ライフサイエンスコーパス: agalactiae

1 id detection of Streptococcus agalactiae (S. agalactiae).

2 ure alone for the detection of Streptococcus agalactiae.

3 genomic region including the pur genes in S. agalactiae.

4 activity and other cellular functions of S. agalactiae.

5 genes specific to the streptococci and to S. agalactiae.

6 n used to distinguish GAS from Streptococcus agalactiae.

7 : Streptococcus pneumoniae and Streptococcus agalactiae.

8 S. anginosus group, 35 S. pyogenes,and 20 S. agalactiae.

9 eria Staphylococcus aureus and Streptococcus agalactiae.

10 3 clinical blood cultures with Streptococcus agalactiae.

11 conserved in Gap homologs from Streptococcus agalactiae.

12 pected of being S. pseudoporcinus and not S. agalactiae.

13 ts of Streptococcus mutans and Streptococcus agalactiae.

14 The most frequent species was Streptococcus agalactiae (34%), and 52% of all cases were hematogenous

15 ltered the expression of several genes in S. agalactiae 874391 that encode key virulence factors, inc

16 We found that covR-deficient serotype III S. agalactiae 874391 was significantly attenuated for colon

17 Streptococcus bovis group (5), Streptococcus agalactiae (9), the Streptococcus anginosus group (1), S

18 otential site of perinatal acquisition of S. agalactiae, a major cause of neonatal sepsis.

19 th in human urine observed in ABU-causing S. agalactiae (ABSA) that was not seen among uropathogenic

20 affect the persistence or progression of S. agalactiae ABU.

21 th-enhanced PCR nominally detected 10 CFU S. agalactiae after 4 h of carrot broth incubation with com

22 enzymes, from S.pneumoniae and Streptococcus agalactiae, allowed for insights into this enzyme's mole

23 corresponding homologues from Streptococcus agalactiae also interacted with each other and formed a

24 Streptococcus agalactiae, also known as Group B Streptococcus (GBS) is

25 secondary immunization with conjugate and S. agalactiae, although not S. pneumoniae, results in a boo

26 , vancomycin, and ceftriaxone, Streptococcus agalactiae, ampicillin, and cefotaxime, Escherichia coli

27 detection of Candida albicans, Streptococcus agalactiae and Chlamydia trachomatis with a single bioch

28 Detection of Streptococcus agalactiae and detection of bacteremia at <1 CFU/ml were

29 perform a similar function in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the

30 ctobacillus iners, Gardnerella vaginalis, S. agalactiae and F. nucleatum to vaginal epithelial cells

31 presence of a supragenome for Streptococcus agalactiae and Haemophilus influenzae, it appears that t

32 h a putative peptidoglycan hydrolase from S. agalactiae and S. pneumoniae, indicative of a role in mu

33 th, as well as in Streptococcus pyogenes, S. agalactiae and S. suis.

34 Gap1 homologues from Streptococcus agalactiae and Staphylococcus aureus also interacted wit

35 Furthermore, the Gap1 homologues from S. agalactiae and Streptococcus sanguinis rescued the Fap1

36 homologous to the CAMP factor genes from S. agalactiae and Streptococcus uberis.

37 nce between the crystal structures of the S. agalactiae and the S. pneumoniae hyaluronate lyases.

38 y comparing the crystal structures of the S. agalactiae and the Streptococcus pneumoniae enzymes, and

39 s), group B streptococci (GBS; Streptococcus agalactiae), and Streptococcus pneumoniae were designed

40 Staphylococcus lugdunensis, 10 Streptococcus agalactiae, and 10 Enterococcus faecalis) were analyzed

41 m, Neisseria meningitidis, and Streptococcus agalactiae, and 3% of the residues in its deduced amino

42 ecium, Streptococcus pyogenes, Streptococcus agalactiae, and Streptococcus pneumoniae with K(d) value

43 calis, Streptococcus pyogenes, Streptococcus agalactiae, and viridans streptococci.

44 iv) dissemination of antibiotic-resistant S. agalactiae appears to include both clonal spread of resi

45 Group B Streptococcus (GBS) or Streptococcus agalactiae are beta-hemolytic gram-positive bacteria tha

46 Group B streptococci (GBS; Streptococcus agalactiae) are a major cause of invasive infections in

47 Group B streptococci (GBS) (Streptococcus agalactiae) are a major cause of sepsis and meningitis i

48 Group B streptococci (GBS; Streptococcus agalactiae) are beta-hemolytic, Gram-positive bacteria t

49 he group B streptococcus (GBS) Streptococcus agalactiae, are an important cause of systemic disease,

50 s which appear to synthesize glutathione (S. agalactiae ATCC 12927, S. pyogenes ATCC 8668, and Entero

51 Group B Streptococcus agalactiae bacteria (group B streptococci [GBS]) are the

52 S. agalactiae binding to chondroitin sulphate C oligosaccha

53 f the laboratories that tested Streptococcus agalactiae by disk diffusion, 17% reported nonsusceptibl

54 Streptococcus agalactiae can cause urinary tract infection (UTI).

55 ctor containing a promoterless Streptococcus agalactiae cat gene was constructed.

