ライフサイエンスコーパス: alachlor

1 ighted exposure-days (p for trend = 0.03) to alachlor.

2 Incubations of alachlor (0-1000 microM) with human albumin and bovine s

3 xhibited a high catalytic activity to reduce alachlor, a chlorinated herbicide, in the aqueous medium

4 Maximal adduction levels of 613-1130 pmol alachlor-albumin adducts/mg protein were observed, with

5 uate the exposure-response relations between alachlor and cancer incidence controlled for the effects

6 More importantly, both alachlor and PCA could be effectively mineralized in thi

7 to identify the degradation intermediates of alachlor and then proposed a possible alachlor degradati

8 ordane, heptachlor, dichlorvos, trichlorfon, alachlor, and cyanazine) for which the odds of diabetes

9 ee chloroacetanilide herbicides (acetochlor, alachlor, and metolachlor) and one chloroacetamide herbi

10 he herbicides S-ethyl dipropylthiocarbamate, alachlor, and metolachlor.

11 dings suggest a possible association between alachlor application and incidence of lymphohematopoieti

12 emoglobin (Hb)- and serum-protein adducts of alachlor as potential biomarkers of alachlor exposure, a

13 Male CD rats treated with alachlor at 150 mg/kg body wt/day ip for 0, 1, 2, and 3

14 acid, 3-phenoxybenozic acid, 4-nitrophenol, alachlor, atrazine, azoxystrobin, chlorpyrifos, diazinon

15 sis of a herbicide test mixture (composed of alachlor, atrazine, butachlor, hexachlorocyclopentadiene

16 The herbicides, atrazine and alachlor, but not 2,4-D, were associated with wheeze.

17 In the assay, treatment of alachlor-cysteinyl protein adducts by methanesulfonic ac

18 uic acid (PCA) can significantly promote the alachlor degradation in the Fe(III)/H2O2 Fenton oxidatio

19 tes of alachlor and then proposed a possible alachlor degradation mechanism in this novel Fenton oxid

20 on of protocatechuic acid could increase the alachlor degradation rate by 10000 times in this Fenton

21 This dramatic enhancement of alachlor degradation was attributed to the complexing an

22 Among alachlor-exposed applicators, the authors found a signif

23 e increased among applicators in the highest alachlor exposure category.

24 ducts of alachlor as potential biomarkers of alachlor exposure, a genotoxic and carcinogenic herbicid

25 A linear concentration response of alachlor-Hb adducts was observed when whole blood from f

26 A maximal binding of 2250 pmol alachlor-Hb adducts/mg globin was observed.

27 Maximal binding was 1860 pmol alachlor-Hb adducts/mg globin.

28 among pesticide applicators with exposure to alachlor in the Agricultural Health Study, a prospective

29 blood from female CD rats was incubated with alachlor in vitro at concentrations up to 300 microM.

30 ay provides a new approach for biomonitoring alachlor levels in experimental animals and has the pote

31 cides (DDT and lindane) and four herbicides (alachlor, metolachlor, 2,4-D, and pendimethalin).

32 [ring-13C6]Alachlor-N-acetylcysteine was added as an internal stand

33 is; 26,510 applicators (53%) reported use of alachlor on the enrollment questionnaire.

34 increased lifetime days of pesticide use for alachlor (p = 0.002), 2,4-dichlorophenoxyacetic acid (2,

35 the observation that cleavage of S-cysteinyl alachlor-protein adducts by methanesulfonic acid gave th

36 rbofuran (1.41;0.99-2.01)], four herbicides [alachlor (RR = 1.40;95%CI 0.99-1.98), metolachlor (1.57;

37 lide herbicides metolachlor, acetochlor, and alachlor, the chloroacetamide herbicide dimethenamid, an

38 Alachlor was reduced in deschloroalachlor, its dechlorin