ライフサイエンスコーパス: alcohol

1 n with 4-methoxybenzyl alcohol (4-MBA, anise alcohol).

2 y metabolites (terpenes, phenolic compounds, alcohol).

3 (Au, Pt, and SiO(2)) and fuels (H(2)O(2) and alcohols).

4 onist, PF-5190457, when co-administered with alcohol.

5 and with greater access to takeaway food and alcohol.

6 tocytes in fueling the cGAS-IRF3 response to alcohol.

7 advanced polycyclic intermediate to form an alcohol.

8 utein and zeaxanthin, magnesium, copper, and alcohol.

9 is derived from polymerization of coniferyl alcohol.

10 nts, and assigning a new proton signal as an alcohol.

11 and reduced conditioned place preference for alcohol.

12 replacement therapy, and use of tobacco and alcohol.

13 e-membered and six-membered cyclic secondary alcohols.

14 ha-branched aldehydes with different allylic alcohols.

15 ucts to generate the corresponding diterpene alcohols.

16 not induced to an appreciable level by other alcohols.

17 reacting with CO(2) to produce aldehydes and alcohols.

18 native coupling, starting from 2-aminobenzyl alcohol/1-phenylethanol and diamine/diol, respectively,

19 re 3-methyl-1-butanol and eugenol, phenethyl alcohol, 2-phenethyl acetate, acetic, isobutyric, isoval

20 ) by its esterification with 4-methoxybenzyl alcohol (4-MBA, anise alcohol).

21 ysfunction was associated with fewer days of alcohol abstinence and also predicted greater number hea

22 daptations are affected by number of days of alcohol abstinence and whether they influence heavy drin

23 tracer [(18)F]FPEB during early and extended alcohol abstinence.

24 Alcohol abuse and alcohol dependence are key factors in

25 , metabolic and genetic diseases, as well as alcohol abuse can also influence both hepatic and cardio

26 e to increased susceptibility to anxiety and alcohol abuse in men.

27 nerally associated with tobacco consumption, alcohol abuse or both, whereas pharynx cancers are incre

28 similarly to people with addictions such as alcohol abuse or smoking(6,7).

29 ods identified treatment barriers including, alcohol abuse, family conflicts, and mental health issue

30 abetes mellitus; 2.22 (95% CI 1.58-3.11) for alcohol abuse; 2.37 (95% CI 1.53-3.68) for liver disease

31 we identify disulfiram, a drug for treating alcohol addiction, as an inhibitor of pore formation by

32 ped tolerance to the sedation after repeated alcohol administrations.

33 , INSIG2, LPL, MYB, NXPH1, PER2, TNFA), gene-alcohol (ALDH2, APOA5, APOC3, CETP, LPL), gene-smoking (

34 ibited a broad reaction scope with different alcohols, allowing primary and secondary alcohols to be

35 Furfuryl alcohol and 2-furoic acids were measured in a selection

36 he sp(3) C-H alkylation of 9H-fluorene using alcohol and a Ru catalyst via the borrowing hydrogen con

37 were less likely to be listed due to active alcohol and opioid use.

38 Alcohol and other addictive substances can remold neural

39 f the 18 policies except for those targeting alcohol and physical activity.

40 survey, the National Epidemiologic Survey on Alcohol and Related Conditions (NESARC), we used structu

41 rbed acetaldehyde and ethanol to form crotyl alcohol and water.

42 ondary alcohols including cyclic and acyclic alcohols and a wide variety of arylacetonitriles bearing

43 re class dependent with best performance for alcohols and acetates, moderate performance for ketones,

44 s and 1H-xanthen-1-ones from o-hydroxybenzyl alcohols and beta-dicarbonyl compounds is provided.

45 es was mainly observed in primary long chain alcohols and, to a minor extent, in long-chain wax ester

46 of birth, father's occupation, ever-consumed alcohol, and asset index.

47 psychiatric vulnerability (e.g., marijuana, alcohol, and caffeine misuse, perceived stress, and impu

48 y drugs, protonic pump inhibitors, exercise, alcohol, and fasting) were considered.

49 ocosahexaenoic acid, omega-3 fatty acid, and alcohol-and 3 nutrients were associated with increased r

50 of propulsion under ambient conditions with alcohols as fuels.

