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1 r osseous replacement occurs more often than adipocytic.
2  of PPARgamma at Ser-112 is required for its adipocytic activity, whereas phosphorylation of RUNX2 at
3 actors, resulting in activation of PPARgamma adipocytic and suppression of RUNX2 osteoblastic activit
4 in the osseous case but were absent from the adipocytic cases.
5 ular lipid metabolism, we selected the SW872 adipocytic cell line as a model.
6  pool and allocation to the osteoblastic and adipocytic cell lineages.
7  ie, sharing a similar expression profile to adipocytic cells at a corresponding stage of differentia
8                             These studies in adipocytic cells strongly support a novel role for CETP
9 T4 protein and gives rise to a population of adipocytic cells that take up glucose in direct response
10 tribute to normal cholesterol homeostasis in adipocytic cells.
11 nduced to differentiate exclusively into the adipocytic, chondrocytic, or osteocytic lineages.
12 hanced contractility despite no reduction in adipocytic commitment or increased expression of TGF-bet
13 ted a redirected lineage choice that favored adipocytic commitment over fibroblast or myofibroblast d
14 scular adipocytes exhibit a reduced state of adipocytic differentiation as compared with adipocytes d
15                                       Normal adipocytic differentiation is associated with an early a
16 rget-gene transcription that normally drives adipocytic differentiation of 3T3-L1 cells, lipid accumu
17 n contribute to inhibition of the process of adipocytic differentiation of 3T3-L1 cells.
18 L1 alleviates the confluency requirement for adipocytic differentiation of precursor cells.
19 ced osteoblast differentiation and repressed adipocytic differentiation to a similar extent.
20  Together, these data indicate that terminal adipocytic differentiation was induced in these malignan
21 and metabolic program that combines impaired adipocytic differentiation with augmented cytokine expre
22 hese MSCs showed that they were committed to adipocytic differentiation, but unable to terminally dif
23 phenotypic changes that were consistent with adipocytic differentiation, suggesting that the effects
24 ell as functionally in terms of its specific adipocytic differentiation-based response to ET-743.
25 roup of malignant mesenchymal tumors showing adipocytic differentiation.
26 activation in response to heregulin leads to adipocytic differentiation.
27 ich correlated with CEBPalpha expression and adipocytic differentiation.
28    Liposarcomas are rare malignant tumors of adipocytic differentiation.
29 n of a gene that is normally associated with adipocytic differentiation.
30 dlk expression and increased insulin-induced adipocytic differentiation.
31 on but had no effect on or slightly enhanced adipocytic differentiation.
32 EK1 or overexpressed MAPK displayed impaired adipocytic differentiation.
33 cells (Q-HSCs), which exhibit epithelial and adipocytic features, into myofibroblastic-HSC (MF-HSCs)
34 4% and approximately 100%, respectively, for adipocytic genes, in accordance with an experimental des
35  fusion protein TLS:CHOP as well as by mixed adipocytic histopathology.
36 ersely, bone-resident cells committed to the adipocytic lineage inhibit hematopoiesis and bone healin
37  the molecular identity of the bone-resident adipocytic lineage, and they establish its involvement i
38  commitment of mesenchymal stem cells to the adipocytic lineage.
39 can differentiate along the chondrocytic and adipocytic lineages in vivo, these cells were inoculated
40 erentiating to osteocytic, chondrocytic, and adipocytic lineages when stimulated under appropriate co
41  neighborhood for early myelopoiesis, and an adipocytic localization for early hematopoietic stem and
42 s and the increase in mRNA expression of the adipocytic markers peroxisome proliferator-activated rec
43 sarcomas are compared to their corresponding adipocytic maturing cells, we identified a group of gene
44  the tyrosine protein kinase activity of rat adipocytic membrane fragments in the absence of insulin;
45 multiple cell lineages, such as melanocytic, adipocytic, osteocytic, and chondrocytic lineages, which
46 n members of the osteogenic, myoblastic, and adipocytic pathways, 176 new genes in addition to 28 ori
47 own under osteogenic conditions developed an adipocytic phenotype after 3 days of complete inhibition
48  suggesting that GJC inhibition may favor an adipocytic phenotype.
49 icient for leptin to repress the epithelioid/adipocytic program.
50 r results reveal the developmental origin of adipocytic properties and the pathophysiological contrib
51 on is required for transition of epithelioid/adipocytic Q-HSCs into MF-HSCs.
52 ase of the myocardium characterized by fibro-adipocytic replacement of myocytes, predominantly in the
53 ements under stress conditions, including an adipocytic skewing of perivascular cells.
54 ancer at the Klb locus was also bound by the adipocytic transcription factor peroxisome proliferator-
55     Lipid droplets, a morphologic feature of adipocytic tumors, are strongly regulated by associated