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1 ls in the tumour core and edge differ due to alternative RNA splicing.
2 f DDX39B, plays a similar role in regulating alternative RNA splicing.
3  a function of O-glycosylation in regulating alternative RNA splicing.
4  vary at their C termini as a consequence of alternative RNA splicing.
5 w viral E6 and E7 expression is regulated by alternative RNA splicing.
6  expression events through the regulation of alternative RNA splicing.
7 re B cells express together with IgM through alternative RNA splicing.
8  steroid receptor-mediated transcription and alternative RNA splicing.
9 h are generated by alternative promoters and alternative RNA splicing.
10 e-specific isoforms and is also regulated by alternative RNA splicing.
11 nsmembrane glycoprotein CD44 are produced by alternative RNA splicing.
12 f an RNA recognition motif (RRM) and affects alternative RNA splicing.
13 -bound (mIg) B cell receptors (BCRs) through alternative RNA splicing.
14 licing, no helicases have been implicated in alternative RNA splicing.
15 M-RAR molecules are expressed as a result of alternative RNA splicing.
16 r from a single promoter in association with alternative RNA splicing.
17 s numbers of EGF-like domains as a result of alternative RNA splicing.
18  biologically active isotypes resulting from alternative RNA splicing.
19                   This isoform resulted from alternative RNA splicing.
20 P1L) and short (CD2BP1S) variants arising by alternative RNA splicing.
21 ence suggests that the deletion is caused by alternative RNA splicing.
22 espectively, and these appear to result from alternative RNA splicing.
23 oplasm and plays a role in the regulation of alternative RNA splicing.
24 specific mRNA isoforms would be generated by alternative RNA splicing.
25  and 206 amino acids/monomer) resulting from alternative RNA splicing.
26  is known to regulate homeotic genes through alternative RNA splicing.
27  gene yields various isoforms as a result of alternative RNA splicing.
28  and investigates the potential link between alternative RNA splicing and autism-related behaviors in
29 DNA binding domain and the leucine zipper by alternative RNA splicing and differential polyadenylatio
30 tio-temporal patterns of GluR expression, by alternative RNA splicing and editing and by targeting an
31  pp52 and S37 mRNA isoforms are generated by alternative RNA splicing and establish that they are ind
32 eome has millions of protein variants due to alternative RNA splicing and post-translational modifica
33 tion factors and RNA-binding proteins affect alternative RNA splicing and synaptic development, poten
34 is elegans gene mec-8 encodes a regulator of alternative RNA splicing and that mec-8 null mutants hav
35                      Entropy calculated from alternative RNA splicing and transcription start/end pre
36  to base pair with its pre-mRNA and regulate alternative RNA splicing and/or A-to-I RNA editing.
37 s, usage of non-canonical initiation codons, alternative RNA splicing, and defective ribosomal RNA pr
38 rent genes (NFI-A, NFI-B, NFI-C, and NFI-X), alternative RNA splicing, and protein heterodimerization
39                Functionally, AGGF1 regulates alternative RNA splicing as it decreases the exon skippi
40 evere midline facial clefting and widespread alternative RNA splicing (AS) changes.
41                                 Misregulated alternative RNA splicing (AS) contributes to the tumorig
42                                              Alternative RNA splicing (AS) regulates proteome diversi
43  Oct2 alpha and beta isoforms are derived by alternative RNA splicing; as determined by Southern anal
44                                              Alternative RNA splicing can generate distinct protein i
45 mmunoprecipitation-chip studies, analysis of alternative RNA splicing, characterization of the methyl
46 tional activity also directly participate in alternative RNA splicing decisions.
47 known as A2BP1), as a prominent regulator of alternative RNA splicing during heart failure.
48             The latter mutation has the same alternative RNA splicing effect as a reported synonymous
49                    Here we describe a unique alternative RNA splicing event that occurs during the de
50  of higher organisms, for example, detecting alternative RNA splicing events and oncogenic chromosoma
51 e transmembrane domain and are the result of alternative RNA splicing events between exons IV and VII
52 ons of these RNA-binding proteins influenced alternative RNA splicing events within key autism-relate
53 TRX, and TP53 mutations cooperatively impact alternative RNA splicing events, particularly suppressio
54 roteomic diversity is frequently achieved by alternative RNA-splicing events that can be fine-tuned i
55                    L4-33K is a virus-encoded alternative RNA splicing factor which activates splicing
56 includes a variety of molecules generated by alternative RNA splicing from 10 variant exons (v1-v10).
57 forms (alpha and beta) that are generated by alternative RNA splicing from a single SpOtx gene.