56 we show that crude extracts of Streptococcus agalactiae catalyze the gamma-GCS and GS reactions and c

57 Escherichia coli and Streptococcus agalactiae cause about 35% of cases of early-onset neona

58 Streptococcus agalactiae causes both symptomatic cystitis and asymptom

59 Streptococcus agalactiae causes severe invasive disease in humans and

60 ve and highly sensitive quantification of S. agalactiae cells in a concentration range of 10(1)-10(7)

61 ominis, Neisseria gonorrhoeae, Streptococcus agalactiae, Chlamydia trachomatis, Trichomonas vaginalis

62 uC homologue from serotype III Streptococcus agalactiae complements DeltaneuC.

63 aromyces cerevisiae and one of Streptococcus agalactiae constructed using the KEGG database.

64 sular polysaccharide (PPS14) and type III S. agalactiae containing a PPS14 core capsule identical to

65 S. agalactiae CovR promotes bladder infection and inflammat

66 I sequences were compared to those of the S. agalactiae cpsIa locus, and the primary difference betwe

67 dis, Streptococcus pneumoniae, Streptococcus agalactiae, cytomegalovirus, enterovirus, herpes simplex

68 The S. agalactiae detection rate by early-aliquot carrot broth-

69 e group B streptococcal (iGBS; Streptococcus agalactiae) disease in infants is unknown.

70 Here we describe Streptococcus agalactiae DPC7040, a human faecal isolate, which exhibi

71 ered from the bottles with S. pneumoniae, S. agalactiae, E. coli, N. meningitidis, or H. influenzae i

72 One of these new structures resembles the S.agalactiae enzyme conformation, and provides evidence of

73 ys with Staphylococcus aureus, Streptococcus agalactiae, Escherichia coli and Klebsiella pneumonia.

74 0% for Neisseria meningitidis, Streptococcus agalactiae, Escherichia coli K1, Listeria monocytogenes,

75 aecalis, Enterococcus faecium, Streptococcus agalactiae, Escherichia coli, Klebsiella pneumoniae, and

76 oniae, Streptococcus pyogenes, Streptococcus agalactiae, Escherichia coli, Klebsiella pneumoniae, Hae

77 The putative gene for S. agalactiae gamma-GCS was identified and cloned, and the

78 e loop region in GSH binding, chimeras of S. agalactiae gamma-GCS-GS were made containing gamma-GCS d

79 Streptococcus agalactiae (GBS) is a major cause of serious newborn bac

80 macrophages induced by group B Streptococcus agalactiae (GBS) is likely an important virulence mechan

81 Streptococcus agalactiae (GBS) is the leading cause worldwide of neona

82 cies equisimilis; SDSE) and B (Streptococcus agalactiae; GBS) was demonstrated.

83 The role of the S. agalactiae global virulence regulator, CovR, in UTI path

84 i), and recombinant SCPB, from Streptococcus agalactiae (group B streptococci), were compared in the

85 shown that the human pathogen Streptococcus agalactiae (Group B Streptococci, GBS) encodes a single

86 The Gram-positive bacteria Streptococcus agalactiae (group B streptococci, GBS) is an important h

87 S. agalactiae (Group B streptococci, GBS), E. faecalis, S.

88 Human isolates of serotype III Streptococcus agalactiae (group B streptococcus [GBS]) can be divided

89 Streptococcus agalactiae (group B Streptococcus [GBS]) causes serious

90 Streptococcus agalactiae (group B streptococcus [GBS]) colonizes the r

91 e characterized 31 isolates of Streptococcus agalactiae (group B Streptococcus [GBS]) from several ge

92 Streptococcus agalactiae (group B Streptococcus [GBS]) has not been de

93 y-onset neonatal sepsis due to Streptococcus agalactiae (group B Streptococcus [GBS]) infection is on

94 Streptococcus agalactiae (group B streptococcus [GBS]) is a Gram-posit

95 Streptococcus agalactiae (group B streptococcus [GBS]) is a leading ca

96 Neonatal infection with Streptococcus agalactiae (group B Streptococcus [GBS]) is a leading ca

97 Streptococcus agalactiae (group B Streptococcus [GBS]) is a leading ca

98 rnal vaginal colonization with Streptococcus agalactiae (Group B Streptococcus [GBS]) is a precursor

