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1 ripheral nerve block or spinal clonidine, an alpha2-adrenergic agonist.
2 o assess anesthetic depth in the presence of alpha2-adrenergic agonists.
3          Stimulated release was inhibited by alpha(2)-adrenergic agonists.
4  guanine nucleotide-sensitive binding of the alpha2-adrenergic agonist, [3H]UK-14,304.
5 ta-blockers, 4.01 (CrI, 0.48 to 7.43); 1995: alpha2-adrenergic agonists, 5.64 (CrI, 1.73 to 9.50); 19
6     In this review the authors will focus on alpha2-adrenergic agonists, a new class of sedative/anal
7 analogs (NO-PGA), EP2 agonists, unoprostone, alpha(2)-adrenergic agonists (AAAs), beta-adrenergic blo
8                    Our findings suggest that alpha2-adrenergic agonists act via a Gi family member on
9       In the present study, we show that the alpha(2)-adrenergic agonist (alpha(2)-agonist) clonidine
10 as to investigate the effects of a selective alpha2-adrenergic agonist, alpha-methylnorepinephrine (a
11                                The selective alpha2-adrenergic agonist, alphaMNE, was as effective as
12                                              Alpha2-adrenergic agonists also reduced stimulation of E
13  that Muller cells are unique in response to alpha2-adrenergic agonists and imply a role for Muller c
14 0 microM NE or 62% by 2 microM UK-14,304, an alpha2-adrenergic agonist, and reduced 63% by 10 microM
15                                              Alpha2 adrenergic agonists are used in the anesthetic ma
16                                          The alpha2-adrenergic agonists are unique.
17        Incubation with norepinephrine or the alpha2-adrenergic agonist BHT 920 also caused dose-depen
18 romoter (Thy1-CFP mice) to determine how the alpha2-adrenergic agonist brimonidine influences loss of
19 Similar actions were evoked by the selective alpha2 adrenergic agonist clonidine.
20 mg/kg i.p.) after chronic treatment with the alpha2-adrenergic agonist clonidine (200 microg/kg per d
21     We therefore examined the effects of the alpha2-adrenergic agonist clonidine on mean arterial pre
22 amezole to rats chronically treated with the alpha2-adrenergic agonist clonidine triggers a powerful
23 onist, phenylephrine (PHE; 0.5 mg/kg BW), an alpha2-adrenergic agonist, clonidine (CLON; 0.6 mg/kg BW
24 pha2-adrenergic receptors because a specific alpha2-adrenergic agonist could substitute for epinephri
25                                The selective alpha(2)-adrenergic agonist dexmedetomidine is a sedativ
26 techniques, we show that the clinically used alpha(2)-adrenergic agonist dexmedetomidine that enhance
27 ions of loss of consciousness (LOC) with the alpha2-adrenergic agonist dexmedetomidine and return of
28 inoimidazolidines including analogues of the alpha2 adrenergic agonist drug clonidine is elaborated.
29 viously, we found that sedation induced with alpha2-adrenergic agonists (e.g., dexmedetomidine) and s
30 nd atomoxetine than for the extended-release alpha2-adrenergic agonists guanfacine or clonidine (no s
31                We examine the effects of the alpha2 adrenergic agonist, guanfacine HCl, on responses
32                                              Alpha2-adrenergic agonists have specific and selective e
33 ntageous for treating hypertension over pure alpha(2)-adrenergic agonists (i.e., guanabenz) due to it
34 necrosis factor-alpha (TNF) as well as to an alpha(2)-adrenergic agonist in an area of the central ne
35 gonists and imply a role for Muller cells in alpha2-adrenergic agonist-induced photoreceptor protecti
36                          Dexmedetomidine, an alpha2 adrenergic agonist, is a useful sedative but can
37                          Dexmedetomidine, an alpha2-adrenergic agonist, is not approved by the Food a
38 es of this work were to study the effects of alpha2-adrenergic agonists on the inflammatory response
39                          Dexmedetomidine, an alpha2-adrenergic agonist, produces sedation and reduces
40 i.m. treatment with xylazine or UK14304, two alpha2-adrenergic agonists, reduced neutrophil migration
41               Immune modulation by selective alpha2-adrenergic agonists represents a novel, to our kn
42 ults indicate that systemically administered alpha2-adrenergic agonists selectively activate ERKs in
43                Intrathecal administration of alpha(2) adrenergic agonists, such as clonidine, is capa
44 xhibited similar sensitivity to the specific alpha(2)-adrenergic agonist UK 14,304, to serotonin, and
45 raarterial hindlimb infusion of the specific alpha2-adrenergic agonist UK 14,304 during KATP channel
46 ate the role and mechanisms of action of the alpha(2)-adrenergic agonist UK14,304 in the regulation o
47                Epinephrine and the selective alpha2-adrenergic agonist UK14304 reduced insulin secret
48 c signalling promotes anti-nociception, with alpha(2)-adrenergic agonists used clinically.
49 ood flows by its alpha1 (alpha1) and alpha2 (alpha2)-adrenergic agonist vasopressor actions.
50 l trauma, and systemic administration of the alpha(2)-adrenergic agonist xylazine, all of which have
51                                              Alpha2-adrenergic agonists xylazine and clonidine were a