ライフサイエンスコーパス: alcohol drinking

1 negative responses to stress following heavy alcohol drinking.

2 dependent C57BL/6J mice following 24 days of alcohol drinking.

3 g a potential mechanistic basis for habitual alcohol drinking.

4 ptations gate or drive excessive, compulsive alcohol drinking.

5 also selectively reduced aversion-resistant alcohol drinking.

6 ior and the abstinence-induced escalation of alcohol drinking.

7 id receptor (MR) activation drive compulsive alcohol drinking.

8 ms mediate environmental changes that affect alcohol drinking.

9 an rhythmicity and explored its relevance to alcohol drinking.

10 y of the ACC during withdrawal and excessive alcohol drinking.

11 knockout protected against anxiety caused by alcohol drinking.

12 essary roles for mGluR5/Homer2/PI3K in binge alcohol drinking.

13 gh alcohol consumption (SHAC) model of binge alcohol drinking.

14 al tegmental area (VTA) negatively regulates alcohol drinking.

15 ng in a genetically selected rodent model of alcohol drinking.

16 s responses to predator odor following heavy alcohol drinking.

17 e concentrated sweet solutions and excessive alcohol drinking.

18 eases, alcohol dependence, and escalation of alcohol drinking.

19 exhibited impaired vasodilation after light alcohol drinking.

20 of dmPFC prevented escalation of compulsive alcohol drinking.

21 sful shock in a familiar context followed by alcohol drinking.

22 od that mice transition to heavy, persistent alcohol drinking.

23 olol, but not atenolol, decreased binge-like alcohol drinking.

24 outcomes observed with moderate versus heavy alcohol drinking.

25 reverse the effect of intranasal oxytocin on alcohol drinking.

26 e., it would not reach the brain), decreased alcohol drinking.

27 dual variance in vulnerability to compulsive alcohol drinking.

28 ort for a neuroimmune mechanism of excessive alcohol drinking.

29 gets reduces excessive and harmful levels of alcohol drinking.

30 environmental contexts associated with prior alcohol drinking.

31 with Daun02 produced a long-term decrease in alcohol drinking.

32 n 2.17 (1.70-2.76), those reporting moderate alcohol drinking 1.76 (1.21-2.57), and those with increa

33 gs indicated a protective effect of moderate alcohol drinking (2-30 drinks/month for women and 2-60 d

34 Binge alcohol drinking, a behavior characterized by rapid repe

35 Chronic alcohol drinking accelerates liver fibrosis in patients

36 king period was terminated by 20 min of free alcohol drinking access that achieved significant blood

37 the authors investigated whether history of alcohol drinking affected risks of NHL and multiple myel

38 ated pathways in the regulation of excessive alcohol drinking after a history of dependence.

39 reclinical animal models of either excessive alcohol drinking, alcohol-seeking, or relapse-like drink

40 However, how chronic alcohol drinking alters cognitive flexibility through CI

41 he first time that Grm7 is a risk factor for alcohol drinking and a new target in addiction therapy.

42 Repeated cycles of binge alcohol drinking and abstinence are key components in th

43 ociated with body mass index (BMI), smoking, alcohol drinking and age at menarche (all nominal P < 0.

44 tegmental nucleus (RMTg) decreased voluntary alcohol drinking and alcohol self-administration.

45 tegmental nucleus (RMTg) decreased voluntary alcohol drinking and alcohol self-administration.

46 y the selective agonist pioglitazone reduces alcohol drinking and alcohol-seeking behavior in rats.

47 hese neuroadaptations mediate the heightened alcohol drinking and anxiety-like behavior observed duri

48 Furthermore, alcohol drinking and anxiety-like behaviors in CREB-hapl

49 icotropin-releasing factor (CRF) drive binge alcohol drinking and anxiety.

50 Associations between alcohol drinking and cardiovascular disease mortality co

51 The authors evaluated alcohol drinking and cigarette smoking in relation to ri

52 DLS of rodents results in the escalation of alcohol drinking and compulsive alcohol intake.

53 ent with mifepristone also blocked escalated alcohol drinking and compulsive responding during protra

54 ficacy of naltrexone (50 mg/day) in reducing alcohol drinking and craving among FHP drinkers with ben

55 ationship between plasma aldosterone levels, alcohol drinking and craving was investigated in alcohol

56 ase mortality could be confounded by diet if alcohol drinking and diet are related.

