ライフサイエンスコーパス: allergic reaction

1 No children developed allergic reaction.

2 s is a potentially life-threatening systemic allergic reaction.

3 olymerization of protein, which may alter an allergic reaction.

4 naphylaxis is a potentially life-threatening allergic reaction.

5 ssarily experience severe symptoms during an allergic reaction.

6 ly given during the provocation phase of the allergic reaction.

7 sensitization and elicitation phases of the allergic reaction.

8 ts received influenza vaccination without an allergic reaction.

9 influenza vaccination without an allergic reaction.

10 ases referred with a suspected perioperative allergic reaction.

11 ssess the contribution of pollensomes to the allergic reaction.

12 llergens, with potential implications in the allergic reaction.

13 s, which might have been due an IgE-mediated allergic reaction.

14 phils and CD4(+) T cells, all features of an allergic reaction.

15 they displayed decreased chemically induced allergic reaction.

16 xis is a severe, potentially fatal, systemic allergic reaction.

17 Twenty patients experienced >1 systemic allergic reaction.

18 ross the intestinal monolayer and trigger an allergic reaction.

19 od allergens that are capable of inducing an allergic reaction.

20 ) syndrome, which is characterized by severe allergic reactions.

21 s a frequent elicitor of severe IgE-mediated allergic reactions.

22 winia) substitution was approved in 2011 for allergic reactions.

23 of inflammatory mediators that contribute to allergic reactions.

24 peanut, but not placebo, during acute peanut allergic reactions.

25 licy was associated with complete absence of allergic reactions.

26 Mast cells are key effector cells in allergic reactions.

27 RII mediates cell type-dependent function in allergic reactions.

28 There were no reports of severe systemic allergic reactions.

29 f food allergens may be a key determinant of allergic reactions.

30 s histamine and leukotrienes, which initiate allergic reactions.

31 fy those most at risk of severe food-induced allergic reactions.

32 irculating factor responsible for triggering allergic reactions.

33 oglobulin G (IgG) plays a regulatory role in allergic reactions.

34 FcepsilonRI plays an important role in acute allergic reactions.

35 ter enrolment of 3232 patients due to severe allergic reactions.

36 se excipient of Inavir could cause immediate allergic reactions.

37 ergen in Mediterranean areas causing diverse allergic reactions.

38 rently formulated, is associated with severe allergic reactions.

39 wan, and 60% of the exposed subjects develop allergic reactions.

40 weight compounds that modulate and stimulate allergic reactions.

41 There were no thrombotic events or severe allergic reactions.

42 bsolute requirement for IgE glycosylation in allergic reactions.

43 sible for most life-threatening food-induced allergic reactions.

44 acerbation of experimental IgE-mediated food allergic reactions.

45 od allergen triggers and acute management of allergic reactions.

46 Poultry meat can induce severe allergic reactions.

47 th canonical and TI variants elicit clinical allergic reactions.

48 mast cells contribute to the development of allergic reactions.

49 e for endophthalmitis because of concern for allergic reactions.

50 ecially at hapten threshold levels eliciting allergic reactions.

51 not L-ASP, is the major antigen that causes allergic reactions.

52 elial barrier integrity in profilin-mediated allergic reactions.

53 is, a severe life-threatening consequence of allergic reactions.

54 lts could lead to severe or life-threatening allergic reactions.

55 -food allergies often associated with severe allergic reactions.

56 asparaginase group) and asparaginase-related allergic reactions (14 [6%] vs 6 [3%]).

57 king the study powder because of a confirmed allergic reaction (25 of 407 [6.1%] compared with 6 of 4

58 oteins and lipids, causes an unusual delayed allergic reaction 3 to 6 hours after ingestion of mammal

59 -Cefaz) as well as the greatest frequency of allergic reactions (3.0% vs 2.4% for Hx-Cefaz and 1.7% f

60 ritic skin lesions, caused by type-I/type-IV allergic reactions accompanied by prominent eosinophil i

61 eters to visualize and quantify the regional allergic reaction after segmental endobronchial allergen

62 She also developed a systemic allergic reaction after sting from an unknown creature w

63 ulin E (IgE) results in the life-threatening allergic reaction anaphylaxis.

