ライフサイエンスコーパス: alcohol abuse

1 duodenal biopsies from patients with chronic alcohol abuse.

2 isorders including depression, epilepsy, and alcohol abuse.

3 nt of future therapeutic strategies to treat alcohol abuse.

4 king behaviors and behaviors associated with alcohol abuse.

5 a new therapeutic direction for treatment of alcohol abuse.

6 proteins that regulate HCV infection during alcohol abuse.

7 vity with potential for use as treatments of alcohol abuse.

8 effects in animal models relevant to drug or alcohol abuse.

9 functional responses associated with chronic alcohol abuse.

10 onsumption that could underlie some forms of alcohol abuse.

11 and was negatively correlated with years of alcohol abuse.

12 offer unexplored targets in the treatment of alcohol abuse.

13 brain is one of the major targets of chronic alcohol abuse.

14 ic view of brain alterations associated with alcohol abuse.

15 heart disease, hyperlipidemia, obesity, and alcohol abuse.

16 the aging process, therapeutics, and drug or alcohol abuse.

17 l stress is an environmental risk factor for alcohol abuse.

18 and seeking in preclinical rodent models of alcohol abuse.

19 tic stress disorder (PTSD), head injury, and alcohol abuse.

20 rcuitry to predispose individuals for future alcohol abuse.

21 atitis, especially in those who are prone to alcohol abuse.

22 tion of additional miR-9 targets relevant to alcohol abuse.

23 naive subject can predict the likelihood of alcohol abuse.

24 ategies, can reduce the population burden of alcohol abuse.

25 VTA circuitry that contributes to subsequent alcohol abuse.

26 ective drug development for the treatment of alcohol abuse.

27 al gambling, cigarette smoking, and drug and alcohol abuse.

28 se, valvular heart disease, tobacco use, and alcohol abuse.

29 bserved in those subjects with substance and alcohol abuse.

30 (BDNF) might be linked with vulnerability to alcohol abuse.

31 stenosis, aortic valve disease, smoking, or alcohol abuse.

32 lumes than did patients without a history of alcohol abuse.

33 borative care (CC) intervention for PTSD and alcohol abuse.

34 tween subjects with and without a history of alcohol abuse.

35 oms only among those who screen positive for alcohol abuse.

36 nteraction of HIV infection and a history of alcohol abuse.

37 itive men with and without a past history of alcohol abuse.

38 s, corticotropin-releasing factor (CRF), and alcohol abuse.

39 to these cardiac diseases in the setting of alcohol abuse.

40 development of new therapeutic approaches to alcohol abuse.

41 mong 14,727,591 patients, 268,084 (1.8%) had alcohol abuse.

42 common polymorphism (MAOA-LPR) and risk for alcohol abuse.

43 eural mechanism for genetic predilection for alcohol abuse.

44 enuation parameter >=248 dB/m, in absence of alcohol abuse.

45 ual-level risk factors for violence, such as alcohol abuse.

46 en dampened MAOA expression, elevated DA and alcohol abuse.

47 ons such as renal failure, diabetes, HIV and alcohol abuse.

48 ders, anxiety disorders, substance abuse, or alcohol abuse.

49 coholic individuals and patients with active alcohol abuse.

50 clinical liver disease, liver cirrhosis, or alcohol abuse.

51 ess also had a higher lifetime prevalence of alcohol abuse (10.1% vs 3.8%, P < .001) and drug abuse (

52 (20%), hypernatremia (12.6%), and extensive alcohol abuse (11.4%).

53 ave had cirrhosis (59.8%), diabetes (27.2%), alcohol abuse (17.7%), hepatocellular carcinoma (17.5%),

54 g management of suicide risk (3 indicators), alcohol abuse (2 indicators), and elderly patients; asse

55 One-year prevalence rates of alcohol abuse (2.3% vs. 3.9%; Z=-2.04; p<0.05) and depen

56 abetes mellitus; 2.22 (95% CI 1.58-3.11) for alcohol abuse; 2.37 (95% CI 1.53-3.68) for liver disease

57 lipidemia (8%), metabolic syndrome (9%), and alcohol abuse (21%).

