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1 treatment in cells grown in the presence of alpha-difluoromethylornithine.
2 s a more potent inhibitor of the enzyme than alpha-difluoromethylornithine.
3 ying exogenous putrescine in the presence of alpha-difluoromethylornithine.
4 , or by preventing putrescine formation with alpha-difluoromethylornithine.
5 TM activation is polyamine dependent because alpha-difluoromethylornithine, a specific inhibitor of O
9 In addition, in vivo inhibition of ODC with alpha-difluoromethylornithine also exacerbated the colit
12 ; (ii) depletion of cellular polyamines with alpha-difluoromethylornithine, an inhibitor of ornithine
15 owth arrest induced by a 72 h treatment with alpha-difluoromethylornithine, an ornithine decarboxylas
16 itor of cationic amino acid transport, or by alpha-difluoromethylornithine, an ornithine decarboxylas
17 utrescine was inhibited by the ODC inhibitor alpha-difluoromethylornithine, and L-proline generation
19 on of intracellular polyamine synthesis with alpha-difluoromethylornithine (DFMO) also increased Mito
21 roblastoma (NB) cells with the ODC inhibitor alpha-difluoromethylornithine (DFMO) depleted polyamine
22 Inhibition of polyamine biosynthesis with alpha-difluoromethylornithine (DFMO) has been shown to i
27 ned treatment with the Odc suicide inhibitor alpha-difluoromethylornithine (DFMO) or Odc heterozygosi
29 tic action of the NO donor agents as well as alpha-difluoromethylornithine (DFMO), a known ODC inhibi
30 tment of ODC/Ras double transgenic mice with alpha-difluoromethylornithine (DFMO), a specific inhibit
32 w significantly higher potency in vitro than alpha-difluoromethylornithine (DFMO), a U.S. Food and Dr
33 y to determine the chemopreventive effect of alpha-difluoromethylornithine (DFMO), an enzyme-activate
34 duced by zinc deficiency can be inhibited by alpha-difluoromethylornithine (DFMO), an enzyme-activate
35 n polyamine levels in COS-7 cells induced by alpha-difluoromethylornithine (DFMO), an inhibitor of or
36 were cultured in the presence or absence of alpha-difluoromethylornithine (DFMO), an inhibitor of th
40 itor of cationic amino acid transport, or by alpha-difluoromethylornithine (DFMO), an ODC inhibitor.
41 herapy, we hypothesized that the addition of alpha-difluoromethylornithine (DFMO), an ornithine decar
42 , i.e. N1,N11-diethylnorspermine (DENSPM) or alpha-difluoromethylornithine (DFMO), have synergistic e
43 ibitors of polyamine biosynthesis, including alpha-difluoromethylornithine (DFMO), inhibit tumor grow
46 shown that the cancer chemopreventive agent alpha-difluoromethylornithine (DFMO), known to inhibit t
50 ministration of the suicide inhibitor of ODC alpha-difluoromethylornithine (DFMO, 0.5% w/v) in the dr
51 s by inhibiting ornithine decarboxylase with alpha-difluoromethylornithine dramatically enhanced the
52 Treatment with the biosynthesis inhibitor alpha-difluoromethylornithine during tetracycline remova
54 position to that observed for putrescine and alpha-difluoromethylornithine in previous T. brucei ODC
55 nhibiting ornithine decarboxylase (ODC) with alpha-difluoromethylornithine increased the levels of AT
58 vation, the polyamine biosynthesis inhibitor alpha-difluoromethylornithine inhibits the ability of MT
59 either the ornithine decarboxylase inhibitor alpha-difluoromethylornithine or the S-adenosylmethionin
61 administration of the suicidal ODC inhibitor alpha-difluoromethylornithine reduced UVB-induced BCCs i
62 with the ornithine decarboxylase inhibitor L-alpha-difluoromethylornithine sensitized all of these le
63 nstream target of Akt, was also increased in alpha-difluoromethylornithine-treated cells, which was p