64 dy aims to compare estimation of the risk of allergic reaction and associated uncertainty using diffe

65 ents' age, ethnicity, nature of the putative allergic reaction and coexisting clinical diseases such

66 t the optimal method to estimate the risk of allergic reaction and the associated uncertainty.

67 The severity of the allergic reaction and the threshold dose during OFCs to

68 Mast cells and basophils are main drivers of allergic reactions and anaphylaxis, for which prevalence

69 nisms by studying mast cells, key players in allergic reactions and anaphylaxis.

70 Reported allergic reactions and consumption of tree nuts and sesa

71 se and the cause of severe, life-threatening allergic reactions and death.

72 ute amount of contamination may cause strong allergic reactions and even death.

73 aucity of new therapeutic targets to control allergic reactions and forestall the rising trend of all

74 Ig isotype, yet it plays a critical role in allergic reactions and host protection from helminth inf

75 is an important immunomodulator involved in allergic reactions and inflammatory responses.

76 There were no infusional or allergic reactions and no difference in treatment emerge

77 There were no infusional or allergic reactions and no difference in treatment-emerge

78 the commonest cause of life-threatening food-allergic reactions and significantly affect quality of l

79 to be used to provide information regarding allergic reactions and therefore potentially aid in more

80 s, shrimps are the most predominant cause of allergic reactions and thus more extensively studied.

81 ved HCP advice/support following their worst allergic reaction, and 28% had not been prescribed an ad

82 llergic swelling (mouth or throat), systemic allergic reactions, and asthma-related treatment-related

83 Mast cells are key effectors in allergic reactions, and decades of research have suggest

84 with eosinophilic disorders, such as asthma, allergic reactions, and fungal and helminthic infections

85 mmune cells associated with type 2 immunity, allergic reactions, and host defense against parasite in

86 Mast cells are primary effectors in allergic reactions, and may have important roles in dise

87 e-resident immune cells that are involved in allergic reactions, and their numbers are increased in t

88 IgE antibodies may elicit potent allergic reactions, and their production is tightly cont

89 orrhage (anticoagulants), moderate to severe allergic reactions (antibiotics), and hypoglycemia with

90 Late allergic reactions are common in the course of allergen-

91 nce and preparedness for prompt treatment of allergic reactions are considered dual standard of care.

92 is consistent with the observation that most allergic reactions are in connection with roasted peanut

93 peanuts are frequently consumed, but severe allergic reactions are rare.

94 Since allergic reactions are triggered by mast cell-mediated h

95 reactions against microbial pathogens, host allergic reactions, as well as tissue repair.

96 yet almost none will experience a fatal food-allergic reaction; asthma is not, in itself, a strong pr

97 ation >= 30 mm Hg (to 37 mm Hg) and 1 had an allergic reaction attributed to the study drug; each res

98 stressful for the patient and avoids severe allergic reactions, BAT ought to precede challenge testi

99 chanistic studies reveal that the ability of allergic reaction blockade by the low-affinity anti-IgE

100 t mediator of protective immunity as well as allergic reaction, but how high affinity IgE antibodies

101 otent as omalizumab at blocking IgE-mediated allergic reactions, but does not induce FcgammaR-depende

102 MCs) are best known as key effector cells of allergic reactions, but they also play an important role

103 ficient suppression of FcepsilonRI-dependent allergic reactions by ligelizumab in vitro as well as in

104 Immunoglobulins E (IgEs) trigger allergic reactions by specifically binding the allergens

105 An acute MC-dependent allergic reaction can lead to systemic shock, but the ea

106 Allergic reactions can be considered as maladaptive IgE

107 Allergic reactions can range from mild local symptoms to

108 Anaphylaxis is a life-threatening allergic reaction caused by cross-linking of high-affini

109 Food-induced anaphylaxis is a serious allergic reaction caused by Fcepsilon-receptor activatio

110 scribed the frequencies and risk factors for allergic reactions caused by accidental allergen ingesti

