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1 promyelocytic leukemia (ie, not of the upper aerodigestive tract).
2 plasms that arise in the mucosa of the upper aerodigestive tract.
3  has developed a second primary tumor in the aerodigestive tract.
4 HN) is the most common neoplasm in the upper aerodigestive tract.
5  CYP1B1 in cell lines derived from the human aerodigestive tract.
6 he epithelium but also the mesenchyme of the aerodigestive tract.
7 of tobacco smoke-related malignancies of the aerodigestive tract.
8  neoplasm arising in the mucosa of the upper aerodigestive tract.
9 ma originally described to involve the upper aerodigestive tract.
10 ctivity in certain malignancies of the upper aerodigestive tract.
11 known to cause cancers of the lung and upper aerodigestive tract.
12 opharyngeal cancers and cancers of the upper aerodigestive tract.
13 over time throughout the mucosa of the upper aerodigestive tract.
14 ans developed a squamous cancer of the upper aerodigestive tract.
15 e presence of cancer cells shed in the upper aerodigestive tract.
16 ent of squamous cell carcinomas of the upper aerodigestive tract.
17 ons and a high risk to develop cancer of the aerodigestive tract.
18 diverse group of tumours affecting the upper aerodigestive tract.
19 terized by growth of papillomas in the upper aerodigestive tract.
20 l samples which reflect the continuum of the aerodigestive tract.
21 arly for carcinomas that derive not from the aerodigestive tract.
22 iated with a relative's cancers in the upper aerodigestive tract (2.14) and lung (1.74).
23      We eliminated tap water exposure to the aerodigestive tract among high-risk patients, and the in
24 he reflux of stomach contents into the upper aerodigestive tract and is often linked to allergic rhin
25 eart disease, COPD, and cancers of the upper aerodigestive tract and lung.
26 s between TNC and VNC in patients with upper aerodigestive tract cancer (UATC) and to evaluate the po
27 ed by ATRA via retinoid receptors in certain aerodigestive tract cancer cells, and its induction by A
28 onstrated activity in the chemoprevention of aerodigestive tract cancer.
29           Twenty-four patients with advanced aerodigestive tract cancers were enrolled; 79% had non-s
30 ssociated with decreased risk of a number of aerodigestive tract cancers, including esophageal cancer
31 has been shown to be a risk factor for upper aerodigestive tract cancers.
32 apillomatosis (RRP) is a rare disease of the aerodigestive tract caused by the Human Papilloma Virus
33 sed interventions focus on the prevention of aerodigestive tract colonization (avoidance of unnecessa
34  arises from the mucosal lining of the upper aerodigestive tract, demonstrates squamous differentiati
35 ng adenotonsillitis (3.29, 2.41-4.50); lower aerodigestive tract diseases, including asthma (2.69, 2.
36          Unique anesthetic complications and aerodigestive tract disorders have been identified.
37 his region that is expressed in normal upper aerodigestive tract epithelium.
38 netic basis for "field cancerization" of the aerodigestive tract has also remained obscure.
39  studies suggest that many SPTs in the upper aerodigestive tract have a common clonal origin, challen
40                                              Aerodigestive tract inflammatory injuries, such as decli
41 be managed conservatively; however, owing to aerodigestive tract injury and other associated injuries
42  the action of RA on the mucosa of the upper aerodigestive tract is mediated via downregulation of st
43                                    The upper aerodigestive tract is predisposed to the formation of m
44  oral cavity, an essential part of the upper aerodigestive tract, is believed to play an important ro
45 the breast, endometrium, lung, kidney, upper aerodigestive tract, liver, and esophagus but not for pr
46 management of regional metastases from upper aerodigestive tract malignancies has shifted from the tr
47 stantial declines in the prevalence of upper aerodigestive tract malignancies.
48                         Individuals with one aerodigestive tract malignancy have a high incidence of
49 clearly related to malignancies of the upper aerodigestive tract, may also increase risk of cancer at
50 clinical replacement of non-functional upper aerodigestive tract mucosa are regulated as medicinal pr
51    Field cancerization predisposes the upper aerodigestive tract mucosa to the formation of multiple
52                                    The upper aerodigestive tract mucosal lining is often disrupted du
53 nd neck cancer surgery for a malignant upper aerodigestive tract neoplasm from 2001 to 2011.
54 ter methylation in cells exfoliated from the aerodigestive tract of current and former smokers.
55 ty for gene promoter hypermethylation in the aerodigestive tract of smokers.
56 ms or presenting masses that may involve the aerodigestive tract or vascular structures require a con
57 UGT1A10, extrahepatic enzymes present in the aerodigestive tract, preferentially formed both BPD-7R-G
58 sly expressed with highest expression in the aerodigestive tract, prostate, adrenal gland, and saliva
59 val, 1.10 to 1.91), and cancers of the upper aerodigestive tract (relative risk, 2.02; 95 percent con
60 eous collection of malignancies of the upper aerodigestive tract, salivary glands and thyroid.
61 (Ecad-lo) and high vimentin within the upper aerodigestive tract SCCs.
62  arises in the lining or mucosa of the upper aerodigestive tract, specifically the oral cavity, oroph
63 romosome arm 3p is a frequent event in upper aerodigestive tract squamous cell carcinoma (UADT SCC).
64 e associations were observed for lung, upper aerodigestive tract, stomach, and prostate cancer mortal
65 st peak observed, whereas in microsomes from aerodigestive tract tissues, peak 2 (both BPD-7R-Gluc an
66 iated with abnormal proximal exposure of the aerodigestive tract to refluxate; these patients respond
67 ulting from field-wide exposure of the upper aerodigestive tract to tobacco smoking.
68 CE-inhibitor-induced angioedema of the upper aerodigestive tract to treatment with 30 mg of subcutane
69 inoids are reported to reduce second primary aerodigestive tract tumors in patients with prior lung o
70 ter vascularized reconstruction of the upper aerodigestive tract (UADT) are associated with considera
71  identified as an oncogene involved in upper aerodigestive tract (UADT) carcinomas.
72  human HNSCC tumors, but not in normal upper aerodigestive tract (UADT) epithelia.