ライフサイエンスコーパス: aged mouse

1 progeny in the ischemic brain of the middle-aged mouse.

2 ischemic striatum and corpus callosum in the aged mouse.

3 progeny in the ischemic brain of the middle-aged mouse.

4 levels decreased significantly in NMJs from aged mouse.

5 xpression significantly decreases in healthy-aged mouse Achilles tendons.

6 lung regeneration and attenuated fibrosis in aged mouse after injury.

7 ads to impaired Ca(2+) signal propagation in aged mouse (aged >22 months) epidermis and human (aged >

8 of macrophage activation in middle-aged and aged mouse and human cochleas, along with transcriptomic

9 ations in the lymphoid progenitor content of aged mouse bone marrow (BM) have been described, irradia

10 the chronic presence of type I interferon in aged mouse brain impedes cognitive ability by altering m

11 8-specific E3 ligase, Ube3a, is decreased in aged mouse brain.

12 dicate an almost 50% aneuploidy frequency in aged mouse brain.

13 udy the properties of senescent cells in the aged mouse brain.

14 as of each ChP in the developing, adult, and aged mouse brain.

15 f chemoattractant factors, accumulate in the aged mouse brain.

16 transcriptomic profiling of young adult and aged mouse brains at acute (3 d) and chronic (14 d) stag

17 cell RNA sequencing (scRNA-seq) of young and aged mouse brains following stroke and found that interf

18 re extensive expression of NT-3 in adult and aged mouse brains with cortical expression apparent at 4

19 ansplantation of MG/MPhi from young, but not aged, mouse brains into the cerebral cortices of aged st

20 L1 is disrupted in human OA cartilage and in aged mouse cartilage.

21 is a decline in eIF2alpha phosphorylation in aged mouse cerebral cortex that is associated with highe

22 sed Duplex Sequencing on eight tissues of an aged mouse cohort to detect >89,000 independent somatic

23 F-actin disruption in aged mouse eggs exacerbated untimely dissociation of sis

24 In moderately aged mouse epidermis, we find that abnormal acidificatio

25 imary-cultured NPCs from the young adult and aged mouse forebrain.

26 ious ages, i.e., juvenile, adult, and middle-aged mouse groups.

27 Aged mouse hepatocytes exhibit transcriptional suppressi

28 We observe that BECs in the aged mouse hippocampus express an inflammatory transcrip

29 RNA inductions after fornix transections in aged mouse hippocampus.

30 egulated by histone deacetylase HDAC3 in the aged mouse hippocampus.

31 level by transplanting individual young and aged mouse HSCs labeled with barcoded viral vector, foll

32 etion and glucose tolerance are preserved in aged mouse islets by the heightened metabolic sensitivit

33 In this study, aged mouse kidneys displayed a reduced epithelial prolif

34 that the ratio of ASK1/Trx-ASK1 increases in aged mouse livers and that this correlates with the incr

35 We find that nuclear extracts from normal aged mouse livers have decreased p42(C/EBPalpha) levels

36 In Sirt7 knockout and aged mouse livers, NUCKS1 was bound at the promoters and

37 an LPS challenge is delayed significantly in aged mouse livers.

38 ory cell recruitment and pulmonary oedema in aged mouse lung during infection.

39 ury contributed to impaired AEC2 recovery in aged mouse lungs after injury.

40 In aged mouse lungs, H3K9me2 loss coincided with fewer alve

41 Using an aged mouse model and high-throughput sequencing, this st

42 effects of low-dose lithium treatment in an aged mouse model expressing a parkin mutation within dop

43 y demonstrates successful gene therapy in an aged mouse model of human genetic deafness.

44 Using an aged mouse model of RSV pathogenesis, we found that aged

45 Here, we used an aged mouse model to investigate the protective efficacy

46 Using aged mouse models and human progenitors, we demonstrate

47 In aged mouse models of AD (APPswe/PS1DeltaE9 overexpressin

48 ed conditional knockout [cKO]) and naturally aged mouse models.

49 n of active zones in developing, mature, and aged mouse NMJs by immunohistochemical detection of the

50 duced the accumulation of senescent cells in aged mouse organs.

51 or-dependent survival of neurons and, in the aged mouse, paradoxical upregulation of myelin and ephri

52 esions were 30-100-fold more frequent in the aged mouse paravertebral SCG than in the prevertebral ce

53 f both the mitochondria-injured and in vitro-aged mouse platelets.

54 positively affects memory mechanisms in the aged mouse, possibly by acting on the septohippocampal c

55 we show that lower VLC-PUFA abundance in the aged mouse retina is accompanied by a reduction in visua

56 oprotein is expressed in neonatal but not in aged mouse skin.

57 libraries from apparently healthy young and aged mouse ventricular cardiac muscle cells.

58 Treatment of the ischemic middle-aged mouse with Sildenafil increased nestin expressing n