111 Most agents typically cause IgE-mediated allergic reactions, causing a distinct form of food alle

112 Immunoglobulin (Ig)E antibodies mediate the allergic reaction characterized by immediate hypersensit

113 We demonstrate that the magnitude of the allergic reaction correlates with the number of CD8(+) e

114 25% of the children experienced an allergic reaction due to a non-pre-packed food from bake

115 ed to estimate the severity and threshold of allergic reactions during OFCs.

116 However, not all allergic reactions end up in the most severe form of ana

117 environmental chamber to stimulate an ocular allergic reaction followed by application of artificial

118 om allergy is a potentially life-threatening allergic reaction following a honeybee, vespid, or ant s

119 ent Reporting System has received reports of allergic reactions following immunization with egg-free

120 These results suggest that allergic reactions following influenza vaccination are n

121 ntify the allergens responsible for systemic allergic reactions following ingestion of pizza in two p

122 lactose-containing dry powder inhalers cause allergic reactions for patients with cow's milk allergy

123 ll differences in sensitization and reported allergic reactions for select tree nuts, with levels bei

124 ied to automate the process of measuring the allergic reactions found by developing systems and algor

125 number of physiological processes including allergic reactions, gastric acid secretion, neurotransmi

126 diator involved in the hypersensitivity-type allergic reaction has gained greater recognition.

127 Food-mediated allergic reactions have emerged as a major health proble

128 Mechanisms driving acute food allergic reactions have not been fully characterized.

129 nctional role as well as the implications on allergic reactions, however, are not clear yet.

130 s labor intensive and could result in severe allergic reactions; however, it reduced dietary restrict

131 s are significantly involved in IgE-mediated allergic reactions; however, their roles in health and d

132 Outcome measures included allergic reactions (ie gastrointestinal, dermatological,

133 from all studies, the eliciting dose for an allergic reaction in 1% of the population estimated for

134 for exposure to an allergen to result in an allergic reaction in specific IgE-positive patients.

135 d in milk are IgE reactive and can induce an allergic reaction in vitro.

136 rate that EPIT markedly reduced IgE-mediated allergic reactions in a mouse model of cashew allergy, w

137 ypoallergenic Cyp c 1 mutant protect against allergic reactions in a murine model of fish allergy.

138 might be responsible for the unusual delayed allergic reactions in alpha-Gal allergic patients and id

139 sponses in lungs and to exacerbate pulmonary allergic reactions in animal models.

140 (CM) formulas are available for avoidance of allergic reactions in CM-allergic children and for preve

141 entomophagy has unfortunately been linked to allergic reactions in Europe with people with profession

142 Because BSA can cause allergic reactions in humans the World Health Organizati

143 s on rates of epinephrine administration for allergic reactions in Massachusetts public schools.

144 is, Gal d 1, is associated with IgE-mediated allergic reactions in most egg-allergic children.

145 al irritants but that also trigger exuberant allergic reactions in patients with allergies.

146 ped us to estimate the severity and types of allergic reactions in patients.

147 ion of time in school but characteristics of allergic reactions in schools are not well studied.

148 can represent a potential risk for eliciting allergic reactions in sensitised individuals.

149 We sought to characterize severe allergic reactions in terms of elicitors, symptoms, emer

150 In contrast, it might result in frequent allergic reactions in the community considering that man

151 nts investigated for suspected perioperative allergic reactions in the Danish Anaesthesia Allergy Cen

152 Ps) represent a major cause of systemic food allergic reactions in the Mediterranean area.

153 ability to severe outcomes from food-induced allergic reactions in the second and third decades.

