1 veolar pressure/area curves, plus changes in alveolar duct and airway size, and the elastic forces in

2 ed marked epithelial hyperplasia in proximal alveolar ducts and adjacent alveoli with associated cent

3 oblasts - ductal myofibroblasts, surrounding alveolar ducts and marked by CDH4, HHIP and LGR6, which

4 al cells lining the terminal bronchioles and alveolar ducts as well as macrophages and alveolar type

5 mice showed prominent lesions at bronchiolar-alveolar duct (BAD) junctions where asbestos fibers depo

6 t the number of proliferating cells at first alveolar duct bifurcations and in adjacent terminal bron

7                 Staining was most evident at alveolar duct bifurcations and within bronchiolar epithe

8 1 (SFRP1) is a critical molecule involved in alveolar duct formation in the lung and here we demonstr

9 nchymal cell types, airway smooth muscle and alveolar duct myofibroblasts.

10  fibers deposit initially at the bronchiolar-alveolar duct regions and alveolar macrophages accumulat

11 were clearly up-regulated at the bronchiolar-alveolar duct regions by 24 hours after the single asbes

12 ithelial cells lining distal bronchioles and alveolar ducts, sites of initial lung deposition of asbe