ライフサイエンスコーパス: adipogenic

1 and 2 of the 7 predicted negatives were also adipogenic.

2 (VmSCs) that are either immunomodulators or adipogenic.

3 Thus, the adipogenic action of exogenous UAG in tibial marrow is d

4 ease TTF-1 expression contributes to its pro-adipogenic action.

5 Resveratrol displayed significant anti-adipogenic activities as exhibited by the ability to ant

6 components and identification of agents with adipogenic activities.

7 PPARgamma agonists while also displaying pro-adipogenic activities.

8 The adipogenic activity in house dust occurred at concentrat

9 This study sought to evaluate the adipogenic activity of indoor house dust extracts and a

10 to test the hypothesis that the pro- or anti-adipogenic activity of phytoestrogen chemicals is relate

11 vels (<20 mug of dust/well), and significant adipogenic activity was also exhibited by 28 of the SVOC

12 ng lipid and glucose metabolism with reduced adipogenic activity, that may be used as a model for a s

13 oxPi, and asked how well the ToxPi predicted adipogenic activity.

14 progenitor cell line that lacks osteogenic, adipogenic and angiogenic potential but is capable of di

15 tial for Wnt secretion, alleviates both anti-adipogenic and anti-lipogenic effects of Hh in cell cult

16 The pool of adipogenic and antimicrobial dFBs declined after birth,

17 bition of the TGF-beta receptor restored the adipogenic and antimicrobial function of dFBs in culture

18 embryonic and adult dFBs and inhibited their adipogenic and antimicrobial function, was identified as

19 study, for the first time, reveals the brown adipogenic and browning effects of apelin and suggests a

20 aired osteogenic differentiation, but normal adipogenic and chondrogenic differentiation.

21 und that developmental PDGFRalpha+ cells are adipogenic and differentiated into mature adipocytes, an

22 We previously showed that GO adipogenic and fibrotic phenotypes could be modelled in

23 e embryonic stem cells survival, is robustly adipogenic and induces postnatal adipose tissue formatio

24 Expression levels of adipogenic and lipogenic genes in adipose tissues were a

25 ere capable of colony-forming efficiency and adipogenic and osteo/dentinogenic differentiation under

26 nalogous beta-catenin-independent defects in adipogenic and osteogenic differentiation, and knockdown

27 DDT enhanced both adipogenic and osteogenic differentiation, which was con

28 cter of transduced hMSCs was investigated by adipogenic and osteogenic differentiation.

29 re we specify the cellular identities of the adipogenic and osteogenic lineages of the bone.

30 contributing exclusive differentiation along adipogenic and osteogenic lineages, respectively.

31 levels, and FM 550 exposure with changes in adipogenic and osteogenic pathways.

32 a more general switch-like mechanism between adipogenic and profibrotic molecular states.

33 from human neck fat and characterized their adipogenic and thermogenic differentiation.

34 We analyzed the expression of adipogenic and thermogenic genes and proteins in the iWA

35 ation by increasing the expressions of brown adipogenic and thermogenic transcriptional factors via t

36 ls are more osteogenic, softer gels are more adipogenic, and cell spreading areas increase with the s

37 including desmoplakin, predominantly in the adipogenic but not fibrogenic subsets.

38 0 knockout ADSCs have dramatically decreased adipogenic capabilities associated with downregulation o

39 Excessive RA-mediated activity impedes the adipogenic capability of ASCs at early but not late stag

40 However, hiPSC-BAPs display a low adipogenic capacity compared to adult-BAPs when maintain

41 besity demonstrates long-term effects on the adipogenic capacity of progenitor cells in offspring adi

42 enchymal stem cells (mBMSCs) to evaluate the adipogenic capacity of ToxCast chemicals.

43 lture-associated decreases in osteogenic and adipogenic capacity were partially protected by LIV in b

44 offspring BAT demonstrated attenuated brown adipogenic capacity.

