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1 f neurodevelopmental and mental disorders in adulthood.
2 erns established in adolescence persist into adulthood.
3 brain expression of sex chromosome genes in adulthood.
4 erences among all disorders in childhood and adulthood.
5 tal exposures contribute to liver disease in adulthood.
6 18.9% of the participants had fatty liver in adulthood.
7 se to new neurons and astrocytes all through adulthood.
8 in children with FASD and persist into early adulthood.
9 ter system altered youths' trajectories into adulthood.
10 lamus (mPFC->pPVT) during social exposure in adulthood.
11 y distributed throughout childhood and early adulthood.
12 enerative disease with onset in mid- to late adulthood.
13 rimary congenital glaucoma (PCG) patients in adulthood.
14 vere retrograde blood flow that persist into adulthood.
15 oligodendrocytes, whereas others remain into adulthood.
16 commonly during the most productive years of adulthood.
17 ight or obesity during adolescence and young adulthood.
18 eating childhood hypertension on outcomes in adulthood.
19 nic conditions in offspring that manifest in adulthood.
20 to abnormal function in early childhood and adulthood.
21 induced obesity and fasting hyperglycemia in adulthood.
22 ive feeding and obesity in childhood through adulthood.
23 manage the ACE-induced abnormal behaviors in adulthood.
24 d alcohol withdrawal and following stress in adulthood.
25 nded to decline during adolescence and young adulthood.
26 f grandchildren during adolescence and young adulthood.
27 adolescence promotes resilience to stress in adulthood.
28 ith a moderately higher risk of mortality in adulthood.
29 resenting the clinical hallmark during early adulthood.
30 r and increased likelihood of fatty liver in adulthood.
31 n the mdx mouse has largely focused on early adulthood.
32 k effects on larval herbivory or survival to adulthood.
33 AMs in Greece shapes their trajectories into adulthood.
34 ung adulthood as well as opioid use in young adulthood.
35 ect lateral line progression and survival to adulthood.
36 nal/education, and mental health outcomes in adulthood.
37 iple cell types, during both development and adulthood.
38 to enhance specific perceptual abilities in adulthood.
39 ignificantly distinct diets in childhood and adulthood.
40 0.25/-0.25 (-12 to +2.75/-13.25 to +2.63) in adulthood.
41 rt- and long-term adverse consequences up to adulthood.
42 hout childhood and educational attainment in adulthood.
43 time points during embryonic development and adulthood.
44 microbial metabolic profiles persisted into adulthood.
45 try from postnatal day 2.5 (P2.5) throughout adulthood.
46 functional capacity to regulate behavior in adulthood.
47 cal spine density in knockouts persists into adulthood.
48 to 10 for approximately ten years into young adulthood.
49 uring mammalian embryonic development and in adulthood.
50 dolescence, the period between childhood and adulthood.
51 function can be overcome by interventions in adulthood.
52 terature on the transition from childhood to adulthood.
53 features emerge in late adolescence or early adulthood.
54 -like molecular biomarkers that persist into adulthood.
55 y altering tissues with impacts lasting into adulthood.
56 effect of prenatal famine exposure on WC in adulthood.
57 evelopment of risk factors of fatty liver in adulthood.
58 ons and some glial cells, which persist into adulthood.
59 ucibility of NMDAR-LTD is greatly reduced in adulthood.
60 in the neocortex throughout development and adulthood.
61 isease might emerge decades earlier in young adulthood.
62 sociated with poor mental health outcomes in adulthood.
63 meningitidis peaks in adolescence and young adulthood.
64 ring adolescence persist or recover in young adulthood.
65 ted by a lack of supportive relationships in adulthood.
66 idden layer' of diversity that persists into adulthood.
67 overweight or obese in adolescence and early adulthood.
68 ethality, and Thm2 loss allows survival into adulthood.
69 ncies associated with long-term effects into adulthood.
70 al structural plasticity and regeneration in adulthood.
71 ropic defects that manifest predominantly at adulthood.
72 edictive of reversal-learning performance in adulthood.
73 ildhood and 20/22/20/20 (0.05/0.0 logMAR) in adulthood.
74 emales' glucocorticoid concentrations across adulthood.
75 9 developmental stages from embryogenesis to adulthood.
76 ild with onset of symptoms in adolescence or adulthood.
77 interrupted mitosis, which persists through adulthood.
78 nt plasticity and recovery from amblyopia in adulthood.
79 f LBW in humans and reduced lung function in adulthood.
80 s, and ultimately clues to their function in adulthood.
81 ), the most common malignant brain tumour in adulthood.