51 l practice, particularly among patients with alcohol-associated or NASH-related cirrhosis and those n

52 dinating carbonyl-directed coupling of allyl alcohols at the C-4 position of indole derivatives under

53 pendent plasma-supported oxidation of benzyl alcohol, benzaldehyde and phenol in the presence of mole

54 nitroaniline derivatives, in the presence of alcohols, benzimidazoles can be obtained in 64-72% yield

55 a range of differentially substituted benzyl alcohols, benzylamines and aniline and can also be perfo

56 mined DNA methylation predictors of smoking, alcohol, body mass index, serum proteins, and cell propo

57 sDREADD in DMS dMSNs but not iMSNs increases alcohol but not saccharin intake.

58 n dMSNs, but not in iMSNs, reduces excessive alcohol but not saccharin intake.

59 iators for the electrocatalytic oxidation of alcohols by an iridium amido dihyride complex (PNP)Ir(H)

60 almost all methods for the incorporation of alcohols by C-H oxidation require the use of the alcohol

61 Using PhFCl/AgNO(3), a primary alcohol can be protected in good yield in the presence o

62 -directed remote C-H activation reactions of alcohols, carboxylic acids, sulfonates, phosphonates, an

63 t the isomerization of epoxides into allylic alcohols catalyzed by supported Au nanoparticles proceed

64 y enhance the selectivity to the unsaturated alcohols compared to monometallic catalysts.

65 hols by C-H oxidation require the use of the alcohol component as a solvent or co-solvent.

66 Determination of ethyl alcohol concentration was performed in 167 samples of al

67 ternational normalized ratio, elderly, drugs/alcohol concomitantly) score was 3.0 +/- 1.1.

68 r the simple 2-phenoxy-1-phenylethane or its alcohol congener, 2-phenoxy-1-phenylethanol, the benzyli

69 Addiction to drugs and alcohol constitutes one of the significant factors under

70 Although both arms reduced average grams of alcohol consumed per week from baseline to 6 and 12 mont

71 he patient should be asked about the current alcohol consumption (i.e. active vs. abstinence, determi

72 inactivity (p < 0.0005 for both genders) and alcohol consumption (p < 0.0005 for men).

73 of education (P = 0.26), smoking (P = 0.11), alcohol consumption (P = 0.52), history of cardiovascula

74 ts enrolled between 2006 and 2010, we tested alcohol consumption (stratified by the Centers of Diseas

75 of a causal relationship between smoking and alcohol consumption and 19 site-specific cancers.

76 TSPAN5 SNPs were also associated with alcohol consumption and alcohol use disorder (AUD) risk.

77 population is low, with a poor knowledge of alcohol consumption and dietary guidelines.

78 in the basolateral amygdala (BLA) decreased alcohol consumption and reduced conditioned place prefer

79 an important role of NBCn1 in regulation of alcohol consumption and sensitivity to alcohol-induced s

80 Alcohol consumption and smoking, 2 major risk factors fo

81 for the difference in the effect of moderate alcohol consumption between alleles).

82 Furthermore, chronic alcohol consumption caused NBCn1 downregulation in the h

83 mechanisms that prompt or mitigate excessive alcohol consumption could be partly explained by metabol

84 f-report severe periodontitis was noted when alcohol consumption exceeding > 20 g/day for women and >

85 The causal role of alcohol consumption for other cardiovascular diseases re

86 th psychiatric disorders, whereas aspects of alcohol consumption have shown stronger links to metabol

87 Moderate alcohol consumption is associated with reduced severity

88 It is well known that alcohol consumption is associated with type 2 diabetes m

89 e association study-defined genomic risk for alcohol consumption is enriched for genes that are prefe

90 Excessive alcohol consumption leads to overproduction of urates an

91 demonstrate that the genetic architecture of alcohol consumption only partially overlaps with the gen

92 including 23 CpGs previously associated with alcohol consumption or alcohol use disorder.

93 Alcohol consumption showed a dose-dependent risk increas

94 Genetically predicted alcohol consumption was consistently associated with str

95 haviour) and an independent causal effect of alcohol consumption when controlling for smoking (IVW OR

96 nce of associations of genetically predicted alcohol consumption with heart failure (OR, 1.00 [95% CI

97 smoking, body mass index, physical activity, alcohol consumption, Alternative Healthy Eating Index, a

98 67:TA was amplified by increasing adiposity, alcohol consumption, and genetic risk of fatty liver dis

99 family history of dementia, non-smoking, low alcohol consumption, depression, daytime somnolence, epi

100 s included in the models included; age, sex, alcohol consumption, IBS diagnosis, family history of ga

101 In comparison, after adjusting for alcohol consumption, smoking retained its association wi

102 ialty, waist circumference, body mass index, alcohol consumption, tobacco smoking, and physical activ

103 wn of Lmo4 in the nucleus accumbens enhanced alcohol consumption, whereas knockdown in the basolatera

104 abstain from alcohol or continue their usual alcohol consumption.

