ライフサイエンスコーパス: air pollutant

1 emit black carbon (BC), a climate forcer and air pollutant.

2 ation of tropospheric ozone (O3), a powerful air pollutant.

3 may modify susceptibility to the effects of air pollutants.

4 egression to account for colinearity between air pollutants.

5 concentrations of fungal spores, pollen, and air pollutants.

6 ward large multiwell pads, release hazardous air pollutants.

7 -km resolution were estimated for 11 ambient air pollutants.

8 lic concerns regarding exposure to hazardous air pollutants.

9 lwood usage, and climate and health-relevant air pollutants.

10 ure to hazardous concentrations of smoke and air pollutants.

11 ay potentiate the adverse effects of oxidant air pollutants.

12 ons and concerns about exposure to hazardous air pollutants.

13 he observed associations were independent of air pollutants.

14 ng CH4 emissions while controlling for other air pollutants.

15 considered effects from exposure to ambient air pollutants.

16 d drug to modulate responsiveness to oxidant air pollutants.

17 tion and may be useful for analysis of other air pollutants.

18 calculate an average exposure parameter for air pollutants.

19 n increased population exposure to unhealthy air pollutants.

20 mortality and long-term exposure to several air pollutants.

21 is a key oxidant in the degradation of most air pollutants.

22 inesses, or dump sites, is a large source of air pollutants.

23 predict long-term average concentrations of air pollutants.

24 ion properties commonly observed in ambient air pollutants.

25 ensive, coproduces carbon dioxide, and emits air pollutants.

26 The estimates slopes for air pollutants (28 days cumulated) were higher in partic

27 Very little is currently known about air pollutants' adverse effects on neurodegenerative dis

28 Nitrogen dioxide (NO2) is an environmental air pollutant and endogenously generated oxidant that co

29 Ozone is a highly toxic air pollutant and global health concern.

30 There is a pressing need to verify air pollutant and greenhouse gas emissions from anthropo

31 NO is a precursor to tropospheric ozone, an air pollutant and greenhouse gas.

32 tions of ground-level ozone (O3 ) - a common air pollutant and phytotoxin - currently being experienc

33 We aimed to investigate whether ambient air pollutant and traffic exposures in early gestation c

34 Associations between air pollutants and ACOS were evaluated using Cox regress

35 We investigated criteria air pollutants and air toxics during the period before c

36 d associations between perinatal exposure to air pollutants and autism spectrum disorder (ASD) in chi

37 The association between mixtures of air pollutants and birthweight z-scores (standardized fo

38 studies have evaluated associations between air pollutants and cardiac arrhythmia.

39 esearch has examined the association between air pollutants and catecholamines.

40 al association between childhood exposure to air pollutants and changes in diet among adolescents.

41 Associations between exposure to ambient air pollutants and childhood asthma incidence (up to age

42 Inhalation exposure to indoor air pollutants and cigarette smoke increases the risk of

43 ease energy security and reduce emissions of air pollutants and CO2 from coal use, China is attemptin

44 tive associations between a diverse group of air pollutants and cognitive functioning in children and

45 Associations between air pollutants and ECG parameters in the concurrent and

46 fects models to estimate the main effects of air pollutants and effect modification by DNA methylatio

47 ical studies addressing maternal exposure to air pollutants and fetal growth during gestation as asse

48 is a key oxidant involved in the removal of air pollutants and greenhouse gases from the atmosphere.

49 pes of oxidized lipids formed in response to air pollutants and how this occurs and their relevance t

50 We tested associations between air pollutants and immune markers and adult-onset asthma

51 While associations between exposure to air pollutants and increased morbidity and mortality are

52 cent progress in the granular measurement of air pollutants and individual-level exposures, and how t

53 to evaluate whether the associations between air pollutants and livebirth were modified by folate int

54 of folate on the associations between other air pollutants and livebirth.

55 association between exposure to 43 household air pollutants and lung cancer.