82 subjects (40%) in childhood and 13 (43%) in adulthood.
83 starting as early as P1 and persisting into adulthood.
84 pathway(s) of larvae and grew the embryos to adulthood.
85 ested behaviorally at late adolescence/early adulthood.
86 excessive loss of local excitatory inputs in adulthood.
87 subjects (67%) in childhood and 22 (73%) in adulthood.
88 8 (27%) subjects in childhood vs 11 (37%) in adulthood.
89 entration through late adolescence and young-adulthood.
90 that a large fraction of neurons is added in adulthood.
91 principles of extinction learning studied in adulthood.
92 h numbers subsequently increasing throughout adulthood.
93 despair-, and schizophrenia-like behavior in adulthood.
94 ristics, and (vi) adverse experiences during adulthood.
95 ty of proteostasis-related pathways in early adulthood.
96 eece as UAMs but had since transitioned into adulthood.
97 and higher sedentary time from childhood to adulthood.
98 lux as the only activity-dependent source in adulthood.
99 ural stem cells (NCSs) and neurogenesis into adulthood.
100 s in distinct forms with onset in infancy or adulthood.
101 nerable to age-alcohol interactions in later adulthood.
102 ion of FASD in childhood as well as in young adulthood.
103 r during homeostasis in postnatal stages and adulthood.
104 behavior of despair in adolescence and young adulthood.
105 as well as clues to episodic functioning in adulthood.
106 th syndrome but normal cardiac function into adulthood.
107 gulation, from early developmental stages to adulthood.
108 including IL-1beta, IL-6, and TNF-alpha, in adulthood.
109 th global or segmental glomerulosclerosis in adulthood.
110 rse, resulting in extranuclear expression by adulthood.
111 eomic endotype for AD FA+ that persists into adulthood.
112 ypertension in childhood and hypertension in adulthood.
113 vestibular function were sustained well into adulthood.
114 embles second "natural" language learning in adulthood.
115 a neuroprotective effect that persists into adulthood.
116 th status, and phenotype information in late adulthood.
117 chanisms that change in adolescence and into adulthood.
118 t, and this expression is maintained through adulthood.
119 pocampal structure and memory performance in adulthood.
120 centrations, and examined their offspring in adulthood.
121 lterations to the hippocampus that last into adulthood.
122 the offspring's behavioural responses during adulthood.
123 V1), once wired up in development, change in adulthood?
124 dolescence [10-18 years of age] and emerging adulthood [19-30 years of age]) investigating emotion re
126 ession diminishes as individuals mature into adulthood, a process that requires conserved regulators
127 the enhanced anxiety-like behaviors in their adulthood, accompanied by increased GABAergic transmissi
130 n sharply increases between days 6 and 12 of adulthood, after the worms have reproduced, as individua
131 ive effects by reducing adolescent and young adulthood (age 25) behaviors that precede these deaths,
133 altreatment histories made once they reached adulthood, along with extensive psychiatric assessment.
134 pal place area and retrosplenial complex) by adulthood, already show domain-specific patterns of func
135 ed brain editing changes are not observed in adulthood, although MIA-induced behavioral deficits are
136 rations in early gut microbiota persist into adulthood and are associated with increased susceptibili
138 the mPFC increases from early adolescence to adulthood and correlates negatively with Dcc levels.
140 ression in all three striatal regions before adulthood and disappears in a region- and sex-specific t
141 (LP), which allows digestion of lactose into adulthood and enables the milk-based, high-protein, low-
142 development of overweight from childhood to adulthood and having overweight at both ages were associ
143 esulted in diminished CD8 T cell response in adulthood and impaired control of tumor growth and bacte
144 ne-cell gene regulation that emerge by young adulthood and may help explain social disparities in the
145 FR during the transition from adolescence to adulthood and overestimation of GFR in young adults.
146 fspring from 3 weeks of age, maintained into adulthood and present also within juvenile F2 offspring.