58   The structure-function relationship of the alternative RNA splicing-generated NH2-terminal variable
59                  We have now determined that alternative RNA splicing gives rise to at least five dif
60                                              Alternative RNA splicing greatly expands the repertoire
61                                              Alternative RNA splicing greatly increases proteome dive
62                                              Alternative RNA splicing has been reported at three site
63    Since its discovery in 1977, the study of alternative RNA splicing has revealed a plethora of mech
64 d trans-acting factors contributing to HPV18 alternative RNA splicing have been discovered in this st
65 generation sequencing and analysis of global alternative RNA splicing identified that the mRNA splici
66                 Dysregulation in patterns of alternative RNA splicing in cancer cells is emerging as
67              We report on the involvement of alternative RNA splicing in generating multiple function
68 NA isoform sequencing (scISO-Seq) can reveal alternative RNA splicing in individual cells, it suffers
69 the RUNX1/RUNX1T1 oncogene is a regulator of alternative RNA splicing in leukemic cells.
70 nisms by which prenatal BPA exposure affects alternative RNA splicing in the prefrontal cortex and in
71                                              Alternative RNA splicing is an essential and dynamic pro
72                                              Alternative RNA splicing is an essential process to yiel
73       Collectively, our findings reveal that alternative RNA splicing is an important mechanism of ge
74  Taking these data together, we propose that alternative RNA splicing is involved in hypothalamic dev
75  The molecular cloning of Rgh3 suggests that alternative RNA splicing is needed for cell differentiat
76                                              Alternative RNA splicing is now known to be pervasive th
77 n was performed to identify tumor-associated alternative RNA splicing isoforms.
78 ggest that posttranscriptional regulation by alternative RNA splicing may play an important role in M
79                                              Alternative RNA splicing may provide unique opportunitie
80 ssing results primarily from a novel form of alternative RNA splicing mediated by multiple exonic spl
81 hanges in the two isozymes, originating from alternative RNA splicing, occur at a stretch of 55 amino
82 group of polypeptide factors that arise from alternative RNA splicing of a single gene.
83 e of CAPER coactivators in the regulation of alternative RNA splicing of an endogenous cellular gene
84       Therefore, we investigated whether the alternative RNA splicing of Bcl-x pre-mRNA was modulated
85 D-containing NMDA receptors are modulated by alternative RNA splicing of GluN1.
86 stinguish these cells is IgD, which, through alternative RNA splicing of H chain transcripts, begins
87 ese data provide the first evidence that the alternative RNA splicing of HPV18 pre-mRNAs is subject t
88 study provides important observations on how alternative RNA splicing of HPV18 pre-mRNAs is subject t
89                     Jmjd6 is shown to change alternative RNA splicing of some, but not all, of the en
90               Exclusion of the alpha-exon by alternative RNA splicing of the fibroblast growth factor
91                     There is no evidence for alternative RNA splicing of this gene product.
92    Finally, we show that Srsf3 regulates the alternative RNA splicing of transcripts encoding protein
93    Expression of HPV18 genes is regulated by alternative RNA splicing of viral polycistronic pre-mRNA
94 ch correlated with the observed sex-specific alternative RNA splicing patterns.
95 s troponin T (TnT) isoforms are generated by alternative RNA splicing primarily in its N-terminal hyp
96                    Developmentally regulated alternative RNA splicing produces TnT isoforms differing
97 tudy was to investigate chicken TERT (cTERT) alternative RNA splicing profiles of samples varying for
98                                              Alternative RNA splicing provides an important means to
99 d trans-acting factors contributing to HPV18 alternative RNA splicing remain unknown.
100 F) A is generated as two isoform families by alternative RNA splicing, represented by VEGF-A165a and
101      We review recent findings pertaining to alternative RNA splicing, the formation of multiple dist
102 ral variability of NRG isoforms generated by alternative RNA splicing, the use of multiple promoters
103 olycistronic pre-mRNAs that are regulated by alternative RNA splicing to produce a repertoire of vira
104         RGMc is a 4-exon gene that undergoes alternative RNA splicing to yield 3 mRNAs with 5' differ
105 POT1 gene encodes four other variants due to alternative RNA splicing (variants v2, v3, v4, and v5),
106            AKT-phosphorylated IWS1 regulates alternative RNA splicing via a pathway that is active in
107 thin the positively selecting CD69+DP cells, alternative RNA splicing was dysregulated in the double
108 s in Htr2c RNA editing profiles, while Htr2c alternative RNA splicing was unchanged.
109 identify cis-acting elements regulating this alternative RNA splicing, we sequenced the 3' end of Mhc
110  SC35, SRp40, SRp55, and SF2/ASF involved in alternative RNA splicing were predicted in exon 12.
111                      These results correlate alternative RNA splicing with the expression of two GPHe
112  and deletion of one or more coding exons by alternative RNA splicing would not shift the downstream

 
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