57 nd BMI, from the perspective of the roles of alcohol drinking and dietary factors in a rural populati

58 mice were given limited access to voluntary alcohol drinking and ethanol intake escalation was induc

59 CeA CRF1 receptors regulate alcohol drinking and have served as a therapeutic target

60 POE) locus modulates the association between alcohol drinking and LDL cholesterol.

61 s selectively recruited in response to binge alcohol drinking and limits further drinking behavior.

62 Alcohol drinking and obesity are associated with an incr

63 nd Nociceptin/Orphanin FQ (N/OFQ) attenuates alcohol drinking and relapse in rodents, suggesting that

64 mediate the effects of PPARgamma agonism on alcohol drinking and seeking in msP rats.

65 diating the effects of PPARgamma agonists on alcohol drinking and seeking.

66 Substantial evidence links alcohol drinking and serotonin (5-HT) functioning in ani

67 The effects of alcohol drinking and smoking on pancreatic ductal adenoc

68 ronal ensemble in the CeA reversed excessive alcohol drinking and somatic signs of alcohol dependence

69 T We examined the relationship between binge alcohol drinking and spike timing-dependent plasticity i

70 ts selectively block emotionality, excessive alcohol drinking and stress-induced reinstatement of alc

71 ed the negative association between lifetime alcohol drinking and superior frontal gyrus volume.

72 al oxytocin administration blocked escalated alcohol drinking and the enhanced motivation for alcohol

73 which neuroimmune pathways mediate excessive alcohol drinking and these findings will help to priorit

74 animal models were used to examine excessive alcohol drinking and to discover genes that may contribu

75 NAc and CeA is a major contributor to binge alcohol drinking and to the genetic propensity to consum

76 after a history of repeated cycles of binge alcohol drinking and withdrawal.

77 ut not in the BLA, are crucial in regulating alcohol-drinking and anxiety-like behaviors in rats.

78 on and participates in mechanisms underlying alcohol-drinking and reconsolidation of alcohol-related

79 , a well established animal model of chronic alcohol drinking, and a combination of longitudinal rest

80 literature showing CRF knockout (KO) induces alcohol drinking, and central administrations of CRF red

81 of sexual partners, use of hormonal creams, alcohol drinking, and condom use by a sexual partner.

82 uronal ensemble, decreases the escalation of alcohol drinking, and decreases the intensity of somatic

83 for age, body mass index, cigarette smoking, alcohol drinking, and dietary nutrient intake, the diffe

84 body mass index, higher uric acid, smoking, alcohol drinking, and hiatal hernia were found to be sig

85 were adjusted for age, sex, tobacco smoking, alcohol drinking, and hypertension.

86 evel, body mass index, status of smoking and alcohol drinking, and intracranial volume.

87 , the manner by which STDP responds to binge alcohol drinking, and its sensitivity to dopamine recept

88 DAR modulators can reduce aversion-resistant alcohol drinking, and support testing of D-serine and D-

89 rther show that the effects of FGF2-FGFR1 on alcohol drinking are mediated via activation of the PI3K

90 ption, and that the effects of FGF2-FGFR1 on alcohol drinking are mediated via the phosphoinositide 3

91 from an alcohol-naive state to a pathologic alcohol drinking are not well understood.

92 autophosphorylation in the establishment of alcohol drinking as an addiction-related behavior in mic

93 Nicotine produced an early escalation of alcohol drinking associated with compulsive alcohol drin

94 Escalations in alcohol drinking associated with experiencing stressful

95 We identified far more alcohol-drinking associated bacterial species than tradi

96 ted the association of maternal and paternal alcohol drinking before and early in pregnancy with infa

97 dings indicate that the hPer1 gene regulates alcohol drinking behavior during stressful conditions an

98 s) in hPer1 were tested for association with alcohol drinking behavior in 273 adolescents and an adul

99 ification of candidate genes associated with alcohol drinking behavior in human populations.

100 e difference may contribute to the disparate alcohol drinking behavior of the P and NP rats.

101 GDNF expression ablated the return to alcohol drinking behavior over a 12-month period of repe

102 cilitating mechanism in the establishment of alcohol drinking behavior with changing the DA-5-HT bala

103 d stimuli (CSs), need to be dissociated from alcohol drinking behavior.

104 erstanding of genetic mechanisms influencing alcohol drinking behavior.