45 , which renders progenitor cells with a high adipogenic capacity.

46 This study compares several adipogenic cell culture systems under a variety of condi

47 t the importance of balancing stromal versus adipogenic cell expansion during white adipose tissue de

48 ate that Wnt/beta-catenin signaling controls adipogenic cell fate within the lower dermis, which pote

49 CD54+) committed preadipocytes and a related adipogenic cell population marked by Clec11a and F3/CD14

50 Using adipogenic cells from the brown adipose tissues of LSD2-

51 Of these 9 adipogenic chemicals, 3 activated PPARgamma, and 1 activ

52 tocol significantly impacts the detection of adipogenic chemicals, and therefore, influences reproduc

53 ficant impact on potencies and efficacies of adipogenic chemicals.

54 by treating confluent preadipocytes with the adipogenic cocktail, which activates transcription facto

55 adult-BAPs when maintained in a traditional adipogenic cocktail.

56 l feedback regulator of both white and beige adipogenic commitment and differentiation, and resistanc

57 rotein, the latter regulating precursor cell adipogenic commitment and PPARgamma induction by BMP4.

58 a maintains DPP4+ cell identity and inhibits adipogenic commitment of DPP4+ and CD142+ cells.

59 ted state, whereas cytosolic WISP2 regulates adipogenic commitment.

60 ased on lineage tracing that mural cells are adipogenic, contrasting with the conclusions of a recent

61 ression of PPARgamma2, is able to rescue the adipogenic defect caused by DPP8/9 inhibition in preadip

62 dipogenic genes, our method recapitulates an adipogenic developmental pathway through successive meso

63 latory regions to repress expression of anti-adipogenic developmental regulators.

64 we show that neonatal skin was enriched with adipogenic dFBs and immature dermal fat that highly expr

65 suggest a novel role for FAP-mediated fibro-adipogenic diaphragm remodeling in obesity-associated re

66 Last, ORO in adipogenic differentiating cells was positively correlat

67 rkers and have chondrogenic, osteogenic, and adipogenic differentiation ability.

68 preadipocytes significantly increases their adipogenic differentiation ability.

69 f tribbles homolog 3 (Trib3) that suppressed adipogenic differentiation and inflammatory responses by

70 repancy, we investigated the role of EZH2 in adipogenic differentiation and lipid metabolism using pr

71 iR-130 during DIO may contribute to impaired adipogenic differentiation and obesity-related metabolic

72 ocyte progenitors in the fat of offspring to adipogenic differentiation and subsequent depletion, whi

73 Genetic ablation of HDAC9 improves adipogenic differentiation and systemic metabolic state

74 ngs suggest that APCDD1 positively regulates adipogenic differentiation and that its down-regulation

75 e of the PPARgamma2 locus after the onset of adipogenic differentiation and the mechanisms by which i

76 differentiation of hASCs while it suppresses adipogenic differentiation as evidenced by upregulation

77 l stromal cells indicated that Dab2 promoted adipogenic differentiation by modulation of MAPK (Erk1/2

78 MSCs, with enhanced osteogenic and decreased adipogenic differentiation capacities, as compared with

79 aracter demonstrated by their osteogenic and adipogenic differentiation capacity and their proliferat

80 Impaired adipogenic differentiation during diet-induced obesity (

81 gesting that miR-130 contributes to impaired adipogenic differentiation during DIO by repressing APCD

82 ery because its DR makes cells more prone to adipogenic differentiation even in the absence of the ad

83 potential of Sca1(+) fibroblasts to undergo adipogenic differentiation ex vivo.

84 on of 55 and 49 small GTPases accompanied by adipogenic differentiation in 3T3-L1 and C3H10T1/2 cells

85 regulator of preadipocyte proliferation and adipogenic differentiation in ASAT.