56 Wildfire smoke contains numerous hazardous air pollutants and many studies have documented populati

57 unclear on how PM(2.5) interacts with other air pollutants and meteorological factors at different t

58 es suggests that exposure to traffic-related air pollutants and particulate matter less than 2.5 um i

59 rtant for tightly controlling the release of air pollutants and preventing more serious diseases.

60 gies typically reduce emissions for specific air pollutants and sectors to maintain pollutant concent

61 kin aging, but no direct link between indoor air pollutants and skin aging manifestations has ever be

62 relationship between early-life exposure to air pollutants and subsequent asthma diagnosis.

63 e commuters experience increased exposure to air pollutants and that traffic characteristics, land us

64 relations between ambient concentrations of air pollutants and the production rates and concentratio

65 eview, we focus on the major constituents of air pollutants and their impacts on chronic respiratory

66 te associations between exposures to ambient air pollutants and TL in African American children and a

67 t moist meteorological conditions in winter, air pollutants and water vapour accumulate in a shallow

68 is paper, we assess the relationship between air pollutants and weather conditions with outpatient vi

69 Snow scavenges air pollutants, and after snow melting, it can induce an

70 diameter <2.5 mum [PM2.5] or <10 mum [PM10]) air pollutants, and heart failure hospitalisations or he

71 ion costs, health damages caused by criteria air pollutants, and methane leakage from the natural gas

72 ess to neurologists and exposure to selected air pollutants, and restricting to never movers and urba

73 pose time series data of PM(2.5), five other air pollutants, and six meteorological factors, as well

74 e elderly is associated with traffic-related air pollutants, and this phenotype differs from genetica

75 is growing evidence that multiple genes and air pollutants are associated with asthma.

76 Ambient air pollutants are important determinants of bronchitis

77 Land use regression (LUR) models for air pollutants are often developed using multiple linear

78 hat neither noise levels nor traffic-related air pollutants are responsible for this relationship.

79 resented here suggest that emissions of many air pollutants are significantly underestimated in curre

80 Traditional depictions of MB for criteria air pollutants are such that each additional ton of emis

81 rotocol, we assessed residential exposure to air pollutants as annual average concentrations of parti

82 oring site that is instrumented for criteria air pollutants, associated gases, and particle compositi

83 We evaluated the lag structure of air pollutant associations with lung function and potent

84 While air pollutants at historical levels have been associated

85 rotection Agency-modeled levels of hazardous air pollutants at the time and place of birth and ASD in

86 o estimate concentrations of traffic-related air pollutants based on nitrogen oxides (NOx) at partici

87 surement platform to characterize a suite of air pollutants (black carbon (BC), particle-bound polycy

88 Residential exposures to traffic-related air pollutants (black carbon, particulate matter <2.5 mu

89 s-sectionally), including indoor and outdoor air pollutants, built environment, green spaces, tobacco

90 itting conditions in the truck fleet affects air pollutants by 20% to 65%; although these estimates a

91 ease, and post-translational modification by air pollutants can enhance the allergenic potential of p

92 Exposure to air pollutants can influence cardiac autonomic tone and

93 sions (CO(2), CH(4), and N(2)O) and criteria air pollutant (CAP) emissions (VOC, CO, NO (x), SO(2), P

94 lyzing the greenhouse gas (GHG) and criteria air pollutant (CAP) emissions associated with hydrogen p

95 ehicle greenhouse gas (GHG) and conventional air pollutant (CAP) emissions to speed, weight, age, and

96 It is not known whether exposure to air pollutants causes systemic oxidative stress in child

97 eway closure on traffic and several criteria air pollutants (CO, NO2, O3, PM10, PM2.5).

98 ve a higher information gain than other gene-air pollutant combinations.

99 attenuation factor," the ratio of the indoor air pollutant concentration to the pollutant concentrati

100 Residence-specific air pollutant concentrations (O3, PM2.5, NOx, and black

101 thickness and long-term exposure to ambient air pollutant concentrations (PM2.5, NOX, and black carb

102 t model (CMAQ) simulations to estimate daily air pollutant concentrations across Georgia.

103 ing fine scale spatial variations in average air pollutant concentrations across urban areas.