105 ty that contributes to mechanisms underlying alcohol drinking behavior.

106 be functionally involved in, mediating high alcohol drinking behavior.

107 y involved in alcohol reinforcement and high alcohol drinking behavior.

108 amatergic neurotransmission is implicated in alcohol-drinking behavior in animal models.

109 may be associated with the predisposition to alcohol-drinking behavior seen in Lewis rats.

110 sm underlies high anxiety-like and excessive alcohol-drinking behavior.

111 ere is substantial individual variability in alcohol drinking behaviors in the population, the neural

112 one alone, would have a greater influence on alcohol drinking behaviors.

113 e and opioid systems play important roles in alcohol drinking behaviors.

114 a neural circuit responsible for individual alcohol drinking behaviors.

115 neuron burst activity in HAD mice decreases alcohol drinking behaviors.

116 Our findings suggest that GDNF reduces alcohol-drinking behaviors by reversing an alcohol-induc

117 sed DSD, thereby increasing anxiety-like and alcohol-drinking behaviors in control rats.

118 The present investigation evaluated alcohol-drinking behaviors in mice that are haplodeficie

119 gnaling pathway involved in anxiety-like and alcohol-drinking behaviors in rats.

120 however, the causal role of the CREB gene in alcohol-drinking behaviors is unknown.

121 n maintaining the high anxiety and excessive alcohol-drinking behaviors of P rats.

122 EB) in genetic predisposition to anxiety and alcohol-drinking behaviors using alcohol-preferring (P)

123 Anxiety-like and alcohol-drinking behaviors were measured after infusion

124 y in a part of the brain (DMS) that controls alcohol-drinking behaviors.

125 the causal role of HDAC2 in anxiety-like and alcohol-drinking behaviors.

126 in neuroadaptations that underlie excessive alcohol-drinking behaviors.

127 ntributor to molecular mechanisms underlying alcohol-drinking behaviors.

128 ol dependence and comorbidity of anxiety and alcohol-drinking behaviors.

129 molecular mechanisms underlying anxiety and alcohol-drinking behaviors.

130 f the CREB gene is associated with increased alcohol-drinking behaviors.

131 vide evidence for the involvement of FGF2 in alcohol-drinking behaviors.

132 rocesses as well as in mechanisms underlying alcohol-drinking behaviors.

133 and GSK-3beta, that in turn drives excessive alcohol-drinking behaviors.

134 ubiquitous Arf6 in the PFC to modulate human alcohol-drinking behaviors.

135 ytes isolated from healthy subjects after an alcohol drinking binge showed enhanced apoptosis (before

136 y DREADD by CNO injection reduced binge-like alcohol drinking, but CNO injection did not alter alcoho

137 nimal models of PTSD have shown increases in alcohol drinking, but effects of stress history on subse

138 Y1R) activation in the BNST suppressed binge alcohol drinking by enhancing inhibitory synaptic transm

139 ated that ERs in the VTA regulate binge-like alcohol drinking by female, but not male, mice.

140 The signal was sustained during alcohol drinking by increased expression of corticotropi

141 estradiol, E2) are associated with increased alcohol drinking by women and experimentally in rodents.

142 Furthermore, alcohol drinking causally mediated increased later depre

143 It is well established that chronic heavy alcohol drinking (CHD) results in significant organ dama

144 Age, male sex, alcohol drinking, cigarette smoking, elevated alanine am

145 Compulsion-like alcohol drinking (CLAD), where consumption continues des

146 -inhibition task in matched heavy- and light-alcohol-drinking college students.

147 Early alcohol drinking confers greater risk for alcohol use di

148 for age, race, sex, study center, education, alcohol drinking, current smoking, prevalent coronary he

149 t be phosphorylated by ERK exhibit excessive alcohol-drinking, despite greater behavioral signs of al

150 as associated with higher levels of smoking, alcohol drinking, diabetes, lipid disorders, and HDI.

151 Lastly, a history of alcohol drinking did not alter synaptic transmission in

152 memory impairments associated with excessive alcohol drinking during acute (24-72 h) but not protract

153 icotropin-releasing factor) are modulated by alcohol drinking during cohabitation.

154 of the glucocorticoid receptor in excessive alcohol drinking during protracted alcohol abstinence.

