ライフサイエンスコーパス: alexia

1 s afferents impairs word recognition ("pure" alexia).

2 ing text reading are disrupted ("hemianopic" alexia).

3 82 patients had anomia, and 23 patients had alexia.

4 ve been extended to patients with hemianopic alexia.

5 ects in participants with poststroke central alexia.

6 portions of these tracts is associated with alexia.

7 nts might improve our diagnostic approach to alexia.

8 ontext of semantic dementia underlie surface alexia.

9 same area was damaged in a patient with pure alexia ("alexia without agraphia") and no hemianopia, wh

10 reading training in nine patients with pure alexia, an acquired reading disorder caused by damage to

11 en normal subjects, patients with hemianopic alexia and patients with an incomplete right homonymous

12 , we focus on the classification of acquired alexia and the current theory and practice underlying th

13 toms including right homonymous hemianopsia, alexia, and simultanagnosia with preserved memory.

14 that dictate reading ability in the acquired alexias are becoming better understood; this, in turn, h

15 auses loss of semantic knowledge and surface alexia as a syndromic diagnosis is unusual after stroke,

16 Central alexia (CA) is an acquired reading disorder co-occurring

17 ed therapy site for patients with hemianopic alexia, called Read-Right.

18 aches to the rehabilitation of patients with alexia caused by stroke, there is still a preponderance

19 ural systems damaged in the various forms of alexia favours a rather different view of reading and th

20 reading in normal subjects and patients with alexia following a left occipital infarct, using PET.

21 A left occipital stroke may result in alexia for two reasons, which may coexist depending on t

22 iour of patients with established hemianopic alexia (>6 months post deficit), a condition affecting l

23 Diagnosis of alexia has historically focused on syndromes such as sur

24 stroke, semantic reading deficits in stroke alexia have not previously been examined systematically.

25 , based on studies of patients with acquired alexia, hypothesizes functional linkages between the ang

26 elative contributions of pure and hemianopic alexia in individual patients needs to be assessed, as t

27 We studied two patients with progressive alexia in order to determine whether their reading defic

28 In patients with pure alexia, iReadMore increased feedback from left inferior

29 the commonest deficit associated with "pure" alexia is a right homonymous field defect; an impairment

30 Progressive alexia is an acquired reading deficit caused by degenera

31 Our results suggest that progressive alexia is associated with a dysfunctional visual word fo

32 in reading network in many cases of acquired alexia is mirrored by its functional disconnection in de

33 he functional deficit is controversial, pure alexia is the result of an inability to map a percept of

34 nt of either reading or naming, resulting in alexia or anomia.

35 urologist Jules Dejerine suggested that pure alexia resulted from an occipital lesion that selectivel

36 neither of the two patients with progressive alexia showed any evidence for a selectivity gradient or

37 gh multivariate, process-based approaches to alexia.SIGNIFICANCE STATEMENT Whereas fMRI evidence for

38 In 'pure' alexia, techniques are usually aimed at improving whole-

39 In hemianopic alexia, the most 'peripheral' of the acquired alexias, w

40 lexia, the most 'peripheral' of the acquired alexias, where text reading speed is determined largely

41 rically focused on syndromes such as surface alexia, which capture discrete patterns of reading defic

42 was damaged in a patient with pure alexia ("alexia without agraphia") and no hemianopia, who read wo

43 Two patients had reading-provoked paroxysmal alexia without motor symptoms, associated with prolonged