86 Bcl2l13 expression also increased during adipogenic differentiation in mouse ear mesenchymal stem

87 ithin this lineage significantly reduced its adipogenic differentiation in the context of exogenous,

88 ce of Wnt/beta-catenin signaling controlling adipogenic differentiation in the developing reticular d

89 of osteoblast differentiation, T63 inhibited adipogenic differentiation in the pluripotent mesenchyma

90 ion and oncogenesis, was reported to promote adipogenic differentiation in vitro by catalyzing trimet

91 us and visceral adipose tissues and impaired adipogenic differentiation in vitro Mechanistically, we

92 and (iii) exhibit markedly increased ex vivo adipogenic differentiation into brown adipocytes.

93 HFD-induced impaired adipogenic differentiation is associated with elevated e

94 esults in decreased myogenic activity, while adipogenic differentiation is significantly increased.

95 plete growth, osteogenic differentiation, or adipogenic differentiation medium, with the longest cili

96 Conversely, activation of GPR30 enhanced adipogenic differentiation of 3T3-L1 preadipocytes.

97 Deletion of GPR30 reduced adipogenic differentiation of adipose tissue-derived str

98 ntribution of short-chain fatty acids to the adipogenic differentiation of adipose-derived stem cells

99 DEG-BA into mouse ears markedly enhanced the adipogenic differentiation of ADSCs, leading to dermal a

100 Atrial myocardium secretome induces the adipogenic differentiation of adult mesenchymal epicardi

101 es osteoblast differentiation and suppresses adipogenic differentiation of AMSCs.

102 and enzymatic hydrolysates (CPHs) to inhibit adipogenic differentiation of C3H10T1/2 murine mesenchym

103 ubstrate deformations, or the osteogenic and adipogenic differentiation of human adipose-derived stro

104 difications that occur during osteogenic and adipogenic differentiation of mouse bone marrow-derived

105 knockdown of HOTAIR inhibited or stimulated adipogenic differentiation of MSCs, respectively.

106 er-donor variability of their osteogenic and adipogenic differentiation potential, as well as their a

107 oach to simulate the experimentally observed adipogenic differentiation processes over 15 days in les

108 encing GREM1 and/or adding BMP4 during white adipogenic differentiation reactivated beige/brown marke

109 transcription factor Prep1 is a repressor of adipogenic differentiation since its down-regulation (DR

110 sate demonstrated a higher reduction in anti-adipogenic differentiation through quantitation by oil-r

111 Here we report a novel role for APCDD1 in adipogenic differentiation via repression of Wnt signali

112 capacity of these stem cells, osteogenic and adipogenic differentiation was achieved.

113 The BA-DEG-BA-induced adipogenic differentiation was mediated via peroxisome p

114 Adipogenic differentiation was not affected by the prese

115 es from these mice likewise exhibit impaired adipogenic differentiation, and this phenotype persists

116 ted LMNA p.R482W mutation is known to impair adipogenic differentiation, but the mechanisms involved

117 educed cellular cholesterol is important for adipogenic differentiation, evidenced by increased induc

118 e that a chronic high-fat diet (HFD) impairs adipogenic differentiation, leading to accumulation of i

119 n of SPP1 and LEP, markers of osteogenic and adipogenic differentiation, respectively.

120 enhanced, whereas an miR-130 mimic blunted, adipogenic differentiation, suggesting that miR-130 cont

121 (FTO-4) mice exhibit increased potential for adipogenic differentiation, while MEFs derived from FTO

122 D6, Ppargamma2 and Cebpalpha expression, and adipogenic differentiation, yet had no effect on C/EBPbe

123 tiated adipocytes with blunted expression of adipogenic differentiation-specific genes.

124 hibitor of Wnt signaling, a key regulator of adipogenic differentiation.

125 s-mediated overexpression of APCDD1 enhanced adipogenic differentiation.

126 g transcription factor that is essential for adipogenic differentiation.

127 , Lipe, Plin1, Pparg, and Cebpa genes during adipogenic differentiation.

128 ffspring, which was correlated with enhanced adipogenic differentiation.