147 cal exposure causes metabolic dysfunction in adulthood and reprograms histone marks in the developing
148 uce distinct phenotypes in mice during young adulthood and suggest sex-based differences in the susce
149 e continued synthesis of collagen throughout adulthood and the conventional transcriptional/translati
150 a slower increase from adolescence to middle adulthood, and by a lack of substantial decline from mid
151 differentiation of brain regions until early adulthood, and compositional changes cause dedifferentia
152 tightly linked during adolescence and young adulthood, and reduced DMN connectivity may reflect loca
153 red early postnatally, is maintained through adulthood, and requires ongoing type I IFN signaling to
154 and influence neuronal functions throughout adulthood, and reveals a potent effect of colony stimula
155 s constantly changing, from birth throughout adulthood, and several variables affect its development
156 ional level in mid-pupae and maintained into adulthood, and this response is partially dependent on d
157 ome communities shortly after birth, through adulthood, and up to 1 y of life in a controlled manner,
158 ICE measures during early childhood and adulthood approximated racial/ethnic and economic dimens
159 e alpha-Gal syndrome is commonly acquired in adulthood as a consequence of tick bites and has a regio
160 the transition between adolescence and early adulthood as a critical period and the populations that
161 highlight the transition from adolescence to adulthood as a period of dynamic maturation of tissue ir
163 opils during development but die just before adulthood, as well as neuronal subtypes that partition d
164 fic disruption of UPF2, an NMD component, in adulthood attenuates learning, memory, spine density, sy
165 t (1st-2nd weeks) and slowly flattened up to adulthood; (b) Layer A1 exhibits increases in SMI-32-pos
166 easures of association between childhood and adulthood blood pressure; reduction of childhood blood p
167 eatures of developing cartilage persist into adulthood, both in peripheral chondrocytes and in cells
168 e birth and self-maintain locally throughout adulthood, but are regenerated by bone marrow (BM) under
169 nile social isolation reduces sociability in adulthood, but the underlying neural circuit mechanisms
170 ng obesity and dyslipidemia in childhood and adulthood, but this association has not been studied in
172 tuation in Drosophila, which closes early in adulthood can, like the critical period for ocular domin
174 and preserved lung function in mice reaching adulthood.Conclusions: Nanoparticle delivery of FOXM1 or
175 Screening for the MEPE LoF mutations before adulthood could potentially prevent osteoporosis and fra
176 genetic stimulation of mPFC->pPVT neurons in adulthood could rescue the sociability deficits caused b
178 (LF/LF) mice, Psen1(LF/LF) rats survive into adulthood despite loss of gamma-secretase activity.
179 expression of 14 genes; in contrast, during adulthood, despite a much larger sample size, only two g
180 tability, synaptic plasticity, and memory in adulthood, despite the lack of requirement for neuronal
183 ith a higher level of depressive symptoms in adulthood during all follow-up periods (beta = 0.07, P =
185 experience-mediated biological embedding in adulthood, even in the conventionally memory-less innate
186 We assessed whether early childhood and adulthood experiences of neighborhood privilege, measure
187 of childhood socioeconomic disadvantage with adulthood fatty liver and tested adulthood risk factors
190 ed trajectories across adolescence and early adulthood for 2 major dietary patterns and their associa
191 , those who used them for >=2 months in late adulthood had increased risks of all-cause mortality (ha
193 the dorsal striatum, protein restriction at adulthood has no impact on dopamine release but the same
195 growth retardation, excessive weight gain in adulthood, heterotaxia, pre-axial polydactyly, neural tu
196 ension can result in adverse outcomes during adulthood; identifying and treating primary and secondar
197 cardiovascular outcomes during transition to adulthood in a national longitudinal cohort study of bir
198 and self-rated health, obesity, and pain in adulthood in a retrospective cohort study of data from t
199 faster cortical thinning linearly throughout adulthood in a similar anatomical distribution, with dif
200 78 organ donor pancreata from birth through adulthood in control subjects and those at various stage
204 tic experiences from childhood through early adulthood in the general population and examined predict
205 to bHR/bLR phenotype across development and adulthood in the hippocampus, a region critical for emot
206 lsivity, and hyperactivity that persist into adulthood in the majority of the diagnosed children.
208 avily enriched in development but reduced in adulthood, including the transcriptional regulator Hmga1
209 bolic diseases, antibiotic use during middle adulthood, indication for use, demographic factors, and
210 cohort studies spanning childhood and young adulthood, integrated with prospective biospecimen colle
211 ing overweight in childhood, adolescence, or adulthood is associated with asthma in their offspring.
212 strategy for triggering robust plasticity in adulthood is to reproduce one of the hallmark physiologi
214 e fashion at any point during development or adulthood, is compatible with sequential barcoding, and
215 P), the continued expression of lactase into adulthood, is the most strongly selected single gene tra
216 neurons continue to sprout growth cones into adulthood, leading to a highly ramified nervous system.