105 isms were used as instrumental variables for alcohol consumption.

106 isms underlying the development of excessive alcohol consumption.

107 regulatory and tolerance-inducing effect of alcohol consumption.

108 sed on histologic analysis, compared with no alcohol consumption.

109 Parameters such as acetic acid and alcohol content (higher in control wines), and anthocyan

110 ain stable for tenths of seconds only if the alcohol content is close to 50%.

111 ly differed in terms of terpenes, esters and alcohols content, thus proving that the yeast-inoculatin

112 Ethyl alcohol contributes to its strong and pungent smell and

113 ymptoms and associated secondary outcomes of alcohol craving and mood, anxiety, and sleep disturbance

114 gloeosporioides) incidence, without inducing alcohol dehydrogenase activity, which suggests that coat

115 alphaA-crystallin or alphaB-crystallin with alcohol dehydrogenase or citrate synthase by applying th

116 : oxidation of retinol into retinaldehyde by alcohol dehydrogenases (ADHs) or retinol dehydrogenases

117 nes including two alkane monooxygenases, two alcohol dehydrogenases, two aldehyde dehydrogenases, a f

118 Alcohol abuse and alcohol dependence are key factors in the development of

119 ed the role of microglia in a mouse model of alcohol dependence using a colony stimulating factor 1 r

120 study, using a well-established rat model of alcohol dependence, ex vivo electrophysiology and ISH, p

121 These regions are commonly affected in alcohol-dependent adults, raising the possibility that c

122 ion: Reduced cerebral mGluR5 availability in alcohol-dependent patients recovers during abstinence an

123 Results: During abstinence, alcohol-dependent patients showed sustained recovered mG

124 nds via catalytic beta-scission of aliphatic alcohol derivatives with both aryl and alkyl halides is

125 trand crosslinks caused by an endogenous and alcohol-derived metabolite, and identify an excision-ind

126 Alcohol diagnosis, diabetes, hypertension, sleep apnea,

127 ter of behavioural factors (ie, tobacco use, alcohol, diet, physical activity, and sodium intake), me

128 The process of diagnosing hazardous alcohol drinking (HAD) is based on self-reported data an

129 n 2.17 (1.70-2.76), those reporting moderate alcohol drinking 1.76 (1.21-2.57), and those with increa

130 y the selective agonist pioglitazone reduces alcohol drinking and alcohol-seeking behavior in rats.

131 estradiol, E2) are associated with increased alcohol drinking by women and experimentally in rodents.

132 tor blockers have been administered to heavy alcohol drinking individuals.

133 ring protracted withdrawal from intermittent alcohol drinking resulted in enhanced prefrontal cortex

134 negative responses to stress following heavy alcohol drinking.

135 By identifying alcohol effects on brain systems involved in performance

136 ped from easily available benzylic secondary alcohols employing thiophenol-stabilized iodonium ion as

137 al but different minor QTLs in the 0 and 14% alcohol environments.

138 ere identified, quantified and classified as alcohols, esters, acids, aldehydes, lactones, phenols, h

139 ltilayer metalized (MET), and ethylene vinyl alcohol (EVOH) based pouches and stored at 25, 35 and 45

140 Alcohol-exposed mice have reduced Bcl6 and PD-1 expressi

141 tored abnormal responses to predator odor in alcohol-exposed mice.

142 oltage optical mapping in intact hearts from alcohol-exposed or aged mice (where JNK2 is activated).

143 Fetal alcohol exposure (FAE) is the leading preventable develo

144 s mechanistic insights into how both chronic alcohol exposure and protracted withdrawal dysregulate 5

145 s are reported to be killed by developmental alcohol exposure because of activation of microglial imm

146 Alcohol-fed mice demonstrated increased hepatic expressi

147 n alpha-defensin 5 (HD5) was orally given to alcohol-fed mice to test the therapeutic potential.

148 nistically, the dehydrogenation of secondary alcohol follows clean pseudo-first-order kinetics and ex

149 fic focus is placed on fatty acids and fatty alcohols for their natural availability, low toxicity, b

150 within the n-alkane, aromatic, n-ketone, and alcohol fractions can be identified.