129 l a previously unrecognized role of Rab32 in adipogenic differentiation.

130 nzyme required for PPARgamma2 activation and adipogenic differentiation.

131 (HDAC9), an endogenous negative regulator of adipogenic differentiation.

132 tin for transcriptional reprogramming during adipogenic differentiation.

133 ymal stem cells during biochemically-induced adipogenic differentiation.

134 is distinctively involved in brown and beige adipogenic differentiation.

135 Knockdown of SMAD4 promotes adipogenic differentiation.

136 al mitophagy receptor, as being critical for adipogenic differentiation.

137 Wnt proteins, was found to prevent the anti-adipogenic effect of 5-Aza-dC in 3T3-L1 preadipocytes an

138 further demonstrated that the mitogenic and adipogenic effect of ghrelin were mainly dependent on th

139 Finally, the adipogenic effect of ibm-infused UAG was completely abol

140 Surprisingly, the adipogenic effects of intra-bone marrow (ibm)-infused ac

141 ting DNA binding of transcription factors to adipogenic enhancers, in particular C/EBPbeta.

142 family, member 10B (Wnt10b)/beta-catenin and adipogenic ERK/MAPK signaling pathways.

143 ion sites on C/EBPbeta, enhances these early adipogenic events.

144 ptide secreted by atrial myocytes is a major adipogenic factor operating at a low concentration by bi

145 induce the expression of Wnt6, a known anti-adipogenic factor, in fat depots of the mouse.

146 on profiling identified keratinocyte-derived adipogenic factors that are induced by beta-catenin acti

147 eduction in the actin turnover shortly after adipogenic (few minutes) and chondrogenic (3-4 hours) co

148 We report that JMJD6 is required for adipogenic gene expression and differentiation in a mann

149 e lamin A p.R482W hot spot mutation prevents adipogenic gene expression by epigenetically deregulatin

150 pocytes showed not only greater induction of adipogenic gene expression during differentiation but al

151 s indicate dual roles for JMJD6 in promoting adipogenic gene expression program by post-transcription

152 osteogenic and decreased PPARgamma-dependent adipogenic gene expression.

153 To elucidate further components of the adipogenic gene regulatory network, we performed a large

154 proliferation and their expression of a key adipogenic gene, peroxisome proliferator-activated recep

155 en, whereas in immobile MPCs, regions around adipogenic genes are open.

156 transcriptional programs: the expression of adipogenic genes common to both brown fat (BAT) and whit

157 actin depletion affects induction of several adipogenic genes during transcriptional reprograming.

158 ired for stimulation of several GR-regulated adipogenic genes in 3T3-L1 preadipocytes by glucocortico

159 This impaired regulation of adipogenic genes is linked to reduced expression of the

160 on of nucleosomal H4K5 and H3 to re-activate adipogenic genes to reverting adipogenesis.

161 arg) and white (Fabp4, Pnpla2, AdipoQ, Fasn) adipogenic genes, and glucose metabolism genes (Glut4, I

162 earlier and increased expression of specific adipogenic genes, consequent to the increased response o

163 Without overexpression of exogenous adipogenic genes, our method recapitulates an adipogenic

164 activation of C/EBPbeta-primed enhancers of adipogenic genes.

165 trast, more lipid deposition was observed in adipogenic-induced smoker PDLSC.

166 edly bypassed by prolonged treatment with an adipogenic inducer, 3-isobutyl-1-methylxanthine (IBMX).

167 c differentiation even in the absence of the adipogenic inducers.

168 Even following adipogenic induction, both male and female MFR adipocyte

169 with overexpression of the MIR355 gene after adipogenic induction.

170 Cs readily differentiated into adipocytes on adipogenic induction.

171 ine 3T3-L1 preadipocytes were used to assess adipogenic induction.

172 nes were up-regulated in LSD2-KD cells under adipogenic induction.