104 ictors in estimating on-road traffic-related air pollutant concentrations and GAMs perform better for

105 ities of air quality (including both average air pollutant concentrations and high pollution episodes

106 s are fundamental in understanding hazardous air pollutant concentrations and ozone formation.

107 ed provides daily spatial field estimates of air pollutant concentrations and uncertainties that are

108 mum in diameter (PM2.5) and traffic-related air pollutant concentrations are associated with cardiov

109 Associations of annual average air pollutant concentrations at participants' residences

110 eased birth weight associated with increased air pollutant concentrations during pregnancy.

111 Contributions from vaping to air pollutant concentrations in the home did not exceed

112 s not known whether exposure to contemporary air pollutant concentrations is associated with progress

113 Air pollutant concentrations near major highways are usu

114 s is needed to achieve targets for household air pollutant concentrations or fuel savings.

115 captures the response of spatially resolved air pollutant concentrations to changes in electricity-g

116 associations of SES measures with predicted air pollutant concentrations, demonstrating the importan

117 e investigated short-term changes in ambient air pollutant concentrations, including speciated partic

118 To assess determinants of variability in air pollutant concentrations, we develop land use regres

119 his risk because a wide range of deleterious air pollutants contribute to the pathogenesis of asthma

120 ndings suggest that prenatal exposure to PAH air pollutants contributes to slower processing speed, a

121 Acute controlled exposure to air pollutants did not increase the short-term risk of a

122 hort-term elevations in the level of ambient air pollutants does not increase the incidence of UGIB s

123 date found that exposure to certain ambient air pollutants during pregnancy is negatively associated

124 t analyses, higher concentrations of several air pollutants during pregnancy or childhood were associ

125 omarker of oxidative stress, and exposure to air pollutants during various time windows of pregnancy.

126 We investigated maternal exposures to air pollutants during weeks 2-8 of pregnancy and their a

127 sistance of human lung cells (e.g., SAEC) to air pollutants (e.g. DEPs).

128 Community-level regional air pollutants [e.g., nitrogen dioxide (NO2), elemental

129 -level covariates, including traffic-related air pollutants [e.g., ultrafine particles (UFPs) and nit

130 Adding a traffic-related air pollutant (elemental carbon) to models explained 20-

131 Air pollutant emission levels in China remain substantia

132 Difficulty in obtaining air pollutant emission permits and the cost of air pollu

133 rado O&G activities, production volumes, and air pollutant emission rates from two Colorado basins to

134 Our analysis of air pollutant emissions and controls can assist biorefin

135 development programs, often lack historical air pollutant emissions data, which can pose challenges

136 missions, fossil energy demand, and criteria air pollutant emissions for the GTW-biodiesel process, i

137 lity of federal air regulations and quantify air pollutant emissions for two feasibility-level bioref

138 health risk by reducing "business as usual" air pollutant emissions from economic activities.

139 ever, the effect of fuel moisture content on air pollutant emissions from solid-fuel cookstoves remai

140 ts based on social costs of carbon and other air pollutant emissions highlights the importance of gro

141 We show that 50-60% of China's air pollutant emissions in 2007 were associated with goo

142 Here, we estimate air pollutant emissions in Africa from future (2030) ele

143 However, reducing air pollutant emissions may also have an important co-ha

144 s (WTW) greenhouse gases (GHGs) and criteria air pollutant emissions of MHD BEVs with their conventio

145 impacts of declining transportation-related air pollutant emissions on disparities in exposure have

146 ive for climate policy through reductions in air pollutant emissions that occur when targeting shared

147 The effects of international trade on air pollutant emissions, air quality and health have bee

148 that climate change, exclusive of changes in air pollutant emissions, can significantly impact ozone

149 mass combustion have become major sources of air pollutant emissions, with substantial spatial and te

150 ated external costs due to energy-associated air pollutant emissions.

151 Diesel vehicles are a major source of air pollutant emissions.

152 12 per cent (411,100 deaths) were related to air pollutants emitted in a region of the world other th

153 Mercury is a toxic air pollutant, emitted from the combustion of coal.

154 sociations with concentrations of 11 ambient air pollutants estimated by combining Community Multisca

155 investigated associations between 11 ambient air pollutants, estimated by combining Community Multisc

156 rely on complete and accurate spatiotemporal air pollutant estimates.