154 It has been associated with allergic reactions in the skin and lungs but also homeos

155 There were no documented allergic reactions in this cohort of patients with syste

156 ystemic anaphylaxis, the most severe form of allergic reaction, in which an acute vascular response p

157 with strong desaturation after an immediate allergic reaction including erythema, abdominal pain, vo

158 ed 12-week MSSA cure, recurrence, and death; allergic reactions including major, minor, and potential

159 hrombotic or thromboembolic events, systemic allergic reactions (including anaphylaxis), or neutralis

160 e the secular trends of hospitalizations for allergic reactions, including anaphylaxis, among childre

161 lar trend of hospitalizations for paediatric allergic reactions, including anaphylaxis, are scarce.

162 individuals to develop severe IgE-associated allergic reactions, including fatal anaphylaxis, on subs

163 ronic inflammatory skin disease triggered by allergic reactions involving IgE antibodies directed tow

164 An allergic reaction is rapidly generated when allergens bi

165 ntifying the allergens responsible for these allergic reactions is challenging because of the high an

166 gnosis to prevent potential life-threatening allergic reactions is crucial.

167 Anaphylaxis, a life-threatening allergic reaction, is dependent on high affinity allerge

168 ntal (47.6%) and self (21.8%) concerns about allergic reactions, lack of referrals (33.6%), parents u

169 the time course of the early (EAR) and late allergic reaction (LAR).

170 The incidence of allergic reactions leading to hospitalizations between 1

171 vities to avoid the annoyingly intense itchy allergic reactions, leading to significant worsening of

172 Blue dye has, however, been associated with allergic reactions, long-term staining of skin, and incr

173 y sources of mediators responsible for acute allergic reactions, notably including anaphylaxis, a sev

174 ted; 1 nonserious treatment-related systemic allergic reaction occurred (assessed as moderate intensi

175 with PE was 0.01 mg, while at 0.3 mg MPE no allergic reaction occurred.

176 local allergic swellings or serious systemic allergic reactions occurred in subjects with asthma trea

177 to 4500 mg of peanut protein until objective allergic reactions occurred.

178 ed symptoms depend on the organ in which the allergic reaction occurs but can include red itchy eyes,

179 anut allergy (PA), but it involves a risk of allergic reactions of unpredictable severity.

180 an trigger fatal outcomes, but the impact of allergic reactions on the cardiovascular system in nonfa

181 oxicillin and clavulanic acid reported other allergic reactions, one of which was reported as a serio

182 cells and prevent allergies without inducing allergic reactions or allergic sensitization.

183 No patients exhibited any systemic or local allergic reactions or complications after intravitreal i

184 We observed no allergic reactions or deaths at 30 day follow-up.

185 There were no acute allergic reactions or nonsignificant excesses of 6-month

186 No investigator-assessed anaphylactic allergic reactions or reactions requiring epinephrine we

187 was no evidence of increased TEAEs, systemic allergic reactions, or severe local allergic swellings i

188 It is a pleiotropic cytokine involved in allergic reactions, parasitic infections, autoimmune inf

189 1999 and 2011 with the primary diagnosis of allergic reaction per International Classification of Di

190 is is the most severe and frightening of the allergic reactions, placing patients at high risk and de

191 beyond their classic role as master cells of allergic reactions, play a role in other settings, as we

192 Anaphylaxis is a systemic allergic reaction, potentially life-threatening that can

193 phase of a pan-European registry for severe allergic reactions provides for the first time data on a

194 s underlying the variable severity of peanut-allergic reactions remain unclear.

195 re were no serious adverse events, including allergic reaction, reported in either the antibiotic gro

196 press the recruitment of immune cells during allergic reactions, representing potential new drugs for

197 The antibiotic abuse may cause allergic reactions, resistance in microorganisms and gen

198 Peanut-induced allergic reactions resulted in a significant decrease in

199 ethods already predict the probability of an allergic reaction resulting from an accidental exposure

200 acute, severe, life-threatening multisystem allergic reaction resulting from the sudden systemic rel

201 ildren who were hospitalized due to systemic allergic reaction (SAR) and food anaphylaxis were recrui

202 immunomodulatory role in the development of allergic reactions such as asthma during environmental e

203 nt of cashew nut allergen may cause a severe allergic reaction, suggesting high potency comparable wi

204 dhood immunity culminating in sensitivity to allergic reactions, susceptibility to microbial infectio

205 emia, pancreatitis, pancreatic neoplasms, or allergic reactions than was placebo.