173 ing frizzled-like sequences bound the potent adipogenic inhibitor, Wnt10b, in vitro.

174 Therefore, miR-155 deletion increases adipogenic, insulin sensitivity, and energy uncoupling m

175 These studies indicate an adipogenic lifestyle alone is sufficient for the develop

176 enitor identity and potency, promoting white adipogenic lineage commitment.

177 -fat diet feeding activates expansion of the adipogenic lineage, an effect that is significantly enha

178 ts can be differentiated down osteogenic and adipogenic lineages, giving rise to self-supporting mono

179 tro, including osteogenic, chondrogenic, and adipogenic lineages.

180 to differentiate into osteo/odontogenic and adipogenic lineages.

181 Progenitor recruitment and adipogenic lipolysis contribute to the anabolic actions

182 Here we show that FAPs cause the adipogenic loss of dysferlin deficient muscle.

183 revents FAP adipogenesis, protecting against adipogenic loss of dysferlinopathic muscle while exogeno

184 erved elevated fibrosis, adipocyte size, and adipogenic marker expression in SCAT and VAT from INSTI-

185 d by higher lipid accumulation and increased adipogenic marker expression.

186 Additionally, an elevated expression of the adipogenic marker genes PPARgamma and Cebpalpha with a c

187 up-regulate expression of cyclin D3 and two adipogenic markers (CCAAT/enhancer binding protein alpha

188 red lipid accumulation and expression of key adipogenic markers in differentiating progenitors expose

189 that pairs H3K4me3 with H3K9me3 to maintain adipogenic master regulatory genes (Cebpa and Pparg) exp

190 Treatment with adipogenic medium induced a significant increment of lip

191 RCC cells increased lipid storage induced by adipogenic medium.

192 s the ability of lamin A to repress the anti-adipogenic miR-335, providing a potential molecular mech

193 eregulating long-range enhancers of the anti-adipogenic MIR335 microRNA gene in human adipocyte proge

194 uencing, we revealed that bta-miR-23a was an adipogenic miRNA mediating bovine adipogenesis in skelet

195 Adipogenic muscle loss is a feature of limb girdle muscu

196 died in muscle diseases, the role of FAPs in adipogenic muscle loss is not well understood.

197 c variants of ternatin, a cytotoxic and anti-adipogenic natural product whose molecular mode of actio

198 The adipogenic nature of the Iberian pig defines many qualit

199 the mechanisms underlying the regulation of adipogenic or osteoblastogenic development focus on tran

200 entiated into various cells lineages such as adipogenic, osteogenic, and chondrogenic.

201 w stromal cells) to a metabolically stressed adipogenic pathway that induces synthesis of a hyalurona

202 he commitment of adipocyte progenitors to an adipogenic pathway.

203 negative regulatory factor, have effects on adipogenic pathways and cellular metabolism in resident

204 induced to differentiate down osteogenic or adipogenic pathways by controlling the content of foulin

205 the cooperative DNA binding behavior of the adipogenic peroxisome proliferator-activated receptor ga

206 genesis by decreasing the recruitment of the adipogenic peroxisome proliferator-activated receptor ga

207 rt of genes was selected to characterize the adipogenic phenotype in primary cell cultures from three

208 tion, a propensity for cell death, and a pro-adipogenic phenotype.

209 ased impact on insulin secretion and reduced adipogenic potential but with preservation of anti-infla

210 , IUGR programs WAT preadipocytes to greater adipogenic potential in males.

211 d hyperplastic adipose tissue, with enhanced adipogenic potential of the stromal vascular fraction an

212 ased osteogenic potential, possess decreased adipogenic potential, form dentin pulp-like complexes, a

213 genic support function (PC-M), while lacking adipogenic potential.

214 ased phagocytic capacity, and acquisition of adipogenic potential.

215 f smaller adipocytes, along with an improved adipogenic potential.

216 n-6/n-3 ratios on offspring adipogenesis and adipogenic potential.