58 deaths [mean 1740] EU-wide) and deaths from alcohol abuse (28.0%, 12.30-43.70; 1550-5490 potential e

59 (40%), peripheral vascular disorders (39%), alcohol abuse (36%), valvular disease (32%), liver disea

60 t of psychiatric disorders; (3) substance or alcohol abuse; (4) compliance with medical regimens; (5)

61 oronary heart disease; 6) hyperlipidemia; 7) alcohol abuse; 8) tobacco use disorder; and 9) random ef

62 1 [0.82]; P = .02), substance use disorders (alcohol abuse, 96.5 [0.67]; P < .001; drug abuse, 97.6 [

63 Understanding the relationship between alcohol abuse, a common and theoretically modifiable con

64 risk factors, such as severe mental illness, alcohol abuse, a painful loss, exposure to violence, or

65 In multivariable analysis, a history of alcohol abuse (adjusted OR, 2.41; P=0.01) and increased

66 ed damage to cholangiocytes, but not ongoing alcohol abuse, affected liver stiffness.

67 Chronic alcohol abuse alters the molecular structure and functio

68 on was associated with a period of decreased alcohol abuse, although recurrence of the alcohol-use di

69 2-fold increased risk of inpatient care for alcohol abuse among patients who had GBS compared with t

70 was no difference in inpatient treatment of alcohol abuse among patients who underwent gastric bypas

71 omic impact worldwide, pharmacotherapies for alcohol abuse and addiction remain limited.

72 Tolerance is a well described component of alcohol abuse and addiction.

73 l attractive targets for novel treatments of alcohol abuse and addiction.

74 ed sensitivity, is an important component of alcohol abuse and addiction.

75 Alcohol abuse and alcohol dependence are key factors in

76 US National Institute on Alcohol Abuse and Alcoholism (AA012388, AA017168, AA0059

77 ctions of alcohol, the National Institute on Alcohol Abuse and Alcoholism and the National Heart, Lun

78 Alcohol abuse and alcoholism are major health problems a

79 feeding mice alcohol (National Institute on Alcohol Abuse and Alcoholism binge model) or chow diets

80 nkers according to the National Institute on Alcohol Abuse and Alcoholism criteria.

81 erview Schedule of the National Institute on Alcohol Abuse and Alcoholism in a large representative s

82 h was organized by the National Institute on Alcohol Abuse and Alcoholism in collaboration with the O

83 mption to sex-specific National Institute on Alcohol Abuse and Alcoholism safe drinking levels.

84 eport published by the National Institute on Alcohol Abuse and Alcoholism showed that liver cirrhosis

85 ug Administration, the National Institute on Alcohol Abuse and Alcoholism, and the National Institute

86 nd could determine individual differences in alcohol abuse and alcoholism, as well as represent a the

87 National Institute on Alcohol Abuse and Alcoholism, US National Institutes of

88 bute to the development of new therapies for alcohol abuse and alcoholism.

89 US National Institute on Alcohol Abuse and Alcoholism.

90 velopment of numerous pathologies, including alcohol abuse and anxiety.

91 sought to investigate the associations among alcohol abuse and atrial fibrillation (AF), myocardial i

92 The main causes of pancreatitis are alcohol abuse and biliary disease.

93 or perpetuate existing patterns of excessive alcohol abuse and can enhance the probability of relapse

94 individuals HCV/HIV co-infection, older age, alcohol abuse and CD4/CD8 ratio seem to correlate with f

95 ote public education about the risks between alcohol abuse and certain types of cancer; * Support pol

96 Alcohol abuse and dependence (alcohol use disorders, AUD

97 Alcohol abuse and dependence disorders are common in the

98 ening tests to improve the identification of alcohol abuse and dependence disorders, the epidemiology

99 Early recognition of alcohol abuse and dependence is necessary and should pro

100 Alcohol abuse and dependence remain highly prevalent and

101 Alcohol abuse and dependence were the most common disord

102 ncident alcohol-related problems, and DSM-IV alcohol abuse and dependence).

103 he study of ethanol-related traits including alcohol abuse and dependence, and behavioral responses t

104 for DSM-III-R provided lifetime diagnoses of alcohol abuse and dependence, and onset and recency ques

105 Inventory of Consequences-Recent, and DSM-IV alcohol abuse and dependence.

106 ikelihood of meeting diagnostic criteria for alcohol abuse and dependence.

107 ol during adolescence is predictive of adult alcohol abuse and dependence.

108 hiatric comorbidity, and treatment of DSM-IV alcohol abuse and dependence.

109 Lifetime and 12-month DSM-IV alcohol abuse and dependence.

110 hin the reward circuitry of the brain during alcohol abuse and dependence.

