ライフサイエンスコーパス: alveolar epithelial cell

1 ical stretch (A549, Calu-3, or human primary alveolar epithelial cells).

2 goblet, Clara, ciliated, type I and type II alveolar epithelial cells.

3 lted in increased adherence of yeast to A549 alveolar epithelial cells.

4 ortant role in the ubiquitination of ENaC in alveolar epithelial cells.

5 ly regulated by the p38 MAP kinase in murine alveolar epithelial cells.

6 zation and inhibiting TGF-beta1 signaling in alveolar epithelial cells.

7 eptor (CNTFR), was expressed only in type II alveolar epithelial cells.

8 rally expressed Na(+)/K(+)-ATPase in type II alveolar epithelial cells.

9 y studied by transfecting the human MMP19 in alveolar epithelial cells.

10 localization predominantly in the nucleus of alveolar epithelial cells.

11 ired for SARS-CoV-2 viral infection of human alveolar epithelial cells.

12 2, in both A549 cells and in primary type II alveolar epithelial cells.

13 an increase in ER stress of resident type II alveolar epithelial cells.

14 for hypoxia-induced AMPK-PKC zeta binding in alveolar epithelial cells.

15 twork and its local mechanical properties in alveolar epithelial cells.

16 the cell that help maintain the integrity of alveolar epithelial cells.

17 led bacterial growth in both macrophages and alveolar epithelial cells.

18 to Candida albicans filaments and A549 human alveolar epithelial cells.

19 vated protein kinase (AMPK) at Thr172 in rat alveolar epithelial cells.

20 ces p53-dependent apoptosis in primary human alveolar epithelial cells.

21 ltered differentiation of type I and type II alveolar epithelial cells.

22 cts as a growth and anti-apoptotic factor on alveolar epithelial cells.

23 li to PC, and type I (WI-26 VA4) and type II alveolar epithelial cells.

24 acterial binding to pulmonary surfactant and alveolar epithelial cells.

25 fimbriae mediate bacterial binding to human alveolar epithelial cells.

26 decreased B7-H3 expression on bronchial and alveolar epithelial cells.

27 sed on unstimulated tracheal, bronchial, and alveolar epithelial cells.

28 cells and ICOS-L expression on bronchial and alveolar epithelial cells.

29 H and cystatin B were colocalized in type 2 alveolar epithelial cells.

30 s in K8, leading to the disassembly of IF in alveolar epithelial cells.

31 the presence of the Ad hexon antigen within alveolar epithelial cells.

32 med in vitro by analysis of isolated type II alveolar epithelial cells.

33 regulate proinflammatory cytokine release in alveolar epithelial cells.

34 or T3 stimulation of Na,K-ATPase activity in alveolar epithelial cells.

35 and its cell surface expression in adult rat alveolar epithelial cells.

36 use of increased proliferation of ductal and alveolar epithelial cells.

37 ith effects on both alveolar macrophages and alveolar epithelial cells.

38 roxic mice, predominantly in macrophages and alveolar epithelial cells.

39 ) and 31% (p < 0.001), respectively, in A549 alveolar epithelial cells.

40 rated that Pneumocystis is not cytotoxic for alveolar epithelial cells.

41 ctly interferes with purinergic signaling in alveolar epithelial cells.

42 vasion of, and persistence within, bronchial alveolar epithelial cells.

43 positive signals of the 2 miR expression in alveolar epithelial cells.

44 ell as TGF-beta-mediated permeability across alveolar epithelial cells.

45 r CXCR3 and its ligand CXCL9 modulate EMT in alveolar epithelial cells.

46 ercapnia)-induced Na,K-ATPase endocytosis in alveolar epithelial cells.

47 n of large cytoplasmic lipid droplets in the alveolar epithelial cells.

48 promoting the endocytosis of Na,K-ATPase in alveolar epithelial cells.

49 of cells identifiable as functional type II alveolar epithelial cells.

