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1 n whole blood and carrageenan-challenged rat air pouch.
2 licited by casein injection into a preformed air pouch.
3 oduction or neutrophil infiltration into the air pouch.
4 on, such as neutrophil infiltration into the air pouch.
5 NTES-induced recruitment of leukocytes in an air pouch.
6 ex-mediated inflammation in the subcutaneous air pouch.
7 nduced leukocyte recruitment into the dorsal air pouch.
8 ed PGs in the lipopolysaccharide-induced rat air pouch.
9 lated in the inflamed (carrageenan, 2 mg/ml) air pouch.
10 flammation were investigated in mouse dorsal air pouches.
11 jected into mouse synovial-like subcutaneous air pouches.
12 ageenan-induced inflammation in footpads and air pouches.
13 jected into mouse synovium-like subcutaneous air pouches.
14 umulation of naive T cells into sterile s.c. air pouches.
15 nules, but failed to induce extravasation in air pouches.
16  resulted in the recruitment of DCs into the air pouches.
17 cells and recruits T cells into subcutaneous air pouches.
18 eir migration into chemokine-challenged s.c. air pouches.
19 cause monocyte accumulation in vivo in mouse air pouches.
20 ngivalis was introduced within murine dorsal air pouches.
21                                    In murine air pouches, 15R/S-methyl-LXA4 dramatically inhibited TN
22 orphonuclear leukocyte recruitment to dorsal air pouch (4 microg/kg).
23  the E species (PGE(2)) and cytokines in the air pouch and elevated levels of these inflammatory medi
24 oreover, total leukocyte infiltration in the air pouch and IL-1beta production were attenuated in Cry
25 tration in two acute models of inflammation (air pouch and LPS challenge).
26 3-dependent rodent models of inflammation in air pouch and peritoneum.
27 splayed oral efficacy in the rat carrageenan air pouch and rat carrageenan-induced paw edema models.
28 In two mouse models of inflammation, zymosan air pouch and thioglycolate-induced peritonitis models,
29    Mononuclear cell recruitment in vivo into air pouches and injured muscles depends on the heterocom
30 rker gene (rAAV-LacZ) were injected into the air pouches, and the tissue was harvested 7 days after v
31 s and ex vivo using mice with a subcutaneous air pouch as a model for inflammation.
32 The number of leukocytes recruited to murine air pouches by the mutant sequence was lower than that r
33 selectively in myeloid cells infiltrating an air pouch cavity upon injection of carrageenan failed be
34                                       In the air pouch cavity, P. gingivalis elicited leukocyte infil
35 ochemical and packaging design of a micro-Al-air pouch cell battery is optimized for two operating po
36 Inhibition was evident by either local intra-air pouch delivery (approximately 77% inhibition) or sys
37 a mixture of a mutant and wt CCL7 within the air pouch elicited no significant cell accumulation.
38  Peripheral adenosine levels, as measured in air pouch exudates, decreased markedly in inflamed pouch
39 e normal recruitment of leukocytes to murine air pouches filled with unmodified CCL2.
40 ited the recruitment of leukocytes to murine air pouches filled with wild-type CXCL12.
41 umor necrosis factor alpha into subcutaneous air pouches formed on the backs of mice.
42 accharide (LPS) into a sterile, subcutaneous air pouch (i.po.) in rats evoked an increase in body tem
43 hese toxins are injected into a subcutaneous air pouch (I.PO.) in rats.
44 e (LPS; 100 microg kg-1) into a subcutaneous air pouch (I.PO.) of rats induced a significant increase
45 esses acute inflammation in the subcutaneous air pouch in mice and chronic joint inflammation charact
46           Leukocyte influx in a subcutaneous air pouch in response to TNF-alpha was reduced by 67% an
47 phages and colon cancer cells and in the rat air pouch in vivo.
48 ate crystals were injected into subcutaneous air pouches in mice that expressed or lacked the murine
49 ta decreased by >80% in CD14(-/-) mouse s.c. air pouches in vivo.
50 did not restore leukocyte recruitment to the air pouch, indicating a role for endothelial CD47.
51  and development of full-blown neutrophilic, air pouch inflammation in response to MSU crystals.
52 lipopolysaccharide (LPS) into a subcutaneous air pouch (intrapouch, i.p.o.) that does not lead to LPS
53 ls was increased in response to CXCL1 in the air pouch model and in the excisional wound beds of beta
54                Two animal models, the dorsal air pouch model and the excisional wound healing model,
55 s hypothesis was tested in the synovial-like air pouch model injected with carrageenan using arthriti
56 ent anti-inflammatory activity in the murine air pouch model of carrageenan- or IL-1-induced acute in
57      DFV890 ((R)-1) was also effective in an air pouch model of gout.
58 ckout mice were subjected to the carrageenan-air pouch model of inflammation and then treated with D-
59 s from M(r) 5,000 to 1,500,000 in the rodent air pouch model of inflammation to determine their poten
60 methotrexate and sulfasalazine in the murine air pouch model of inflammation, and injection of APCP,
61                       In the rat carrageenan air pouch model of inflammation, CAPE (10-100 mg/kg) cau
62                                        In an air pouch model of inflammation, CCL20 triggered recruit
63     Compound 20 was orally active in the rat air pouch model of inflammation, inhibiting white blood
64 ays) on leukocyte accumulation in the murine air pouch model of inflammation.
