ライフサイエンスコーパス: alcohol intoxication

1 rapeutic compounds in the treatment of acute alcohol intoxication.

2 ver-damaging diseases, such as infection and alcohol intoxication.

3 characterized by repeated episodes of heavy alcohol intoxication.

4 to obtain ethanol, and are more sensitive to alcohol intoxication.

5 s may contribute to the cognitive effects of alcohol intoxication.

6 y United States police officers to determine alcohol intoxication.

7 insensitivity to the aversive properties of alcohol intoxication.

8 nduced hepatic TNF-alpha production in acute alcohol intoxication.

9 ssion in AcbC, CeMPV and EW and the level of alcohol intoxication.

10 , in the initiation of hepatic injury during alcohol intoxication.

11 e warranted for treating PWID who experience alcohol intoxication.

12 iation of regular drinking, and age at first alcohol intoxication.

13 els in alcoholic pancreatitis but not during alcohol intoxication.

14 he ED, especially in the setting of drug and alcohol intoxication.

15 atients admitted to intensive care for acute alcohol intoxication.

16 r (AUD) differ in their sensitivity to acute alcohol intoxication.

17 dicated to caring for patients with drug and alcohol intoxication.

18 cascade that plays a critical role in acute alcohol intoxication.

19 ependent cardiac dysfunction following acute alcohol intoxication.

20 this hypothesis, we compared the effects of alcohol intoxication (0.75 g/kg alcohol vs placebo) on b

21 o-controlled cross-over design with moderate alcohol intoxication (~0.6 g/kg), 35 young healthy adult

22 Median FAEEs were similarly elevated during alcohol intoxication (205 nmol/L; 95% confidence interva

23 ched in circulation during moderate-to-heavy alcohol intoxication (50-100 mM) modify Ca(2+)- and volt

24 erate consensus for investigation: depicting alcohol intoxication (73%; 35/48), violating patient con

25 .21; P < 0.001), CVD (AHR, 0.70; P = 0.001), alcohol intoxication (AHR, 0.52; P = 0.003), and violenc

26 with an earlier onset and increased risk of alcohol intoxication, alcohol-related problems during la

27 Alcohol intoxication alters coordination and motor skill

28 n is often implicated in pathology following alcohol intoxication and burn injury.

29 association between the subjective degree of alcohol intoxication and changes in [(11)C]PBR28 V(T).

30 rified fatty acids (NEFAs) precursors during alcohol intoxication and clinical alcoholic pancreatitis

31 BARs as critical targets underlying low-dose alcohol intoxication and demonstrate that subtle changes

32 or the development of novel therapeutics for alcohol intoxication and dependence.

33 cellular mechanism of DHM for counteracting alcohol intoxication and dependence.

34 and microvascular leakage following combined alcohol intoxication and hemorrhagic shock, in a dose-re

35 rinking, despite greater behavioral signs of alcohol intoxication and reduced anxiety, and are insens

36 is one of the central proteins that mediate alcohol intoxication and tolerance across species.

37 the hypothalamic-pituitary-adrenal axis) by alcohol intoxication and withdrawal and consequent alter

38 opulation has been explained as misdiagnosed alcohol intoxication and withdrawal effects mistaken for

39 tionship between AUD and chronic pain across alcohol intoxication and withdrawal.

40 ducation level, marital status, age at first alcohol intoxication, and history of other psychiatric o

41 rs following prolonged exposure to cycles of alcohol intoxication, and is associated with persistent,

42 Neuronal mechanisms underlying alcohol intoxication are unclear.

43 the excellent performance of our device for alcohol intoxication assessment in real-time.

44 anol and test sensor system capabilities for alcohol intoxication assessment.

45 Alcohol intoxication at early ages is a risk factor for

46 Clinical and animal studies demonstrate that alcohol intoxication at the time of injury worsens postb

47 vascular disturbances, extra-axial injuries, alcohol intoxication at time of injury, and presence of

48 Alcohol intoxication can impact learning and this may co

49 Hangovers resulting from alcohol intoxication can lead to adverse effects ranging

50 Alcohol intoxication can lead to both positive and negat

51 Alcohol intoxication caused liver injury in association

52 che, vomiting, an age over 60 years, drug or alcohol intoxication, deficits in short-term memory, phy

53 tration six times lower than the legal blood-alcohol intoxication (driving) limit in most states (0.0

54 ase (CVD); respiratory disorders; neoplasms; alcohol-intoxication; drug intoxication; and violence-re

55 social cognition assessments when examining alcohol intoxication effects.

56 is commonly used in cases of suspected acute alcohol intoxication, especially in the emergency room.

57 Our results show that alcohol intoxication exacerbated hemorrhagic shock and r

58 Alcohol intoxication has a dangerous effect on human hea

59 We directly investigated how alcohol intoxication impacts hemorrhagic shock and resus

60 Alcohol intoxication impairs neutrophil function and inc

61 paired ability to recognize modest levels of alcohol intoxication in a range of life situations.

62 Attributing low GCS to alcohol intoxication in TBI patients may delay necessary

63 creener, with each alcohol outcome (incident alcohol intoxication, incident alcohol-related problems,

64 Acute alcohol intoxication inhibited the increase in the numbe

65 posure on the granulopoietic response, acute alcohol intoxication (intraperitoneal injection of 5 g a

66 l evidence indicates that one consequence of alcohol intoxication is a reduction in retinoic acid (RA

67 During the binge/intoxication stage, alcohol intoxication leads to a faster sleep onset, but

68 Therefore, acute alcohol intoxication leads to decreased MRSA clearance i

69 was derived from factor analysis of lifetime alcohol intoxications, lifetime cannabis use, use of nic

70 ions during implementation (860 [73.0%] with alcohol intoxication; median breath or blood concentrati

71 ization for alcoholic pancreatitis (n = 31), alcohol intoxication (n = 25), and in normal controls (n

72 rgency inpatient or outpatient treatment for alcohol intoxication or misuse (HR = 1.98, 95% CI 1.76-2

73 eliorate the microvascular leakage following alcohol intoxication plus hemorrhagic shock and resuscit

74 Among young men, low sensitivity to alcohol intoxication predicts subsequent alcohol abuse a

75 It is commonly assumed that alcohol intoxication reduces GCS, thus limiting its util

76 significant reduction in frequent past-year alcohol intoxication self-reported by men (adjusted odds

77 Alcohol intoxication strongly attenuates early visual re

78 complications reported in adults with acute alcohol intoxication, such as electrolyte disturbances a

79 Alcohol intoxication suppressed ERK activation in granul

80 Chronic alcohol consumption plus acute alcohol intoxication suppressed the increase in blood gr

81 During alcohol intoxication, the human brain increases metaboli

82 Repeated alcohol intoxications trigger inflammatory processes and

83 Acute alcohol intoxication was induced in BALB/c mice 30 min b

84 While alcohol intoxication was significantly associated with f

85 presenting for any cause and presenting with alcohol intoxication were eligible (N = 2108); 1764 were

86 'll feel queer" regarding moderate-to-severe alcohol intoxication, whereas subjective signs of progre