50 rally localized Na(+)-K(+)-ATPase in type II alveolar epithelial cells.

51 e did not activate PKA in human bronchial or alveolar epithelial cells.

52 pulmonary fibroblasts and in airway, but not alveolar, epithelial cells.

53 in 2.8-fold more PGE2 than control mice, and alveolar epithelial cells (2.7-fold), AMs (125-fold), an

54 in vitro model consisting of human PMNs and alveolar epithelial cells (A549) grown on inverted Trans

55 Exposure of the human-type II cell alveolar epithelial cells (A549) to DPM derived from FBC

56 cidosis and hypoxia in fibroblasts (N12) and alveolar epithelial cells (A549).

57 n histone acetylation:deacetylation in human alveolar epithelial cells (A549).

58 (Adora2b(loxP/loxP) VE-cadherin Cre(+)), or alveolar epithelial cells (Adora2b(loxP/loxP) SPC Cre(+)

59 a direct link between injury to the type II alveolar epithelial cell (AEC) and the accumulation of i

60 nitase-2 (Aco-2) each reduce oxidant-induced alveolar epithelial cell (AEC) apoptosis, but it is uncl

61 Alveolar epithelial cell (AEC) apoptosis, proliferation,

62 c depletion approaches, we demonstrated that alveolar epithelial cell (AEC) GM-CSF mediates recovery

63 Rationale: Alveolar epithelial cell (AEC) injury and dysregulated r

64 Alveolar epithelial cell (AEC) injury is a key step that

65 Alveolar epithelial cell (AEC) mitochondrial dysfunction

66 Alveolar epithelial cell (AEC) trans-differentiation is

67 urine cell line derived specifically from an alveolar epithelial cell (AEC) was utilized.

68 he clinical context of ARDS, its effect upon alveolar epithelial cell (AEC) wounding and repair remai

69 lls (AEC II) trans-differentiate into type I alveolar epithelial cells (AEC I) during lung recovery a

70 oliferate and transdifferentiate into type I alveolar epithelial cells (AEC I) when the normal AEC I

71 Type II alveolar epithelial cells (AEC II) proliferate and trans

72 s-differentiation is a process where type II alveolar epithelial cells (AEC II) trans-differentiate i

73 ch, causes disassembly of keratin IF in lung alveolar epithelial cells (AEC) and that this disassembl

74 nt to high altitude and pulmonary edema, the alveolar epithelial cells (AEC) are exposed to hypoxic c

75 tivated by integrin alphavbeta6 expressed on alveolar epithelial cells (AEC) continuously inhibits th

76 -beta1 (TGF-beta1) interactions in pulmonary alveolar epithelial cells (AEC) in the context of EMT an

77 genous pulmonary cytokine produced by normal alveolar epithelial cells (AEC) that is a key defender o

78 ly benefit from reproducible availability of alveolar epithelial cells (AEC).

79 -adrenergic regulation of the Na,K-ATPase in alveolar epithelial cells (AEC).

80 many viral antigens were detected in type II alveolar epithelial cells (AEC-IIs) within autopsied lun

81 Type 2 alveolar epithelial cells (AEC2s) are stem cells in the

82 anoid colony formation and found that type 2 alveolar epithelial cells (AEC2s), the stem cell-contain

83 RT in epithelial cells, we generated type II alveolar epithelial cell (AECII)-specific TERT condition

84 ial epithelial cells (HBEC), primary type II alveolar epithelial cells (AECII), and Calu-3 cells.

85 murine lower airway tissues, primary type II alveolar epithelial cells (AECIIs), and the mouse lung c

86 ing CO(2)-induced Na,K-ATPase endocytosis in alveolar epithelial cells (AECs) and alveolar epithelial

87 Human alveolar epithelial cells (AECs) and alveolar macrophage

88 ese responses reflect the cross-talk between alveolar epithelial cells (AECs) and resident alveolar m

89 We confirmed the expression of IRAK-M in alveolar epithelial cells (AECs) and showed that hyperox

90 lters Na,K-ATPase expression and function in alveolar epithelial cells (AECs) and the ability of the

91 a type III intermediate filament protein, in alveolar epithelial cells (AECs) are unknown.