155 ody mass index, district, cigarette smoking, alcohol drinking, education, occupation, use of vitamin

156 Binge alcohol drinking elevated p(Ser729)-PKCepsilon levels in

157 at, in male mice, a history of chronic binge alcohol-drinking elevates BNST levels of the mGlu5-scaff

158 ate oxytocin's effects on dependence-induced alcohol drinking, enhanced motivation for alcohol, and a

159 6-April 2002) using Medical Subject Headings alcohol drinking, ethanol, cerebrovascular accident, cer

160 However, Nf1(+/-) mice failed to escalate alcohol drinking following chronic intermittent ethanol

161 exone showed only modest effects in reducing alcohol drinking for the 12 weeks of treatment.

162 A 30-day history of binge alcohol drinking (for example, 4-5 g kg(-1) per 2 h(-1))

163 ed according to age, sex, race, smoking, and alcohol drinking) for the presence of mutant p53 protein

164 al activity predict the future escalation of alcohol drinking from casual to compulsive?

165 ntly significant in the never-smoker and non-alcohol drinking groups.

166 suggest unique neuroadaptations between the alcohol drinking groups.

167 according to age, gender, body composition, alcohol drinking habits, and hangover frequency.

168 The process of diagnosing hazardous alcohol drinking (HAD) is based on self-reported data an

169 ctedly, VTA dopamine neuron activity in high alcohol drinking (HAD) mice does not differ from alcohol

170 Alcohol-preferring (P) rats and high alcohol-drinking (HAD) rats are selectively bred for hig

171 Alcohol drinking has been extensively studied in relatio

172 Excessive alcohol drinking has been shown to modify brain circuitr

173 s, but a causal role for estrogen in driving alcohol drinking has not been established.

174 x, study center, education, tobacco smoking, alcohol drinking, hepatitis B surface antigen, and/or an

175 and that the altered pattern is specific to alcohol drinking history.

176 FTI-276, produced a robust decrease of rats' alcohol drinking; however, sucrose consumption was unalt

177 o attenuation of adult anxiety and excessive alcohol drinking in a rat model of adolescent alcohol ex

178 ozapine-N-oxide injection reduced binge-like alcohol drinking in a similar manner as systemic adminis

179 rom alcohol is causally related to excessive alcohol drinking in alcohol-dependent rats, whereas a si

180 monstrated that naltrexone (50 mg/d) reduces alcohol drinking in alcohol-dependent subjects.

181 le during abstinence significantly decreased alcohol drinking in both groups.

182 ic responses wherein PDYN knockout decreased alcohol drinking in both male and female mice, whereas K

183 tween NPY and CRF in the regulation of binge alcohol drinking in both mice and monkeys.

184 FosB was investigated during acquisition of alcohol drinking in C57BL/6J mice.

185 ses Arc expression, and produces anxiety and alcohol drinking in control rats.

186 s been hypothesized to induce high levels of alcohol drinking in dependent rats.

187 alcohol drinking associated with compulsive alcohol drinking in dependent, but not in nondependent r

188 tic strategy for the treatment of compulsive alcohol drinking in humans carrying the Met66BDNF allele

189 ifically, a mouse paradigm that mimics binge alcohol drinking in humans produced a robust reduction i

190 IN OXT selectively inhibited alcohol drinking in male, but not female, animals.

191 endent vGAT-shRNA-based AAV strategy reduces alcohol drinking in male, but not female, mice.

192 ped a method to assess long-term patterns of alcohol drinking in mice housed in groups using the Inte

193 -the-Dark (DID) paradigm to model binge-like alcohol drinking in mice that underwent spared nerve inj

194 For long-term studies of alcohol drinking in mice we used IntelliCages.

195 Apremilast also reduced excessive alcohol drinking in models of stress-facilitated drinkin

196 are part of an endogenous system that keeps alcohol drinking in moderation.

197 during abstinence is required for excessive alcohol drinking in nondependent rats that binge drink a

198 tudies have shown that nicotine can increase alcohol drinking in nondependent rats, yet it is unknown

199 antages to MOP-selective drugs, can decrease alcohol drinking in nonhuman primates.

200 nic nicotine will speed up the escalation of alcohol drinking in rats and that this effect will be ac

201 Sustained voluntary alcohol drinking in rats has been associated with an upr

202 ctively, these data indicate that compulsive alcohol drinking in rats is associated with alterations

203 because increased CRF system activity drives alcohol drinking in rodents, we examined whether it pred

204 ved in neuroadaptations that drive escalated alcohol drinking in rodents.

