ライフサイエンスコーパス: allostimulation

1 hibited the response of recipient T cells to allostimulation.

2 -regulates DC activation and its function in allostimulation.

3 ells in the alloreactive pool after repeated allostimulation.

4 on occurs in all Vbeta expressing T cells by allostimulation.

5 on occurred in all Vbeta expressing cells by allostimulation.

6 pattern altered dramatically after repeated allostimulation.

7 l ligand/s (CD28 and/or CD154) could restore allostimulation.

8 duce normal cytokine levels upon third-party allostimulation.

9 ine expression following in vitro or in vivo allostimulation.

10 splenocytes and increased in both cases upon allostimulation.

11 r beta secretion, IgG production, and T-cell allostimulation.

12 However, IL-6(-/-) BMDC induced less T cell allostimulation and Ag-specific T cell activation, but o

13 xpression and subsequent alloanergization by allostimulation and concomitant blockade of CD28-mediate

14 ubset was less potent than GFP(-) pDC2s in T allostimulation and production of tumor necrosis factor

15 -3, were enhanced most by the combination of allostimulation and rATG, than either stimulus alone.

16 Tregs were phenotypically normal, anergic to allostimulation, and suppressed proliferation of allogen

17 biological activity following polyclonal or allostimulation; and 2) tumor-specific immunosuppression

18 cells, and that both ligands participate in allostimulation, autologous proliferation, as well as sp

19 <0.001) impaired the release of sCD30 during allostimulation but did not alter the levels of ADAM10 a

20 lood T lymphocytes (CBTL) respond to primary allostimulation but they do not proliferate upon rechall

21 han 97% of migratory LC in vitro and inhibit allostimulation by LC up to 95%.

22 expression, each subset displayed different allostimulation capability in mixed lymphocyte reaction

23 in precursor frequency in response to donor allostimulation compared with the untreated control grou

24 s exhibited enhanced IFN-gamma production to allostimulation compared with young naive T cells.

25 eactive T cell clones, we performed in vitro allostimulation cultures for a cohort of patients underg

26 After allostimulation, distinct proliferating Eomes(lo)CTLA4(h

27 2) We show that allostimulation has dichotomous effects on replication o

28 etectable levels during the first 48 h after allostimulation in MLCs.

29 In vivo allostimulation in the absence of IL-2 led to exaggerate

30 n vivo during anti-CD3/anti-CD28 stimulation/allostimulation in the late phase of the alloimmune resp

31 oantigen-specific anergy in donor T cells by allostimulation in the presence of costimulatory blockad

32 graft, produce both IFN-gamma and IL-4 after allostimulation in vitro, prohibit the expansion of allo

33 transfection of DC with caTLR4 RNA enhanced allostimulation of CD4(+) T cells.

34 Allostimulation of CD4+ and CD8+ T cells by BxCM-treated

35 CB with CTLA4Ig during allostimulation of CD8+ T cells reduced CTLA4 but not Eo

36 Allostimulation of human CD4(+) T cells showed that impr

37 sed an in vitro model of alloanergization by allostimulation of human donor T cells with irradiated u

38 , Tc, T-memory and CD28 receptors, following allostimulation of peripheral blood leukocytes from 20 n

39 sion pattern of miRs and mRNAs following the allostimulation of T cells and a high correlation betwee

40 In contrast, 24-hr allostimulation of T cells from naive mice resulted in I

41 minated blood, we have tested the effects of allostimulation on HIV-1 infection.

42 o induce naive Th-cell proliferation through allostimulation or anti-CD3 monoclonal antibody stimulat

43 sponse during anti-CD3/anti-CD28 stimulation/allostimulation, prevents Treg generation, inhibits Treg

44 Allostimulation results in the up-regulation of the T-ce

45 in BTLA+/+ donor cells after 1 wk of in vivo allostimulation was not observed in BTLA-/- donor CD4+ c

46 n mixed lymphocyte reactions (MLRs) to model allostimulation, we analyzed responder B cells using tra

47 Poor T cell responses to allostimulation were corrected by the activation and exp

48 CD8(+) T cells expanded by allostimulation were identified as CD8(+), CFSE(low) cel

49 ("alloanergization") in donor bone marrow by allostimulation with costimulatory blockade before haplo