157 epigenome-wide association studies (EWAS) on air pollutants exist, and none have been done on transpo

158 ally related to observations linking ambient air pollutant exposure and adverse neurological/neurodev

159 Long-term low- to moderate-level air pollutant exposure is associated with a greater risk

160 even greater importance that the effects of air pollutant exposure on human skin be investigated.

161 Recent studies link air pollutant exposure to adverse neurodevelopmental out

162 ession (LUR) models have been used to assess air pollutant exposure, but limited evidence exists on w

163 halation, which is the model traffic-related air pollutant exposure, is associated with vascular dysf

164 males-to the deleterious effects of prenatal air pollutant exposure, which may be due to a synergism

165 t racial, ethnic, and economic inequality in air pollutant exposure.

166 birth, we assessed long-term traffic-related air pollutant exposures (represented by nitrogen dioxide

167 nd n-6 polyunsaturated fatty acids [PUFAs]), air pollutant exposures (residence-specific third-trimes

168 fects models to examine associations between air pollutant exposures and SBP, taking into account mea

169 ovide modest support for the hypothesis that air pollutant exposures are related to sympathetic nervo

170 for estimating associations between CVDs and air pollutant exposures by providing clues about the und

171 ssess the interaction of maternal asthma and air pollutant exposures in relation to PTB risk.

172 Acute and long-term air pollutant exposures merit further study in relation

173 findings outline the lung-brain axis, where air pollutant exposures result in circulating, cytokine-

174 Air pollutant exposures were analyzed as both quintiles

175 r (10), before adjusting for risk factors or air pollutant exposures.

176 n children was associated with higher annual air pollutant exposures.

177 ciations between short-term exposure to main air pollutants (fine particulate matter [PM2.5] and ozon

178 ions of short-term exposure to major ambient air pollutants-fine particles (PM(2.5)), inhalable parti

179 ric ozone is considered the most detrimental air pollutant for vegetation at the global scale, with n

180 organic pollutants and the beneficial use of air pollutants for the removal of water pollutants since

181 c aromatic hydrocarbons (PAHs) are hazardous air pollutants formed during incomplete combustion.

182 r when meteorological inversions concentrate air pollutants from oil and gas activities, but when sol

183 Exposure in pregnancy and childhood to air pollutants from tailpipe and non-tailpipe emissions

184 ement results and indicated the transport of air pollutants from wildfires burning in southern Califo

185 mental coexposures including traffic-related air pollutants, greenness, and neighborhood walkability.

186 and production sites the major stressors are air pollutants, ground and surface water contamination,

187 Although a common indoor air pollutant, guidelines for control of incense use hav

188 d that women with higher exposure to ambient air pollutants had lower fertility treatment success but

189 timate annual concentrations of 40 hazardous air pollutants (HAPs) across the continental United Stat

190 However, there are many gas-phase hazardous air pollutants (HAPs) identified by the Environmental Pr

191 nogen in outdoor air among the 187 hazardous air pollutants (HAPs) identified by the U.S. Environment

192 on (HC) species, including several hazardous air pollutants (HAPs).

193 Near-road exposure to air pollutants has been associated with decreased lung f

194 actions between GSTP1 and different types of air pollutants have a higher information gain than other

195 pulmonary effects of the individual criteria air pollutants have been well investigated, but little i

196 We updated the model to add criteria air pollutants, hazardous air pollutants, releases to wa

197 cts have been hypothesized as a mechanism of air pollutant health effects, though scant prior epidemi

198 The case study illustrates how air pollutant health impacts can be cost-effectively min

199 or smaller than 2.5 mum, the most prominent air pollutant in Chinese cities).

200 tality risk related to long-term exposure to air pollutants in a large prospective US cohort.

201 and NO2 (collectively called NOx) are major air pollutants in automotive emissions.