205 g-Evans rats at 48-h withdrawal from chronic alcohol drinking in the intermittent schedule.

206 le in establishing a high level of voluntary alcohol drinking in these mouse models.

207 Accordingly, alcohol drinking increased alpha-amino-3-hydroxy-5-methy

208 Alcohol drinking increased the expression of genes invol

209 rovides evidence that a history of excessive alcohol drinking increases signaling through the metabot

210 Moderate alcohol drinking increases the activity and function of

211 ality was seen for frequency of non-beverage alcohol drinking independent of volume of beverage ethan

212 tor blockers have been administered to heavy alcohol drinking individuals.

213 therapeutic targets for HCV infection and/or alcohol drinking-induced liver injury.

214 Despite the fact that binge alcohol drinking (intake resulting in blood alcohol conc

215 ncluded age, sex, education, race/ethnicity, alcohol drinking intensity, cigarette smoking duration a

216 rhesus macaques underwent over 20 months of alcohol drinking interspersed with three 30-day forced a

217 Chronic alcohol drinking is a major risk factor for alcohol-asso

218 Binge alcohol drinking is a tremendous public health problem b

219 stulate that a genetic predisposition toward alcohol drinking is accompanied by increased responsiven

220 Alcohol drinking is an established risk factor for sever

221 Additionally, chronic alcohol drinking is associated with a dose dependent hei

222 A key feature of compulsive alcohol drinking is continuing to drink despite negative

223 ol use disorder in women than men, and binge alcohol drinking is correlated with high estrogen levels

224 sults indicate that the ability OT to reduce alcohol drinking is mediated by signaling at OTR in the

225 er that initiates with episodes of excessive alcohol drinking known as binge drinking.

226 rophysiological techniques, we find that low alcohol drinking (LAD) mice have dramatically higher ven

227 ely, alcohol-nonpreferring (NP) rats and low alcohol-drinking (LAD) rats.

228 Furthermore, chronic alcohol drinking led to persistent alterations in Y1R fu

229 center, education, body mass index, smoking, alcohol drinking, level of physical activity, energy int

230 d H&N cancer patients, especially those with alcohol drinking, lower BMI, and advanced stage of prima

231 past smoking (pack-years <5), no or moderate alcohol drinking (</=1 drink/d for women, </=2 drinks/d

232 ed and its expression downregulated in heavy alcohol drinking macaques.

233 These results suggest that heavy alcohol drinking may increase the risk for stroke in Chi

234 Early binge-like alcohol drinking may promote the development of hazardou

235 GABAergic strength in DMS D1- and D2-MSNs of alcohol-drinking mice and control mice.

236 the amygdala proteome in mice after moderate alcohol drinking (n = 12/group) followed by behavioral s

237 l, we determined the impact of preconception alcohol drinking of the mother on offspring stress respo

238 The effect of alcohol drinking on LDL-cholesterol concentrations is un

239 In addition, the role of alcohol drinking on outcomes in patients with cancer is

240 f dopamine in mediating the effects of binge alcohol drinking on synaptic plasticity of NAc MSNs diff

241 n of the PVT(BNST) projection promoted binge alcohol drinking only in female mice, while activation r

242 in the CeA is causally related to excessive alcohol drinking or if it represents a consequence of ex

243 ), smoking (p < 0.0001), evidence of harmful alcohol drinking (p = 0.0001), and ART non-adherence (p

244 reased risk of breast cancer associated with alcohol drinking (P for interaction = 0.02).

245 NSCLC, and with a greater number of years of alcohol drinking (P=0.02) in HNSCC.

246 ween the number of drinks consumed during an alcohol drinking paradigm (ADP) before and after 1 week

247 periods, separated by a 6-day washout, and 3 alcohol drinking paradigm (ADP) sessions.

248 a for alcohol dependence participated in two alcohol drinking paradigms (ADPs) separated by a week of

249 human Met66BDNF allele (Met68BDNF) and used alcohol-drinking paradigms in combination with viral-med

250 e two common genetic variants greatly affect alcohol drinking patterns.

251 mer2-PI3K signaling predisposes a high binge alcohol-drinking phenotype.

252 mily history of diabetes, cigarette smoking, alcohol drinking, physical activity, and diet.