99 Streptococcus agalactiae (group B Streptococcus [GBS]) is an important

100 Streptococcus agalactiae (group B Streptococcus [GBS]) is an important

101 Streptococcus agalactiae (group B Streptococcus [GBS]) remains a leadi

102 Escherichia coli, E. faecalis, Streptococcus agalactiae (group B streptococcus [GBS]), or Streptococc

103 Streptococcus agalactiae (group B Streptococcus or GBS) is a common ca

104 Streptococcus agalactiae (group B Streptococcus or GBS) is a common co

105 Streptococcus agalactiae (group B streptococcus) causes invasive disea

106 Streptococcus agalactiae (Group B Streptococcus) is a commensal of the

107 The exception was Streptococcus agalactiae (group B Streptococcus), for which non-contam

108 with the homologous gene from Streptococcus agalactiae (Group B Streptococcus), Streptococcus equi s

109 Streptococcus agalactiae (Group B Streptococcus, GBS) causes life-thre

110 Streptococcus agalactiae (group B streptococcus, GBS) causes neonatal

111 Streptococcus agalactiae (group B streptococcus, GBS) expresses either

112 Streptococcus agalactiae (Group B Streptococcus, GBS) is a commensal o

113 Streptococcus agalactiae (group B Streptococcus, GBS) is a leading cau

114 Streptococcus agalactiae (group B Streptococcus, GBS) is the predomina

115 igh abundance by the bacterium Streptococcus agalactiae (Group B Streptococcus, GBS).

116 Streptococcus agalactiae (group B streptococcus, or GBS) is a common c

117 Streptococcus agalactiae (Group B Streptococcus; GBS) is a common caus

118 Streptococcus agalactiae (group B Streptococcus; GBS) is a significant

119 Streptococcus agalactiae (group B Streptococcus; GBS) produces a CPS t

120 by the Gram-positive bacteria Streptococcus agalactiae (Group B Streptococcus; GBS) type III (GBSIII

121 otentially pathogenic bacteria Streptococcus agalactiae (Group-B-Streptococci; GBS) and Escherichia c

122 Streptococcus agalactiae [group B Streptococcus (GBS)] is a leading ca

123 Streptococcus agalactiae, (Group B Streptococcus (GBS)), is a common c

124 In Streptococcus agalactiae, GSH synthesis is catalyzed by a single enzym

125 Based on their core genomes, S. agalactiae had the highest relative rate of recombinatio

126 he burden of disease caused by Streptococcus agalactiae has increased significantly among older adult

127 Skizzle (SkzL), secreted by Streptococcus agalactiae, has moderate sequence identity to streptokin

128 ulosis (also using GeneXpert), Streptococcus agalactiae, herpes simplex virus (types 1 and 2), varice

129 Streptococcus agalactiae hyaluronate lyase degrades primarily hyaluron

130 Streptococcus agalactiae hyaluronate lyase is a virulence factor that

131 exasaccharide hyaluronan complex with the S. agalactiae hyaluronate lyase was determined at 2.2 A res

132 ides a discriminatory subtype analysis of S. agalactiae; (ii) most human invasive and bovine S. agala

133 itis and peptic ulcer disease, Streptococcus agalactiae, implicated in neonatal meningitis, and sever

134 s a rapid detection and quantification of S. agalactiae in environmental samples but also opens up ne

135 (ABU); however, growth characteristics of S. agalactiae in human urine have not previously been repor

136 atment of women colonized with Streptococcus agalactiae include vancomycin prophylaxis for those with

137 Group B streptococcus (GBS; Streptococcus agalactiae) induces apoptosis of macrophages, and this m

138 Neonatal Streptococcus agalactiae infections cause significant morbidity and mo

139 ction as rare complications of Streptococcus agalactiae infective endocarditis.

140 act Neisseria meningitidis nor Streptococcus agalactiae inhibited the OVA-specific IgG response.

141 Group B streptococcus (GBS) or Streptococcus agalactiae is a beta-hemolytic, Gram-positive bacterium

142 Streptococcus agalactiae is a primary cause of neonatal morbidity and

143 Streptococcus agalactiae is the leading cause of bacterial sepsis and

144 roup B Streptococcus (GBS), or Streptococcus agalactiae, is a pathogen that causes preterm births, st

145 Streptococcus agalactiae isolates (n = 1,056) were highly susceptible

146 We conclude that (i) human and bovine S. agalactiae isolates represent distinct populations; (ii)

147 tiae; (ii) most human invasive and bovine S. agalactiae isolates represent distinct subtypes, suggest

148 characteristics of 52 human and 83 bovine S. agalactiae isolates.