151 We observed a lower diversity in the alcohol groups compared with controls.

152 be highly selective for the formation of cis alcohol (>90%), irrespective of the size of the hydride

153 hanism for this tolerance-inducing effect of alcohol, however, is unknown.

154 ma iodide are incorporated onto an aliphatic alcohol in a single operation.

155 n amino acid, aromatic aldehyde, and primary alcohol in alkaline solutions under microwave-assisted c

156 g can then furnish the desired Z-homoallylic alcohol in high enantiomeric purity.

157 density and viscosity of sorbitol as a sugar alcohol in the ([mmim](MeO)(2)PO(2)) ionic liquid (IL) w

158 l reports, 2,518 (25.9%) had been exposed to alcohol in utero.

159 used to quantitatively analyze a mixture of alcohols in a quasi-online manner.

160 totic lipid hydroperoxides to nontoxic lipid alcohols in a tissue-specific manner.

161 and oxetanes yielding secondary and tertiary alcohols in excellent yields and regioselectivities.

162 ducts with lower molecular weight, such as n-alcohols in O(1) class and subsequently to n-fatty acids

163 Alcohols, in particular polyols, are well-known for the

164 Various secondary alcohols including cyclic and acyclic alcohols and a wid

165 While reports indicate that alcohol increases excitatory neurotransmission exclusive

166 c deficiency of AR (knockout mice) prevented alcohol-induced increase in harmful AR metabolites, toxi

167 Taken together, the study suggests that alcohol-induced PC alpha-defensin dysfunction is mediate

168 ckout mice revealed increased sensitivity to alcohol-induced sedation and developed tolerance to the

169 on of alcohol consumption and sensitivity to alcohol-induced sedation.

170 findings identify a novel mechanism by which alcohol induces TGFbeta1 in the lung.

171 adjustment for potential covariates, ages at alcohol initiation (AAIs) of 18.1-29.0 years, 29.1-39.0

172 Alcohol intake >30 g/day yielded increased risk estimate

173 f GalphasDREADD in DMS dMSNs or iMSNs alters alcohol intake and observed that CNO-dependent activatio

174 udy provides evidence in support of limiting alcohol intake in adherence to the USDGA recommendations

175 ats, it remains unknown how NAc MSNs control alcohol intake in either sex.

176 Alcohol intake influences plasma lipid levels, and such

177 mic risk stratification, and to test whether alcohol intake or body mass index interacts with polygen

178 ved regulatory mechanism linking anxiety and alcohol intake that might contribute to increased suscep

179 of abstinence period, and quantification of alcohol intake), and if the patient is an active drinker

180 d on deterrent health factors, like smoking, alcohol intake, cheese consumption and average systolic

181 p with low mental health index score and low alcohol intake, HRs (95% CIs) for all-cause mortality we

182 core combined with light, moderate, and high alcohol intake, respectively.

183 ealth 2000), with available data on baseline alcohol intake.

184 y youthful drinking may be vulnerable to age-alcohol interactions in later adulthood.

185 d controlled trials have tested clinic-based alcohol interventions.

186 Repeated alcohol intoxications trigger inflammatory processes and

187 lkylation of secondary alcohols with primary alcohols is reported.

188 ow inositol levels in response to high blood alcohol levels support a mechanism of reversible osmolar

189 s of acrylamide and predominant formation of alcohol-like compounds.

190 tion based setting, drinking high volumes of alcohol may contribute to the prevalence of sleep proble

191 volatiles (such as acetate esters and higher alcohols) may be indicative of microbial spoilage.

192 race/ethnicity, education, income, smoking, alcohol, menopausal hormone therapy, and hysterectomy st

193 variant in ADH1B is associated with a higher alcohol metabolizing activity compared to the ancestral

194 perience of IPV and (b) their male partner's alcohol misuse among couples in urban Zambia.

195 AUD and aspects of alcohol misuse have been shown to overlap strongly with

196 ce subscale, and the secondary outcome, male alcohol misuse, by the Alcohol Use Disorders Identificat

197 ish the stereochemistry of the vicinal amino alcohol motif embedded within the targets.

198 noparticles is introduced into the polyvinyl alcohol networks, building hybrid hydrogel evaporators i

199 generated by direct homolytic activation of alcohol O-H bonds through a proton-coupled electron-tran

200 nd by reducing the pK(a) of the nucleophilic alcohol of a serine.