92 We and others have shown that activated alveolar epithelial cells (AECs) directly contribute to

93 eviously unrecognized subpopulation of mouse alveolar epithelial cells (AECs) expressing the laminin

94 Human alveolar epithelial cells (AECs) form the key line of lu

95 Apoptosis of alveolar epithelial cells (AECs) has been implicated as

96 MFs), bronchial epithelial cells (BECs), and alveolar epithelial cells (AECs) in this strain of mice.

97 When alveolar epithelial cells (AECs) lacked Sting or gap jun

98 We hypothesized that alveolar epithelial cells (AECs) may serve as a source o

99 Alveolar epithelial cells (AECs) participate in the path

100 enchymal transition, involving transition of alveolar epithelial cells (AECs) to pulmonary fibroblast

101 Indeed, alveolar epithelial cells (AECs) undergo EMT in vivo dur

102 We treated HO-exposed mice and primary alveolar epithelial cells (AECs) with the novel TREK-1 a

103 omechanical properties of the KIF network in alveolar epithelial cells (AECs), independent of other c

104 In alveolar epithelial cells (AECs), the membrane-anchored

105 vidence of herpesvirus antigen expression in alveolar epithelial cells (AECs), which correlated with

106 ts an important role of aberrantly activated alveolar epithelial cells (AECs), which produce a large

107 tion involves the attachment of organisms to alveolar epithelial cells (AECs).

108 bition blocked both pY654 and EMT in primary alveolar epithelial cells (AECs).

109 c response is driven by abnormally activated alveolar epithelial cells (AECs).

110 endoplasmic reticulum (ER) stress in type II alveolar epithelial cells (AECs).

111 se-dependent injury and death in healthy rat alveolar epithelial cells (AECs).

112 dance in the plasma membrane and activity in alveolar epithelial cells (AECs).

113 ighly regulated by the adhesion molecules on alveolar epithelial cells (AECs).

114 that miR-17 and miR-548b were upregulated in alveolar epithelial cells after CI/EVR, which merit furt

115 with GM-CSF, an agent we have shown protects alveolar epithelial cells against apoptosis, decreased F

116 the expression of Egr-1, CTGF, and Cyr61 to alveolar epithelial cells, airway epithelial cells, and

117 rs throughout the lung, including airway and alveolar epithelial cells, airway smooth muscle cells, a

118 These alveolar epithelial cells also expressed the correspondi

119 uctance regulator regulates fluid balance in alveolar epithelial cells and appears to modulate the in

120 The binding of P. carinii to alveolar epithelial cells and extracellular matrix const

121 idence indicates that aberrant activation of alveolar epithelial cells and fibroblasts in an aging lu

122 ice also demonstrated increased apoptosis of alveolar epithelial cells and greater numbers of fibrobl

123 ffect of Eyjafjallajokull ash on primary rat alveolar epithelial cells and human airway epithelial ce

124 the alveolar surface as a product of type II alveolar epithelial cells and includes PC as the major c

125 stry analysis of IL-6 and IL-8 revealed that alveolar epithelial cells and macrophages and a few inte

126 we detected CLF-1 expression in both type II alveolar epithelial cells and macrophages.

127 ion in airway epithelium, as well as type II alveolar epithelial cells and macrophages.

