ライフサイエンスコーパス: air pollution

1 re to nPM, a nano-sized subfraction of urban air pollution.

2 tive function, especially in areas with high air pollution.

3 rs for early-onset ischaemic stroke, such as air pollution.

4 onounced effect on atmospheric chemistry and air pollution.

5 use of a tax on carbon, methane leakage, and air pollution.

6 n considering climate change, economics, and air pollution.

7 le approaches directed at particulate matter air pollution.

8 o investigated potential causal mediation by air pollution.

9 ive effect was 1.33 in nutrition and 1.16 in air pollution.

10 reproductive consequences of traffic-related air pollution.

11 nce indicating they are a novel component of air pollution.

12 levation, extreme weather events, and rising air pollution.

13 ower folate intake and/or higher exposure to air pollution.

14 ly tightened with the goal of reducing urban air pollution.

15 ty from exposure to higher levels of ambient air pollution.

16 the respiratory mucosal immune responses to air pollution.

17 ributor to fine particulate matter (PM(2.5)) air pollution.

18 xposure-response relationships for household air pollution.

19 ncreases are not mediated by the increase in air pollution.

20 burden associated with exposure to household air pollution.

21 ors: 1) the role of differential exposure to air pollution across populations as an explanation for s

22 Outdoor air pollution adversely affects human health and is esti

23 Here we quantify the exchange of air pollution among the contiguous United States, and as

24 mune markers mediate the association between air pollution and adult-onset asthma.

25 the immune system in the association between air pollution and adult-onset asthma.

26 pospheric ozone (O(3)) is a key component of air pollution and an important anthropogenic greenhouse

27 an Heart Association scientific statement on air pollution and cardiovascular disease in 2010, unequi

28 iation between long-term exposure to ambient air pollution and cardiovascular disease mortality risk.

29 discharge, leading to increased exposure to air pollution and chemical hazards.

30 Air pollution and climate change have a significant impa

31 the potential mechanisms between exposure to air pollution and COVID-19 and the opportunity to clearl

32 nd opportunities regarding the links between air pollution and COVID-19 are discussed with a focus on

33 ation between short-term exposure to ambient air pollution and daily hospital admissions for depressi

34 approach to assess the associations between air pollution and daily hospital admissions.

35 Emissions of aromatic compounds cause air pollution and detrimental health effects.

36 luate the prospective association between PM air pollution and developing depression assessed using t

37 fforts to characterize exposure to household air pollution and for other contexts.

38 recent years, threatening targets for local air pollution and global carbon emissions.

39 models did not yield any association between air pollution and immune markers.

40 Individuals are exposed to air pollution and ionizing radiation from natural source

41 two time points of the Swiss Cohort Study on Air Pollution and Lung and Heart Diseases in Adults (SAP

42 2,757, aged 39-67 years; and Swiss Study on Air Pollution and Lung and Heart Diseases in Adults: n =

43 tion between fine particulate matter (PM2.5) air pollution and lung cancer.

44 We collected hourly air pollution and meteorological data from fixed urban b

45 current understanding of combined effects of air pollution and meteorological variables on adverse bi

46 e associations between long-term exposure to air pollution and MI incidence, adjusting for road traff

47 ohesion, and reducing stress and exposure to air pollution and noise.

48 a form of pneumoconiosis commonly caused by air pollution and other environmental factors, is a new

49 ce, which could contribute to disparities in air pollution and other nuisance exposures.

50 un exposure, temperature, relative humidity, air pollution and photo-oxidation caused by ground level

51 ension and education), others (eg, household air pollution and poor diet) vary by a country's economi

52 gate potentially causal associations between air pollution and poor mental health.

53 son spread treated as natural experiments on air pollution and population health.

54 n models to explore the associations between air pollution and public transport strikes and between p

55 Substantial quantities of air pollution and related health impacts are ultimately

56 ed the association between long-term outdoor air pollution and rhinitis severity in adults.

57 Although the association between ambient air pollution and risk of depression has been investigat

58 ion between individual long-term exposure to air pollution and severity of rhinitis.

59 ment (UNGD) to adverse health has implicated air pollution and stress pathways.

60 "macro-level" environmental factors such as air pollution and the built environment.

61 our time, as, for example, the reduction of air pollution and the redistribution of scarce resources

62 ascular disease side effect profiles, indoor air pollution), and encourage the inclusion of relevant

63 characteristics, exposure to traffic-related air pollution, and airport-related noise.

64 dietary risks, high fasting plasma glucose, air pollution, and high LDL cholesterol).

