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1 addition of an antimuscarinic therapy to an alpha-blocker.
2 verine, and solifenacin), with or without an alpha-blocker.
3 of disease progression during treatment with alpha-blocker.
4 riamterene and hydrochlorothiazide, and 1 an alpha-blocker.
5 lls could be a marker for treatment with TNF-alpha blockers.
6 ing these cell types unlikely targets of TNF-alpha blockers.
7 are normalized in patients treated with TNF-alpha blockers.
8 enzyme inhibitors, and 1 of 1 RCT (100%) of alpha-blockers.
13 population, such that an interaction between alpha-blockers and prostate cancer risk is clinically re
14 inhibitors, calcium channel blockers (CCBs), alpha-blockers, and angiotensin receptor blockers-was si
18 ent (calcium-channel antagonists, diuretics, alpha-blockers, beta-blockers, and centrally acting agen
19 e inhibitors, angiotensin-receptor blockers, alpha-blockers, beta-blockers, calcium channel blockers,
20 ical centers were analyzed, including use of alpha-blockers (both selective and nonselective), IFIS,
22 t declines in the use of doxazosin and other alpha-blockers coincided with the early termination of t
24 , Enbrel), which is a tumour necrosis factor alpha blocker currently used to treat rheumatoid arthrit
26 between 1996 and 2002, including changes in alpha-blocker drug prices, generic conversion, drug prom
27 therapy for CP/CPPS with urinary symptoms is alpha-blockers (eg, tamsulosin, alfuzosin; DeltaNIH-CPSI
28 cted comparing the safety and efficacy of an alpha blocker-finasteride combination versus placebo in
30 ior prostate surgery and men who were taking alpha-blockers for urinary tract symptoms, 708 participa
31 Patients given calcium-channel blockers or alpha blockers had a 65% (absolute risk reduction=0.31 9
34 r, this relationship is complex as different alpha-blockers have divergent effects in laboratory stud
35 tors (ACEIs), calcium antagonists (CCBs) and alpha-blockers in preventing one or more major clinical
36 e of antimuscarinics, in combination with an alpha-blocker, in men with an overactive bladder and sum
38 cantly to this decline although cessation of alpha-blocker marketing may have hastened the decline.
39 n patients who were not premedicated with an alpha-blocker (n = 5) had a higher level of systolic blo
41 her risk of OD was found with beta-blockers, alpha-blockers, opioids, antiemetics, antivertiginosa or
48 ed during healing, administration of the TNF-alpha blocker reduced apoptosis of bone-lining cells by
49 increase in risk associated with exposure to alpha-blockers require replication and warrant further i
50 whether treatment with tumor necrosis factor alpha blockers results in further increased incidence of
51 ta are provided, doxazosin, and probably all alpha-blockers, should no longer be used as first-line a
52 ies have shown the efficacy of new selective alpha-blockers (silodosin and naftopidil); however, ther
54 ies include 5-alpha reductase inhibitors and alpha-blockers that are only partially effective and pos
55 veractive bladder) following therapy with an alpha-blocker, the addition of an antimuscarinic therapy
56 ungal therapy and the safety of resuming TNF-alpha blocker therapy after successful treatment of hist
61 nercept and infliximab have showed these TNF-alpha blockers to be well tolerated and effective in the
65 t, there was a slightly higher prevalence of alpha-blocker use in case vs control patients (32% vs 30
67 trials in which calcium-channel blockers or alpha blockers were used to treat ureteral stones were e
68 trials in which calcium-channel blockers or alpha blockers were used to treat urinary stone disease.