253 domestic product; and prevalence of smoking, alcohol drinking, physical inactivity, obesity, diabetes

254 bottle choice paradigm that models excessive alcohol drinking produces a mobilization of DLS p75 neur

255 stores dopaminergic dysfunction in long-term alcohol-drinking rats and shows promise as a novel treat

256 At 48 h of withdrawal, alcohol-drinking rats showed anxiety- and depression-lik

257 etic stimulation of mPFC-NAcore terminals in alcohol-drinking rats, similar to reported effects of th

258 Our analysis suggests that chronic alcohol drinking, regardless of dose alters resting tran

259 -preferring, alcohol-nonpreferring, and high-alcohol-drinking replicate 1 line of rats (Indiana Unive

260 ring protracted withdrawal from intermittent alcohol drinking resulted in enhanced prefrontal cortex

261 Chronic alcohol drinking results in a significant increased meth

262 BP5 inhibitors were administered on the last alcohol drinking session and prior to each trauma-relate

263 ogether, our results indicate that the first alcohol drinking session induces synaptic plasticity in

264 hibition of mTORC1 activity during the first alcohol drinking session reduced alcohol consumption and

265 sex-specific mechanisms that drive excessive alcohol drinking.SIGNIFICANCE STATEMENT Estrogen has pot

266 t FGF2 in the DMS is a positive regulator of alcohol drinking.SIGNIFICANCE STATEMENT Long-term alcoho

267 For different smoking and alcohol drinking status, only subjects who are both curr

268 Smoking and alcohol-drinking status were not associated with unilate

269 ion in the mPFC increased aversion-resistant alcohol drinking, supporting a mechanistic role of Syt2

270 isk of developing uncontrolled and excessive alcohol drinking that can be reversed by directly activa

271 lectively blocks the enhanced motivation for alcohol drinking that develops in alcohol dependence lik

272 ice were tested in two models of free-choice alcohol drinking: the continuous and intermittent access

273 inking in the dark (DID) as a model of binge alcohol drinking to assess its effects on spike timing-d

274 ting robust and reproducible stress-enhanced alcohol drinking to examine the role of dynorphin/kappa

275 titis B virus, cirrhosis, diabetes, obesity, alcohol drinking, tobacco smoking, and host genetic poly

276 on of diabetes, diabetes treatment, smoking, alcohol drinking, total energy intake, and physical acti

277 behavior-at age 18 years was associated with alcohol drinking trajectories over 3 years, in this stud

278 ealthy behaviors: current smoking, high-risk alcohol drinking, unhealthy weight, physical inactivity,

279 l Ox1R inhibition did not alter female mouse alcohol drinking, unlike in males.

280 nd long term (30 years) associations between alcohol drinking (volume and hazardous drinking) and sle

281 Age, sex, education, smoking status, alcohol drinking, waist circumference, dental visit freq

282 ogic characteristics, cigarette smoking, and alcohol drinking was abstracted from hospital charts.

283 In contrast, heavy alcohol drinking was associated with higher cardiovascul

284 Current and former alcohol drinking was associated with higher odds among o

285 We found that alcohol drinking was initially diminished in alphaCaMKII

286 serum LPS, sCD14, sCD163 and the quantity of alcohol drinking was observed after adjusting for covari

287 Conversely, alcohol drinking was predicted by gonadal phenotype inde

288 ng continuous or intermittent access or when alcohol drinking was tested in the context of aversive o

289 ing in the rural area, cigarette smoking and alcohol drinking were associated with insomnia.

290 relations with total amount and frequency of alcohol drinking were more evident among women.

291 eptidase (GGTP), a liver enzyme indicator of alcohol drinking, were determined.

292 y by a selective receptor antagonist reduces alcohol drinking, when given systemically or directly in

293 ) are critical for promoting compulsion-like alcohol drinking, where rats consume alcohol despite pai

294 r binding exhibited a 50% reduction in binge alcohol drinking, which was related to reduced NAC basal

295 e within this brain region induces excessive alcohol-drinking, which reflects a selective insensitivi

296 deprivation effect (ADE) model in long-term alcohol drinking Wistar rats, two behavioral models for

297 ted that the beneficial associations between alcohol drinking with meals and T2D were mainly driven b

298 ation of alcohol consumption and duration of alcohol drinking with type 2 diabetes mellitus among Chi

299 Systemic D-serine reduced aversion-resistant alcohol drinking, without altering consumption of quinin

300 e duct cancers) in 69,310 nonsmoking and non-alcohol-drinking women.