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1                              Moreover, their allostimulatory abilities are impaired compared with tho
2 kocyte reactions were performed to assess DC allostimulatory ability and also the function of putativ
3 transduced ODN DCs exhibit markedly impaired allostimulatory ability and promote apoptosis of activat
4  gel shift assay results, and showed reduced allostimulatory ability of the CsA-treated cells compare
5 , adenosine-differentiated DCs have impaired allostimulatory activity and express high levels of angi
6                       They exhibit very poor allostimulatory activity and similar migration character
7 mined by flow cytometric analysis, and their allostimulatory activity assessed in primary mixed leuko
8 iated with marked impairment of their T-cell allostimulatory activity but with only modest prolongati
9 panded CD34+-selected cells showed increased allostimulatory activity compared to both cultured CD34+
10 0, CD80, CD86, and IAb, and augmented T cell allostimulatory activity compared with controls.
11 s) from PD-L1(-/-) mice exhibited a stronger allostimulatory activity compared with that in wild-type
12 ing DC differentiation significantly reduced allostimulatory activity in a MLR using naive CD4(+) T c
13 howed pronounced impairment (>80%) of T-cell allostimulatory activity in vitro.
14      In addition, PorB not only enhanced the allostimulatory activity of DC, but also augmented the a
15 nd CD86 expression and up-regulated the poor allostimulatory activity of the DCp assessed by MLR and
16                                              Allostimulatory activity of the gene-transduced DCp was
17 and costimulatory molecules and naive T cell allostimulatory activity than migratory wild-type (wt) C
18  costimulatory molecules (CD80 and CD86) and allostimulatory activity than those from controls.
19                                        Their allostimulatory activity was assessed in vitro by MLR an
20                                        Their allostimulatory activity was further diminished by addit
21 /CD14- cells, a 42-fold increase (median) of allostimulatory activity was observed as compared with s
22 nistration and the resulting augmentation of allostimulatory activity within host lymphoid tissue, is
23 ndocytosis properties, cell surface markers, allostimulatory activity, and cytokine production follow
24 acity to produce IL-12, did not acquire full allostimulatory activity, and rapidly underwent apoptosi
25 rIL-4 were functionally immature in terms of allostimulatory activity, but this activity increased af
26 immature phenotype was linked to poor T cell allostimulatory activity, indicative of DC progenitors.
27  immature DCs partially restored the reduced allostimulatory activity, whereas alcohol given only dur
28                           There was impaired allostimulatory activity.
29 icient in IFN-gamma still exhibited vigorous allostimulatory activity.
30 deed, Stat4(-/-) DC similarly displayed poor allostimulatory activity.
31 uclear translocation of RelB, and developing allostimulatory activity.
32 sed the impact of transgene expression on DC allostimulatory activity.
33 g HLA-DR, CD1a, CD83, CD80, CD86, and potent allostimulatory activity.
34 They also possess substantial phagocytic and allostimulatory activity.
35 nd epidermal cell suspensions were devoid of allostimulatory activity.
36  tolerogenic role of immature DC, the T cell allostimulatory and Th1-biasing function of CD14+ LC pre
37        rAd-transduced DC exhibited increased allostimulatory capacity and levels of interleukin-6 (IL
38 crophages, but acquire, upon maturation, the allostimulatory capacity and surface phenotype of classi
39 from gammadelta-/- recipients exhibited less allostimulatory capacity compared to wt DCs after irradi
40 L-treated animals exhibited increased T cell allostimulatory capacity compared with controls.
41 nally by production of IL-12 and an enhanced allostimulatory capacity in a MLR.
42                                      Reduced allostimulatory capacity in HCV core- and NS3-treated im
43 11c as determined by flow cytometry, and the allostimulatory capacity of 5azaD/TSA-expanded cells as
44 ether, our studies indicate that the reduced allostimulatory capacity of 5azaD/TSA-expanded cells is
45 of IL-10, during MLR ameliorated the reduced allostimulatory capacity of alcohol-treated DCs.
46 In summary, lack of CCR2 interferes with the allostimulatory capacity of DC and promotes the generati
47 on and G-CSF mainly acted on the suppressing allostimulatory capacity of DCs.
48                        We have separated the allostimulatory capacity of endothelium from that of hem
49 lta T cells exacerbate GVHD by enhancing the allostimulatory capacity of host antigen-presenting cell
50 d NS3 (not E2) inhibited differentiation and allostimulatory capacity of immature DCs similar to defe
51 In our current studies, we have assessed the allostimulatory capacity of the 5azaD/TSA-expanded graft
52                                       T-cell allostimulatory capacity of the DC, including DC exposed
53 cal to that of interdigitating DC and potent allostimulatory capacity were released from FSDDC-A with
54 d costimulatory molecule expression and poor allostimulatory capacity when interacting with T cells,
55  loci in germline configuration and show low allostimulatory capacity, but produce type I IFN upon vi
56 ich are markedly impaired in Foxp3(-) T cell allostimulatory capacity, to favor the stimulation of mu
57 cipients of islet allografts, display lesser allostimulatory capacity.
58 dy weight) in normal volunteers inhibited DC allostimulatory capacity.
59 ion of proinflammatory cytokines, and T-cell allostimulatory capacity.
60 edly lowered rate of Ag uptake and increased allostimulatory capacity.
61 oglobulin (Ig) production, and CD4(+) T-cell allostimulatory capacity.
62  mitigated the enhanced in vitro and in vivo allostimulatory capacity.
63 (CD40, CD80, CD86), and MHC class II Ag, and allostimulatory capacity.
64 es exhibited dendritic morphology and potent allostimulatory capacity.
65 ctivation by danger signals and their T cell allostimulatory capacity.
66 tial augmentation of the number of potential allostimulatory cells in donor organs before transplanta
67  and CD40 cross-linking increased the T-cell allostimulatory function of BM DCs, only LPS stimulation
68                                          The allostimulatory function of DCs was assessed by mixed le
69 s C virus (HCV) has been shown to affect the allostimulatory function of DCs.
70  a time-dependent manner and were capable of allostimulatory function.
71 phenotype, and mixed lymphocyte reaction for allostimulatory function.
72                            Consequently, the allostimulatory functions of DCs were impaired by TPT tr
73 PS was superior to CpG ODN in increasing the allostimulatory potential of lung DCs and their expressi
74 TSA-expanded cells possesses relatively less allostimulatory response but is still capable of generat
75 d CD80, and reduced the in vitro and in vivo allostimulatory responses induced by the DCs.
76         Thus, although normal livers provide allostimulatory signals sufficient to elicit an antidono
77 ory and maturation molecules induce a robust allostimulatory T-cell response.
78                 These data suggest that some allostimulatory white cells are filter adherent, whereas