65 the effects of indoor air pollution, outdoor air pollution, and subsequent climate change on asthma a

66 ular morbidity and mortality associated with air pollution are discussed.

67 whether pregnancy and childhood exposures to air pollution are related to white matter microstructure

68 s that are chronically exposed to unmodified air pollution as a whole have yet to be studied.

69 fuels results in up to 65% net reduction in air pollution as we approach 2050, even after accounting

70 and disease burden associated with household air pollution, as measured by disability-adjusted life-y

71 ealth impacts from tobacco smoke and ambient air pollution, as well as the hazards of certain occupat

72 of air pollutants operate, the causality of air pollution-associated shifts in birthweight is better

73 Air pollution at any given time is unequally distributed

74 mates of students' exposure to, and dose of, air pollution at population scales have to rely on simul

75 We sought to examine the association between air pollution, atopy, and eczema in adulthood.

76 on in rural areas with substantial levels of air pollution attributed to solid fuel burning for house

77 9) and postnatal (n = 128) exposures include air pollution, built environment, meteorology, natural s

78 ssociated health outcomes, and estimated the air pollution burden associated with the use of solid fu

79 n-generating sources bear a disproportionate air-pollution burden.

80 Air pollution by nitrogen oxides, NO(x), is a major prob

81 ucing dispersed coal consumption and related air pollution by promoting the use of clean and low-carb

82 Air pollution can induce oxidative stress.

83 Particulate matter (PM) air pollution causes cardiopulmonary mortality via macro

84 tify metabolites and classes associated with air pollution changes.

85 ally take long periods to properly match the air pollution characteristics from monitoring stations t

86 ve a stronger negative response under severe air pollution compared with Chinese observers.

87 maternal outcomes from exposure to household air pollution, compared with no exposure.

88 men), 5780 were assigned outdoor residential air pollution concentrations in the year of their baseli

89 is has reduced tropospheric and ground-level air pollution concentrations, using satellite data and a

90 Although air pollution contains a heterogeneous mixture of gases,

91 Exposure to PM2.5 air pollution contributes to lung cancer incidence and i

92 Air pollution contributes to type 2 diabetes and cardiov

93 DE), an established model of traffic-related air pollution, contributes significantly to the global b

94 s a function of both coal rank and installed air pollution control devices.

95 stimates of TE partitioning within installed air pollution control processes.

96 National Research Program for Key Issues in Air Pollution Control; and the National Natural Science

97 nia Children's Health Study over a period of air pollution decline.

98 Outdoor air pollution derived from traffic and other human activ

99 emerging epidemiological associations among air pollution, diabetes, and lung disease.

100 onmental exposures to ultraviolet radiation, air pollution, disruptions in the food and water supply,

101 ration during physical activity may increase air pollution dose, which may attenuate the benefits of

102 Rationale: Exposure to air pollution during intrauterine development and throug

103 Lower exposures to air pollution during pregnancy and childhood were associ

104 ater in life health effects from exposure to air pollution during the prenatal period have not been n

105 onal outcomes and find that mean exposure to air pollution during the prenatal stage is associated wi

106 These data suggest that adverse air pollution effects might be exacerbated in people rec

107 ve Programme on Assessment and Monitoring of Air Pollution Effects on Forests] Forests dataset), we c

108 rived from the European Study of Cohorts for Air Pollution Effects project, at the participants' resi

109 ndia for the year 2030 under two contrasting air pollution emission pathways for two different climat

110 Similar to that of air pollution, emission control is recognized as an effi

111 Air pollution epidemiology studies have primarily invest

112 Indian subcontinent is greatly vulnerable to air pollution, especially during the winter season.

113 large part of students' diurnal exposure to air pollution, especially in cities in developed countri

114 educe the adverse health impact of household air pollution, especially in LMICs.

115 ion during 2013-2017, short-term exposure to air pollution, especially PM2.5, was associated with inc

116 (2.5) concentrations frequently cause severe air pollution events in Delhi.

117 Analyses of the frequency distribution of air pollution events indicate that Fort McKay is regular

118 However, on typical air pollution events, fire emissions contributed as high

119 nd post-harvest fires-a precursor of extreme air pollution events, has been further demonstrated in p

120 tor compounds were used to isolate sustained air pollution "events".

121 nt tests, and replicated previously reported air pollution EWAS signals.

122 disease mortality associated with long-term air pollution exposure ( P-interaction<0.05).