217 exhibited reduced myogenic (Myf5 and -6) and adipogenic (Pparg, Cebpa, and Lep) gene expression, wher

218 tein-4 (BMP4) plays a key role in regulating adipogenic precursor cell commitment and differentiation

219 kers, suggesting the presence of multipotent adipogenic precursor cells.

220 in mesenchymal stem cells, fibroblasts, and adipogenic precursor cells.

221 in muscle diseases and muscle-resident fibro/adipogenic precursors (FAPs) are implicated in this proc

222 results suggest that the need for MCE in the adipogenic process is independent from the requirement f

223 te that polyamines are required early in the adipogenic process.

224 range of activation of genes involved in the adipogenic process.

225 data suggest a role for Foxc1 in inhibiting adipogenic processes in CAR progenitors.

226 mononuclear cell types, including two fibro-adipogenic progenitor (FAP) cell subtypes.

227 ponse in this injury model and reduces fibro/adipogenic progenitor cell proliferation while simultane

228 lso abrogate TGFbeta signaling, reduce fibro/adipogenic progenitor cells and reduce fibrogenesis of m

229 constellation of markers diagnostic of fibro/adipogenic progenitor cells and were often associated wi

230 e hair follicles, suggesting a new source of adipogenic progenitor cells.

231 c progenitor cells, embryonic stem cells and adipogenic progenitor cells.

232 Maternal obesity reduces adipogenic progenitor density in offspring adipose tissu

233 al pathway through successive mesodermal and adipogenic progenitor stages.

234 nt PDGFRa(+) SCA-1(+) cells as cardiac fibro/adipogenic progenitors (cFAPs) and show that they respon

235 cytes, we isolated satellite cells and fibro/adipogenic progenitors (FAPs) from muscle; satellite cel

236 Moreover, intradiaphragmatic fibro-adipogenic progenitors (FAPs) proliferate with long-term

237 Upon injury, muscle-resident fibro/adipogenic progenitors (FAPs) proliferated and gave rise

238 ulates a population of muscle-resident fibro/adipogenic progenitors (FAPs) that play a supportive rol

239 Unexpectedly, Tppp3(-)Pdgfra(+) fibro-adipogenic progenitors coexist in the tendon stem cell n

240 uring the differentiation of brown and beige adipogenic progenitors in mice.

241 results identify perivascular cells as fibro/adipogenic progenitors in WAT and show that PDGFRalpha t

242 els of pro-inflammatory markers, APOD+ fibro-adipogenic progenitors, TPPP3/PRG4+ chondrogenic cells,

243 s deletion does not impair the classic brown adipogenic program but rather induces premature activati

244 ates AKT signaling to drive and maintain the adipogenic program in the skin.

245 Accordingly, Nck2 deficiency promotes an adipogenic program that not only enhances adipocyte diff

246 Here we use the dynamic adipogenic program that occurs during hair growth to unc

247 vity is required for up-regulating the brown adipogenic program.

248 R1), fundamentals for the PPARgamma-mediated adipogenic program.

249 Although the altered adipogenic programming following MFR was detectable at P

250 l nutrition in facilitating the sex-specific adipogenic programming in the IUGR offspring.

251 e possible mechanism underlying sex-specific adipogenic programming in the IUGR offspring.

252 tional profiling, we determined sex-specific adipogenic programming of WAT progenitors isolated from

253 odel, bone marrow stem cells showed enhanced adipogenic programming; however, the effect of IUGR on w

254 ipocytes were programmed to exhibit stronger adipogenic propensity than females.

255 The inherent adipogenic properties of human adipose-derived stem cell

256 The adipogenic property of the atrial secretome was enhanced

257 reased adipose PHD levels and decreased anti-adipogenic protein levels by increasing their ubiquitina

258 inhibitor) prevented the degradation of anti-adipogenic proteins and retarded RIAD.