201 night may be related to the acute effects of alcohol on GABAergic systems that are associated with sl

202 st meta-analysis to explore acute effects of alcohol on the human brain.

203 signed in a 1:1 ratio to either abstain from alcohol or continue their usual alcohol consumption.

204 odel was used to model daily abstinence from alcohol over the 21 days after ketamine infusion.

205 of obesity, nonalcoholic liver disease, and alcohol overuse worldwide.

206 Electrochemical alcohol oxidation by NiOOH has been understood since the

207 Use of nonsterile gloves and an alcohol pad does not significantly impact the contaminat

208 drink containing approximately 12 g of pure alcohol) per week and who had paroxysmal or persistent a

209 lure, myocardial infarction, thyrotoxicosis, alcohol, pericarditis, pulmonary embolism, and myocardit

210 cture, the as-obtained anisotropic polyvinyl alcohol/polyaniline hydrogel can work as a stretching/co

211 CMP, a combination of painful conditions and alcohol problems had the largest effect size on mortalit

212 subsequently reacts with mCPBA to afford the alcohol product and regenerate the aroyloxy radical.

213 mate homeostasis in the NA core of cocaine + alcohol rats relative to rats consuming cocaine alone, s

214 that there will be a dramatic rising tide of alcohol relapse, admissions for decompensated ALD, and a

215 s2642438 was associated with reduced risk of alcohol-related cirrhosis (adjusted odds ratio, 0.76; P=

216 052 was associated with an increased risk of alcohol-related cirrhosis (adjusted odds ratio, 1.30; P=

217 c associations with the risks for developing alcohol-related cirrhosis and HCC were determined using

218 gment our knowledge of mechanisms underlying alcohol-related cognitive/motor impairment and inform in

219 arch parsing what AUD patients remember from alcohol-related episodes is lacking.

220 Microbial dysbiosis is associated with alcohol-related hepatitis (AH), with the mechanisms yet

221 the identification of mechanisms underlying alcohol-related impairment as well as factors reinforcin

222 ally affect the risk for developing advanced alcohol-related liver disease.

223 framework of how emotional dysregulation and alcohol-related microbiome dysbiosis could accelerate th

224 eral that have been previously overlooked in alcohol research.

225 An understanding of alcohol's acute neural effects could augment our knowled

226 mOP-LTD at cortical inputs was sensitive to alcohol's deleterious effects.

227 hamphetamine, morphine, heroin, nicotine, or alcohol seeking, as assessed in rodent models.

228 st pioglitazone reduces alcohol drinking and alcohol-seeking behavior in rats.

229 , hypertension, diagnoses including obesity, alcohol, sleep apnea, diabetes, chronic obstructive pulm

230 uit-vegetable intake ameliorating effects of alcohol-smoking-fatty food.

231 uction of GalA to the oxidation of the sugar alcohol sorbitol that has a higher reduction state compa

232 pothalamus in a postnatal rat model of fetal alcohol spectrum disorders.

233 n of the catalytic dehydration of tert-butyl alcohol (TBA) used to probe the activities of OH groups

234 acid and target volatile compounds (esters, alcohols, terpenols, volatile acids and volatile phenols

235 For more sterically congested alcohols, the use of a recently-developed isoselenourea

236 iscuss the major contribution of obesity and alcohol to the ten most common cancers as well as measur

237 ent alcohols, allowing primary and secondary alcohols to be employed as nonhazardous and greener alky

238 he alpha-alkylation of nitriles with primary alcohols to selectively synthesize nitriles by a well-de

239 and 2-methylfuran by dehydration of furfuryl alcohol under dry conditions.

240 pursuit response to very low levels of blood alcohol, under the conditions of our study.

241 t injecting (OR, 0.98; 95% CI, 0.94-1.02) or alcohol use (OR, 0.99; 95% CI, 0.95-1.04).

242 d insula GMVs were associated with increased alcohol use across samples.

243 e are evident, some open questions regarding alcohol use and its consequences in the US population re

244 ed, novel brain regions that are involved in alcohol use and novel biomarkers of alcohol use need to

245 of the amygdala plays a significant role in alcohol use and other affective disorders; however, the

246 ce experience and recent suicidal behaviour, alcohol use and recent suicidal behaviour, illicit drug

247 le the increased health risks resulting from alcohol use are evident, some open questions regarding a

248 Hepatitis C virus (HCV) and alcohol use are patient risk factors for accelerated fib

249 imitations of using quantitative measures of alcohol use as proxy measures for AUD, and we outline ho

250 18q23 in regulating neural connectivity and alcohol use behavior, potentially via dysregulated myeli

251 lammation-related genes were associated with alcohol use behaviors.