128 PF, CLF-1 is a selective stimulus of type II alveolar epithelial cells and may potentially drive an a

129 ry interface of the living human lung, human alveolar epithelial cells and microvascular endothelial

130 neage maturation into proximal cells, type I alveolar epithelial cells and morphologically mature typ

131 r cell polarity (PCP) pathway is required in alveolar epithelial cells and myofibroblasts for alveolo

132 D4 (LTD4) on the function of Na,K-ATPase in alveolar epithelial cells and on alveolar fluid clearanc

133 ties of TH are associated with protection of alveolar epithelial cells and restoration of mitochondri

134 YapV acted as an adhesin for alveolar epithelial cells and specific extracellular mat

135 microparticles, respectively, for uptake by alveolar epithelial cells and subsequent inhibition of S

136 an autocrine trophic factor for human adult alveolar epithelial cells and that under situations of p

137 ng SOD1 prevented the PM2.5-induced death of alveolar epithelial cells and the associated increase in

138 ll viability were performed using A549 human alveolar epithelial cells and THP-1 monocyte-derived mac

139 and invasion of human-derived HeLa and A549 alveolar epithelial cells and to its inability to surviv

140 was expressed in ciliated airway and type II alveolar epithelial cells and was targeted for cell-spec

141 V-2, as demonstrated in human leukocytes and alveolar epithelial cells, and rhesus macaques, without

142 is unknown, it is characterized by extensive alveolar epithelial cell apoptosis and proliferation of

143 strated increased pulmonary inflammation and alveolar epithelial cell apoptosis compared to controls.

144 Although alveolar epithelial cell apoptosis has been documented a

145 flammatory responses or direct inhibition of alveolar epithelial cell apoptosis would improve surviva

146 ll viability in O(2), decreased O(2)-induced alveolar epithelial cell apoptosis, and accelerated alve

147 how amelioration of lung function, decreased alveolar epithelial cell apoptosis, and fibroblast proli

148 reducing extracellular matrix production and alveolar epithelial cell apoptosis.

149 ough mice lacking c-Jun specifically in lung alveolar epithelial cells appear normal at the age of 6

150 ells decreases the expression of markers for alveolar epithelial cells (Aqp5 and Sftpc), Clara cells

151 These findings suggest that human alveolar epithelial cells are a target of Bacillus anthr

152 ls lining the respiratory units of the lung, alveolar epithelial cells, are a target of lethal toxin

153 These data reveal alveolar epithelial cells as progenitors for fibroblasts

154 xic DNA double-strand breaks (DSBs) in human alveolar epithelial cells, as indicated by ataxia telang

155 reduction in invasion of human bronchial and alveolar epithelial cells at 1, 3, and 6 h postinfection

156 d club cells' capacity to differentiate into alveolar epithelial cells at the single-cell level.

157 tenuated mitochondria-regulated apoptosis in alveolar epithelial cells both in vivo and in vitro.

158 eceptor expressed on both type I and type II alveolar epithelial cells but not vascular endothelium,

159 -8 activation, which was localized to type 1 alveolar epithelial cells by flow cytometric analysis.

160 These studies indicate that bronchial and alveolar epithelial cell C5aR is up-regulated and greatl

161 lens cells, and recent studies indicate that alveolar epithelial cells can be derived from hematopoie

162 Primary alveolar epithelial cells can be derived from human lung

163 Extensive infection of alveolar epithelial cells caused apoptosis and leakage o

164 he genetic deletion of c-Jun specifically in alveolar epithelial cells causes progressive emphysema w

165 expression of Fas on the surface of type II alveolar epithelial cells; conversely, infection with P.

166 Ex vivo, primary alveolar epithelial cells cultured on provisional matrix

167 In patients with severe ARDS, alveolar epithelial cell death is a major mechanism that

168 Similar results in A549 lung alveolar epithelial cells demonstrated the biological re

169 in alpha5 in signaling pathways that promote alveolar epithelial cell differentiation and VEGF expres

170 d by disrupted secondary septation, abnormal alveolar epithelial cell differentiation, excessive coll

171 00/beta-catenin interaction, concurrent with alveolar epithelial cell differentiation.

