosine receptor antagonist failed to block renal IPC,
65                              In contrast, an adenosine receptor antagonist failed to prevent protecti
66                                          The adenosine receptor antagonist had a modest vasoconstrict
67                  Finally, the application of adenosine receptor antagonists has been implicated in pr
68  appears to be an effective and selective A1 adenosine receptor antagonist in humans.
69 ith the synthesis of an A2-subtype selective adenosine receptor antagonist in only two steps.
70                     Treatment with selective adenosine receptor antagonists indicated a contribution
71 r adenosine, and by aminophylline, a general adenosine receptor antagonist, indicating that they are
72 in models, oral intake of caffeine, a potent adenosine receptor antagonist, interferes with acupunctu
73 n attractive scaffold for the preparation of adenosine receptor antagonists, is the low water solubil
74 IA) and was reduced following treatment with adenosine receptor antagonist isobutylmethylxanthine (IB
75                                           A1 adenosine receptor antagonists may be useful for the pre
76 the food-, beverage-, and medication-derived adenosine receptor antagonists may modify an individual'
77                                              Adenosine receptor antagonists MRS1706 and DPCPX with kn
78 latory effect on migration was blocked by an adenosine receptor antagonist, MRS1754, ARL67156, an ect
79 or antagonist, or a selective A(2A) or A(2B) adenosine-receptor antagonist, negated the resistance to
80 ts to investigate the effect of selective A1 adenosine receptor antagonists on ischemia-reperfusion i
81 on was to examine the effect of caffeine (an adenosine receptor antagonist) on whole-body insulin-med
82                                 Studies with adenosine receptor antagonists or the adenosine uptake b
83 eophylline (SPT [50 mumol/L], a nonselective adenosine receptor antagonist), or calphostin C (100 nmo
84 tment with a CD73 antagonist, a nonselective adenosine-receptor antagonist, or a selective A(2A) or A
85               Prospective (anti-IgE therapy, adenosine receptor antagonists, phosphodiesterase inhibi
86 l/L 8-(p-sulfophenyl) theophylline (SPT), an adenosine receptor antagonist; preconditioned hearts als
87 ata and a general pharmacophore model for A3 adenosine receptor antagonists recently proposed, we app
88       We also show that treatment with an A1-adenosine receptor antagonist reduces the severity of lu
89 trolled Randomized Study of the Selective A1 Adenosine Receptor Antagonist Rolofylline for Patients H
90 trolled Randomized Study of the Selective A1 Adenosine Receptor Antagonist Rolofylline for Patients H
91 Controlled Randomized Study of the Selective Adenosine Receptor Antagonist Rolofylline for Patients H
92 after prolonged ischemia and reperfusion, A1 adenosine receptor antagonists should provide a protecti
93                                           A1 adenosine receptor antagonists significantly reduce isch
94  27.1 +/- 4.8% following the A(2A) selective adenosine receptor antagonist, similar to that observed
95 behaviors could be blocked by cannabinoid or adenosine receptor antagonists, suggesting a mixed mecha
96 ot affected by administration of the general adenosine receptor antagonist theophylline and the A1 an
97 zyme therapy and also through the use of the adenosine receptor antagonist theophylline, implicating
98 4 mg/kg, per os) that are reversible with an adenosine receptor antagonist (theophylline).
99                The effects of 2 nonselective adenosine receptor antagonists, theophylline and caffein
100 transport small therapeutic amounts of an A1 adenosine receptor antagonist to the respiratory centers
101 restingly, the drug aminophylline, a general adenosine receptor antagonist used to block retinal wave
102                  Three novel families of A2B adenosine receptor antagonists were identified in the co
103  while P2Y-selective receptor antagonists or adenosine receptor antagonists were ineffective.
104 enyl)-theophylline) (8-SPT), a non-selective adenosine receptor antagonist which has been found to be
105 y reduced by 8-phenyltheophylline (8-PT), an adenosine receptor antagonist which is non-selective bet
106 e reversed completely by two different A(2A) adenosine receptor antagonists without affecting T cells
107                    However, the nonselective adenosine receptor antagonist xanthine amine congener co
108                             The selective A1 adenosine receptor antagonists xanthine amine congener (
109                            The selective A2A adenosine receptor antagonist ZM241385 (3 mg kg(-1), I.V

 
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