202 ssessing the relative importance of specific air pollutants in different regions of the world, showin

203 mias during and after controlled exposure to air pollutants in healthy volunteers and patients with c

204 to monitor the spatiotemporal variations of air pollutants in large scales, but it has been challeng

205 Prenatal exposure to multiple air pollutants in the first trimester was associated wit

206 thacholine (MCh) and particulate matter (PM) air pollutants, in the absence of underlying inflammatio

207 Concentrations of 17 air pollutants including nitrogen oxides (NOX), particul

208 Higher short-term exposure to air pollutants, including PM2.5, black carbon, and parti

209 The transient concentration of the other air pollutants, including PM2.5, PM10, and O3, did not s

210 Although exposure to ambient air pollutants increases cardiovascular disease risk in

211 ty income, or prenatal exposure to hazardous air pollutants, indicating that spatial variation is not

212 ine atmosphere, where continental and marine air pollutants interact and atmospheric oxidative capaci

213 Increased levels of urban air pollutants interacting with parental stress have bee

214 The air pollutant is highly variable over time and space, wh

215 Exposure to air pollutants is a well-established cause of asthma exa

216 Exposure to traffic-related air pollutants is an important public health issue.

217 ed the hypothesis that perinatal exposure to air pollutants is associated with ASD, focusing on pollu

218 iologic literature suggests that exposure to air pollutants is associated with fetal development.

219 c studies that increased exposure to ambient air pollutants is associated with olfactory dysfunction.

220 Sequestration of air pollutants is one of the main ecosystem services tha

221 Findings suggest that particle air pollutant levels in Canadian metros are substantiall

222 ssociations between the risk of CRAO and the air pollutant levels in the days preceding each event.

223 repeated biomarker measurements and ambient air pollutant levels.

224 that diabetes alters pulmonary responses to air pollutants like ozone (O3).

225 two putative biological mechanisms by which air pollutants may adversely affect the brain.

226 Air pollutants may impact anatomy and/or physiological f

227 Ambient air pollutants may increase preterm birth (PTB) risk, bu

228 Perinatal exposure to air pollutants may increase risk for ASD.

229 g in utero, environmental factors, including air pollutants, may permanently organize these systems t

230 Land use regression (LUR) models rely on air pollutant measurements for their development, and ar

231 Regional air pollutant measures were based on the Environmental P

232 Prenatal exposure to ambient air pollutants might cause adverse birth outcomes; howev

233 ntile to the median level of exposure to the air pollutant mixture could occur.

234 vent-based" approach to characterize complex air pollutant mixtures was applied in the Oil Sands regi

235 We identified air pollutant monitoring stations located near these pat

236 ns to mitigate the adverse health impacts of air pollutants must include the reduction of emissions t

237 del concentrations of mainly traffic-related air pollutants (nitrogen oxides (NOx), particulate matte

238 r concentrations of a transportation-related air pollutant, nitrogen dioxide (NO2), in the United Sta

239 Nitrous oxide (N(2) O) is an air pollutant of major environmental concern, with agric

240 effects of prenatal exposure to mixtures of air pollutants of particulate matter (PM), PM(2.5), PM(1

241 papers to estimate national mean effects of air pollutants on daily deaths in time-series analyses.

242 The effects of ECV and air pollutants on repeated wheeze (>/= 2 episodes) were

243 The effect of indoor air pollutants on respiratory morbidity among patients w

244 High concentrations of air pollutants on roadways, relative to ambient concentr

245 relationship between suicide and exposure to air pollutants on the day of the suicide and during the

246 tifying mechanisms through which mixtures of air pollutants operate, the causality of air pollution-a

247 These scenarios reflect air pollutant outcomes of implementations of certain ide

248 of air humidity, darkness, and pulses of the air pollutant ozone (O3), involves the SLOW ANION CHANNE

249 Inhalation of the ambient air pollutant ozone causes lung inflammation and can sup

250 to estimate average annual exposure to five air pollutants: ozone, nitrogen dioxide (NO(2)), sulfur

251 estimate on-road concentrations of four key air pollutants, particle-bound polycyclic aromatic hydro