172 ommunication between macrophages and type II alveolar epithelial cells during influenza infection whe

173 (FABP5) is expressed in lung cells, such as alveolar epithelial cells (ECs) and alveolar macrophages

174 dent AMs can blunt inflammatory signaling in alveolar epithelial cells (ECs) by transcellular deliver

175 Using LA4 murine lung alveolar epithelial cells, effects of doxycycline and er

176 The role of alveolar epithelial cells, endothelial cells, and alveol

177 al resting lung interstitial macrophages and alveolar epithelial cells express high levels of RGMb mR

178 Alveolar epithelial cells expressed lethal toxin recepto

179 Type II alveolar epithelial cell expression of IL-4Rs in mice se

180 alysis, we demonstrate that specification of alveolar epithelial cell fate begins concomitantly with

181 Apoptosis of alveolar epithelial cells, followed by abnormal tissue r

182 ularensis also invades and replicates within alveolar epithelial cells following inhalation in a mous

183 there was no change in COX-1/COX-2 levels in alveolar epithelial cells following treatment with CT an

184 To more definitively test the capacity of alveolar epithelial cells for EMT, mice expressing beta-

185 To overcome the scarcity of primary human alveolar epithelial cells for lung research, and the lim

186 lines (MLE-15, LA-4) and in primary type II alveolar epithelial cells, FRH enhanced apoptosis in res

187 By contrast, alveolar epithelial cells from bid(-/-) mice were resist

188 cells as well as in freshly isolated type II alveolar epithelial cells from different species.

189 sure to PM(2.5) induced apoptosis in primary alveolar epithelial cells from wild-type but not Noxa(-/

190 In alveolar epithelial cells, G-protein coupled-receptors a

191 e, expresses Pdgfralpha, and is critical for alveolar epithelial cell growth and self-renewal.

192 d surfactant proteins, it is unknown whether alveolar epithelial cells have distinct roles in innate

193 interstitial cells, and additionally type I alveolar epithelial cells immunostained for green fluore

194 tes caspase-3 in macrophages, monocytes, and alveolar epithelial cells in a Dot/Icm-dependent manner

195 phage-derived MVs were fully internalized by alveolar epithelial cells in a time-, dose-, and tempera

196 increased Na,K-ATPase activity of adult rat alveolar epithelial cells in a transcription-independent

197 In this study, we exposed alveolar epithelial cells in culture and mice to fine pa

198 ound efficient transduction of bronchial and alveolar epithelial cells in hDSG2-transgenic mice.

199 Atypical alveolar epithelial cells in IPF express molecular marke

200 epithelial cells, with reduced expression in alveolar epithelial cells in IPF lungs.

201 at miR-34a expression is increased in type 2 alveolar epithelial cells in neonates with respiratory d

202 was selectively deleted from bronchiolar and alveolar epithelial cells in Stat3(DeltaDelta) mice.

203 e initially believed merely to assist type 2 alveolar epithelial cells in surfactant production durin

204 mediated an increased bacterial adherence to alveolar epithelial cells in the presence of Vn.

205 TES production in macrophages and cocultured alveolar epithelial cells in vitro.

206 hypoxia, and hypoxia directly induced DCK in alveolar epithelial cells in vitro.

207 ung is determined by their interactions with alveolar epithelial cells, in particular alveolar type 1

208 Alveolar epithelial cell infection was normal, but alveo

209 rms of pathogenesis, IPF is characterized by alveolar epithelial cell injury and activation with inte

210 y sublethal hyperoxic insult, accompanied by alveolar epithelial cell injury and increased pulmonary

211 ratory distress syndrome is characterized by alveolar epithelial cell injury, edema formation, and in

212 mice from hyperoxic lung injury by limiting alveolar epithelial cell injury.

213 nism for this deterioration may be increased alveolar epithelial cell injury.

214 he ATCC 19606(T) type strain with A549 human alveolar epithelial cells is independent of the producti

215 Whether these effects are mediated by alveolar epithelial cells is unclear.

216 that differentiation of type II cells, also alveolar epithelial cells, is normal.