123 at we were not able to study associations of air pollution exposure and infant mortality during the d

124 t all studies suggest an association between air pollution exposure and infant mortality.

125 However, the associations between air pollution exposure and the risk of noncommunicable d

126 studies, long-considered the "holy grail" of air pollution exposure assessment.

127 Our aim was to investigate associations of air pollution exposure at birth and at the time of later

128 Air pollution exposure does not seem to increase the ove

129 Both extreme heat and air pollution exposure during pregnancy have been associ

130 ims to quantify changes in outdoor (ambient) air pollution exposure from different migration patterns

131 Air pollution exposure has been linked to coronary heart

132 Rationale: Although elevated air pollution exposure impairs lung-function development

133 Increased prenatal household air pollution exposure is associated with impaired infan

134 e cardiometabolic and respiratory effects of air Pollution Exposure on healthy and prediabetic indivi

135 nmental agents, in the influence of prenatal air pollution exposure on neurodevelopment and behavior

136 lication of that statement, evidence linking air pollution exposure to cardiovascular health has cont

137 Household air pollution exposure was defined as use of polluting f

138 Prenatal household air pollution exposure was indexed by serial maternal ca

139 hens evidence for an association of smoking, air pollution exposure, and characteristics of the built

140 e RR meta-estimates, prevalence of household air pollution exposure, and disease-specific mortality a

141 e these interventions in the face of ongoing air pollution exposure.

142 y and country to country as a function of PM air pollution exposure.

143 Air pollution exposures are novel contributors to the gr

144 nction at ages 8 and 15 years in relation to air pollution exposures during pregnancy, infancy, and c

145 We previously assessed indoor and personal air pollution exposures in this area; however, the influ

146 Our results indicate that air pollution exposures may contribute to obesogenic beh

147 ith source-specific transportation noise and air pollution exposures, with distinct and shared enrich

148 Bs, independent of noise and traffic-related air pollution exposures.

149 s), and the majority had multiple sources of air pollution exposures.

150 luded the unavailability of data on personal air pollution exposures.

151 independent EWAS on transportation noise and air pollution exposures.

152 mental health, and reductions in exposure to air pollution, extreme temperatures, and noise.

153 ntal forces including an altered microbiome, air pollution, food allergens in a changed diet, and che

154 goal was to evaluate fine particulate matter air pollution ([Formula: see text]) and mortality using

155 Air pollution from fossil fuel burning and traffic-relat

156 Air pollution from indoor burning of mosquito repellants

157 Associations between exposure to household air pollution from solid fuel stoves and telomere length

158 Exposure to air pollution from solid-fuel cookstoves is a leading ri

159 hs occur annually from exposure to household air pollution from the use of biomass cooking fuels.

160 educe the greenhouse gas emissions and local air pollution from transportation.

161 om 2000, mortality associated with household air pollution had reduced by 36% (95% CI 29-43) and dise

162 the realm of environmental health: household air pollution (HAP) and poor water, sanitation, and hygi

163 Unsafe drinking water and household air pollution (HAP) are major causes of morbidity and mo

164 urations, which were used to model household air pollution (HAP) in terms of estimated particulate ma

165 The resulting household air pollution (HAP) is a leading environmental risk fact

166 suite and variety of biomarkers in household air pollution (HAP) studies in concert with exposure and

167 Outdoor air pollution has also been viewed mostly as an urban pr

168 Prenatal exposure to air pollution has been associated with childhood respira

169 Traffic-related air pollution has been linked to higher risks of inferti

170 Air pollution has been related to brain structural alter

171 maternal diseases associated with household air pollution has declined worldwide but remains high in

172 bustion and household ventilation on outdoor air pollution has not been assessed.

173 Efforts to mitigate air pollution have focused mainly on the relationship be

174 Policies to address air pollution have reduced criteria pollutant emissions,

175 household stove improvement to reduce indoor air pollution have resulted in higher outdoor air pollut

176 y, exposure to the world's highest levels of air pollution, highly prevalent pretransition forms of H

177 These results show that outdoor air pollution in a rural region of China was associated

178 Severe events of wintertime particulate air pollution in Beijing (winter haze) are associated wi

179 Air pollution in Beijing has been improving through impl

180 Reduced exposure to air pollution in childhood might help to preserve TL.

181 High levels of air pollution in China may contribute to the urban popul

182 aking aimed at protecting public health from air pollution in China.

183 nding ports are a major contributor to urban air pollution in coastal and inland riverside cities.

184 mes are a major source of exposure to indoor air pollution in developing countries.