219 predispose offspring to metabolic disease in adulthood, likely due to a combination of the effects of
220 trajectory with FEV(1) below normal in early adulthood (low maximally attained FEV(1) trajectory).Obj
222 usions: Long-term use of antibiotics in late adulthood may be a risk factor for all-cause and cardiov
224 assessed at 3 life epochs (childhood, young adulthood, midlife) via questionnaire (2001-2002) and su
226 t independent effects of early childhood and adulthood neighborhood privilege on preterm delivery and
228 e feature of the striatal regions is that in adulthood neurons exclusively express membrane but not n
229 om normal maximally attained FEV(1) in early adulthood (normal maximally attained FEV(1) trajectory)
231 hypertension and abnormal blood pressure in adulthood (odds ratios, 1.1-4.5; risk ratios, 1.45-3.60;
232 es of four species and decreased survival to adulthood of two species; it had no effect on adult mass
235 ct of stress exposure during adolescence and adulthood on the activity of putative pyramidal neurons
236 ein restriction either during adolescence or adulthood on the function of the mesolimbic (nucleus acc
238 mass index (weight (kg)height (m)2) >=30) in adulthood (OR = 1.32, 95% CI: 1.06, 1.64; P = 0.01).
239 US mortality related to CHD from infancy to adulthood over the past 19 years and determine differenc
240 SD17B3 via gene-delivery to Sertoli cells in adulthood partially rescues the adult phenotype, showing
241 colleagues present compelling evidence that adulthood physical activity is beneficial across the ris
243 s to novelty-induced exploration deficits in adulthood, potentially involving the dopamine system.
244 onal types during development and throughout adulthood, rather than ones restricted to the dopaminerg
249 antage with adulthood fatty liver and tested adulthood risk factors of fatty liver as possible mediat
252 women whose BMI normalized from childhood to adulthood: RR was 2.04 (95% CI: 0.93, 4.47) for gestatio
253 plasticity as well as functional recovery in adulthood.SIGNIFICANCE STATEMENT The decline of cortical
255 strength decreases from adolescence to early adulthood Somatosensory cortical responses in youth with
256 s of myocardial and vascular health in young adulthood specify known/novel pathways of metabolic dysf
258 pt myelin formation during development or in adulthood, such as multiple sclerosis and peripheral neu
259 reshold-spiking somatostatin interneurons in adulthood, suggesting a circuit mechanism underlying soc
260 ected, these enhancers are active throughout adulthood, suggesting that their formation in early life
261 abolic disease risk factors collected in mid-adulthood: systolic (SBP) and diastolic blood pressure (
264 ad a markedly higher mortality risk in early adulthood than that of other children, which requires pu
265 ntal conditions during a sensitive period in adulthood (the first day post-emergence), but the role o
266 icting the persistence of ADHD symptoms into adulthood, the genetic architecture underlying the traje
269 ve induction of small heat shock proteins in adulthood, thereby protecting against age-related proteo
271 egulate vulnerability to traumatic stress in adulthood through region-specific epigenetic activity an
272 ciation of cumulative BP exposure from young adulthood to midlife with gait and cognitive function in
273 lonizing the cortex and became stationary by adulthood to occupy the same vascular space for nearly 2
274 ts, and then reared larvae from egg hatch to adulthood under diurnally variable regimens including da
275 fe accelerate epigenomic aging, and later in adulthood unlock metabolically restricted epigenetic rep
278 enetic suppression of mPFC->pPVT activity in adulthood was sufficient to induce sociability deficits
279 3.6%; 95% CI 1.2-6.0; P = 0.027) and larger adulthood WC (d = 2.72 cm; 95% CI 0.20-5.24; P = 0.034).
283 eizure activity, seven weeks of treatment in adulthood were able to correct electrographic, neuroanat
284 women patterns of allergic sensitization in adulthood were not found to differ between women with cu
285 al days (PND) 25-27, EE from PND 35 to early adulthood, when behavioural testing and assessment of AH
286 demonstrate that PI5P4Kalpha is expressed in adulthood, whereas PI5P4Kbeta is expressed early in deve
287 he NF186 ectodomain aberrantly persists into adulthood, whereas the cytoplasmic domain/transport-depe
290 ytes before birth, but self-maintain through adulthood with contribution from peripheral monocytes.
291 to other brain regions and was associated in adulthood with decreased anxiety-like behavior, increase
294 Two tKO mice survive and grow normally to adulthood with smaller myofiber diameter, diminished phy
295 ficant decline of pathogenic bacteria before adulthood, with an increase of the commensal Staphylococ
296 rises an expanding continuum from infancy to adulthood, with early evidence of genotype-phenotype cor
297 ctory inflammatory syndrome develops in late adulthood, with fevers, cytopenias, characteristic vacuo
298 2 expression throughout development and into adulthood, with greater impact on the Schwann cell-speci
300 e to develop normally and survive into early adulthood without thrombin activity will provide importa