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2 der and the hepatic alpha-TTP, but not brain alpha-TTP gene expression are important in determining a
3 entrations do not appear to be determined by alpha-TTP expression which was undetectable in all brain
5 of alpha-TTP by comparing mice that express alpha-TTP with mice that are genetically unable to expre
8 esults show that both gender and the hepatic alpha-TTP, but not brain alpha-TTP gene expression are i
9 ormality (ataxia) develops only very late in alpha-TTP-KO mice in spite of the severe alpha-tocophero
12 To test the hypothesis that VitE, not just alpha-TTP, is necessary for nervous system development,
15 tudied immunohistochemically the presence of alpha-TTP in the brains of a patient with AVED, normal s
18 ed by the alpha-tocopherol transfer protein (alpha-TTP) and is transferred to plasma, while the other
19 ations in alpha-tocopherol transfer protein (alpha-TTP) gene and it can be experimentally generated i
21 o hepatic alpha-tocopherol transfer protein (alpha-TTP), but this hypothesis has not been tested.
22 rolled by alpha-tocopherol transfer protein (alpha-TTP), which is a 278 amino acid protein encoded on
23 protein [alpha-tocopherol transfer protein (alpha-TTP)] in zebrafish embryos causes death within 24
24 ice, respectively (P < 0.0001), showing that alpha-TTP preferentially selects 2R-alpha-tocopherols fo