185 Cardiopulmonary Impact of Particulate Matter Air Pollution in High Risk Populations, held on May 29 t

186 are limited epidemiological data on outdoor air pollution in rural areas with substantial levels of

187 disease, may be associated with exposure to air pollution in settings where household biomass stoves

188 Increasing air pollution in South Asia has serious consequences for

189 Urban outdoor air pollution in the developing world, mostly due to par

190 urce characterization and attribution of the air pollution in the Indigenous community of Fort McKay,

191 on model to identify long-range transport of air pollution in the nocturnal residual layer.

192 pe contribute to a diversity of responses to air pollution in the population at the individual and gr

193 cluding India and Nepal, suffers from severe air pollution, including high concentrations of aerosols

194 reastfeeding, crowding, malnutrition, indoor air pollution, incomplete immunisation, and paediatric H

195 ng factors are often investigated to explain air pollution-induced cardiovascular dysfunction, yet br

196 nd bioactive circulating milieu may underlie air pollution-induced cardiovascular dysfunction.

197 identified particulate and nitrogen dioxide air pollution inside the home, urine cotinine levels ind

198 The Household Air Pollution Intervention Network (HAPIN) trial is a li

199 e exposure data collection for the Household Air Pollution Intervention Network (HAPIN), a multicount

200 of asthma incidence under each hypothetical air pollution intervention with incidence under the obse

201 mate the potential public health benefits of air pollution interventions in exposed populations.Objec

202 Air pollution involving particulate matter smaller than

203 Outdoor air pollution is a growing public health concern, partic

204 Ambient air pollution is a known risk factor for adverse birth o

205 Exposure to ambient particulate matter (PM) air pollution is a leading risk factor for morbidity and

206 ong-term exposure to particulate matter (PM) air pollution is associated with all-cause mortality and

207 suggest that short-term exposure to ambient air pollution is associated with an increased risk of da

208 Air pollution is associated with detrimental effects on

209 ns (THC) and volatile organic compounds from air pollution is associated with risk of coronary heart

210 ially in cities in developed countries where air pollution is caused predominantly by vehicle traffic

211 glected sources contributing to the regional air pollution is garbage burning.

212 We show that outdoor air pollution is just as severe in nonurban regions as i

213 are modifiable factors, whereas exposure to air pollution is less so.

214 The World Health Organization estimates that air pollution is responsible for 7 million deaths per an

215 These results show that air pollution is significantly associated with increased

216 The neurotoxicity of air pollution is undefined for sex and APOE alleles.

217 Air pollution is well recognized as a major risk factor

218 ile such knowledge is crucial to address the air pollution issue more effectively.

219 ees, Apis dorsata, at locations with varying air pollution levels in Bangalore, India.

220 ir pollution have resulted in higher outdoor air pollution levels.

221 Fine particulate air pollution <2.5 mum in diameter (PM(2.5)) is a major

222 A few studies suggest that air pollution may decrease fertility, but prospective st

223 Obesity and air pollution may have synergistic effects on the develo

224 posures, including urban and traffic-related air pollution, may shorten telomeres.

225 Mobile air pollution measurements can provide information on fi

226 We also explored whether air pollution mediated those associations.

227 For air pollution, minimal data exist on the exposure-risk r

228 eveal the sectors that should be targeted by air pollution mitigation policy.

229 nding potential health impacts, prioritizing air pollution mitigation strategies, and enabling global

230 hina enabled assessment of the efficiency of air pollution mitigation.

231 eralizability and public health relevance of air pollution models and the accuracy of personal monito

232 o nPM, suggesting a critical role of NRF2 in air pollution neurotoxicity.

233 -2016 on strikes, health events, and ambient air pollution (nitrogen dioxide, nitrogen monoxide, part

234 Vs could increase some health risks (such as air pollution, noise, and sedentarism); however, if prop

235 Evidence regarding the impact of air pollution on acute respiratory distress syndrome (AR

236 The chemical influence of air pollution on aeolian dust contributes to the aerosol

237 empirical evidence supporting the effects of air pollution on cardiovascular health are examined, pot

238 The impacts of air pollution on circulatory and respiratory systems hav

239 partnerships to reduce the adverse impact of air pollution on current and future cardiovascular healt

240 ort-term exposure to particulate matter (PM) air pollution on human cognitive performance via a doubl

241 While the impact of air pollution on human health is well studied, mechanist

242 ance of research to understand the impact of air pollution on human health, commenting on a study by

243 Although the physical effects of air pollution on humans are well documented, there may b

244 ntitative analysis on the current impacts of air pollution on insects, and indicates the urgency for

245 The effects of prenatal household air pollution on lung development are unknown.

246 alth is well studied, mechanistic impacts of air pollution on wild systems, including those providing

247 en DMPs were associated with traffic-related air pollution or secondhand smoke.