252 at were robust to adjustment by other common air pollutants (particles <2.5 mum in diameter, ozone, n

253 ngs suggest that early exposures to criteria air pollutants, particularly from transport emissions, a

254 of asthma exacerbation in children; whether air pollutants play a role in the development of childho

255 climate change and reduce exposure to toxic air pollutants, policy assessments have considered few i

256 We monitor air pollutants pre- and post-renovation and quantify eff

257 Formaldehyde is an air pollutant present in both indoor and outdoor atmosph

258 From a mechanistic perspective, air pollutants probably cause oxidative injury to the ai

259 t a large potential public health benefit of air pollutant reduction in reduced incidence of childhoo

260 el to add criteria air pollutants, hazardous air pollutants, releases to water, releases to land, and

261 A large portion of air pollutants result from residential combustion of sol

262 entified as an important source of ultrafine air pollutants resulting in elaborated treatment systems

263 alibration method was proposed for low-level air pollutant sampling using this high capacity membrane

264 tact inhaled inflammatory insults, including air pollutants, smoke, and microorganisms.

265 d by a substantial increase of certain other air pollutants, specifically the number of ultrafine par

266 though long-term exposure to traffic-related air pollutants such as nitrogen dioxide has been linked

267 (e.g., changes in humidity or other similar air pollutants such as NO (x), SO(2), H(2)S, or acetylen

268 raffic, make a large contribution to harmful air pollutants such as ozone and fine particulate matter

269 otosynthesis, loss of water, and entrance of air pollutants such as ozone.

270 ontinuously for months and release hazardous air pollutants such as particulate matter and volatile o

271 With the potential of air pollutants such as particulate matter to not only mo

272 deling, and episodic exacerbations caused by air pollutants, such as particulate matter (PM; PM <2.5

273 ur heavy fuel oil are an important source of air pollutants, such as sulfur dioxide and particulate m

274 iatives that lead to lower concentrations of air pollutants, such as urban traffic restrictions.

275 Transportation Lden and air pollutants tendentially associated with decreased an

276 oad transport remains an important source of air pollutants that are linked with acute and chronic he

277 n 10,000 L of air that contains a variety of air pollutants that can pose negative consequences to lu

278 Diesel exhaust particles (DEPs) are major air pollutants that lead to numerous human disorders, es

279 ictors to resolve the spatial variability of air pollutants together with complementary national esti

280 This interaction controls thermal and air pollutant transport and distribution.

281 cific associations between breast cancer and air pollutants using Cox regression models, adjusting fo

282 litan regions, long-term exposure to ambient air pollutants was significantly associated with increas

283 nt method), and confounding by other gaseous air pollutants were also investigated.

284 od exposures to regional and traffic-related air pollutants were associated with increased consumptio

285 er exposures to regional and traffic-related air pollutants were associated with intake of a high-tra

286 Several ambient air pollutants were associated with preterm birth; assoc

287 Complete monitoring data for the ambient air pollutants were collected from Taiwan Environmental

288 ent for coexposure to noise, traffic-related air pollutants were not associated with the incidence of

289 Both particulate and gaseous air pollutants were positively associated with prevalent

290 In 2012 air pollutants were responsible of seven million human d

291 In conclusion, concentrations of all air pollutants were significantly elevated in passenger

292 Concentrations of UFP and other air pollutants were simultaneously monitored in and out

293 hweight z-scores and exposure to mixtures of air pollutants, where up to -0.21 or approximately a 96

294 Nitrogen dioxide is a common air pollutant with growing evidence of health impacts in

295 .5 mum in diameter (PM2.5) and other ambient air pollutants with adverse birth outcomes; yet, to our

296 exposure to house dust endotoxin and ambient air pollutants with asthma outcomes.Methods: We analyzed

297 We aimed to assess associations of air pollutants with prevalent and incident DSPN in a pop

298 PM2.5 mortality costs per tonne of inorganic air pollutants with the 36 km x 36 km spatial resolution

299 models, the effect estimates of all included air pollutants, with the exception of O(3), were signifi

300 Tropospheric ozone is a serious air-pollutant, with large impacts on plant function.