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1 tivity (the latter against the TRPA1 agonist allyl isothiocyanate).
2 neurons do not respond to the TrpA1b agonist allyl isothiocyanate.
3 g electrophilic sensory irritants, including allyl isothiocyanate.
4 ed antimicrobial packaging incorporated with allyl isothiocyanate (AIT) and carbon nanotube (CNT), an
5     Moreover, unlike the broad TRPA1 agonist allyl isothiocyanate (AITC) (an active ingredient of tea
6 s acrolein (tear gas), allicin (garlic), and allyl isothiocyanate (AITC) (mustard oil).
7 alenol (alpha-ZOL) on a solution model using allyl isothiocyanate (AITC) and also determines the bioa
8                                      Inhaled allyl isothiocyanate (AITC) evoked atropine-sensitive br
9 packaging based on the controlled release of Allyl isothiocyanate (AITC) from mustard seed was design
10 dPGJ(2) evoked currents similar to equimolar allyl isothiocyanate (AITC) in the nominal absence of ca
11 e of cabbage that accounted for the enhanced allyl isothiocyanate (AITC) in the volatile oils of the
12 psulation and controlled release of volatile allyl isothiocyanate (AITC) molecules.
13                                              Allyl isothiocyanate (AITC) occurs in many commonly cons
14 activated by electrophilic compounds such as allyl isothiocyanate (AITC) through covalent modificatio
15                (1)H and (13)C NMR spectra of allyl isothiocyanate (AITC) were measured, and the excha
16  TRPA1 activators, including cinnamaldehyde, allyl isothiocyanate (AITC), and 4-hydroxynonenal, incre
17 ctive ingredients of these plants - menthol, allyl isothiocyanate (AITC), and capsaicin, respectively
18                    Pungent chemicals such as allyl isothiocyanate (AITC), cinnamaldehyde, and allicin
19                                The effect of allyl isothiocyanate (AITC), in combination with low tem
20 intravenous injection of the noxious stimuli allyl isothiocyanate (AITC).
21 aicin, acid (hydrogen chloride, pH 3.5), and allyl isothiocyanate (AITC).
22                                              Allyl isothiocyanate (AITC; aka, mustard oil) is a power
23 h increasing doses of the chemical irritants allyl isothiocyanate (AITC; also known as mustard oil) o
24        Using calcium imaging, TRPA1 agonists allyl isothiocyanates (AITC) and hydrogen peroxide were
25 of Botrytis cinerea in tomatoes by releasing allyl-isothiocyanate (AITC) from mustard seeds at room t
26            The pungent component of mustard, allyl-isothiocyanate (AITC), activates the extreme cold
27 e neurons while capsaicin (TRPV1 agonist) or allyl-isothiocyanate (AITC, TRPA1 agonist) elicited resp
28                                   Apart from allyl isothiocyanate also 3-(methylthio)propyl isothiocy
29                           The TRPA1 agonists allyl isothiocyanate and cinnamaldehyde and the TRPV4 ag
30 hey responded to the electrophilic compounds allyl isothiocyanate and cinnamaldehyde as well as heat.
31 , and some pungent plant ingredients such as allyl isothiocyanate and cinnamaldehyde.
32                                         Both allyl isothiocyanate and THC mediate their excitatory ef
33 s, which also responded to the TRPA1 agonist allyl isothiocyanate and the TRPV1 agonist capsaicin.
34 oducing a significant potentiating effect on allyl isothiocyanate- and diclofenac-induced currents of
35 d to capsaicin, menthol, and/or mustard oil (allyl isothiocyanate) at concentrations found in foods a
36  chemical products of enzymatic degradation (allyl isothiocyanate, benzyl isothiocyanate and 4-hydrox
37                               Treatment with allyl isothiocyanate, cinnamaldehyde, or GSK1016790A cau
38  (EC(50) = 34 microM) than the TRPA1 agonist allyl isothiocyanate (EC(50) = 400 microM) or the TRPV3
39 gredients of pungent natural products (e.g., allyl isothiocyanate), environmental irritants (e.g., ac
40  humans find pungent and irritating, such as allyl isothiocyanate (in wasabi) and acrolein (in cigare
41  neurons that also responded to mustard oil (allyl isothiocyanate), indicating a presynaptic action.
42  FIH-mediated hydroxylation may be linked to allyl isothiocyanate-induced channel activation.
43 s capable of desensitizing TRPA1, and unlike allyl isothiocyanate, it failed to induce nocifensive be
44 gredients of pungent natural products [e.g., allyl isothiocyanate (ITC), cinnamaldehyde, allicin, and
45  treatment (LC(50) = 5.29 ppm) compared with allyl isothiocyanate (LC(50) = 19.35 ppm) and 4-hydroxyb
46 ically activated by natural products such as allyl isothiocyanate (mustard oil), cinnamaldehyde (cinn
47 ed responses triggered by the TRPA1 agonists allyl-isothiocyanate (mustard oil), carvacrol, and polyu
48                      Here we show that AITC (allyl isothiocyanate; mustard oil) and menthol represent
49 lencing TRPV1(+) or TRPA1(+) neurons allowed allyl isothiocyanate or capsaicin, respectively, to evok
50 nd corresponding quenching when activated by allyl isothiocyanate or heat suggest lipid bilayer-indep
51 Here we show that allicin and DADS excite an allyl isothiocyanate-sensitive subpopulation of sensory
52 the nociceptive and inflammatory response to allyl isothiocyanate (the agonist of TRPA1) and reversed
53  and DADS share structural similarities with allyl isothiocyanate, the pungent ingredient in wasabi a
54                                              Allyl isothiocyanate, the pungent principle of wasabi an
55          Topical application of mustard oil (allyl isothiocyanate) to the skin activates underlying s
56 ely, activation of TRPA1 by the electrophile allyl isothiocyanate was abolished by glutathione, but w
57                  Unlike the covalent agonist allyl isothiocyanate, which elicits channel desensitizat
58 n of KS(13)CN with allyl bromide to give the allyl isothiocyanate, which upon Trofimov pyrrole synthe