248 ublic transport strikes and either increased air pollution or worse population health.

249 This review summarizes the effects of indoor air pollution, outdoor air pollution, and subsequent cli

250 ntal cobenefits with regard to human health, air pollution, ozone depletion, acidification, and land

251 the damaging effects of a known component of air pollution (particulate matter) on human primary derm

252 topics included climate and weather change, air pollution, particulates, greenhouse gasses, traffic,

253 th India and explored the mediating roles of air pollution, physical activity, and stress in these as

254 the oxidative potential of fine particulate air pollution (PM(2.5)).

255 f the causal role of fine particulate matter air pollution (PM(2.5), or particulate matter <=2.5 mum

256 e aimed to evaluate the relationship between air pollution, PM components, and breast cancer risk in

257 apportionment is important for a successful air pollution policy.

258 In MICs and LICs, household air pollution, poor diet, low education, and low grip st

259 been improving through implementation of the Air Pollution Prevention and Control Action Plan (2013-1

260 Women's daily exposures to air pollution ranged from [Formula: see text] for [Formu

261 ts of several realistic scenarios for future air pollution reductions on lung growth.Methods: Mixed-e

262 We show that an air pollution regulation limiting ozone precursors emiss

263 that in addition to protecting human health, air pollution regulations have previously unrecognized a

264 an-urban migrant groups have higher rates of air pollution-related deaths.

265 mal for large-scale biomonitoring studies of air pollution relevant to global health as well as occup

266 Most air pollution research has focused on assessing the urba

267 low, and increased after the Olympics, when air pollution returned to normal (high) levels.

268 tracts can quantitatively characterize urban air pollution source patterns and are applicable to deve

269 Diverse urban air pollution sources contribute to spatially variable a

270 We provide large-scale evidence that air pollution, specifically ozone, is associated with de

271 ies categorized the exposure, whereas 89% of air pollution studies analyzed the exposure as a continu

272 We surveyed nutritional and air pollution studies that found statistically significa

273 ta from the Cincinnati Childhood Allergy and Air Pollution Study (n = 762) birth cohort were used to

274 b the staggering health burden attributed to air pollution, the sustainable solution for India would

275 Community efforts can significantly reduce air pollution, thereby reducing greenhouse gas emission

276 Indoor air pollution threats anticipated from climate changes i

277 the growing epidemiological evidence linking air pollution to cardiovascular dysfunction in humans.

278 ive a rationale for epidemiologic studies of air pollution to consider sex interactions with APOE all

279 usceptible to the effects of traffic-related air pollution (TRAP) exposure.

280 Traffic-related air pollution (TRAP) is made up of complex mixtures of p

281 roadways expose students to traffic-related air pollution (TRAP).

282 Within the Brain Development and Air Pollution Ultrafine Particles in School Children (BR

283 )Po levels and exposure parameters for urban air pollution using linear regression models adjusted fo

284 , we characterize the impacts of COVID-19 on air pollution using NO(2) and Aerosol Optical Depth (AOD

285 Household air pollution was associated with 1.8 million (95% CI 1.

286 Ambient air pollution was associated with 13.9% of the PAF for c

287 d whether early-life exposure to residential air pollution was associated with leukocyte telomere len

288 Exposure to air pollution was associated with shorter TLs in nonasth

289 Air pollution was associated with stroke and AF onset, e

290 The findings suggested that air pollution was causally associated with mortality, ev

291 eased during the 2008 Beijing Olympics, when air pollution was low, and increased after the Olympics,

292 Air pollution was not consistently associated with IgE s

293 Household air pollution was positively associated with asthma (RR

294 Associations with air pollution were stronger in carriers of fewer risk al

295 Considering environmental concerns regarding air pollution which is induced by burning fossil fuels t

296 There is an emerging link between air pollution, which is ubiquitous in our environment, p

297 A causal association of air pollution with mental diseases is an intriguing poss

298 CI: 93.2, 179.4) premature deaths related to air pollution, with rural-urban producing the highest ri

299 -populated or that had more crowded housing, air pollution, women, 20-49-year-olds, racial/ethnic min

300 GVs) have been promoted in China to mitigate air pollution, yet our measurements and analyses show th