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1  be reduced by therapy with phentolamine, an alpha-adrenergic antagonist.
2 ty was also delayed by treating mice with an alpha-adrenergic antagonist.
3 inistration of a beta(2) (but not beta(1) or alpha) adrenergic antagonist.
4 nociception partially blocked by intrathecal alpha-adrenergic antagonists, but the mechanism underlyi
5                        We also found that an alpha-adrenergic antagonist can specifically block the Q
6  hormone epinephrine and show that beta- and alpha-adrenergic antagonists can block the bacterial res
7 pamine was administered with and without the alpha-adrenergic antagonist dapiprazole, and its effects
8                                   The use of alpha-adrenergic antagonist for the treatment of pediatr
9 To update the physician regarding the use of alpha-adrenergic antagonists in the management of variou
10 ipulations in vivo, we show that infusion of alpha-adrenergic antagonists into the NIf (nucleus inter
11 tra-arterial infusion of phentolamine (PHEN, alpha-adrenergic antagonist) or phenylephrine (PE, alpha
12               Presurgical treatment with the alpha-adrenergic antagonist phentolamine completely prev
13 ephrine, and this increase is blocked by the alpha-adrenergic antagonist phentolamine.
14                           The application of alpha-adrenergic antagonists (phentolamine or phenoxyben
15  noradrenaline release) and phentolamine (an alpha-adrenergic antagonist), profound vasodilatation wa
16 agonist to the microvessel preparation or an alpha-adrenergic antagonist to the myocytes and was augm
17 e derived vasoconstriction, phentolamine, an alpha-adrenergic antagonist was administered prior to MA
18                       Compared with placebo, alpha-adrenergic antagonists will also aid significantly
19                     OPC-28326 is a selective alpha-adrenergic antagonist with preferential binding to