206 Anaphylaxis is a severe allergic reaction that can be lethal if not treated adeq

207 Anaphylaxis is a severe allergic reaction that can be rapidly progressing and fa

208 Anaphylaxis is a serious allergic reaction that can cause death; however, the act

209 g, life-threatening, generalized or systemic allergic reaction that is classically elicited by antige

210 Anaphylaxis is a severe allergic reaction that is rapid in onset and might cause

211 n acute, potential life-threatening systemic allergic reaction that may have a wide range of clinical

212 st cells (MCs) are central effector cells in allergic reactions that are often mediated by immunoglob

213 heir interactions with specific IgE, trigger allergic reactions that can be life threatening.

214 Several authors have reported on allergic reactions that resulted in presentation to the

215 s can contribute to the immunologic basis of allergic reactions, the molecular underpinnings associat

216 have focused on quantifying the severity of allergic reactions; the impact of food matrix, immunolog

217 However, since they could cause allergic reactions they are included in Allergen Control

218 l injections received PI prophylaxis without allergic reactions, thus demonstrating a lack of true PI

219 develops as the result of a non-IgE-mediated allergic reaction to a variety of organic materials or l

220 nine of 27 patients, and one patient had an allergic reaction to carboplatin.

221 acute infusional toxicity (attributed to an allergic reaction to dimethyl sulfoxide) in the 29 patie

222 her (1) a convincing history of an immediate allergic reaction to egg, milk, or both with a positive

223 Many patients developing an allergic reaction to fruit are diagnosed as having oral

224 with a self-reported egg allergy or previous allergic reaction to inactivated influenza vaccine.

225 tients with a documented history of systemic allergic reaction to labile plant food allergens and on

226 ed additional visits because they had a mild allergic reaction to metronidazole.

227 testing to predict the severity of a future allergic reaction to peanut.

228 sts they are at a low risk for developing an allergic reaction to penicillin.

229 if a particular patient has had an apparent allergic reaction to the medication, potential allergy t

230 mune tolerant (IT) if they had no detectable allergic reactions to a peanut oral food challenge after

231 tentially be applied to inhibit IgE-mediated allergic reactions to any drug/small-molecule allergy.

232 nificantly between the two groups, but fewer allergic reactions to asparaginase were observed in the

233 We studied 387 patients with immediate allergic reactions to beta-lactams and 1124 paired contr

234 Allergic reactions to beta-lactams are among the most fr

235 ncluded in the guideline are epidemiology of allergic reactions to beta-lactams, molecular structure,

236 We conducted a systematic review of (1) allergic reactions to botulinum antitoxin and (2) the pr

237 osures causing food allergy, such as delayed allergic reactions to carbohydrate moieties in mammalian

238 Perioperative allergic reactions to chlorhexidine are often severe and

239 Allergic reactions to drugs are a serious public health

240 Eighteen patients (28%) had a history of allergic reactions to egg, legumes, and nuts and strictl

241 ding perceptions of severe, life-threatening allergic reactions to food by different stakeholders, wi

242 venom-related anaphylaxis also occur during allergic reactions to food in 22 patients with peanut al

243 sed to identify those at most risk of severe allergic reactions to food, and the consequences of misi

244 this cohort have become especially aware of allergic reactions to food.

245 optimizing venous return during significant allergic reactions to food.

246 zes two workshops focused on the severity of allergic reactions to food.

247 e experience of patients suffering real-life allergic reactions to food.

248 de guidance to reduce the risk of accidental allergic reactions to foods in the community.