111 additive and interactive effects of previous alcohol abuse and HIV infection on cognition.

112 ts on cognitive function were found for past alcohol abuse and HIV infection, with significant intera

113 increased hepatic injury in a model of binge alcohol abuse and in response to TNF-alpha treatment.

114 osis is the most common consequence of acute alcohol abuse and may predispose to more severe hepatic

115 sk factors for progression of steatosis were alcohol abuse and overweight/obesity; cumulative exposur

116 hould be to change people's attitudes toward alcohol abuse and seeking professional help.

117 Alcohol abuse and/or dependence (AA/D) and mood disturba

118 ntabuse; a compound used in the treatment of alcohol abuse) and the antifungal agent chlordantoin.

119 e and 21 HIV positive) had a past history of alcohol abuse, and 47 (18 HIV negative and 29 HIV positi

120 eline, 40% of the patients had diabetes, 29% alcohol abuse, and 6% HIV or HBV coinfection.

121 n analysis adjusted for stroke, head trauma, alcohol abuse, and cancer showed 1-year, 2-5 year, and 6

122 is is associated with age, gender, diabetes, alcohol abuse, and coinfection with human immunodeficien

123 Younger age, black race, a history of alcohol abuse, and homelessness were predictors of clust

124 ablished behavioral risk factors of smoking, alcohol abuse, and lack of physical activity, economic (

125 lude HCV infection, HBV infection, diabetes, alcohol abuse, and low CD4+ cell count.

126 it aggressiveness, impaired impulse control, alcohol abuse, and low CSF 5-hydroxyindoleacetic acid.

127 tiation, including non-injection drug abuse, alcohol abuse, and mental illness.

128 After adjusting for age, smoking, alcohol abuse, and nonimmunocompromising comorbidities,

129 umatic stress disorder [PTSD], substance and alcohol abuse, and others) and medical disorders (cardio

130 jor surgery, and history of substance abuse, alcohol abuse, and self-harm/suicide were associated wit

131 Unemployment, alcohol abuse, and smoking were associated with resistan

132 , diabetes (AOR, 1.79; 95% CI, 1.6-2.1), and alcohol abuse (AOR, 1.78; 95% CI, 1.5-2.1), whereas blac

133 other complex disease traits, alcoholism and alcohol abuse are influenced by the combined effects of

134 Use of alcohol abuse as a screening method for alcohol dependen

135 ns are significantly influenced by substance/alcohol abuse as well as postmortem interval; however, t

136 preceded or was concurrent with the onset of alcohol abuse (Bipolar First), and 83 subjects with bipo

137 i in fecal samples from patients with active alcohol abuse but not in controls.

138 red in individuals with a history of chronic alcohol abuse but without ARDS (0.46 ng/mL [0.12-2.75],

139 g is a well-known risk factor for subsequent alcohol abuse, but the neural events underlying this ris

140 s is a well-known risk factor for subsequent alcohol abuse, but the neural mechanisms underlying inte

141 9 consecutive patients with ongoing or prior alcohol abuse, but without known liver disease.

142 , metabolic and genetic diseases, as well as alcohol abuse can also influence both hepatic and cardio

143 (diabetes, insulin resistance, obesity) and alcohol abuse can be influenced before starting treatmen

144 Drug and alcohol abuse can produce a variety of ocular and neuro-

145 3 or more pressors simultaneously, extensive alcohol abuse, cancer history (nonskin), active meningit

146 tors, although an interaction with substance/alcohol abuse cannot be completely ruled out.

147 al abuse, neglect, caregiver's substance use/alcohol abuse, caregiver's depressive symptoms, caregive

148 Alcohol abuse causes half of all deaths from cirrhosis i

149 ABSTRACT: Alcohol abuse causes major social, economic and health-r

150 Alcohol abuse causes widespread changes in gene expressi

151 T) value, Hepatitis C virus (HCV) infection, alcohol abuse, CD4/CD8 ratio and an increased number of

152 as attributable to CV risk factors, IHD, and alcohol abuse combined, whereas among RA subjects, only

153 Thus, alcohol abuse could be a co-factor in progression of HIV

154 measures (including duration and severity of alcohol abuse, craving, and anxiety or depressive sympto

155 ine dependence (4.5%; 95% CI, 3.6% to 5.4%), alcohol abuse/dependence (0.3%; 95% CI, 0.1% to 0.6%), a