217 nduced similar levels cell death in vitro in alveolar epithelial cells isolated from WT and bid(-/-)

218 lial RelA activation, we stimulated a murine alveolar epithelial cell line (MLE-15) with bronchoalveo

219 tion of IL-31 was characterized in the human alveolar epithelial cell line A549 in which the expressi

220 to the keratin IF network in A549 cells (an alveolar epithelial cell line) exposed to 1.5% oxygen.

221 f2 by hyperoxia using a non-malignant murine alveolar epithelial cell line, C10.

222 that Ad7 induces IL-8 release from the A549 alveolar epithelial cell line.

223 g with alveolar macrophages or with the A549 alveolar epithelial cell line.

224 Bronchial and alveolar epithelial cell lines, and primary human bronch

225 that invaded but failed to replicate within alveolar epithelial cell lines.

226 tularensis LVS invaded and replicated within alveolar epithelial cell lines.

227 gen activation upregulated PGE2 synthesis in alveolar epithelial cells, lung fibroblasts, and lung fi

228 sitive correlation between CDC42 and type II alveolar epithelial cells marker SP-A, indicating the po

229 sion of beta-catenin-driven target genes and alveolar epithelial cell markers in the elastase, as wel

230 iciency promotes lung fibrosis by augmenting alveolar epithelial cell mitochondrial DNA damage and ap

231 anical stretch (MS) potentiates LPS-mediated alveolar epithelial cell (MLE-12) expression of the chem

232 NLRP3 improves the integrity of alveolar epithelial cell monolayers by enhancing cellula

233 L-1beta-mediated protein permeability across alveolar epithelial cell monolayers.

234 Alveolar epithelial cells, myofibroblasts and endothelia

235 DCK was elevated in hyperplastic alveolar epithelial cells of patients with IPF and in mi

236 sent in the total lipid extract from type II alveolar epithelial cells of the cell line A549 separate

237 ypes of human cells, such as macrophages and alveolar epithelial cells of the lung.

238 When studied on rat primary alveolar epithelial cells or on immortalized human pulmo

239 g aspects of aberrant wound repair involving alveolar epithelial cells or septal endothelial cells ar

240 tations, IPF patients had short telomeres in alveolar epithelial cells (P < 0.0001).

241 unknown etiology; however, apoptosis of lung alveolar epithelial cells plays a role in disease progre

242 oxygen species generation, and apoptosis of alveolar epithelial cells, potential mechanisms of LYCAT

243 These data provide the first evidence that alveolar epithelial cells produce dopamine and that incr

244 Here we tested whether alveolar epithelial cells produce dopamine and whether i

245 beta1 integrin-deficient alveolar epithelial cells produced excessive monocyte ch

246 l as murine bone marrow macrophages, but not alveolar epithelial cells, produced type I IFNs upon inf

247 markedly reduced due to decreased ductal and alveolar epithelial cell proliferation and decreased sur

248 AL surfactant protein D, a marker of type II alveolar epithelial cell proliferation in a human model

249 ion of pulmonary fibrosis by contributing to alveolar epithelial cell proliferation.

250 ted inflammatory response in IPS-1-deficient alveolar epithelial cells, pulmonary macrophages, and CD

251 ty of IPF, we hypothesized that hyperplastic alveolar epithelial cells regulate the fibrotic response

252 Furthermore, alveolar epithelial cells require H2R to produce CCL24,

253 regulates ADAM10 metalloprotease activity in alveolar epithelial cells, resulting in cleavage of the

254 of the Na,K-ATPase to the plasma membrane of alveolar epithelial cells results in increased active Na

255 This finding was subsequently confirmed in alveolar epithelial cell scratch models.

256 rosis was examined using a DCK inhibitor and alveolar epithelial cell-specific knockout mice.

