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1 urgdorferi sensu stricto, B. garinii, and B. afzelii).
2 ensu stricto, Borrelia garinii, and Borrelia afzelii.
3 of species B. garinii, and one of species B. afzelii.
4 urgdorferi sensu stricto, B. garinii, and B. afzelii.
5 is chronica atrophicans, and infection by B. afzelii.
6 conserved MLa1 pattern characteristic of B. afzelii.
7 urgdorferi sensu stricto, B. garinii, and B. afzelii.
8 gdorferi but not with Arp-deficient Borrelia afzelii.
10 e vls loci present in B. garinii Ip90 and B. afzelii ACAI have characteristics similar to those found
11 e loci of Borrelia garinii Ip90 and Borrelia afzelii ACAI, consisting of 11 vls silent cassettes and
12 cell-per-milliliter range: 5 cells/ml for B. afzelii and 8 cells/ml for Borrelia burgdorferi and Borr
13 mice that had immunizing infections with B. afzelii and B. garinii and then challenged by transplant
14 tic similarity and diversity suggest that B. afzelii and B. garinii are close relatives and were perh
15 i has greater inflammatory potential than B. afzelii and B. garinii, which may account in part for va
17 of Borrelia burgdorferi, along with Borrelia afzelii and Borrelia garinii, possessed a single iron-co
21 on, < 1 nM) against B. burgdorferi, Borrelia afzelii, and Borrelia garinii, the 3 main genospecies en
24 a burgdorferi, Borrelia garinii and Borrelia afzelii, are fascinating and enigmatic bacterial pathoge
25 urgdorferi sensu stricto, B. garinii, and B. afzelii--are associated with different chronic disease m
26 n Eurasia these species are largely Borrelia afzelii, B. garinii, B. burgdorferi, and B. bavariensis
27 ecies tested (B. burgdorferi, B. garinii, B. afzelii, B. japonica, B. valaisiana sp. nov., and B. and
28 t borrelial lymphocytoma can be caused by B. afzelii but also demonstrate an association with another
29 ined expression of BB0405 and showed that B. afzelii CB43 spirochetes significantly and drastically d
31 nce analyses revealed that B. garinii and B. afzelii CspZ orthologs possess a 64-amino-acid N-termina
34 urgdorferi sensu stricto, B. garinii, and B. afzelii genospecies was tested with a panel of sera with
36 urgdorferi sensu stricto, B. garinii, and B. afzelii have been isolated in Europe, whereas only B. bu
37 an Lyme agents Borrelia garinii and Borrelia afzelii; however, some of these plasmids had uniform but
38 the five isolates belonged to the species B. afzelii; however, this species is the predominant patien
40 and levels of antibody in B. garinii- and B. afzelii-infected animals were lower than in B. burgdorfe
41 lopmental stages and in response to Borrelia afzelii infection suggests a role in transmission of thi
43 originally identified on plasmid cp8.3 of B. afzelii IP21 were also analyzed by Southern hybridizatio
45 ia species that cause human Lyme disease, B. afzelii isolates ACA-1 and PKo and B. garinii isolates P
46 erologous B. burgdorferi, B. garinii, and B. afzelii isolates, suggesting that there is synergy betwe
49 This suggests that either B. garinii or B. afzelii might be the etiologic agent of borrelial lympho
50 does not provide cross-protection against B. afzelii, mostly likely due to downregulation of this pro
52 o the B. burgdorferi sensu stricto, Borrelia afzelii, or Borrelia garinii genospecies, the three main
53 10 isolates each of B. burgdorferi, Borrelia afzelii, or Borrelia garinii recovered from erythema mig
55 and N40; Borrelia garinii IP90; and Borrelia afzelii P/Gau was fluorescently labeled and hybridized t
56 . burgdorferi sensu stricto (s.s.) N40 or B. afzelii PKo and then were hyperimmunized with homologous
57 r among 10 B. burgdorferi s.s. N40 and 10 B. afzelii PKo isolates from DbpA hyperimmune mice, compare
58 ces among 9 B. burgdorferi s.s. N40 and 9 B. afzelii PKo isolates from OspC hyperimmune mice or among
59 ces among 4 B. burgdorferi s.s. N40 and 9 B. afzelii PKo isolates from OspC-immunized mice revealed n
60 B. burgdorferi sensu stricto cN40, Borrelia afzelii PKo, and Borrelia garinii PBi) and then treated
61 urgdorferi sensu stricto, B. garinii, and B. afzelii revealed a consistent association of infectivity
63 tive to blots prepared with B. garinii or B. afzelii strain antigens, in particular B. garinii 20047
65 fate binding correlated well, but DbpA of B. afzelii strain VS461 promoted differential binding to de
66 burgdorferi, Borrelia garinii, and Borrelia afzelii strains as well as in relapsing fever spirochete
67 and inflammatory, whereas B. garinii and B. afzelii strains can survive the adaptive immune response
70 relia species, specifically against Borrelia afzelii, the predominant B. burgdorferi sensu lato genos