249 Allergic reactions to gelatin are comparatively rare, bu

250 dults with a history of rapid-onset systemic allergic reactions to honeybee stings and positive speci

251 Allergic reactions to Hymenoptera insect stings remain a

252 Eleven of the 16 CC strains had allergic reactions to intraperitoneal peanut challenge,

253 re conducted on sera from subjects reporting allergic reactions to mammalian meat or IFA.

254 Allergic reactions to mosquito bites are an increasing c

255 , aged 18-50 years with no history of severe allergic reactions to mosquito bites.

256 The aim of this study was to characterize allergic reactions to non-pre-packed foods and to invest

257 rved in school or brought from home reported allergic reactions to nuts.

258 Unexpected allergic reactions to peanut are the most common cause o

259 he discrepancy between IgE sensitization and allergic reactions to peanut could facilitate diagnosis

260 Biomarkers of severity and threshold of allergic reactions to peanut could significantly improve

261 o PA status, severity, and threshold dose of allergic reactions to peanut during OFC.

262 associated with the threshold (P = .020) of allergic reactions to peanut during OFCs.

263 Human allergic reactions to peanut proteins and the associated

264 sitivity is associated with the threshold of allergic reactions to peanut.

265 nges in cardiovascular function during acute allergic reactions to peanut.

266 Risk factors for allergic reactions to PEG-ASP remain unclear.

267 he management of immediate and non-immediate allergic reactions to penicillins and other beta-lactams

268 Central European patients displaying strong allergic reactions to plant-derived food.

269 E antibodies to alpha-gal and delayed type I allergic reactions to the carbohydrate alpha-gal after c

270 The cause might be allergic reactions to the gram-positive pathogen Staphyl

271 ood allergens and aeroallergens, or reported allergic reactions to tree nuts and sesame.

272 t allergen can result in diverse patterns of allergic reactions to various foods.

273 Allergic reactions to walnut can be life-threatening.

274 Hospitalizations for allergic reactions totalled 1987 in Finland and 5433 in

275 the basis of absence or presence of systemic allergic reactions upon sting challenge.

276 There were no systemic allergic reactions (upper 95% CI for population, 1.3%).

277 le the dynamic transcriptome of acute peanut allergic reactions using serial peripheral blood samples

278 lostridium difficile infection and potential allergic reactions (using epinephrine administration as

279 severe local allergic swelling, or systemic allergic reactions versus subjects without asthma.

280 t FcepsilonRII may play a role in regulating allergic reactions via modulating IFN-gamma production i

281 lerated, and only 1 patient with a suspected allergic reaction was noted.

282 Allergic reaction was triggered by a single intraperiton

283 Surgeons' concern for allergic reactions was associated with decreased rates o

284 One adverse event (an allergic reaction) was reported in 1 patient in the PPI

285 f cardiovascular changes during food-induced allergic reactions, we found evidence for significant fl

286 As mast cell mediators trigger allergic reactions, we here determine the function of TR

287 drawal (IT, n = 7), whereas those who had an allergic reaction were categorized as nontolerant (NT; n

288 ugh to August 2013, 1334 (0.51%) episodes of allergic reactions were detected, and 532 (0.20%) episod

289 Allergic reactions were mainly caused by food (children

290 Severe allergic reactions were reported in ten (1%) of 1605 pat

291 lls has been well documented in IgE-mediated allergic reactions, whereas other mast cell functions ar

292 food allergic people, the incidence of food allergic reactions which are coded as anaphylaxis by hea

293 gainst food proteins and a history of severe allergic reactions) who underwent specific food oral imm

294 Anaphylaxis is a serious systemic allergic reaction with rapid onset and potentially life-

295 of allergenic foods and management of acute allergic reactions with antihistamines and epinephrine a

296 consciousness are known to occur with severe allergic reactions with IgE-mediated food allergy, repor

297 This study investigates the risk of an allergic reaction within the milk-, wheat-, hazelnut- an

298 FA diagnosis was based on recognized allergic reactions within 4 hours after exposure to know

299 atory state, and is associated with dampened allergic reactions within the host.

300 is one of the most common elicitors of food-allergic reactions worldwide.