156 UC patients with regard to PTSD (P =.01) and alcohol abuse/dependence (P =.048).

157 Bipolar II disorder predicted both alcohol abuse/dependence and benzodiazepine use and abus

158 showed on average a decrease in the rate of alcohol abuse/dependence of -24.2% (95% CI, -19.9% to -2

159 ar carcinoma and NHL and presence of HCV and alcohol abuse/dependence using International Classificat

160 Alcohol abuse/dependence was diagnosed by using the Alco

161 ement, (2) motivational interviews targeting alcohol abuse/dependence, and (3) evidence-based pharmac

162 of older age, those with obesity, those with alcohol abuse/dependence, and those who lacked insurance

163 ) of combat veterans in which PTSD, lifetime alcohol abuse/dependence, and Vietnam versus Gulf War se

164 ficantly greater risk for the later onset of alcohol abuse/dependence, cannabis use and abuse/depende

165 After adjusting for HCV infection and alcohol abuse/dependence, HIV status was not independent

166 atios of advanced fibrosis or cirrhosis were alcohol abuse/dependence, obesity, and being uninsured.

167 After adjusting for HCV and alcohol abuse/dependence, the IRR for NHL comparing HIV-

168 xplained by the higher prevalence of HCV and alcohol abuse/dependence.

169 cidal ideation/attempt; nicotine dependence; alcohol abuse/dependence; and illicit drug abuse/depende

170 The alcohol-abuse deterrent disulfiram (DSF) is shown to hav

171 oints in a combined temporal sepsis network: Alcohol abuse, Diabetes and Cardio-vascular diagnoses.

172 In DSM-IV, an alcohol abuse diagnosis is preempted by dependence, alth

173 We determined the risk of an alcohol abuse diagnosis on incident AF, MI, and CHF.

174 Alcohol abuse disorders are associated with dysfunction

175 naling could be developed as medications for alcohol abuse disorders.

176 drug may be developed as a new treatment for alcohol abuse disorders.

177 this polymorphism increases the severity of alcohol abuse disorders.

178 lled consumption of alcohol is a hallmark of alcohol abuse disorders; however, the central molecular

179 have an important role in the development of alcohol-abuse disorders and studies from this laboratory

180 opathology; e.g., binge eating and opiate or alcohol abuse, disorders in which muORs and aberrant cor

181 Alcohol abuse does not always accompany alcohol dependen

182 ods identified treatment barriers including, alcohol abuse, family conflicts, and mental health issue

183 of dysthymia, major depressive disorder, and alcohol abuse for sexually abused white participants.

184 ugh posttraumatic stress disorder (PTSD) and alcohol abuse frequently occur among acutely injured tra

185 Acute pancreatitis is associated with alcohol abuse, gallstones and bacterial infection.

186 t severe disorder (P < .001), and those with alcohol abuse had a mean (SE) of 5.6 (1.2) points lower

187 In addition, patients who resumed alcohol abuse had decreased medial frontal gray matter v

188 plexes (mRCC) that are adversely affected by alcohol abuse has not been studied.

189 reports on epigenetic mechanisms involved in alcohol abuse have focus on hepatic and neuronal regions

190 nstrates that HIV infection and a history of alcohol abuse have independent effects on some aspects o

191 cal comorbidity index, dementia, depression, alcohol abuse, head injury, smoking, body mass index, an

192 mans is induced by various insults including alcohol abuse, hepatitis B/C virus infection, autoimmune

193 Donor age, obesity, alcohol abuse, hepatitis serology, liver only donor, ima

194 s remained significant after controlling for alcohol abuse history.

195 ently predicted by higher comorbidity index, alcohol abuse, history of trauma, and acquired abnormal

196 -Stage Liver Disease (MELD) score, diabetes, alcohol abuse, HIV, or HBV coinfection were collected at

197 r, strains and younger age, urban residency, alcohol abuse, homelessness, noninjection drug use, and

198 mechanism for developing myelosuppression in alcohol-abusing hosts with severe bacterial infection.

199 Although it is well recognized that alcohol abuse impairs alveolar macrophage immune functio

200 Alcohol abuse impairs immune defense.

201 the third theme in the paper - screening for alcohol abuse in college settings.

202 s of the galanin (GAL) gene with anxiety and alcohol abuse in different male populations but were una

203 mechanisms for the synergism between HCV and alcohol abuse in liver disease progression.

