ライフサイエンスコーパス: alpha-adrenergic receptor

1 via vasoconstriction mediated by alpha-ARs (alpha-adrenergic receptors).

2 his effect is most probably mediated through alpha-adrenergic receptors.

3 ses in FVC that are exclusively mediated via alpha-adrenergic receptors.

4 ganglion neurons evidencing the presence of alpha-adrenergic receptors.

5 ed by its intrinsic ability to also activate alpha-adrenergic receptors.

6 ined for 4 days in culture, we now show that alpha-adrenergic receptor activation by phenylephrine ca

7 salgia and for increased expression of human alpha-adrenergic receptors after loss of sympathetic act

8 od aspiration followed by instillation of an alpha-adrenergic receptor agonist such as phenylephrine,

9 Treatment with the nonselective alpha-adrenergic receptor agonist, octopamine, and the s

10 ipocytes were treated with phenylephrine, an alpha-adrenergic receptor agonist, to drive HDACS out of

11 rpose of this study was to determine whether alpha-adrenergic receptor agonists have a role in alveol

12 Phentolamine (alpha-adrenergic receptor (alpha-Ad) antagonist) enhance

13 The 2A subtype of the alpha-adrenergic receptor (alpha2A-AR) is necessary for

14 Alpha-adrenergic receptor (alphaAR)-stimulated hypertrop

15 s, pharmacological antagonists selective for alpha-adrenergic receptors (alphaAR), but not beta-adren

16 alpha-adrenergic receptors (alphaARs) are G protein-coup

17 tension, we investigated the role of central alpha-adrenergic receptors and CRF in mediating differen

18 This excitation is mediated by alpha-adrenergic receptors and has a time course reminis

19 nt was dependent on both local activation of alpha-adrenergic receptors and modulation of T-type calc

20 netic variants of beta-adrenergic receptors, alpha-adrenergic receptors, and endothelin receptors (am

21 This study evaluated the role of alpha-adrenergic receptor- and neuropeptide Y (NPY) Y1 r

22 189254 effect was completely reversed by the alpha-adrenergic receptor antagonist phentolamine (i.p.

23 C) for 4 h or by intravenous infusion of the alpha-adrenergic receptor antagonist phentolamine for on

24 from the SPGN terminal, because neither the alpha-adrenergic receptor antagonist phentolamine nor th

25 Treatment of mice with the alpha-adrenergic receptor antagonist phentolamine preven

26 textual fear conditioning, injections of the alpha-adrenergic receptor antagonist prazosin (1.0 or 3.

27 ed with saline, phentolamine, a nonselective alpha-adrenergic receptor antagonist, or propranolol, a

28 Treatment with alpha-adrenergic receptor antagonists potentiated dopami

29 ostatic hyperplasia is commonly treated with alpha-adrenergic-receptor antagonists (alpha-blockers) o

30 inhibited phospholipase C activation by the alpha-adrenergic receptor (AR) agonist phenylephrine, su

31 f the present study was to determine whether alpha-adrenergic receptor (AR) constriction of venular s

32 hetic nervous system mediated principally by alpha-adrenergic receptors because phentolamine, but not

33 As a result, alpha-adrenergic receptor blockade eliminated the signif

34 of studies aimed at examining the effects of alpha-adrenergic receptor blockade on LH secretion and t

35 Complete alpha-adrenergic receptor blockade was demonstrated by t

36 erazinylthiocarbonyl) disulfide (FLA-63), or alpha-adrenergic receptor blockade with phenoxybenzamine

37 Local (intra-femoral artery) alpha-adrenergic receptor blockade with phentolamine evo

38 ing forearm evoke vasodilatation after local alpha-adrenergic receptor blockade, raising the possibil

39 Phentolamine (alpha-adrenergic receptor blocker) not only increased pl

40 and benign prostatic hyperplasia with either alpha adrenergic receptor blockers or 5alpha-reductase i

41 Antimicrobial agents and alpha-adrenergic receptor blockers are frequently used.

42 studies, we considered two receptors in the alpha-adrenergic receptor family, alpha2A and alpha2C, a

43 The parallel activation of alpha-adrenergic receptors imposed a distinctive "signat

44 These results confirm the involvement of the alpha-adrenergic receptor in extinction processes in bot

45 ptors, but little is known about the role of alpha-adrenergic receptors in extinction.

46 the hypothesis that increased expression of alpha-adrenergic receptors in primary afferent neurons i

47 ypothesis proposes the increased presence of alpha-adrenergic receptors in primary afferent neurons t

48 nt, demonstrating a novel mechanism by which alpha-adrenergic receptors may regulate ion channel acti

49 ductance (FVC) and the contribution of local alpha-adrenergic receptor-mediated pathways to the obser

50 o suppression of mutant channel bursting via alpha-adrenergic receptor-mediated stimulation of PKC.

51 earm vascular conductance (FVC) responses to alpha-adrenergic receptor stimulation during rhythmic ha

52 rm vascular conductance (FVC) in response to alpha-adrenergic receptor stimulation during rhythmic ha

53 These results suggest that alpha-adrenergic receptor stimulation inhibits the beta-

54 alpha-Adrenergic receptor stimulation regulates the acti

55 kinase C (PKC) to the KACh channel following alpha-adrenergic receptor stimulation.

56 1-vasopressin receptors, but is dependent on alpha-adrenergic receptor stimulation.

57 athway that attenuates this response through alpha-adrenergic receptor stimulation.

58 Alpha-adrenergic receptor, subtype 2A (alpha2A-AR), acti

59 ubstitution on ligand binding and agonism of alpha-adrenergic receptor subtypes alpha1a, alpha1b, alp

60 Targeting alpha-adrenergic receptors synergizes with isotretinoin

61 After blockade of alpha-adrenergic receptors to prevent sympathetically me

62 Our results demonstrate that post-junctional alpha-adrenergic receptor vasoconstrictor responsiveness

63 alpha-Adrenergic receptors were activated with noradrena

64 of the mu-opioid receptor (MOR), but not of alpha-adrenergic receptors, which activate the same pool

65 current study examined whether antagonism of alpha-adrenergic receptors would impair the consolidatio