149 The Gram-positive pathogen Streptococcus agalactiae, known as group B Streptococcus (GBS), is the

150 Inhibition remained S. agalactiae-like (i.e., very weak).

151 pyogenes (</=0.12 microg/mL), Streptococcus agalactiae (</=0.12 microg/mL), Streptococcus anginosus

152 We recently identified that Streptococcus agalactiae MprF synthesizes lysyl-phosphatidylglycerol (

153 treptococcus iniae (CpsY), and Streptococcus agalactiae (MtaR) that regulate methionine transport, am

154 dder uroepithelial cell models of UTI and S. agalactiae mutants in covR and related factors, includin

155 Streptococcus agalactiae or group B Streptococcus (GBS) is the cause o

156 Streptococcus agalactiae, or group B Streptococcus (GBS), is an import

157 Perinatal infection with Streptococcus agalactiae, or Group B Streptococcus (GBS), is associate

158 eumoniae, Escherichia coli, or Streptococcus agalactiae (P < .001).

159 reservoir available for inclusion in the S. agalactiae pan-genome is vast and that unique genes will

160 The results suggest a role for SkzL in S. agalactiae pathogenesis through fibrinolytic enhancement

161 that an unannotated homodimeric TetR from S. agalactiae (PDB 3KKC) is the bona fide zinc efflux regul

162 S. epidermidis compared to S. aureus and S. agalactiae PJI.

163 Mice infected with covR-deficient S. agalactiae produced less proinflammatory cytokines than

164 ent H10, similar to an unknown Streptococcus agalactiae protein, was present in 31% of middle ear iso

165 ginosa, Klebsiella pneumoniae, Streptococcus agalactiae, Proteus, Enterococcus and Staphylococcus spe

166 ll promote comparative genomic studies of S. agalactiae recovered from diverse sources.

167 Homologous GtfA and GtfB from Streptococcus agalactiae rescued the glycosylation defect in the gtf1g

168 sor for the rapid detection of Streptococcus agalactiae (S. agalactiae).

169 ptococcal species, including S. pyogenes, S. agalactiae, S. dysgalactiae, S. equi, S. mutans, S. pneu

170 m SALSA, bound to Streptococcus pyogenes, S. agalactiae, S. gordonii, and Escherichia coli.

171 ococci, including Streptococcus pyogenes, S. agalactiae, S. pneumoniae, and S. equi.

172 the crystal structures of the Streptococcus agalactiae SAG2603 V/R sortase SrtC1 in two space groups

173 mutant of SCP from group B Streptococcus (S. agalactiae, SCPB) revealed SCPB is composed of five dist

174 The reporting of accurate Streptococcus agalactiae screening results in a short time frame is of

175 se available in databases showed that the S. agalactiae species can be described by a pan-genome cons

176 Srr2, an SRRP from S. agalactiae strain COH1, has been implicated in bacterial

177 e shotgun draft sequence for a Streptococcus agalactiae strain representing multilocus sequence type

178 Many group B Streptococcus agalactiae strains and other pathogenic streptococci exp

179 We conclude that some S. agalactiae strains can grow in human urine, and this rel

180 quenced serotype V strain 2603 V/R and 19 S. agalactiae strains from several serotypes using whole-ge

181 revealed the genetic heterogeneity among S. agalactiae strains, even of the same serotype, and provi

182 cytogenes, Streptococcus spp., Streptococcus agalactiae, Streptococcus anginosus group, Streptococcus

183 Genome comparisons among S. agalactiae, Streptococcus pneumoniae, Streptococcus pyog

184 bination in the pan-genomes of Streptococcus agalactiae, Streptococcus pyogenes and Streptococcus sui

185 ccus aureus in 8 patients, and Streptococcus agalactiae, Streptococcus pyogenes, and Streptococcus sa

186 t populations; (ii) human host-associated S. agalactiae subtypes may occasionally be transmitted to b

187 ,160,267 bp genome sequence of Streptococcus agalactiae, the leading cause of bacterial sepsis, pneum

188 r disease-causing serotypes of Streptococcus agalactiae, the main cause of neonatal infection in huma

189 For S. agalactiae, there was a single false-positive and false-

190 By contrast, in Streptococcus agalactiae, there were a number of cases in which both s

191 i, Fusobacterium nucleatum and Streptococcus agalactiae to oligomannose N-glycans, galactose-terminat

192 rain of group B Streptococcus (Streptococcus agalactiae) type III (GBS-III) that expresses desialylat

193 n additional 26 (12.8%) were positive for S. agalactiae upon subculture.

194 SA) that was not seen among uropathogenic S. agalactiae (UPSA) strains isolated from patients with ac

195 The pathogenesis of S. agalactiae UTI is complex, multifactorial, and influence

196 Streptococcus agalactiae was the most common pathogen (5/15 [33.3%]) a

197 ission of antibiotic-resistant Streptococcus agalactiae, we compared phenotypic and genotypic charact

198 hich only 2 (Escherichia coli, Streptococcus agalactiae) were cultivated.

199 region of a GspB homologue of Streptococcus agalactiae, which is acidic rather than basic, showed no

200 mproved with inocula of 100 and 1,000 CFU S. agalactiae, with the majority of these aliquots demonstr