204 e to increased susceptibility to anxiety and alcohol abuse in men.

205 Alcohol abuse increased the risk of AF, MI, and CHF to a

206 Previous alcohol abuse increases the risk of developing mood diso

207 Chronic alcohol abuse induces liver injury and increases the sev

208 Alcohol abuse is a comorbid factor in many human immunod

209 Chronic alcohol abuse is a comorbid variable of acute respirator

210 Alcohol abuse is a leading cause of liver disease charac

211 Alcohol abuse is a leading cause of pancreatitis, accoun

212 Alcohol abuse is a leading cause of preventable death an

213 Alcohol abuse is a major cause of liver injury.

214 Alcohol abuse is a major cause of pancreatitis in people

215 Alcohol abuse is a major risk factor for pancreatitis an

216 Alcohol abuse is a significant medical and social proble

217 Alcohol abuse is a widespread and serious problem.

218 Relapse to alcohol abuse is an important clinical issue that is fre

219 Chronic alcohol abuse is associated with deficits in cognitive c

220 Alcohol abuse is associated with immunosuppressive and i

221 ovel clinical findings provide evidence that alcohol abuse is associated with significant zinc defici

222 de and other biomarkers for the detection of alcohol abuse is being better established.

223 Ill-health due to alcohol abuse is improving in some nations but deteriora

224 ly increased incidence included substance or alcohol abuse, latent tuberculosis, diabetes mellitus, a

225 Alcohol abuse leads to aberrant forms of emotionally sal

226 ultivariate predictors of mortality included alcohol abuse, low body mass index, and poor social adju

227 equent to comorbidity with chronic diseases, alcohol abuse, malignancy, immune deficient/suppression

228 aggravates the severity of ALD; conversely, alcohol abuse may be a cryptic co-factor in some cases o

229 ALD patients is hefty and the prevalence of alcohol abuse may be increasing in both the developed an

230 for age, sex, socioeconomic status, smoking, alcohol abuse, medication, and comorbidity were estimate

231 Other causes include alcohol abuse, medications, and hepatitis B and C.

232 violations, aggressive driving, sleepiness, alcohol abuse, metabolic disorders, and multiple medicat

233 Thus, efforts to mitigate alcohol abuse might result in meaningful reductions of c

234 creatitis with pancreas divisum (n = 3), and alcohol abuse (n = 2).

235 d trauma, surgery, drug therapy, smoking, or alcohol abuse, nor was there any relevant family history

236 ronmental factors associated with CP include alcohol abuse (odds ratio [OR], 3.1; 95% CI, 1.87-5.14)

237 The population-attributable risk of alcohol abuse on each outcome was of similar magnitude t

238 odel of HIVE, we investigated the effects of alcohol abuse on the clearance of virus-infected macroph

239 nerally associated with tobacco consumption, alcohol abuse or both, whereas pharynx cancers are incre

240 organ disease develops in a minority, due to alcohol abuse or concurrent genetic modifiers that are n

241 (OR, 0.29; 95% CI, 0.90 to 0.92) or lifetime alcohol abuse or dependence (OR, 0.30; 95% CI, 0.15 to 0

242 e disorder (ie, drug abuse or dependence and alcohol abuse or dependence), and any mental disorder (i

243 43 (1.27-1.64) for GAD, 1.20 (1.08-1.35) for alcohol abuse or dependence, 1.39 (1.18-1.67) for PTSD,

244 25.3% for social phobia, 9.1% vs. 25.9% for alcohol abuse or dependence, and 6.7% vs. 17.6% for drug

245 , history of traumatic brain injury, current alcohol abuse or dependence, and current use of psychotr

246 stment for a prior major depressive episode, alcohol abuse or dependence, and drug abuse or dependenc

247 d with an increased risk of depression, GAD, alcohol abuse or dependence, and PTSD and with some patt

248 ving while impaired sample reported 12-month alcohol abuse or dependence, compared with 1.0% and 1.8%

249 ving while impaired sample reported 12-month alcohol abuse or dependence, compared with to 2.0% and 1

250 history of depression did not predict later alcohol abuse or dependence.

251 Manual for Mental Disorders-IV criteria for alcohol abuse or dependence.

252 Alcohol dependence and alcohol abuse or harmful use cause substantial morbidity

253 liver dysfunction, or reported a history of alcohol abuse or recent anemia therapy left 4,940 NHANES

254 He denied alcohol abuse or recreational drug use.

255 similarly to people with addictions such as alcohol abuse or smoking(6,7).