65 bition of prostaglandin E2 production in the air pouch model of inflammation.
66 over 2 without loss of efficacy in the mouse air pouch model of inflammation.
67 ell as neutrophils and monocytes in a dermal air pouch model of TNF-alpha-induced inflammation.
68                             The mouse dorsal air pouch model was used to evaluate the in vivo impact
69 s this question, we employed a murine dorsal air pouch model with wild-type and CB(2)(-/-) 8-12-wk-ol
70  model of internal trauma and repair (murine air pouch model), endogenously produced adenosine releas
71 abolites have inflammatory properties in the air pouch model, and rHuPH20 can instead inhibit some as
72                Finally, with the carrageenan air pouch model, both vagus nerve stimulation and cholin
73                Utilizing the chimeric murine air pouch model, MK2 signaling differentially regulated
74 n response to zymosan injection in the mouse air pouch model.
75 lammation using intravital microscopy and an air pouch model.
76 in response to delivery of CXCL1 through the air pouch model.
77 release of AnxA1 and CRAMP in a subcutaneous air pouch model.
78 AM17 during local inflammation in the murine air pouch model.
79 titanium particle-induced inflammation in an air pouch model.
80 sponse to urate crystals in the subcutaneous air pouch model.
81 ammatory activity in the carrageenan-induced air-pouch model and less gastric toxicity than a standar
82 ied acute inflammation in the generic murine air-pouch model by using wild-type mice and mice rendere
83  vehicle control on acute inflammation in an air-pouch model in A(2A) and A(3) receptor knockout mice
84 nses of 4 inbred mouse strains to MTX in the air-pouch model of acute inflammation.
85 a strong anti-inflammatory effect in the rat air-pouch model of inflammation in vivo.
86 ession in RAW264.7 cells and also in a mouse air-pouch model of inflammation.
87 esponse to tumor necrosis factor-alpha in an air-pouch model of leukocyte migration.
88  HA-induced neutrophilic inflammation in the air-pouch model of synovitis, and they show decreased jo
89                            A humanized mouse air-pouch model showed that intravenous treatment with t
90                           First, in a murine air-pouch model, apoptotic cell supernatants induced a t
91                                        In an air-pouch model, CS-induced neutrophil recruitment is de
92                               Using a dorsal air-pouch model, passive transfer of apoptotic neutrophi
93  facsimile synovium in vivo using the murine air-pouch model.
94 effective antiinflammatory agents in the rat air-pouch model.
95                    Using LPS and carrageenan air pouch models in mice, we found that physiological co
96                      When injected in murine air pouches, nociceptin elicited leukocyte infiltration
97 ion in blood vessels in the walls of healing air pouches of A(2A) receptor knockout mice compared to
98     Furthermore, injection of mEAR2 into the air pouches of mice resulted in the recruitment of DCs i
99 e leukocyte recruitment when added to murine air pouches (p < 0.05).
100       Local application of GDNF in the mouse air pouch reduced SP-induced leukocyte migration.
101                Injection of CRAMP into mouse air pouches resulted in the recruitment predominantly of
102                               In vivo dorsal air pouch studies revealed that RvE1 decreases neutrophi
103 ours after injection of carrageenan into the air pouch using previously described methods.
104 recruitment of monocyte/macrophages into the air-pouch wall, but not in the lumen.
105      In vivo, Th17 cell recruitment into the air pouch was reduced in CD43(-/-) mice in response to C
106 lites detected in carrageenan-challenged rat air pouch were PGE2, 6-keto PGF1 alpha, and TxB2.
107 e and leukocyte accumulation in the inflamed air pouch were similarly reduced in the A(2A) receptor k
108                                              Air pouches were raised by 2 injections of sterile air,
109  local induction of IL-1beta in subcutaneous air pouches were suppressed 6 hours after injection of M
110 lso induced neutrophil infiltration into the air pouch, which was not observed with rHuPH20 treatment
111 neutrophil infiltration into skin or into an air pouch with an ED50 of 0.2 mg/kg.
112                              Challenging the air pouch with LPS or BTH stimulated production of cytok
113  All 3 compounds decreased LTB4 levels in be air pouch, with Bay y 1015 being the most effective.

 
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