259 nd that this leads to the commitment of anti-adipogenic proteins to the ubiquitination-proteasomal pa

260 cellular matrix turnover and shedding of the adipogenic regulator DLK1, but that in adipose tissue in

261 paralleled by upregulated expression of the adipogenic regulator PPARG and its co-activator PPARGC1B

262 ramuscular adipogeneic commitment as an anti-adipogenic regulator which acts by targeting ZNF423.

263 ntrols the expression of most early and late adipogenic regulators, identifying ZEB1 as a central tra

264 gical inhibition of FAP adipogenesis arrests adipogenic replacement and degeneration of dysferlin-def

265 iber repair, chronic muscle inflammation and adipogenic replacement of dysferlinopathic muscle.

266 Muscle loss due to fibrotic or adipogenic replacement of myofibers is common in muscle

267 deficient mice reduced muscle inflammation, adipogenic replacement of myofibers, and improved muscle

268 rlin exhibit chronic muscle inflammation and adipogenic replacement of the myofibers.

269 2 does not result in chronic inflammation or adipogenic replacement of the myofibers.

270 TTF-1 interacts with PPFP to inhibit the pro-adipogenic response to pioglitazone, and that the abilit

271 ntiation, coinciding with enhanced export of adipogenic RNAs.

272 mediated via enhanced expression of the pro-adipogenic short isoform of RUNX1T1, which enhanced adip

273 Here, we show that adipogenic stimuli trigger nuclear translocation of S6K1

274 evelopmental caveats of using antibiotics in adipogenic studies.

275 a at enhancers controlling the expression of adipogenic target genes and continued differentiation.

276 In contrast, the master adipogenic TF peroxisome proliferator-activated receptor

277 lt human atrial epicardial cells were highly adipogenic through an epithelial-mesenchymal transition

278 olecules in modulating white and brown/beige adipogenic tissue development and activity.

279 f myofibres and their replacement with fibro-adipogenic tissue.

280 cells and revealed their specification from adipogenic to fibrogenic cells in the rat model of atria

281 PDGFRalpha, they underwent fate change from adipogenic to fibrotic lineage.

282 differentiation had no obvious influence on adipogenic traits, suggesting that LSD2 functions during

283 bits PPFP target gene expression and impairs adipogenic trans-differentiation.

284 ssion and protein S-glutathionylation of the adipogenic transcription factor CCAAT enhancer-binding p

285 ng activin A, blunted fat loss, and enhanced adipogenic transcription factor expression within 3 week

286 Gcn5/PCAF inhibits expression of the master adipogenic transcription factor gene PPARgamma, thereby

287 ced lipid accumulation and inhibited the key adipogenic transcription factor peroxisome proliferative

288 BDE-47 modestly activated the key adipogenic transcription factor peroxisome proliferator-

289 Thy1 decreases the activity of the adipogenic transcription factor PPARgamma by more than 6

290 CAAT/enhancer-binding protein alpha, a major adipogenic transcription factor, and therefore, they wer

291 Here we show that C/EBPbeta, a key pro-adipogenic transcription factor, is PARylated by PARP-1

292 netic loci resided within binding motifs for adipogenic transcription factors (e.g., PPARG and EGR1).

293 fasting insulin and increased expression of adipogenic transcription factors but lack glucose intole

294 esis by directly promoting the expression of adipogenic transcription factors CCAAT/enhancer-binding

295 F6 acts by exerting translational control of adipogenic transcription factors like C/EBPbeta, C/EBPde

296 ey directly promote the expression of master adipogenic transcription factors peroxisome proliferator

297 gamma (PPARgamma) and C/EBPalpha are master "adipogenic" transcription factors.

298 a AnxA3 negatively modulates the response to adipogenic treatment and may act as negative regulator o

299 PC-A cells were not osteogenic or adipogenic under standard differentiation conditions and

300 aracterized CrF as both pro-fibrotic and pro-adipogenic with a rich milieu of activated immune cells