256 usion (OR 2.75, 95% CI 1.22-6.37, p = .016), alcohol abuse (OR 2.09, 95% CI .88-5.10, p = 0.098), rec

257 ), peripheral vascular disorders (OR = 1.8), alcohol abuse (OR = 1.8), neurological disorders (OR = 1

258 S, coagulopathy, deficiency anemia, obesity, alcohol abuse, or drug abuse) were associated with highe

259 liver illnesses, including viral hepatitis, alcohol abuse, or metabolic disease.

260 posttransplant psychosocial problems such as alcohol abuse, other drug abuse, noncompliance, schizoph

261 In multivariable analysis, living alone, alcohol abuse, perception of medical care as being a sub

262 V-positive subjects, those with a history of alcohol abuse performed more poorly on tests of verbal I

263 e), lifestyle characteristics (BMI, smoking, alcohol abuse, physical inactivity) and social factors (

264 e than double the risk of inpatient care for alcohol abuse postoperatively compared with patients und

265 e (PR = 1.41; 95% CI: 1.21-1.63; P = 0.001), alcohol abuse (PR = 2.57; 95% CI: 2.33-2.84; P = 0.001),

266 that homelessness, intravenous drug use, and alcohol abuse predisposed patients to emm32.2 iGAS disea

267 Alcohol abuse predisposes the host to bacterial infectio

268 of chronic pancreatitis secondary to chronic alcohol abuse presented to the hospital with acute abdom

269 Differences between men and women in alcohol abuse prevalence have long been attributed to so

270 21) subjects had higher risks of drug abuse, alcohol abuse, rapid cycling, and suicide attempts.

271 disturbance, in turn, increases the risk of alcohol abuse relapse.

272 quency of mood disorders among patients with alcohol abuse relapse.

273 e higher IL-8 levels, liver surgery, chronic alcohol abuse, shock, higher peak airway pressure while

274 ated with a period of recovery from comorbid alcohol abuse, suggesting this posthospital time may pro

275 correlation of reduced binding with years of alcohol abuse suggests an involvement of CB1 receptors i

276 all racial groups, caused by drug overdoses, alcohol abuse, suicides, and a diverse list of organ sys

277 y influence the development or expression of alcohol abuse syndromes in animal models or humans.

278 ere lower in feces from patients with active alcohol abuse than controls.

279 more likely to receive a diagnosis of DSM-IV alcohol abuse than their peers not attending college; de

280 promising lead for the development of novel alcohol abuse therapies.

281 drug targets would be clinically relevant in alcohol abuse treatment and may serve to provide a bette

282 Individuals with a history of chronic alcohol abuse underwent bronchoalveolar lavage within 7

283 9H-SAT, men with a history of incarceration, alcohol abuse, use ever of intravenous drugs, younger ag

284 Prevalence of lifetime and 12-month alcohol abuse was 17.8% and 4.7%; prevalence of lifetime

285 After multivariable adjustment, alcohol abuse was associated with an increased risk of i

286 Overall, prevalence of smoking and alcohol abuse was higher among patients with AD than the

287 Current alcohol abuse was more prevalent among men, whites, and

288 Alcohol abuse was nominally associated with advanced epi

289 The relative risk of inpatient care for alcohol abuse was studied before and after surgery.

290 Chronic alcohol abuse weakens alveolar tight junctions, priming

291 ectin levels from the patients with ARDS and alcohol abuse were also significantly elevated compared

292 Symptoms of dysthymia, major depression, and alcohol abuse were assessed using the National Institute

293 Gender, age, nicotine and alcohol abuse were clinical variables associated with al

294 ronic renal failure, diabetes mellitus, HIV, alcohol abuse) were more common in nonwhite sepsis patie

295 in relation to depressive symptomatology and alcohol abuse with conflicting findings.

296 y (2003-2014) to estimate the association of alcohol abuse with liver fibrosis.

297 uated, our method could estimate the risk of alcohol abuse with statistical significance.

298 been reported in animal models of epilepsy, alcohol abuse, withdrawal, and stress.

299 omfort-care), intraabdominal conditions, and alcohol abuse/withdrawal.

300 m of the OPN gene, compared to patients with alcohol abuse without liver disease.