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1 including the m5 muscarinic receptor and the alpha1B adrenergic receptor.
2 te allosteric modulation of the alpha1A- and alpha1B-adrenergic receptors.
3 lls transfected with mutant versus wild-type alpha1b-adrenergic receptors.
4 correlate to inhibition of both alpha1A- and alpha1B-adrenergic receptors.
5                   In contrast, activation of alpha1B-adrenergic receptors (alpha1B-ARs) induced trans
6 ection of NF1/L, NF1/Red1, or NF1/X with the alpha1B adrenergic receptor (alpha1BAR) gene middle (P2)
7  Progress toward elucidating the function of alpha1B-adrenergic receptors (alpha1BARs) in the central
8                                    Since the alpha1B adrenergic receptor and dopamine receptor type 1
9 ]prazosin dissociation from the alpha1A- and alpha1B-adrenergic receptors and noncompetitively inhibi
10 ial-targeted overexpression of the wild-type alpha1B adrenergic receptor (AR) (Tg alpha43), we studie
11              We previously demonstrated that alpha1B-adrenergic receptor (AR) gene transcription, mRN
12 pecific overexpression of the wild-type (WT) alpha1B-adrenergic receptor (AR) using the murine alpha-
13 both the alpha1a-adrenergic receptor and the alpha1b-adrenergic receptor (AR), that account almost en
14           Overall, this study identified the alpha1B adrenergic receptor as a pharmacologic target in
15                    The analogous residues in alpha1b-adrenergic receptors, aspartic acid 125 and lysi
16  type A receptor, dopamine D1A receptor, and alpha1b adrenergic receptor) bound betaarrestin2 with hi
17                    Upon agonist stimulation, alpha1B-adrenergic receptors couple to Gq proteins, calc
18    We investigated the role of this motif in alpha1B-adrenergic receptor function and regulation.
19 and CP1 bind to the major P2 promoter of the alpha1B adrenergic receptor gene to generate footprint I
20 specific regulation of the expression of the alpha1B adrenergic receptor gene.
21                                        Y348A alpha1B-adrenergic receptors in which this sequence was
22 tivation, (ii) provide further evidence that alpha1B-adrenergic receptor internalization can be separ
23                                The expressed alpha1b-adrenergic receptor mutant demonstrated a 6-fold
24 ere obtained for a lysine to a glutamic acid alpha1b-adrenergic receptor mutation.
25                        Taken together, these alpha1b-adrenergic receptor mutations suggest a molecula
26 were also observed for two aspartic acid 125 alpha1b-adrenergic receptor mutations, consistent with t
27 icular traffic were investigated by studying alpha1B-adrenergic receptor-Rab protein interactions, us
28 of radioligand binding sites for two mutated alpha1B-adrenergic receptors reported previously was inv
29                                         This alpha1b-adrenergic receptor salt bridge constraint is th
30  beta2 but not in cells transfected with the alpha1B-adrenergic receptor, suggesting that the PLC bet
31                                         When alpha1B-adrenergic receptors that had been mutated at pr
32 an important role for Tyr348 in coupling the alpha1B-adrenergic receptor to G protein and subsequent
33 Data suggest that protein kinase C modulates alpha1B-adrenergic receptor transfer to late endosomes a