252 use of mortality in the United States, where alcohol use consistently increased over the last decades

253 e expression and hippocampal degeneration in alcohol use disorder (AUD) and other mental diseases is

254 ical and economic consequences of coexisting alcohol use disorder (AUD) in patients with cirrhosis, l

255 Alcohol use disorder (AUD) is a devastating illness defi

256 and palatable fluids.SIGNIFICANCE STATEMENT Alcohol use disorder (AUD) is a major health burden worl

257 ded pedigrees selected for high-densities of alcohol use disorder (AUD) or drug abuse (DA).

258 also associated with alcohol consumption and alcohol use disorder (AUD) risk.

259 n a multicenter observational study, 36 with alcohol use disorder (AUD), and 17 persons without AUD (

260 higher ASCA levels compared to patients with alcohol use disorder and to nonalcoholic controls.

261 ms, autism spectrum disorder, psychosis, and alcohol use disorder compared with the control sample.

262 individuals who met criteria for a past-year alcohol use disorder did.

263 individuals who met criteria for a past-year alcohol use disorder had a psychiatric comorbidity, whil

264 in female mice suggests that treatments for alcohol use disorder in women may need to account for th

265 ents with alcoholic hepatitis, patients with alcohol use disorder, and nonalcoholic controls using fu

266 aptive responses to stress are a hallmark of alcohol use disorder, but the mechanisms that underlie t

267 ndence are key factors in the development of alcohol use disorder, which is a pervasive societal prob

268 s a potential pharmacological agent to treat alcohol use disorder.

269 ously associated with alcohol consumption or alcohol use disorder.

270 condary outcome, male alcohol misuse, by the Alcohol Use Disorders Identification Test (AUDIT).

271 Any alcohol use during pregnancy is associated with subtle y

272 tanding of the neurocircuitry that underlies alcohol use has improved, novel brain regions that are i

273 Alcohol use increased during the period 1999-2014.

274 Our data suggest that alcohol use is associated with differential methylation

275 Excessive alcohol use is the third leading cause of mortality in t

276 e ((18)F-FPEB) PET has revealed that chronic alcohol use leads to decreased limbic mGluR5 availabilit

277 olved in alcohol use and novel biomarkers of alcohol use need to be identified.

278 Reevaluation of Medicaid alcohol use policies may be warranted, to align more clo

279 This study investigates whether alcohol use predicts the periodontal pocket development

280 Genomic risk for alcohol use was enriched in gene sets that were preferen

281 fter adjustment for confounders were harmful alcohol use, 1.4 (0.9-2.0, p = 0.10) and obesity, 1.4 (0

282 city, older age, history of mental disorder, alcohol use, and civil/legal issues.

283 s findings in the literature concerning age, alcohol use, and depression-related changes in brain vol

284 HCC risk was also higher in patients with alcohol use, older age, and infection with HCV genotype

285 eported drug use, and 10.9% reported harmful alcohol use.

286 , we identified replicable GMV correlates of alcohol use.

287 on of carbonyl moieties to the corresponding alcohol using simply hydrazine hydrate has been consider

288 oxidation of primary and secondary aliphatic alcohols using a pair of flavin and dialkylthiourea cata

289 based copolymer molecule containing vanillyl alcohol (VA) that exhibits high sensitivity and specific

290 atalyzed cleavage of C-C bonds in unstrained alcohols via beta-carbon elimination.

291 ced fruitiness, sweetness, fullness/body and alcohol warming sensation (p < 0.05).

292 s is demonstrated here by using an aliphatic alcohol-water system.

293 y or almost daily consumption of tobacco and alcohol were both significantly associated with increase

294 tions to produce enantioenriched homoallylic alcohols with a stereodefined trisubstituted alkene.

295 uted ketones via the alkylation of secondary alcohols with primary alcohols is reported.

296 ronic, heavy use increases the likelihood of alcohol withdrawal symptoms and associated secondary out

297 These data support further evaluation of alcohol withdrawal symptoms as a prognostic indicator of

298 By week 12, participants with high alcohol withdrawal symptoms on prazosin reported 7.07% h

299 ting critically ill patients including pain, alcohol withdrawal syndrome, status epilepticus, and acu

300 (PACAP), and its cognate receptor, PAC1R, in alcohol withdrawal-induced behaviors.