257 grew normally in murine lung macrophages and alveolar epithelial cells, suggesting that legiobactin p

258 elial sodium channel (ENaC) complex from the alveolar epithelial cell surface, leading to persistence

259 s, particularly fibronectin, associated with alveolar epithelial cell surfaces, triggers organism pro

260 wild-type strain PAO1 were more cytotoxic to alveolar epithelial cells than those from quorum-sensing

261 ying a novel integrin alpha6beta4-expressing alveolar epithelial cell that serves as a multipotent pr

262 oncogene and display morphologic changes in alveolar epithelial cells that recapitulate those of pre

263 In alveolar epithelial cells, the overexpression of SOD1 pr

264 activates the intrinsic apoptotic pathway in alveolar epithelial cells through a pathway that require

265 The lung is protected from pathogens by alveolar epithelial cells, tissue-resident alveolar macr

266 Malignant progression of normal alveolar epithelial cells to adenocarcinoma in Kras(LA1)

267 increased with malignant progression (normal alveolar epithelial cells to adenocarcinoma) in K-ras(LA

268 n the suppression of the calcium response of alveolar epithelial cells to ATP, thereby affecting cell

269 jury requires the repair and regeneration of alveolar epithelial cells to restore the integrity of ga

270 odifications regulate Aqp5 expression during alveolar epithelial cell transdifferentiation, suggestin

271 ryonic fibroblasts from ASK1 knock-out mice, alveolar epithelial cells transfected with dominant nega

272 In studies using isolated murine type II alveolar epithelial cells, treatment with GM-CSF greatly

273 hologically and functionally distinct cells: alveolar epithelial cell types I and II (AEC I and II).

274 relationship between distinct differentiated alveolar epithelial cell types in vivo and in single-cel

275 oxygen species generation, and apoptosis of alveolar epithelial cells under bleomycin challenge.

276 nic mice overexpressing TNF-alpha in type II alveolar epithelial cells under the control of the surfa

277 Alveolar epithelial cells undergoing transforming growth

278 30 days and co-localized in bronchiolar and alveolar epithelial cells using an antibody to cytokerat

279 on in the lung, we depleted JAM-A in primary alveolar epithelial cells using shRNA.

280 mation in lung microvascular endothelial and alveolar epithelial cells via small Rho GTPase.

281 In vitro, EphrinB2 preserved alveolar epithelial cell viability in O(2), decreased O(

282 Further, no reduction in alveolar epithelial cell viability was observed, but let

283 Atypical PV expansion in alveolar epithelial cells was attributed in part to defe

284 Using lung microvascular endothelial and alveolar epithelial cells, we demonstrated that N-WASP d

285 In primary rat alveolar epithelial cells, we found that exposure to hyp

286 n A2B adenosine receptor-specific agonist to alveolar epithelial cells, we subsequently performed stu

287 The type II alveolar epithelial cells were capable of surfactant pro

288 In vitro, alveolar epithelial cells were exposed to oxidative stre

289 Alveolar epithelial cells were isolated from rats 24 hou

290 When cultured rat alveolar epithelial cells were subjected to cyclic stret

291 internalization of TGF-beta1 and TbetaRI in alveolar epithelial cells, which inhibited TGF-beta1 sig

292 TGF-beta1 activation mediated by IL-1beta in alveolar epithelial cells, which requires actin stress f

293 of the disease is areas of injury to type II alveolar epithelial cells with attendant accumulation of

294 Treatment of alveolar epithelial cells with bronchoalveolar lavage fl

295 -2 is induced by RSV infection of human lung alveolar epithelial cells with the concomitant productio

296 Treatment of alveolar epithelial cells with the NAD(P)H oxidase inhib

297 is expressed predominantly in bronchial and alveolar epithelial cells, with reduced expression in al

298 o determine the effects of FZD4 signaling on alveolar epithelial cell wound healing and repair, as we

299 ature WNT-5A attenuated canonical WNT-driven alveolar epithelial cell wound healing and transdifferen

300 r epithelial cell apoptosis, and accelerated alveolar epithelial cell wound healing, maintained lung