ライフサイエンスコーパス: alcohol problem

1 to the propensity for excessive drinking and alcohol problems.

2 ned, 57% met criteria for moderate to severe alcohol problems.

3 y influenced and predicts heavy drinking and alcohol problems.

4 n that limiting outlet density will decrease alcohol problems.

5 g was found in former drinkers with lifetime alcohol problems.

6 ntrations are important predictors of future alcohol problems.

7 % of nonremitters had received treatment for alcohol problems.

8 f worry and the risk for incident reports of alcohol problems.

9 ent of depression might reduce risk of later alcohol problems.

10 s, particularly with less severe and chronic alcohol problems.

11 tential to produce benefits in patients with alcohol problems.

12 d contributes to the onset and escalation of alcohol problems.

13 e child generation, and polygenic scores for alcohol problems.

14 in young adults, which might promote future alcohol problems.

15 ons treatment, (2) experience fewer drug and alcohol problems, (3) report fewer physical symptoms rel

16 a KS patients (n = 366) to be diagnosed with alcohol problems (43% v 18%), noninjection drug use (45%

17 ecause of worry predicted the development of alcohol problems among respondents with lifetime anxiety

18 iteria included substantial comorbid drug or alcohol problems and people who lacked capacity to conse

19 ncorporated prior depressive symptoms, prior alcohol problems and sociodemographic variables (age, ra

20 of DA, criminal activity, and psychiatric or alcohol problems) and an environmental risk index (inclu

21 s, including consumption, drinking patterns, alcohol problems, and clinical sequelae.

22 arental college education, family history of alcohol problems, and college status.

23 ave confirmed that adolescents with drug and alcohol problems are also beset by sleep problems, and h

24 Alcohol problems are not consistently managed in hospita

25 ry of divorce, death, criminal activity, and alcohol problems, as well as an adoptive sibling history

26 ol exposure is significantly associated with alcohol problems at 21 years of age.

27 They also reported greater decreases in alcohol problems at follow-up (group x time interaction:

28 ive sibling history of DA and psychiatric or alcohol problems) both strongly predicted the risk for D

29 sessment and treatment services not only for alcohol problems but also for drug use and the other psy

30 al nicotine exposure was not associated with alcohol problems by offspring at 21 years of age.

31 ys in which internists can aid patients with alcohol problems by screening, giving brief advice, and

32 Screening and intervention for alcohol problems can reduce drinking and its consequence

33 Counselling for Alcohol Problems (CAP), a brief intervention delivered b

34 ss and cost-effectiveness of Counselling for Alcohol Problems (CAP), a brief psychological treatment

35 Latent growth curve modeling revealed that alcohol problems decreased over time (slope = -0.059; 95

36 ole of fetal exposure and the development of alcohol problems deserve study.

37 y discounting behavior and family history of alcohol problems, drug problems, depression, mania, schi

38 hat depressive symptoms predicted subsequent alcohol problems for females, whereas alcohol problems p

39 k of confidentiality may deter soldiers with alcohol problems from accessing treatment.

40 n part, the transmission of genetic risk for alcohol problems from parents to children to predict ear

41 Hospital patients with alcohol problems had a much greater risk for alcoholic l

42 CMP, a combination of painful conditions and alcohol problems had the largest effect size on mortalit

43 t or sibling with a history of alcoholism or alcohol problems) had 49% higher odds of obesity than th

44 xcellent sensitivity to the heterogeneity of alcohol problems have been developed; (5) evidence that

45 al relations between depressive symptoms and alcohol problems have been examined infrequently in comm

46 advances in the assessment and treatment of alcohol problems have the following important implicatio

47 may be a risk factor for the development of alcohol problems in humans.

48 suggests enhanced screening and treatment of alcohol problems in PWH is warranted.

49 ls of alcohol intake and a greater number of alcohol problems in the 10 years preceding the diagnosis

50 ors at age 13 to 19 included psychiatric and alcohol problems in the surviving parent, receipt of soc

51 Given the prevalence of alcohol problems in trauma centers, screening, intervent

52 Screening and brief intervention for alcohol problems in trauma patients is cost-effective an

53 Measures of alcohol problems (in the previous year) incorporated an

54 s the clinical presentation of patients with alcohol problems including screening, diagnosis, detoxif

55 tients with an initial hospital contact with alcohol problems (intoxication, harmful use, or dependen

56 Voluntary, community-based screening for alcohol problems is feasible and offers education, scree

57 rstandings of health risks and stigma around alcohol problems may explain why public health messages

58 dence, in whom CCM was associated with fewer alcohol problems (mean score, 10 vs 13; incidence rate r

59 activation mediated an effect of genotype on alcohol problems (n = 104).

60 ependent of the effects of family history of alcohol problems, nicotine exposure, other prenatal expo

61 Chronically homeless individuals with severe alcohol problems often have multiple medical and psychia

62 ory of homelessness, imprisonment, drug use, alcohol problems or mental health problems.

63 Many of those with lifetime alcohol problems or posttraumatic stress disorder no lon

64 substance use patterns and in the course of alcohol problems over 1 year.

65 es, depressive symptoms predicted subsequent alcohol problems over 3 years (odds ratio = 3.04, 95% co

66 equent alcohol problems for females, whereas alcohol problems predicted subsequent depressive symptom

67 isordered individual (e.g., relative with an alcohol problem) reported lower perceived stigma (beta =

68 completed a sleep questionnaire, measures of alcohol problem severity and depression severity, and po

69 n reducing hazardous alcohol use and reduced alcohol problem severity when 2 or more sessions were de

70 The primary outcome was change in alcohol problem severity, measured with the AUDIT total

71 screening, intervention, and counseling for alcohol problems should be routine.

72 t apparent in patients with mild to moderate alcohol problems (SMAST score 3 to 8); they had 21.6+/-4

73 onclusion, parents transmit genetic risk for alcohol problems to their children not only directly, bu

74 Family history of alcohol problems was assessed from interviews with paren

75 Alcohol problems were associated with death from digesti

76 nt home environment mediate genetic risk for alcohol problems within families across generations.