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1 ubtype, after systemic administration of the alpha(2)-agonists 5-bromo-N-(4,5-dihydro-1H-imidazol-2-y
3 inephrine (NE), clonidine (CLON, a selective alpha2-agonist) and phenylephrine (PE, a selective alpha
4 fect profiles when compared with traditional alpha2 agonists, and may thereby allow for more widespre
6 ified into an exposed group, who received an alpha2 agonist as part of their sedation regimen, and an
9 ttens received microinfusions (1 mul) of the alpha2 agonist clonidine (CLON; 1.32 nmol), and half rec
10 ,N-MePhe4, Gly-ol5]enkephalin (DAMGO) or the alpha2 agonist clonidine inhibited voltage-gated Ca2+ cu
12 ed evidence supporting the widespread use of alpha2 agonists (clonidine and dexmedetomidine) in pedia
13 cocaine CPP in the presence and absence of a alpha2-agonist (clonidine), beta-adrenergic receptor ant
14 e show that the alpha(2)-adrenergic agonist (alpha(2)-agonist) clonidine induced resistance to M. avi
16 eatment of HUVECs, but not neutrophils, with alpha2-agonists decreased transendothelial migration, wi
17 yet preliminary evidence indicates that the alpha(2) agonist dexmedetomidine may have distinct advan
20 both norepinephrine and the highly selective alpha2 agonist dexmedetomidine each reversed the VLPO de
23 ial alpha(1)-agonist, phenylephrine, or full alpha(2)-agonist, dexmedetomidine, indicated that the be
24 rine (alpha(1)-agonist) and dexmedetomidine (alpha(2)-agonist) during moderate rhythmic handgrip exer
25 rine (alpha(1)-agonist) and dexmedetomidine (alpha(2)-agonist) during rhythmic handgrip exercise (15%
26 to investigate the role of the alpha2aAR in alpha2 agonist-evoked analgesia and adrenergic-opioid sy
27 ernal cesium and external barium, opioid and alpha2 agonists had no effect at potentials more negativ
31 nhibition of vas deferens contraction by the alpha2 agonist in alpha2A-AR knockout mice was only 42 +
32 phrine (an alpha1 agonist) and clonidine (an alpha2 agonist) in 10 healthy men during rhythmic handgr
34 ission at IPL and that brimonidine and other alpha2 agonists may protect RGCs under disease condition
36 y before naloxone with clonidine (20 microg; alpha2 agonist), MK-801 (3 microg; noncompetitive NMDA a
40 neuroprotection of retinal ganglion cells by alpha2 agonists, such as brimonidine, in animal models o
45 he NE-induced inhibition was mimicked by the alpha2-agonist, UK14,304, but not by the alpha1- or beta
46 (P < 0.05) and sensitivity (P < 0.05) to the alpha2 agonist UK14304 was lower in protein-, but not in
54 e (selective alpha1-agonist), and clonidine (alpha2-agonist) were determined in 10 young (aged 26+/-1
55 fter we will compare and contrast the use of alpha2-agonists with clinically available agents, and sp